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1                                   Crithidia, Trypanosoma and Leishmania are all members of the order
2  (T) confined to pathogenic protozoa such as Trypanosoma and Leishmania JBP1 has two known functional
3 ection system of parasitic protozoa, such as trypanosoma and leishmania parasites.
4 ypanosomatids, which include human pathogens Trypanosoma and Leishmania This makes Paratrypanosoma un
5 estral characters such as peptidases between Trypanosoma and Leishmania, genomic differences that wer
6 ude medically important members of the genus Trypanosoma and Leishmania, the 26/28S large subunit rib
7                        African trypanosomes (Trypanosoma) are vector-borne haemoparasites that surviv
8      Most of the samples were active against Trypanosoma b.
9 ng drug target within the parasitic protozoa Trypanosoma brucei (T. brucei), the causative agent for
10 tal disease caused by the protozoan parasite Trypanosoma brucei (Tb).
11                     Nonetheless, the GMPR of Trypanosoma brucei (TbGMPR) includes a unique structure
12 rominent defence of the unicellular parasite Trypanosoma brucei against the host immune system is a d
13              Structural studies performed in Trypanosoma brucei already highlighted the numerous pecu
14                    The flagellated eukaryote Trypanosoma brucei alternates between the insect vector
15              The compounds were assessed for Trypanosoma brucei and Leishmania major PTR1 inhibition
16 ct life cycle stages of two human parasites; Trypanosoma brucei and Leishmania mexicana.
17 el can be applied to the characterization of Trypanosoma brucei and Leishmania spp. ribosomes as well
18 in two well-studied kinetoplastid parasites, Trypanosoma brucei and Leishmania, focusing on recent wo
19 wledge largely stems from the human pathogen Trypanosoma brucei and mouse experimental models.
20                                           In Trypanosoma brucei and related kinetoplastids, gene expr
21 zation of this channel to acidocalcisomes of Trypanosoma brucei and suggest that caution should be ex
22                   The parasitic trypanosomes Trypanosoma brucei and T. cruzi are responsible for sign
23 roducts exhibit significant activity against Trypanosoma brucei and T. cruzi, featuring favorable dru
24 ickness, is caused by the protozoan parasite Trypanosoma brucei and transmitted through the bite of i
25                              We also compare Trypanosoma brucei and Trypanosoma congolense, parasites
26                             Mutations in the Trypanosoma brucei aquaporin AQP2 are associated with re
27                   Mitochondrial ribosomes of Trypanosoma brucei are composed of 9S and 12S rRNAs, eub
28 cells but were effective in cell cultures of Trypanosoma brucei brucei (EC(50) = 1-15 muM) and elimin
29 h a single-digit micromolar activity against Trypanosoma brucei brucei (EC50 = 2.97 muM), thus being
30 on by several protozoan parasites, including Trypanosoma brucei brucei Endocytosis and acidification
31 ate immunity against the veterinary pathogen Trypanosoma brucei brucei is conferred by trypanosome ly
32 e glycerol kinase and alternative oxidase of Trypanosoma brucei brucei.
