ライフサイエンスコーパス: V

1 V-EoE was more likely to attain histologic remission via

2 butyl viologen as the anolyte to yield a 2.0 V battery.

3 rrent densities of around -10 muA/cm(2) at 0 V vs RHE.

4 vity of 36 % for CO in 0.1 M KHCO(3) at -1.1 V vs. RHE, similar to that of pure Ag NPs.

5 to FETs with the Al layer (V(Dirac) = - 6.1 V and hysteresis = 2.9 V).

6 on transfer into TPP molecules occurs at <+1 V in the presence of mobile ions, enabled by ionic scree

7 anical stimuli (0-16 mN) at a gate bias of 1 V.

8 results in a much smaller Dirac voltage (- 1 V) and hysteresis (0.9 V) when compared to FETs with the

9 oxometalate salt [(C(4)H(9))(4)N](4)H[PMo(10)V(2)O(40)], specially designed to be insoluble in water,

10 ME after 5.5 h processing under 30 V and 15 V, respectively.

11 eleration of 1-5 G at a supply voltage of 15 V.

12 powered device that can operate less than 15 V that leads to significant decline in the infrared tran

13 The single SMFC produces a voltage of 1.16 V, which is too low for practical application.

14 an irreversible cathodic peak (E(pc) = -2.17 V vs SHE).

15 ent absolute test-retest variability for 1TC V (T) (<=5%) and BP (ND) (<=10%).

16 tions and weak electric field strengths (0-2 V cm(-1)).

17 ing ozonation, but a higher-voltage (0.6-1.2 V) LEEFT significantly enhances the ozone inactivation.

18 2) with a total electrolysis voltage of ~2.2 V.

19 dominate the IV cycle of the junction (+/-2 V) in ACN vapor, enhancing the reversible charge storage

20 e, and for mobilities as low as 10(-3) cm(2) V(-1) s(-1) , [Formula: see text] Here, mu(w) is the wei

21 films show a mobility as high as 16.1 cm(2) V(-1) s(-1) , which represents the highest mobility valu

22 terials with mobilities greater than 1 cm(2) V(-1) s(-1) .

23 ed film gave a highest mobility of 1.5 cm(2) V(-1) s(-1) along the column direction, which is a 3 ord

24 t, and relatively high mu(e) of 10(-4) cm(2) V(-1) s(-1).

25 on mobilities ranging from 0.1 to 0.018 cm(2)V(-1)s(-1) between 1.7 to 200 K.

26 Results showed R(2)(V) between 0.28 and 0.89, %LOF < 6%, variance explained

27 ed hole mobility of 10(-5) and 10(-3) cm(2)/(V s), respectively.

28 otocurrent density of 15.1 mA cm(-2) at 1.23 V vs. reversible hydrogen electrode (RHE) with an onset

29 with near unity H(2)O(2) selectivity at 0.27 V vs. RHE.

30 iate in the Mg-V-S compositional space, Mg(3)V(2)S(8), comprising [VS(4)](3-) tetrahedral units, iden

31 e transitions is linked to the lattice V(3+)/V(5+) concentrations of stoichiometric VO(2) and that el

32 6.3% for ME after 5.5 h processing under 30 V and 15 V, respectively.

33 ) to NiOOH requires a high potential of 1.35 V vs. RHE.

34 catalysts can deliver 100 mA cm(-2) at 1.39 V.

35 Our results show that a low-voltage (<0.4 V) LEEFT has no obvious effect on the following ozonatio

36 ctivity but are oxidatively unstable above 4 V, which prevents the use of high-voltage cathodes that

37 A high mass activity (1.946 A mg(-1) at 0.45 V) of Pd(delta+)-OCNT is achieved.

38 iconducting Bi(3.33)(VO(4))(2)O(2) and Bi(46)V(8)O(89) components, and the rectifying contact between

39 elf-polarization in the ion-conducting Bi(46)V(8)O(89) constituent.

