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1 ine protease cruzipain (Cz) protects against Trypanosoma cruzi infection.
2 uestions in detail using the murine model of Trypanosoma cruzi infection.
3 afety of benznidazole in adults with chronic Trypanosoma cruzi infection.
4 e tissue (WAT) and adipocytes are targets of Trypanosoma cruzi infection.
5 CD8(+) T cells are essential for controlling Trypanosoma cruzi infection.
6 diomyopathy, we studied a rat model of acute Trypanosoma cruzi infection.
7 cells are key effectors in immune control of Trypanosoma cruzi infection.
8 ase (DCD) is an enteric neuropathy caused by Trypanosoma cruzi infection.
9 tral to the adaptive immune response against Trypanosoma cruzi infection.
10 o be relevant in the context of experimental Trypanosoma cruzi infection.
11 eviously described in immune response during Trypanosoma cruzi infection.
12 to reach cardiac tissue in vivo, even after Trypanosoma cruzi infection, a known inducer of lymphoid
13 ion of Th17 cells, we used a murine model of Trypanosoma cruzi infection, a protozoan parasite that c
16 response plays a crucial role in controlling Trypanosoma cruzi infection and parasite burden, but it
17 ntral and South America should be tested for Trypanosoma cruzi infection, and family members of peopl
18 es to lymphocytic choriomeningitis virus and Trypanosoma cruzi infections, and a specific defect in C
20 luated with the experimental murine model of Trypanosoma cruzi infection as a means to induce antipar
22 nic epithelial model HCT116 cells respond to Trypanosoma cruzi infection by secreting defensin alpha-
26 at immune responses in subjects with chronic Trypanosoma cruzi infection display features common to o
28 The pathogenesis of tissue damage in chronic Trypanosoma cruzi infection has been a subject of long-s
29 Increasingly during the past few decades, Trypanosoma cruzi infection has been detected in North A
30 mmunochromatographic dipstick test to detect Trypanosoma cruzi infection in 366 human serum samples w
31 or T-bet (Tbx21) regulates Th17 responses to Trypanosoma cruzi infection in an IFN-gamma-independent
33 e required for host control of intracellular Trypanosoma cruzi infection in mice, although the basis
35 The diversity of individuals at risk for Trypanosoma cruzi infection in the United States poses c
39 nic Chagas disease cardiomyopathy, caused by Trypanosoma cruzi infection, is a major cause of heart f
42 loss of certain functional activities during Trypanosoma cruzi infection might result in the inabilit
43 ses tend to be associated with resistance to Trypanosoma cruzi infection, mixed Th1 and Th2 cytokine
48 tablishing a system for drug cure of chronic Trypanosoma cruzi infection, we present the first extens
51 tural selection in a set of genes related to Trypanosoma cruzi infection, which is the pathogen respo