33   RNase H1 loss is tolerated by the parasite Trypanosoma brucei but no work has examined the function
34 rotozoan parasites Plasmodium falciparum and Trypanosoma brucei by microscopy imaging, proving that t
35                                              Trypanosoma brucei causes African sleeping sickness for
36                                              Trypanosoma brucei causes African trypanosomiasis and co
37                                              Trypanosoma brucei causes fatal human African trypanosom
38                                              Trypanosoma brucei causes human African trypanosomiasis
39                                              Trypanosoma brucei causes human African trypanosomiasis
40                         The protist parasite Trypanosoma brucei causes Human African trypanosomiasis
41                       The protozoan parasite Trypanosoma brucei causes the fatal illness human Africa
42                        A defining feature of Trypanosoma brucei cell shape is the lateral attachment
43                                              Trypanosoma brucei CRK9 is an essential cyclin-dependent
44 ly assembled flagellum in the human parasite Trypanosoma brucei depends on the faithful duplication a
45                                  Survival of Trypanosoma brucei depends upon switches in its protecti
46 al metabolic remodeling is a hallmark of the Trypanosoma brucei digenetic life cycle because the inse
47                           The human parasite Trypanosoma brucei does not synthesize heme de novo and
48  generates functional mitochondrial mRNAs in Trypanosoma brucei Editing is catalyzed by three distinc
49                                              Trypanosoma brucei encodes three paralogue single-protei
50                Unlike other type II TKs, the Trypanosoma brucei enzyme (TbTK) is a tandem protein wit
51                                              Trypanosoma brucei evades the host immune system through
52 ad of important trypanosome variants such as Trypanosoma brucei evansi that exploit mechanical transm
53 clusion ensures that the African trypanosome Trypanosoma brucei exclusively expresses only 1 of thous
54  screens were undertaken in bloodstream form Trypanosoma brucei exposed to the antifolates methotrexa
55   The bloodstream form of the human pathogen Trypanosoma brucei expresses oligomannose, paucimannose,
56                                              Trypanosoma brucei faces relentless immune attack in the
57 segmented gene conversion, is fundamental in Trypanosoma brucei for both VSG gene switching and for g
58 ine reductase 1 with phenotypic screening on Trypanosoma brucei for hit identification.
59 st Trypanosoma, including the human pathogen Trypanosoma brucei gambiense and Trypanosoma cruzi paras
60 uman African trypanosomiasis (HAT) caused by Trypanosoma brucei gambiense can be diagnosed in the ear
61       The World Health Organization targeted Trypanosoma brucei gambiense human African trypanosomias
62                           Infection rates of Trypanosoma brucei gambiense in tsetse are extremely low
63 to replace the current treatment regimen for Trypanosoma brucei gambiense infections, following a pos
64      Human African trypanosomiasis caused by Trypanosoma brucei gambiense is a parasitic infection th
65 e of Human African Trypanosomiasis caused by Trypanosoma brucei gambiense.
66  this effect in an important human pathogen, Trypanosoma brucei gambiense.
67 number of constitutive origins mapped in the Trypanosoma brucei genome is less than the minimum requi
68 l for growth and infectivity of the parasite Trypanosoma brucei However, the mechanism by which TbIP(
69 ausative agent of African sleeping sickness, Trypanosoma brucei In mitochondria of this pathogen, mos
70 eptor (TfR) of the bloodstream form (BSF) of Trypanosoma brucei is a heterodimer comprising glycosylp
71                                              Trypanosoma brucei is a kinetoplastid parasite that caus
72                                              Trypanosoma brucei is a parasitic protozoan that undergo
73                                              Trypanosoma brucei is a protist parasite causing sleepin
74                                              Trypanosoma brucei is a protozoan parasite that evades i
75                      The African trypanosome Trypanosoma brucei is a single-celled eukaryote with a s
76                                              Trypanosoma brucei is an extracellular parasite that cau
77         The basal body in the human parasite Trypanosoma brucei is structurally equivalent to the cen
78                                 The parasite Trypanosoma brucei is the causative agent of African sle
79                     The unicellular parasite Trypanosoma brucei is transmitted between mammals by tse
80 The mitochondrion of the parasitic protozoan Trypanosoma brucei lacks tRNA genes and therefore import
81 The mitochondrion of the parasitic protozoan Trypanosoma brucei lacks tRNA genes, and thus imports al
82  screen with 176 individual bloodstream form Trypanosoma brucei lines identified PKs required for pro
83 es, putative stem-loops from L. donovani and Trypanosoma brucei nucleobase transporter mRNAs were not
84                                              Trypanosoma brucei parasites successfully evade the host
85 rican trypanosomiasis is a disease caused by Trypanosoma brucei parasites with limited treatment opti
86 allelic exclusion and antigenic variation in Trypanosoma brucei parasites.
