ライフサイエンスコーパス: VEP

1 VEP acuity in the LCP-supplemented group was significant

2 VEP amplitude was a monotonically increasing function of

3 VEP amplitude was measured as a function of the size of

4 VEP amplitude, RNFL thicknesses provided by Cirrus and S

5 VEP is a method that can be used to assess brain functio

6 VEP latencies at week 16 were shorter in erythropoietin-

7 VEP latencies were found to decrease significantly durin

8 VEP latency and visual function tests that capture optic

9 VEP latency, PERG measurements, and macular thicknesses

10 VEP may be useful for early diagnosis of glaucoma.

11 VEP P100 latency was found superior to color vision and

12 VEP results can be predictive of visual recovery in trau

13 VEP Vernier acuity and grating acuity develop at differe

14 VEP Vernier acuity remains strikingly immature throughou

15 VEP waveforms were nonrecordable in a few subjects or ex

16 VEP-G2P(DD) shows a sensitivity/precision of 97.3%/33% f

17 VEPs can be valuable in diagnosing optic neuropathies, n

18 VEPs in response to pattern-reversing checkerboards were

19 VEPs recorded in humans showed significant phase locking

20 VEPs to checks and ZD stimulation were similar.

21 VEPs were analyzed at the pattern reversal rate using sp

22 VEPs were less pronounced in the infarction model, provi

23 VEPs were recorded from the visual cortices of five maca

24 VEPs were recorded in 16 healthy adults in response to a

25 VEPs were recorded in 35 infants between 5 and 15 months

26 VEPs were recorded to the onset of luminance increments

27 VEPs were repeated in nine subjects with 20/40 or better

28 Here, we investigate the performance of 35 VEPs in the discrimination between pathogenic and putati

29 r response than 10-s stimuli (PhNR-S = 7.5%; VEP-S = 6.2%).

30 ovea, (3) nystagmus intensity, (4) BCVA, (5) VEP asymmetry, (6) skin pigmentation, and (7) hair pigme

31 ith a trend of 60-s stimuli (PhNR-S = 11.6%; VEP-S = 10.4%) producing a larger response than 10-s sti

32 ut procedure of Sutter was used to obtain 60 VEP responses to a scaled checkerboard pattern.

33 ng approach, assessing the performance of 97 VEPs using missense DMS measurements from 36 different h

34 re and 1 month after surgery, visual acuity, VEP, PERG, and 3 repetitions of scans using the RNFL and

35 The presence of cataracts affects VEP amplitude, RNFL, and macular measurements performed

36 At 1 month of age, VEP implicit times were somewhat shorter in Akt-DD trans

37 Beyond 6months of age, VEP latencies were consistently delayed in Akt-DD transg

38 other eye resulted in an increased amplitude VEP response.

39 Resting-state activity and VEP yielded mixed results.

40 Age, severity of amblyopia, and VEP amplitudes of positive peak (P)100, P1, and negative

41 ation instability reduces VEP amplitude, and VEP reliability is therefore low in this important patie

42 tory and visual evoked potentials (BSAEP and VEP) and in sleep patterns.

43 crucial role in the generation of BSAEP and VEP, as well as in sleep disturbances.

44 Magnetic resonance imaging and VEP confirmed absence of decussation of retinofugal fibe

45 During the follow-up, the PERG and VEP differences observed with respect to baseline were e

46 The PERG and VEP mean values observed in LHON eyes were compared (1-w

47 baseline, in all LHON eyes for each PERG and VEP parameter (amplitude and implicit time), the 95% con

48 At baseline, mean values of PERG and VEP parameters detected in the LHON group were significa

49 s showed unmodified (within 95% CL) PERG and VEP values, and mean absolute values of these measures w

50 tingly, while behavioral, physiological, and VEP effects were comparable at immediate and delayed ass

51 Both PhNR-S and VEP-S significantly increased ONH blood flow (p < 0.001)

52 visual electrophysiology stimuli (PhNR-S and VEP-S) induced a demonstrable increase in ONH and peripa

53 Three assays (resting state, faces task, and VEP) show promise in terms of acquisition rates and cons

54 t the development of optomotor threshold and VEP acuity can occur in an experience-independent manner

55 ng methods for accurately estimating VES and VEP is important in designing trials for diseases with a

56 tical auditory evoked potentials (CAEPs) and VEPs and a decrease in both REM sleep and SWS.

57 ic visual functions (motion perception), and VEPs were assessed repeatedly.