40 In addition, CTNNBL1 466V/V Ramos B cells displayed a decreased incidence of SHM t

41 c process (E(pc) = -1.58 V and E(pa) = -1.47 V vs SHE) that is followed by an irreversible cathodic p

42 uction potential of enolized RuBP (near 0.49 V) is compatible with superoxide (O(2) (*-)) production,

43 potential of water in the range -0.4 to -0.5 V.

44 -) showing a characteristic response at +1.5 V vs SCE.

45 LEDs or charge up a 15 muF capacitor to 12.5 V in ~90 s.

46 olyte with electrochemical stability up to 5 V and a low activation energy barrier (<0.2 eV) for micr

47 is at a modest electrode potential of -0.536 V vs. the reversible hydrogen electrode.

48 ctrode (RHE) with an onset potential of 0.55 V vs. RHE and a record high half-cell solar-to-hydrogen

49 a reversible cathodic process (E(pc) = -1.58 V and E(pa) = -1.47 V vs SHE) that is followed by an irr

50 nanosheets array exhibits a voltage of 1.58 V at 30 mA cm(-2) as bifunctional electrode for water sp

51 peak voltage and peak current were 2154+/-59 V and 33.9+/-1.6 A, respectively.

52 Bulk electrolyses of 1 at -1.60 V vs SHE afford flavanone, 2'-hydroxychalcone, 2'-hydrox

53 of 8.87 mA cm(-2) at low potential of -0.65 V versus RHE.

54 y of EBV gH/gL and the EBV gH/gL-N(69)L/S(71)V mutant.

55 with a nearly 10-fold increase at |V| > 0.8 V than at lower bias.

56 A current density of 65.8 mA.cm(-2) at -1.8 V vs. Ag/Ag(+) is observed with a Faradaic efficiency to

57 er Dirac voltage (- 1 V) and hysteresis (0.9 V) when compared to FETs with the Al layer (V(Dirac) = -

58 yer (V(Dirac) = - 6.1 V and hysteresis = 2.9 V).

59 r frequency (TOF) up to 12500 h(-1) at -0.95 V versus the reversible hydrogen electrode (RHE), with a

60 ClC-5 had a 2Cl(-)/H(+) exchange ratio at a V(h) of +40 mV with a [Cl(-)](out) of 104 mm, but the tr

61 y microwave-excited coherent spin waves in a V(TCNE)(x) film can be transferred into an adjacent Pt l

62 in-down AHP, the former frequently induces a V-pattern esotropia requiring reoperation.

63 and 0.20 +/- 0.05, respectively, revealing a V-shaped SF/RH dependence.

64 mechanisms that might contribute to aberrant V(D)J recombination and the development of lymphoid tumo

65 cking beta1,6N-acetylglucosaminyltransferase V (Mgat5), an enzyme responsible for generating beta1,6-

66 primer-probe set described by Corman et al. (V.

67 Degradation of RAD21 eliminated all V(D)J recombination and interactions associated with RAG

68 The control of T6SS genes varies among V. cholerae strains and typically includes inputs from e

69 er Waals materials, such as alpha-MoO(3) and V(2)O(5), support exotic polariton propagation, as their

70 that lead to delocalized polaronic V(3+) and V(5+) cation states.

71 Low Mg/Ca, and high U/Ca, Mo/Ca, and V/Ca potentially suggest a decreased abundance of "cente

72 y reflecting desorption, while higher Cr and V levels were measured in near-neutral pH and oxic groun

73 gree of measurement temperature for R(d) and V(cmax) respectively.

74 in oxidative phosphorylation complexes I and V increased in CLPP2 knockouts, without accumulation of

75 ecreased mitochondrial area, complex III and V expression increased in debanding compared with sham o

76 striatum reveals that both layer II/III and V neurons in the motor cortex express BDNF as a potentia

77 y neurons in cortical layers II-III, IV, and V, as well as the dentate gyrus.

78 well were used to determine enzyme K(m) and V(max) values.

79 ried effects on transport kinetics (K(m) and V(max)) and substrate specificity.

80 Enzymatic parameters (K(M) and V(max)), residual activity, effect of bentonite and elec

81 and Zn) and trace metals (As, Cd, Pb, U and V).

82 rein, we show for the first time that VB and V(3) B(4) have high electrocatalytic HER activity.