87                                              Trypanosoma brucei possesses a highly complex RNA editin
88                                              Trypanosoma brucei PRMT7 (TbPRMT7) is a protein arginine
89 ation of the proliferative cell cycle of the Trypanosoma brucei procyclic life cycle stage, three sub
90 volutionarily early divergent human parasite Trypanosoma brucei proliferates through binary cell fiss
91                                    The major Trypanosoma brucei protein arginine methyltransferase, T
92 Cardiolipin depletion-induced changes in the Trypanosoma brucei proteome.
93           The infectious metacyclic forms of Trypanosoma brucei result from a complex development in
94 ition of the cysteine proteases rhodesain of Trypanosoma brucei rhodesiense and falcipain-2 of Plasmo
95 logues possess micromolar activities against Trypanosoma brucei rhodesiense and Leishmania donovani a
96 ent manner and demonstrated activity against Trypanosoma brucei rhodesiense bloodstream forms, which
97 r proteins that contribute to virulence, and Trypanosoma brucei rhodesiense EVs contain the serum res
98 stance to most African trypanosomes, but not Trypanosoma brucei rhodesiense or T.b. gambiense, which
99 gnized as a strong inhibitor of rhodesain of Trypanosoma brucei rhodesiense, endowed with a k(second)
100 ch as Plasmodium falciparum, Lassa Virus and Trypanosoma brucei rhodesiense, has resulted in elevated
101 c markers in CSF from patients infected with Trypanosoma brucei rhodesiense, using 1H nuclear magneti
102 ite concentrations in patients infected with Trypanosoma brucei rhodesiense, using liquid chromatogra
103                We report X-ray structures of Trypanosoma brucei S-adenosylmethionine decarboxylase al
104 ved following RNAi-mediated silencing of the Trypanosoma brucei SODA ortholog suggests that SODA is e
105                                              Trypanosoma brucei spp. cause African human and animal t
106 sites Trypanosoma cruzi, Leishmania spp. and Trypanosoma brucei spp., respectively.
107                                              Trypanosoma brucei stumpy forms are the only stage that
108                Humans are susceptible to two Trypanosoma brucei subspecies but protected from other t
109 the procyclic and bloodstream form stages of Trypanosoma brucei that yields viable and proliferative
110             Here, we show that in the PAM of Trypanosoma brucei the function of Pam18 has been replac
111 ing the cell cycle of the protozoan parasite Trypanosoma brucei The source of components required to
112 rin uptake, we determined the structure of a Trypanosoma brucei transferrin receptor in complex with
113 ow that cytosine 32 in the anticodon loop of Trypanosoma brucei tRNA(Thr) is methylated to 3-methylcy
114                       Mitochondrial mRNAs in Trypanosoma brucei undergo extensive insertion and delet
115 ghly motile and versatile protozoan pathogen Trypanosoma brucei undergoes a complex life cycle in the
116                                              Trypanosoma brucei undergoes cytokinesis uni-directional
117                                              Trypanosoma brucei uses multiple mechanisms to evade det
118 ve-site residues of PRMT7 from the protozoan Trypanosoma brucei We have designed 26 single and double
119 ulation of glycosomes in live procyclic form Trypanosoma brucei When added to cells, this fluorescent
120                        Here, we identify the Trypanosoma brucei ZC3H39/40 RNA-binding proteins as reg
121 tozoa (Leishmania spp., Plasmodium spp., and Trypanosoma brucei).
122              We examined BBSome functions in Trypanosoma brucei, a flagellated protozoan parasite tha
123                           Differentiation of Trypanosoma brucei, a flagellated protozoan parasite, be
124 rganisms belonging to the phylum euglenozoa: Trypanosoma brucei, a lethal human parasite, and Euglena
125 mography structures of the 96-nm repeat from Trypanosoma brucei, a protozoan parasite in the Excavate
126                                           In Trypanosoma brucei, an ancient unicellular eukaryote, on
127 e biosynthesis pathway of the human parasite Trypanosoma brucei, an early branching eukaryote that la
128                                              Trypanosoma brucei, an early divergent microbial eukaryo
129 ential for growth of the parasitic protozoan Trypanosoma brucei, enabling the study of its function i
130                                           In Trypanosoma brucei, genes are arranged in Polycistronic
131 th sleeping sickness, caused by the parasite Trypanosoma brucei, have disruptions in both sleep timin
132 ed to TbKHC1, an orphan kinesin H chain from Trypanosoma brucei, inhibited T. musculi excreted/secret
133 receptor of the African trypanosome species, Trypanosoma brucei, is expressed when the parasite is in