58 as a significant factor influencing apparent VEP performance, often leading to inflated AUROC values

59 were acquired with a commercially available VEP unit using standard electrode recording techniques.

60 Time-averaged VEP central field latency was shorter by 6.1 ms (95% CI

61 any of the baseline, 1 year or time-averaged VEP variables.

62 A strong correlation was found between VEP latencies and motion perception.

63 The relation between VEP latency and blood glucose was determined.

64 The within-subject relationship between VEP Vernier acuity and grating acuity follows the same d

65 d impaired P1/N2 amplitudes and latencies by VEP were seen in middle and late EAE groups.

66 , and OCT) and demyelination (as measured by VEP).

67 It was possible to record a clear VEP from Akt-DD mice at all ages examined.

68 crucial limitations when used for comparing VEP performance across different genes.

69 Our results suggest that comparing VEP performance against diverse functional assays repres

70 f the main positive component of the control VEP demonstrated around 2-fold prolongation during activ

71 Using only human genetic data VEP-G2P performs well compared to other freely-available

72 Delayed VEP latency, which depends on technical and methodologic

73 Either a delayed VEP is not a good indicator of damaged, as opposed to de

74 The delayed VEP responses of the patients with MAR to luminance incr

75 High-density VEPs evoked by a contrast reversing checkerboard were co

76 animals, we measured NMDA receptor-dependent VEP potentiation ipsilateral to the NDE during MD, which

77 rocessing can be detected with the dichoptic VEP method we describe.

78 Many different VEPs have been released, and there is tremendous variabi

79 monstrated by the PR-VEP latencies, our DRCD-VEP data show that the visual cortex is remarkably ready

80 nse to dynamic random dot correlograms (DRDC-VEP), appears to be at around the same time after birth

81 To ensure attentiveness during VEP assessments, subjects responded with a button press

82 rally presented IC stimuli resulted in early VEP modulation (88-100 msec) over lateral-occipital (LOC

83 primary analysis evaluated vaccine efficacy (VEP) as the percent reduction (vaccine vs placebo) in cu

84 Estimated VEP (mITT) was15.8% (-21.9, 41.8) at 60 months postinfec

85 In this study, we examined VEP plasticity in healthy control subjects and patients

86 h better outcomes across all measures except VEP conduction time; male sex, which predicted worse out

87 DeMAG improves performance over existing VEPs by reaching balanced specificity (82%) and sensitiv

88 In a second experiment, VEPs were obtained using slowed m-sequences (8 and 16 vi

89 Deprived-eye VEPs were no larger in the injected hemisphere than in t

90 ion speed were highly correlated with faster VEP latencies.

91 using full-field visual evoked potential (FF-VEP) versus the unaffected fellow eye at baseline.

92 inform the design of sustainability-focused VEPs and future research to understand the causal pathwa

93 However, VEP testing requires stable fixation, which is impossibl

94 Like the monkey neurons, human VEPs more typically phase locked to stimuli containing s

95 ected fellow eyes, no significant changes in VEP parameters or functional indices were observed.

96 th vernier offsets resulted in a decrease in VEP amplitude for both horizontal and vertical dispariti

97 RTT patients exhibited a similar decrease in VEP amplitude that was most striking in the later stages

98 imulus-locked outcomes were also observed in VEPs across LFP and EEG recordings.

99 howing decreased SEP amplitude and increased VEP amplitude.

100 mately 8.8 Hz, 2 minutes) designed to induce VEP potentiation.

101 s shorter latency than visible light-induced VEP, its amplitude increases with peak irradiance and pu

102 Shock-induced VEP is essential in extinguishing fibrillation but can r

103 Shock-induced VEPs may reinduce arrhythmias via a phase-singularity me

104 restored visual evoked potential latencies (VEP).

105 Sixty local VEP responses were recorded simultaneously.

106 hmic framework bridges the gap between MAVEs/VEPs and clinically actionable variant classification.

107 The MO VEP did not change in peak time (P >= 0.0747) or interpe

108 Motion-onset visual evoked potentials (MO VEPs) are robust to dioptric blur when low contrast and

109 Thus, we demonstrated that MO VEPs evoked by checkerboard, structure containing high s

110 41% of children, whereas an abnormal motion VEP, Worth 4-dot, or positive 4-PD base-out prism respon

111 lly strabismic monkeys had asymmetric motion VEP responses: AI = 0.57 +/- 0.22 in the Delayed Repair

112 The concordance between motion VEP symmetry and normal fusional vergence was significan

113 naturally strabismic monkeys also had motion VEP asymmetries of equivalent magnitude when tested usin

114 Delayed repair causes permanent motion VEP maldevelopment.

115 ric (F2) frequency component from the motion VEP.