83 tive 2-dimensional lattice formed by annexin V on trophoblast surfaces by anticardiolipin, via its in

84 nd BE(2)-C cells was demonstrated by Annexin V/PI staining.

85 h is characterized by the binding of Annexin V, demonstrates that programmed cell death can be promot

86 Antibody V(D)J mutations conferred pathogenicity by causing the a

87 miRNA-mediated mRNA repression, of AR and AR-V expression and the potential these mechanisms hold as

88 Here, we use 490 Argentinian V. cholerae genome sequences to characterise the variati

89 eceive either an ART-only (n=30) or an ART + V + V (n=30) regimen; all 60 participants completed the

90 Z, S, or additional rare variants denoted as V(R).

91 Nuclear processes, such as V(D)J recombination, are orchestrated by the three-dimen

92 ions showed 98-99% removal of As(III) and As(V) in the presence of PNHM/Fe(3)O(4)-40 following pseudo

93 city caused by the reduction of arsenate [As(V)] to arsenite [As(III)].

94 to transform arsenic to the less harmful As(V) state.

95 This result can explain the presence of As(V) in the solid phase.

96 H(*) and the aggregation of the resulting As(V)-Fe(III) polymers was enhanced by the presence of Mn.

97 th-relevant redox-sensitive elements (U, As, V, and Cr) in 1494 groundwater wells across North Caroli

98 N42C Az, with a nearly 10-fold increase at |V| > 0.8 V than at lower bias.

99 Vacuolar type ATPase (V-ATPase) has recently emerged as a promising novel anti

100 The vacuolar-type H(+)-ATPases (V-ATPase) hydrolyze ATP to pump protons across the plasm

101 lland (now Janssen Vaccines and Prevention B.V.), European Union's Horizon 2020 research and innovati

102 ing is to obtain a sufficient amount of beta(V) polymorph of the right size.

103 Associations between V(TOT) and other functional and morphologic measures wer

104 rprising lack of correlation evident between V(on) and equivalent circuit element parameters commonly

105 Molecular pathogenesis is preserved between V-EoE and L-EoE.

106 in situ generation of a uniquely reactive Bi(V) arylating agent from a bench-stable Bi(III) precursor

107 g a high-throughput assay that analyses both V(D)J segment usage and somatic hypermutation profiles,

108 a-cells through voltage-dependent Ca(2+) (Ca(V) ) channels.

109 11)/ P2Y(11)-like receptors, AC5, PKA and Ca(V)1.2 into nanocomplexes at the plasma membrane of human

110 tages of development, which are driven by Ca(V) 1.3 Ca(2+) channels.

111 Changing activity of cardiac Ca(V)1.2 channels under basal conditions, during sympatheti

112 Concerning Ca(V)3.1, the compound did not alter the shape of the insta

113 annel possesses similar features as human Ca(V)2.1 and other Ca(V)2 channels, including high voltage-

114 y (basal) hair cells was also affected in Ca(V) 1.3(-/-) mice, but to a much lesser extent than apica

115 c release sites, and synapse structure is Ca(V)2 independent.

116 caused by beta-adrenergic augmentation of Ca(V)1.2 voltage-gated calcium channels(1-4).

117 ly permits CTCF binding and expression of Ca(V)2.2 channel isoforms with increased opioid sensitivity

118 ilar features as human Ca(V)2.1 and other Ca(V)2 channels, including high voltage-activated currents

119 Reduced Ca(V) 1.3 activity might open new ways to understand sympto

120 Second, active zone proteins may scaffold Ca(V)2s to presynaptic release sites, and synapse structure

121 at TSPAN-7 modulation of beta-cell L-type Ca(V) channels is a key determinant of beta-cell glucose-st

122 TSPAN-7 KD enhanced L-type Ca(V) currents in mouse and human beta-cells.

123 CL-2 secretion was decreased after L-type Ca(V) inhibition.

124 tage-gated ion channels, including Na(V), Ca(V), and K(V) channels.

125 is enriched in subcellular domains where Ca(V) channels reside, such as the cardiac dyad.