134  OM protein of the early diverging protozoan Trypanosoma brucei, is signal-anchored.
135                                           In Trypanosoma brucei, mitochondrial pre-mRNAs undergo 3'-5
136                                           In Trypanosoma brucei, most mitochondrial mRNAs undergo edi
137                                           In Trypanosoma brucei, most mitochondrial mRNAs undergo int
138  the causative agent, the protozoan parasite Trypanosoma brucei, occurs through receptor-mediated end
139 e effects of the sleeping sickness parasite, Trypanosoma brucei, on sleep patterns in mice, under bot
140                                           In Trypanosoma brucei, PE synthesis has been shown to occur
141           To identify new chemotypes against Trypanosoma brucei, previously we identified 797 potent
142                                              Trypanosoma brucei, protozoan parasites that cause human
143 t that CRK1, a G1 cyclin-dependent kinase in Trypanosoma brucei, regulates anterograde protein traffi
144 ains of TR from the basal eukaryotic species Trypanosoma brucei, revealing the ancestry of TR compris
145 ruzi, the causative agent of Chagas disease; Trypanosoma brucei, the causative agent of African sleep
146 13b ortholog in the evolutionarily divergent Trypanosoma brucei, the causative agent of African sleep
147 , which had been identified as a hit against Trypanosoma brucei, the causative agent of human African
148 es (PKs) are a class of druggable targets in Trypanosoma brucei, the causative agent of Human African
149  point for the synthesis of new hits against Trypanosoma brucei, the causative agent of human African
150                           Here, we show that Trypanosoma brucei, the causative agent of human sleepin
151                                 The parasite Trypanosoma brucei, the causative agent of sleeping sick
152                                              Trypanosoma brucei, the causative agent of sleeping sick
153                                              Trypanosoma brucei, the etiologic agent of African Sleep
154            In the sleeping sickness parasite Trypanosoma brucei, the kinetoplast is a chain mail-like
155                                           In Trypanosoma brucei, the proteins that make up the kineto
156                                           In Trypanosoma brucei, the small noncoding TBsRNA-10 was fi
157 resistant strains of the protozoal pathogens Trypanosoma brucei, Trypanosoma congolense and Leishmani
158 nt parasites and include the human pathogens Trypanosoma brucei, Trypanosoma cruzi, and Leishmania sp
159                          The trypanosomatids Trypanosoma brucei, Trypanosoma cruzi, and Leishmania sp
160 nd biogenesis of the array, with emphasis on Trypanosoma brucei, Trypanosoma cruzi, and Leishmania, w
161 pathway and its enzymes have been studied in Trypanosoma brucei, Trypanosoma cruzi, and various Leish
162              The sleeping sickness parasite, Trypanosoma brucei, uses quorum sensing (QS) to balance
163 its application to the pathogenic protozoan, Trypanosoma brucei, using hyperpolarized (13)C1 pyruvate
164                       The protozoan parasite Trypanosoma brucei, which causes devastating diseases in
165                                An example is Trypanosoma brucei, which causes human African trypanoso
166                        Tsetse flies transmit Trypanosoma brucei, which is the parasite that causes hu
167 in the causative agent of sleeping sickness, Trypanosoma brucei, with that of human erythrocytes, and
168 ammalian cells, CL synthesis is essential in Trypanosoma brucei.
169 ndidates, Xpo-t and Xpo-5 for tRNA export in Trypanosoma brucei.
170 ke and the modulation of drug sensitivity in Trypanosoma brucei.