116 Motion VEPs, random dot stereopsis, and bifoveal fusion were me

117 ths after the removal of the goggles, motion VEPs to horizontally oscillating grating stimuli were re

118 the development or maldevelopment of motion VEPs.

119 tric motion visual evoked potentials (motion VEPs).

120 ly Repair monkeys exhibited symmetric motion VEPs (AI < 0.25).

121 erebral cortex, measured as symmetric motion VEPs.

122 luding neuroaxonal loss (as measured by MRI, VEP, and OCT) and demyelination (as measured by VEP).

123 Multifocal VEPs were recorded from both eyes of six normal subjects

124 % to 70% of first-phase voltage) produced no VEP, because of an asymmetric reversal of the first-phas

125 and recommendations for the release of novel VEPs.

126 uction number (R0) and accurate estimates of VEP for higher R0 values.

127 riment 1: although adults showed evidence of VEP amplitude alternations between the eyes for cross-or

128 jor challenges in clinical interpretation of VEP scores persist, highlighting the need for further re

129 The amplitudes and latencies of VEP waveform components were quantified, and were relate

130 r this period, a significant prolongation of VEP latencies occurred in the asymptomatic fellow eye, a

131 study which showed significant shortening of VEP latencies between 6 months and 3 years without signi

132 Positive values of VEP resulted in a prolongation of the action potential d

133 that LTP similarly enhances the amplitude of VEPs, but in a way that generalizes across multiple stim

134 to navigate the selection and application of VEPs.

135 the effects of medication and mood state on VEP plasticity.

136 Multifocal electroretinogram (ERG) or VEP can provide an objective assessment of visual field

137 he mean latencies and amplitudes of the P100 VEP and P3 ERP showed comparable values (p > 0.10 in all

138 In addition, a new pairwise, VEP-centric approach mitigates the impact of missing pre

139 e was observed for LM- or achromatic-pathway VEP latency in subjects with diabetes.

140 The S-pathway VEP latency was correlated significantly with blood gluc

141 le of resolution (logMAR) units, and pattern VEP.

142 referred to as a virtual electrode pattern (VEP).

143 resulted in virtual electrode polarization (VEP).

144 changes in virtual electrode polarizations (VEPs) and propagation delay through the peri-infarct zon

145 ants by using sweep visual evoked potential (VEP) acuity as the functional outcome.

146 suggested that the visual evoked potential (VEP) amplitude can vary with stimulus duration.

147 ION, with a loss of visual evoked potential (VEP) amplitude that persisted in the long term.

148 The P100 visual evoked potential (VEP) and P3 event-related potential (ERP) were compared

149 investigate if the visual evoked potential (VEP) could be used as an unbiased, quantitative biomarke

150 Abnormal visual evoked potential (VEP) findings of increased latencies, reduced amplitudes

151 ade tracers into the vagus evoked potential (VEP) focus in Pf of macaque monkeys.

152 t plasticity of the visual evoked potential (VEP) induced by repeated visual stimulation might reflec

153 ssive shortening of visual evoked potential (VEP) latencies and to determine whether this is associat

154 then shortening of visual evoked potential (VEP) latencies in optic neuritis in MS may identify demy

155 ss with a prolonged visual evoked potential (VEP) latency suggests that acute and persistent demyelin

156 cal examination and visual evoked potential (VEP) measurement, each patient had their optic nerves im

157 e recorded, and the visual evoked potential (VEP) peak-to-peak amplitude (N70 and P100) was calculate

158 than for standard visually evoked potential (VEP) recordings, the eVEP has proven to be a reliable to

159 function, the sweep visual evoked potential (VEP) was used to evaluate cortical responses to grating

160 ionship between the Visual Evoked Potential (VEP), a component of the electroencephalogram elicited b

161 sual acuity (BCVA), visual evoked potential (VEP), and grading of skin and hair pigmentation were use

162 assessed using the visual evoked potential (VEP).

163 e EEG (rsEEG), the visual evoked potentials (VEP) and the visual P300 (P3) from 16 healthy subjects (

164 modulations in the visual evoked potentials (VEP) recorded.

165 and measurement of visual evoked potentials (VEP) was performed on each patient.