126 he analysis of 20 elements (Mg, P, S, K, Ca, V, Cr, Mn, Fe, Co, Cu, Zn, Se, Br, Rb, Sr, Mo, I, Cs, an

127 This process called V(D)J recombination that involves the RAG recombinase bi

128 and inconsequential variant of the canonical V(D)J recombination.

129 ity (maximum rates of Rubisco carboxylation, V(cmax) , and of electron transport, J(max) ) was reduce

130 t 6 months after Hurricane Maria, a category V storm.

131 lyticus T3SS effector VopQ targets host-cell V-ATPase, resulting in blockage of autophagic flux and n

132 f increased post-injury scarring in collagen-V-deficient mice.

133 In contrast, V. fischeri ES114 mutants incapable of aerobactin produc

134 this work, we analyzed the voltage-current (V-I) characteristics of several FCDI units.

135 ion only at extreme membrane depolarization (V(50) ~ +75 mV), in contrast to other TPCs and Na(V) cha

136 We describe dTAG(V)-1, an exclusively selective VHL-recruiting dTAG molec

137 hain that, owing to junctional biases during V(D)J recombination, appear much more frequently than pr

138 e fraction, thick, 3D-structured electrodes (V(2) O(5) cathode and Li metal anode) are realized throu

139 on within, and between, epidemic and endemic V. cholerae.

140 , we show that Escherichia coli Endonuclease V (eEndoV), an inosine-cleaving enzyme, can be repurpose

141 tulating that locally evolving environmental V. cholerae contributes to outbreaks outside Asia remain

142 plaque rupture, we hypothesized that factor V Leiden may be a stronger risk factor for atherothrombo

143 ithout), and inherited thrombophilia (factor V Leiden carriers with a 10-year cumulative incidence of

144 ART in Fiebig I to 15.9 (7.6-29.2) in Fiebig V.

145 virulence regulator, ToxR, was important for V. cholerae resistance to hydrogen peroxide.

146 72 to 0.92 while the corresponding value for V(T) was 0.85.

147 g/V initially tracks the current, increasing ~15-fold from

148 G34R/V may become dispensable for tumor maintenance, whereas

149 The group V patients had the highest expression of IL5, TSLP, and

150 site highlights a single amino acid (I960 -> V) responsible for the potency shift.

151 dness (mechanical stability) value of ~194 H(V) that is significantly higher compared to the pristine

152 Voltage-gated proton channels (H(V)1) are essential for various physiological tasks but a

153 of HCV+ nonviremic (HCV-NV) and viremic (HCV-V) hearts nationally and by UNOS region.

154 neutralizing antibodies similar to the human V(H)1-69 class antibody specific for antigen region 3 we

155 The I-V curves of this single-molecule system also exhibit mem

156 In the subsequent current-voltage (I-V) measurements to examine its breakdown behavior under

157 reaction with common components of gTSSA-II/V/VI recombinant antigen.

158 tructures based on oxide perovskites and III-V, II-VI and transition metal dichalcogenide semiconduct

159 Semiconductor III-V photonic crystal (PC) laser is regarded as a promising

160 Here, we demonstrate ultrasmall III-V PC membrane lasers monolithically grown on CMOS-compat

161 ble on-axis Si (001) substrates by using III-V quantum dots.

162 RAG2, a late evolutionary addition in V(D)J recombination, appears to enforce the sharp kinks

163 protein necessary for curvature formation in V. cholerae.

164 isplayed a significantly reduced increase in V-ATPase activity and assembly upon starvation.

165 non-coherent spin waves on various modes in V(TCNE)(x) is demonstrated and show that the angular mom

166 regulator aphB; however, the role of OmpR in V. cholerae biology outside virulence regulation remaine

167 namic activity of type IV competence pili in V. cholerae as a model system.