171  in tRNAs from Bacillus subtilis, plants and Trypanosoma brucei.
172 orthologue, PNT1, in the parasitic protozoon Trypanosoma brucei.
173  by RNA polymerase I (Pol I) in the parasite Trypanosoma brucei.
174 diting in kinetoplastid protists typified by Trypanosoma brucei.
175 ents in the large ribosomal subunit (60S) of Trypanosoma brucei.
176 e boundaries of previously annotated CDSs in Trypanosoma brucei.
177 As and is unique for kinetoplastids, such as Trypanosoma brucei.
178 P2 in either the cytosol or mitochondrion of Trypanosoma brucei.
179 ith either of two subspecies of the parasite Trypanosoma brucei.
180 ositol pyrophosphate biosynthetic pathway in Trypanosoma brucei: inositol polyphosphate multikinase (
181 of unicellular parasitic flagellate protozoa.Trypanosoma bruceispecies and Trypanosoma cruziare the m
182          We report a way to selectively kill Trypanosoma by blocking glycosomal/peroxisomal import th
183 ection model of zebrafish (Danio rerio) with Trypanosoma carassii.
184  the protozoal pathogens Trypanosoma brucei, Trypanosoma congolense and Leishmania mexicana.
185 o-infections with other trypanosome species (Trypanosoma congolense and Trypanosoma vivax) are common
186 mal African trypanosomiasis (AAT), caused by Trypanosoma congolense and Trypanosoma vivax, remains on
187 fectious diseases, their role in immunity to Trypanosoma congolense has not been investigated.
188                         Here we show that in Trypanosoma congolense this receptor is instead expresse
189 st the principal causative organisms of AAT: Trypanosoma congolense, and Trypanosoma vivax.
190       We also compare Trypanosoma brucei and Trypanosoma congolense, parasites that share a common pa
191                                              Trypanosoma cruzi (T. cruzi) infection is endemic in Lat
192 the authors present a 2.5-A structure of the Trypanosoma cruzi 60S ribosomal subunit and propose a mo
193  disease is caused by the protozoan parasite Trypanosoma cruzi and affects 5-8 million people in Lati
194  time for the simultaneous detection of anti-Trypanosoma cruzi and anti-Leishmania infantum antibodie
195     Chagas disease is caused by the parasite Trypanosoma cruzi and is an important cause of morbidity
196 have been considered as virulence factors of Trypanosoma cruzi and Leishmania spp., and have been dem
197 gondii and kinetoplastid protozoa, including Trypanosoma cruzi and Leishmania spp., cause millions of
198 centrating on Mycobacterium tuberculosis and Trypanosoma cruzi as examples of bacterial and parasitic
199                                          The Trypanosoma cruzi ascorbate peroxidase is, by sequence a
200 ole and posaconazole have not been tested in Trypanosoma cruzi carriers.
201 reference perspective, cruzain, an essential Trypanosoma cruzi cysteine protease, is highly homologou
202                      To successfully infect, Trypanosoma cruzi evades and modulates the host immune r
203           In contrast, as a model eukaryote, Trypanosoma cruzi exhibits two individual putative ortho
204                                The protozoan Trypanosoma cruzi has a complicated dual-host life cycle
205 e extreme genetic diversity of the protozoan Trypanosoma cruzi has been proposed to be associated wit
206                         Oral transmission of Trypanosoma cruzi has gained relevance because of its as
207                     Vertical transmission of Trypanosoma cruzi infection accounts for a growing propo
208                                   Congenital Trypanosoma cruzi infection accounts for an estimated 22
209 response plays a crucial role in controlling Trypanosoma cruzi infection and parasite burden, but it
210                                              Trypanosoma cruzi infection drives the expansion of rema
211 nic Chagas disease cardiomyopathy, caused by Trypanosoma cruzi infection, is a major cause of heart f
212                                   In chronic Trypanosoma cruzi infection, the cause of Chagas disease
213                                              Trypanosoma cruzi infection, which is the etiological ag
214 o be relevant in the context of experimental Trypanosoma cruzi infection.