166 e basis of lesion, visual evoked potentials (VEP), and neuroimaging evidence, others contend that IC

167 inogram (PERG) and visual evoked potentials (VEP), in response to 60' and 15' checks visual stimuli,

168 ing enhancement of visual evoked potentials (VEP).

169 ural feedback in visually evoked potentials (VEP).

170 ured were visual evoked cortical potentials (VEPs) and multifocal (mf)ERGs, with both a standard fast

171 on of prototypical visual evoked potentials (VEPs) across local field potential (LFP) wires and EEG s

172 tials (spikes) and visual-evoked potentials (VEPs) align with the video impulses, particularly when h

173 rtical plasticity [visual evoked potentials (VEPs) and cortical current density].

174 cuity with sweep visually evoked potentials (VEPs) and for optotype acuity (Landolt C) with behaviora

175 stinjection, and visually evoked potentials (VEPs) and single-cell activity were recorded.

176 n the magnitude of visual evoked potentials (VEPs) elicited in layer 4 (L4) by familiar phase-reversi

177 Visual Evoked Potentials (VEPs) following optic neuritis (ON) remain chronically p

178 tern-reversal (PR) visual evoked potentials (VEPs) have been found to be a sensitive indicator of vis

179 ual behavior and visually evoked potentials (VEPs) in binocular visual cortex of the same mice before

180 tagmus (OKN) and visually evoked potentials (VEPs) in one direction than to those in the opposite dir

181 Cortical visually evoked potentials (VEPs) in response to stimuli designed to selectively act

182 of neurons, and visually-evoked potentials (VEPs) in response to task light cues, while increasing c

183 (LGN) can modulate visual evoked potentials (VEPs) in the intact large animal; and to study the impac

184 RECENT FINDINGS: Visual evoked potentials (VEPs) may be useful as an objective measurement of refra

185 In awake mice, visual evoked potentials (VEPs) recorded in layer 4 of binocular visual cortex und

186 vision based on visually evoked potentials (VEPs) recorded in rats implanted with photovoltaic subre

187 Visual evoked potentials (VEPs) to check reversal (163-18 arc min) and onset of si

188 Steady-state visual-evoked potentials (VEPs) to contrast reversing gratings were recorded over

189 Visual evoked potentials (VEPs) were measured in 10 patients with RP and in 10 age

190 Visual evoked potentials (VEPs) were recorded over three occipital sites to the on

191 Structural MRI, visual evoked potentials (VEPs), and optical coherence tomography (OCT) were used

192 menting changes in visual evoked potentials (VEPs), neuronal spiking activity, and oscillations in th

193 etinography (ERG), visual evoked potentials (VEPs), spectral-domain optical coherence tomography (OCT

194 by spatial sweep visually evoked potentials (VEPs).

195 on perception, and visual evoked potentials (VEPs).

196 visual field, and visual evoked potentials (VEPs).

197 MD on its own potentiated VEPs contralateral to the NDE during MD and shifted ocul

198 For PR VEP, the checkerboards were reversed in terms of their c

199 In contrast, for PR VEP, the results showed a decrease in interpeak amplitud

200 amination that included pattern reversal (pr)VEP (2013-2014) and history of ICH based on direct measu

201 PR-VEP latency is not affected by premature birth, demonstr

202 PR-VEP responses were recorded from 81 adults and 137 infan

203 ked response to pattern reversal stimuli (PR-VEP).

204 sual pathway, clearly demonstrated by the PR-VEP latencies, our DRCD-VEP data show that the visual co

205 ed with OCT, OCT-angiography (OCT-A), and PR-VEPs.

206 N70 and P100 latencies were assessed from PR-VEPs.

207 attern-reversal visual evoked potentials (PR-VEPs) in a geographically defined population-based cohor

208 ll-type-specific variant effect predictions (VEPs) as functional annotations.

209 ctices followed by Variant Effect Predictor (VEP) and Annovar.

210 ed using Ensembl's Variant Effect Predictor (VEP) and Jannovar.

211 nted as an Ensembl variant effect predictor (VEP) plugin, COCOS captures amino acid sequence alterati

212 tension to Ensembl Variant Effect Predictor (VEP), VEP-G2P was used to filter both disease-associated

213 and computational variant effect predictors (VEPs) into ACMG/AMP evidence strengths.

214 Computational Variant Effect Predictors (VEPs) provide valuable evidence in variant assessment, b

215 tations, known as variant effect predictors (VEPs), are widely used in the assessment and interpretat

216 At presentation, VEPs were a more sensitive indicator of optic pathway da

217 mal defibrillation waveforms did not produce VEPs because of an asymmetric effect of phase reversal o

218 s with a strong second phase (>70%) produced VEPs of reversed polarity.