168 We demonstrate that cell shape in V. cholerae is regulated by the bacterial second messeng

169 alian RAG1-RAG2 recombinase, which initiates V(D)J recombination, we find that the active site is rec

170 report two cryo-EM structures of the intact V-ATPase from bovine brain with all the subunits includi

171 trochemical performance of PANI-intercalated V(2) O(5) electrode is remarkable improved, exhibiting e

172 As a result, the PANI-intercalated V(2) O(5) exhibits a stable and highly reversible electr

173 scertained that five coordinate amide iodine(V) complexes are unreactive toward redox reactions due t

174 The growth of Cr(2)O(3) on isostructural V(2)O(3) thin film electrodes helps eliminate the existe

175 crus I, paraflocculus, and vermal regions IV/V and VI - highlighting these regions as potential hubs

176 [(11)C]UCB-J V(T) was significantly lower in the frontal and anterior

177 ion channels, including Na(V), Ca(V), and K(V) channels.

178 , which encodes voltage-gated K(+) channel K(V) 1.2.

179 ocker of voltage-gated potassium channels (K(V)1 family) clinically approved for the symptomatic trea

180 g mechanism in voltage-dependent gating of K(V)7.1 as triggered by VSD activations to the intermediat

181 pharmacology, and disease pathogenesis of K(V)7.1, and likely applies to numerous domain-swapped K(V

182 likely applies to numerous domain-swapped K(V) channels.

183 iofluorinated analog of 4AP, also binds to K(V)1 channels and can be used as a PET tracer for the det

184 Here, utilizing K(V)7.1's unique two open states, we report a two-stage E-

185 irst converts 3D images to key-value data (K-V).

186 Then the K-V data is used for 3D array partitioning and data exchan

187 etected in 79% of participants in the Lactin-V group.

188 f phase transitions is linked to the lattice V(3+)/V(5+) concentrations of stoichiometric VO(2) and t

189 ty with maximal enrichment in cortical layer V excitatory neurons.

190 V) when compared to FETs with the Al layer (V(Dirac) = - 6.1 V and hysteresis = 2.9 V).

191 e starvation-dependent increase in lysosomal V-ATPase activity without altering basal activity.

192 e starvation-dependent increase in lysosomal V-ATPase activity, indicating that H89 and dorsomorphin

193 The organic-based magnet [V(TCNE)(x) ; TCNE = tetracyanoethylene; x ~ 2] has shown

194 In particular, a major V. cholerae lineage occasionally grows to large numbers

195 The panel methodologist (V.K.A.) assigned a level of evidence rating to each stud

196 a previously unknown intermediate in the Mg-V-S compositional space, Mg(3)V(2)S(8), comprising [VS(4

197 ein is capable of accessing a high-valent Mn(V)-oxo species which can transfer an O atom to a thioeth

198 ta point to a novel "thiol-blocked" [(PDT)Mo(V)O(S(Cys))(thiolate)](-) structure, which is supported

199 indicating that H89 and dorsomorphin modify V-ATPase activity through other cellular targets.

200 ifically, mutations T407A and S411A in motif V exhibit a hyperactive helicase phenotype, leading to t

201 We tested Motif V mutations in both the replicon and recombinant protein

202 These simulations indicate that Motif V controls communication between the ATP-binding pocket

203 a dominant negative approach against myosin V, spine synapses became stronger compared to controls.

204 analytical and numerical modeling of myosin V diffusion and stepping.

205 motor, kinesin-1, and an actin motor, myosin-V, are essential for osk mRNA posterior localization.

206 Na(V)1.7(-/-) showed substantial scratch reduction mainly t

207 identified, in adult cardiac myocytes, a Na(V)1.5 subpopulation in close proximity to subjacent subs

208 covery and characterization of ST-2262, a Na(V)1.7 inhibitor that blocks the extracellular vestibule

209 A Na(V)Sp1-specific S4-S5(L) peptide, containing the residues

210 ~ +75 mV), in contrast to other TPCs and Na(V) channels that activate between -20 and 0 mV.

211 sequently, multiple Na(V)1.7-specific and Na(V)1.8-specific blockers have undergone clinical trials,

212 sodium channel isoforms, Na(V)1.1-1.6 and Na(V)1.8.