215 eviously described in immune response during Trypanosoma cruzi infection.
216                                   In chronic Trypanosoma cruzi infections, parasite burden is control
217                                              Trypanosoma cruzi infects many cell types but preferenti
218                                              Trypanosoma cruzi is a unicellular parasite and the etio
219                                              Trypanosoma cruzi is an intracellular protozoan parasite
220    The insect-transmitted protozoan parasite Trypanosoma cruzi is the causative agent of Chagas disea
221                                              Trypanosoma cruzi is the causative agent of chronic chag
222                                              Trypanosoma cruzi is the causative pathogen of Chagas di
223                                              Trypanosoma cruzi is the etiological agent of Chagas dis
224  of the protozoans Leishmania tarentolae and Trypanosoma cruzi mitoribosomes.
225                                              Trypanosoma cruzi parasites are the causative agents of
226                             The diversity of Trypanosoma cruzi parasites infecting humans is still po
227 an pathogen Trypanosoma brucei gambiense and Trypanosoma cruzi parasites.
228 s were identified as potential inhibitors of Trypanosoma cruzi replication, a parasite responsible fo
229 es, we determined the 2.5-A structure of the Trypanosoma cruzi ribosome large subunit by single-parti
230                                              Trypanosoma cruzi species is categorized into six discre
231 s from a screen against the related parasite Trypanosoma cruzi Subsequent optimization of the chemica
232                                   Congenital Trypanosoma cruzi transmission is now estimated to accou
233 pest insect that has the ability to transmit Trypanosoma cruzi under experimental laboratory conditio
234 as disease, caused by the protozoan parasite Trypanosoma cruzi, affects 7 million people in Latin Ame
235 gas disease is a chronic infection caused by Trypanosoma cruzi, an intracellular protozoan parasite.
236 infections due to Toxoplasma gondii (n = 3), Trypanosoma cruzi, and Leishmania species.
237      The trypanosomatids Trypanosoma brucei, Trypanosoma cruzi, and Leishmania spp. are flagellate eu
238 lude the human pathogens Trypanosoma brucei, Trypanosoma cruzi, and Leishmania spp., which in humans
239  array, with emphasis on Trypanosoma brucei, Trypanosoma cruzi, and Leishmania, which cause life-thre
240 mes have been studied in Trypanosoma brucei, Trypanosoma cruzi, and various Leishmania species.
241 ng infection with the intracellular parasite Trypanosoma cruzi, as evidenced by transcriptome and cyt
242                         The protozoan agent, Trypanosoma cruzi, comprises six genetic lineages, TcI-T
243 sed by infection with the protozoan parasite Trypanosoma cruzi, is a leading cause of heart disease (
244                    Chagas disease, caused by Trypanosoma cruzi, is a major public health issue.
245 used by the intracellular protozoan parasite Trypanosoma cruzi, is a public health problem affecting
246 ysteine protease of the parasitic protozoan, Trypanosoma cruzi, is an important drug target for Chaga
247      Chagas disease, caused by the protozoan Trypanosoma cruzi, is endemic in Latin America and affec
248       Chagas disease, caused by the parasite Trypanosoma cruzi, is endemic in Latin America.
249 s' disease, caused by the protozoan parasite Trypanosoma cruzi, is the most common cause of cardiac-r
250 y infection with the kinetoplastid parasites Trypanosoma cruzi, Leishmania spp. and Trypanosoma bruce
251 a novel mechanism used by diverse pathogens (Trypanosoma cruzi, Listeria monocytogenes, and adenoviru
252 mice with the myotropic Colombiana strain of Trypanosoma cruzi, parasites persisted in tissue at low
253 uM against neglected Chagas' disease causing Trypanosoma cruzi, respectively.