213 o their mammalian counterparts, bacterial Na(V) channels possess a simpler, fourfold symmetric struct

214 thetics ambroxol and lidocaine block both Na(V)1.7 and NaChBac but affect activation and inactivation

215 tic gain-of-function and loss-of-function Na(V)1.7 mutations have been identified in select individua

216 of the monovalent peptides for the human Na(V)1.4 channel.

217 These findings implicate Na(V)1.7 as a key pharmacotherapeutic target for the treatm

218 nexpected stimulus-dependent diversity in Na(V) channel-mediated itch signalling.

219 Ms structure, induced both an increase in Na(V)Sp1 current density and a negative shift in the activa

220 ral voltage-gated ion channels, including Na(V), Ca(V), and K(V) channels.

221 ng domain targeting toxin BDS-I increases Na(V)1.7 but decreases NaChBac peak currents.

222 over off-target sodium channel isoforms, Na(V)1.1-1.6 and Na(V)1.8.

223 Consequently, multiple Na(V)1.7-specific and Na(V)1.8-specific blockers have under

224 Recently CaM was found to engage part of Na(V) 1.5 that is required for channel inactivation with hi

225 ncy, selectivity, and binding kinetics of Na(V) channel ligands.

226 linical development, and the targeting of Na(V)1.9, although hampered by technical constraints, might

227 e Drosophila, para Despite being the only Na(V) channel in the fly, we show that only 23 +/- 1% of ne

228 and subcellular localization of the sole Na(V) channel in both male and female Drosophila, para Desp

229 d to interact with S6(T) according to the Na(V)Ms structure, induced both an increase in Na(V)Sp1 cur

230 Adaptations in nCa(V) confer unusually sensitive, voltage-dependent inactiv

231 We show that the 26 co-occurring V. cholerae lineages continuously compete for limited sp

232 The potent neutralization activity of V(H)-Fc ab8 combined with good developability properties

233 erials whereby an increased concentration of V(O) sites correlates with a superior OER activity.

234 Concentrations of V and Cr(VI) co-exceeded health recommendations from the

235 martphone-based PD platform for detection of V. cholerae.

236 the archazolids as well as the evaluation of V-ATPases as a novel and powerful class of anticancer ta

237 T-dependent cholera toxin synthesis genes of V. cholerae c2-HDA significantly repressed invasion gene

238 lele, with subsequent feedback inhibition of V(D)J recombination on the other allele.

239 This study identifies mechanisms of V-ATPase assembly and biogenesis that rely on the integr

240 light-organ crypts, where the population of V. fischeri cells resides.

241 resulted in a prolonged culturable state of V. cholerae in artificial sea water at 4 degrees C, wher

242 ion of the oxygen-deficient vanadium oxide, [V(6)O(6)(OC(2)H(5))(12)](1-), was confirmed via independ

243 ition of (18)F-FACBC PET derived parameters (V(T), SUV) to DWI and RAFF derived parameters did not im

244 including the nepenthesin loop of plasmepsin V and a histidine in place of a catalytic aspartate in p

245 aximum out-of-plane piezoresponse is 0.56 pm V(-1) , which is as strong as that observed in conventio

246 (+) pairs that lead to delocalized polaronic V(3+) and V(5+) cation states.

247 de macrolides, which present the most potent V-ATPase inhibitors known to date.

248 Well-defined solid sources of NH(4)Pu(V)O(2)CO(3)(s) were placed in two 5-L lysimeters contain

249 These Pu(V) sources exhibited significantly greater migration tha

250 An 80-min scan was sufficient to quantify V (T) and BP (ND) The test-retest study showed excellent

251 t systemic K(ATP) channel inhibition reduces V O(2) max and critical speed during treadmill running i

252 Mutating region V severely disrupted function without affecting subcellu

253 n, calcium-binding proteins), pH regulation (V-type proton ATPase), and inorganic carbon regulation (

254 X-ray absorption spectroscopy revealed V(V) reduction to V(IV) and formation of bidentate corne

255 well-established that mutations in the LC's V(L) domain are important prerequisites, the mechanisms

256 Treatment with S/V was well-tolerated.

257 sm in cultivated Vitis vinifera ssp. sativa (V. vinifera).

258 We then use seasonal V(cmax,25C) field measurements from 10 sites across dive

259 mpounds from the approved oncology drugs set V library were found to exhibit anticlostridial activity

260 We describe two genetically similar V(H)6-1 bnAb clonotypes from the same individual that ex

261 ecy) in wild Vitis vinifera ssp. sylvestris (V. sylvestris) to hermaphroditism in cultivated Vitis vi

262 cid substitutions observed at positions T91A/V, S195D and M217T in relation to the RotaTeq vaccine we

263 evious work in our laboratory indicated that V. cholerae OmpR functioned as a virulence regulator thr

264 The V(DD)J recombination is currently viewed as an aberrant

265 ated with B cell diversification such as the V(D)J rearrangement process.

266 c roles of the three ion pairs formed by the V(O) defect, including Cu(1+) -Ti(4+) , Ti(3+) -Ti(4+) a

267 e, we investigate whether shared SHMs in the V and J segments of the BCR can be leveraged along with

268 the expanded NSC pool was maintained in the V-SVZ until old age.

269 stinal tract and which are substrates of the V. cholerae RND efflux systems.

270 rch, hopefully stimulating the growth of the V. natriegens research community.

271 t -BMF(lim)) by determining the ratio of the V.Z-products of undigested and digested food.

272 rgy of HCOO* protonation is decreased on the V(O) -rich N-SnO(2) NS, thus enhancing HCOO(-) selectivi

273 portions but with minor differences in their V regions have been demonstrated to interact with FcRn w

274 ective and diffusive O(2) delivery, and thus V O(2) , especially within fast-twitch oxidative skeleta

275 ther to facilitate or to impose a barrier to V(D)J recombination.

276 d long-term outcomes of severe, grade III to V irAE-N at a tertiary care center over 6 years.

277 of R(d) to growth temperature via a link to V(cmax) , and compare predictions to a global set of mea

278 tion spectroscopy revealed V(V) reduction to V(IV) and formation of bidentate corner-sharing surface

279 lsifier, hindered the polymorphic form IV-to-V transition.

280 ator protein total distribution volume (TSPO V(T)), measured with positron emission tomography, mainl

281 U(V) persists on the surface of magnetite and is further r

282 ce-associated U(VI) to form pentavalent U, U(V).

283 Cu, K, Mn, Mo, Na, Ni, P, Pb, Th, Tl, Sb, U, V, Y and Zn) in 73 commercial products marketed in Spain

284 report hydrogenolysis of a terminal uranium(V)-nitride under mild conditions even though it is elect

285 ve either an ART-only (n=30) or an ART + V + V (n=30) regimen; all 60 participants completed the stud

286 e concentration of surface oxygen vacancies (V(O)) in these materials whereby an increased concentrat

287 ctive dopant to modulate the oxygen vacancy (V(O) ) concentration and Ti(3+) formation, which markedl

288 heterojunctions and creating oxygen vacancy (V(O)) in spinel NiCo(2)O(4).

289 ature with a molecular catalyst of vanadium (V)-oxo dimer.

290 g about 100 MHz ac frequencies (with varying V(rms) amplitudes), our method generates an electrotherm

291 mputed over a wide range of shear velocities V.

292 enotypic variation between insect virulence (V) and the mutualistic (M) support of nematode reproduct

293 orrelation between fractional plasma volume (V(p)), a parameter derived from DCE perfusion MRI, and h

294 , mast cell-deficient (W/W(V)) mice, and W/W(V) mice given injections of mast cells derived from wild

295 Reconstitution of mast cells in W/W(V) mice restored the visceral hypersensitivity response.

296 Ptgs2(Y385F) mice), mast cell-deficient (W/W(V)) mice, and W/W(V) mice given injections of mast cells

297 ICCs were moderate between WISC-V tasks (0.663), and relatively modest between NIH Toolb

298 robactin production lose in competition with V. harveyi.

299 uality-adjusted life year (QALY) gained with V-MMRV; and from pound 9,220 to pound 27,101 per QALY ga

300 Aqueous vanadate (H(x)V(V)O(4)((3-x)-)((aq))) concentrations are often control