254  of fire ants against the protozoan parasite Trypanosoma cruzi, the aetiologic agent of Chagas diseas
255              The Triatominae are vectors for Trypanosoma cruzi, the aetiological agent of the neglect
256       Using a murine model of infection with Trypanosoma cruzi, the causal agent of Chagas cardiomyop
257 ted with AT during part of their life cycle: Trypanosoma cruzi, the causative agent of Chagas disease
258                        The release of EVs by Trypanosoma cruzi, the causative agent of Chagas disease
259                                              Trypanosoma cruzi, the causative agent of Chagas disease
260  (NMT), an essential and druggable target in Trypanosoma cruzi, the causative agent of Chagas' diseas
261                                              Trypanosoma cruzi, the causing agent of Chagas disease,
262                          We report here that Trypanosoma cruzi, the etiologic agent of Chagas disease
263          Methods for genetic manipulation of Trypanosoma cruzi, the etiologic agent of Chagas disease
264                                              Trypanosoma cruzi, the etiological agent of Chagas disea
265                                              Trypanosoma cruzi, the etiological agent of Chagas disea
266                                              Trypanosoma cruzi, the parasitic agent of Chagas disease
267  role of CD43 in the immune response against Trypanosoma cruzi, the protozoan parasite that causes Ch
268             Transmitted by a protozoan named Trypanosoma cruzi, this infection triggers a chronic inf
269 g resistance of the Chagas disease parasite, Trypanosoma cruzi, to current therapies are not well und
270 elivers Gzms into three protozoan parasites (Trypanosoma cruzi, Toxoplasma gondii and Leishmania majo
271 C(50) within the low nanomolar range against Trypanosoma cruzi, with very high selectivity toward the
272 ar why only a proportion of children born to Trypanosoma cruzi-infected mothers acquire the infection
273 hmania-like flagellum attachment zone, and a Trypanosoma cruzi-like cytostome are ancestral features,
274  in the activation and functional profile of Trypanosoma cruzi-specific DN T cells.
275 n the infective stages of the human parasite Trypanosoma cruzi.
276 CI 2.2-6.8) were estimated to be infected by Trypanosoma cruzi.
277 the kinetoplastidea including Leishmania and Trypanosoma cruzi.
278 used by the kinetoplastid protozoan parasite Trypanosoma cruzi.
279  T. brucei bloodstream forms, Leishmania and Trypanosoma cruzi.
280  disease is caused by the protozoan parasite Trypanosoma cruzi.
281 gas disease caused by the protozoan parasite Trypanosoma cruzi.
282  easy discrimination of Leishmania major and Trypanosoma cruzi.
283 idate clathrin-associated proteins (CAPs) in Trypanosoma cruzi; the cohort includes orthologs of many
284 llate protozoa.Trypanosoma bruceispecies and Trypanosoma cruziare the major agents of human trypanoso
285  testing identified the infecting species as Trypanosoma evansi.Despite relapsing 6 weeks after compl
286                The parasitic protists of the Trypanosoma genus infect humans and domestic mammals, ca
287 s show significant cellular activity against Trypanosoma, including the human pathogen Trypanosoma br
288 range of available chemotherapeutics against Trypanosoma is limited, and the existing therapies are p
289                                              Trypanosoma is unable to synthesize purines de novo and
290  hosts) trypanosomatid species of the genera Trypanosoma, Leishmania, and Phytomonas, but only fragme
291                                              Trypanosoma lewisi is a member of the Stercorarian group
292 examination of blood revealed infection with Trypanosoma METHODS: Microscopic observation, polymerase
293 ced in macrophages during mouse infection by Trypanosoma musculi, a natural parasite of this host.
294                                              Trypanosoma protists are pathogens leading to a spectrum
295                                              Trypanosoma protozoan parasites are the causative agents
296 identify PEX14 as an "Achilles' heel" of the Trypanosoma suitable for the development of new therapie
297                                              Trypanosoma vivax is a related, livestock pathogen whose
298 panosome species (Trypanosoma congolense and Trypanosoma vivax) are common in animals, generating the
299  (AAT), caused by Trypanosoma congolense and Trypanosoma vivax, remains one of the most important liv
300 rganisms of AAT: Trypanosoma congolense, and Trypanosoma vivax.

 
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