219 ng-edge voltage of the first phase) produced VEPs similar to monophasic shocks.

220 A voluntary environmental program (VEP), such as SolSmart, can encourage local governments

221 ysed using PLINK v1.09, SPSS, R programming, VEP tool, and FUMA GWAS tool.

222 Contrary to previous reports, prolonged VEP delays were present in a minority of patients with g

223 The authors recorded VEPs from 57 healthy full-term infants and 4 adults.

224 We recorded VEPs in a model in which there is complete demyelination

225 We recorded VEPs in response to strobe flash ganzfeld stimuli presen

226 itude and static visual functions recovered, VEP latency remained significantly prolonged, and motion

227 pearance of vernier onset-offset, and reduce VEP amplitude for both horizontal and vertical dispariti

228 ptic nerve pallor, whereas all had a reduced VEP in 1 or both eyes.

229 Fixation instability reduces VEP amplitude, and VEP reliability is therefore low in t

230 or testing, in vivo visual evoked responses (VEP) and single-unit cortical recordings.

231 We recorded pattern-reversal VEPs in Mecp2 heterozygous female mice and 34 girls with

232 ings indicate that the latency of the rodent VEP is sensitive to changes mediated by the increased ex

233 The modulation block resulted in significant VEP plasticity in healthy control subjects.

234 ngs support the introduction of standardized VEP analysis in clinical and research settings to probe

235 nst temporal frequency (TF) for steady state VEP measurements as well as from the transient P1 peak.

236 The steady state VEPs were analyzed with discrete Fourier transforms to o

237 Step VEP acuity was 0.46 (95% CI: -0.13 to 1.06) logMAR units

238 Step VEP and GAC acuities correlated highly (r(2) = 0.60, P =

239 e equation: acuity(GAC) = (0.9 x acuity(step VEP)) - 0.37.

240 cuity assessed five times with both the step VEP and with Glasgow Acuity Cards (GAC).

241 The step VEP provides a rapid, objective means of estimating visu

242 or a visual evoked potential-like stimulus (VEP-S)-each presented in separate 10- and 60-s epochs.

243 Shocks producing strong VEPs resulted in postshock reentrant arrhythmias via a m

244 Sweep VEP acuity was the primary outcome.

245 Sweep VEP data were obtained from 16 healthy observers under b

246 Assessments of optotype acuity and sweep VEP acuity revealed amblyopic deficits in both pseudopha

247 t there is not a significant change in sweep VEP acuity estimates over an 8-second stimulus presentat

248 requency, contrast, and vernier offset sweep VEP tuning functions were measured at 5 to 7 months' cor

249 The sweep VEP acuities for the 16 subjects did not change signific

250 mates obtained by extrapolation of the sweep VEP are altered by this adaptation effect.

251 icantly affect the clinical use of the sweep VEP.

252 avioral testing with optotypes or with sweep VEPs.

253 ne efficacy against progression to symptoms (VEP).

254 This study provides proof-of-concept that VEP amplitude (and therefore prognostic reliability) can

255 Thus, it has been directly confirmed that VEP latencies reflect the myelin status of the optic ner

256 The VEP consists of large adjacent areas of strong positive

257 The VEP may be used as a biomarker to index the neurobiologi

258 The VEP peak-to-peak amplitude was reversibly suppressed rel

259 The VEP plasticity was significantly impaired in patients wi

260 The VEP was strongest lateral and anterior to the habenuloin

261 Immediately preceding the IC effect, the VEP modulated with inducer eccentricity--the configurati

262 results suggested that N1-P2 complex in the VEP could be a neural marker for stereopsis and fNIRS de

263 e faces task latency of the P1 and N170, the VEP amplitude of N1 and P1, and resting alpha power.

264 As animals recovered neurologically, the VEP latencies decreased in association with complete rem

265 g experiences and variation in the P1 of the VEP at 6 months, yet caregiving experience do explain va

266 Current-source density (CSD) analysis of the VEP depth profile shows augmentation of short latency cu

267 previous SRP occludes TBS-induced LTP of the VEP evoked by the experienced stimulus, but not by unfam

268 the first positive component, the P1, of the VEP in relation to cognitive scores on the Mullen Scales

269 We find that the P1 amplitude of the VEP is related to concurrent cognitive performance in ea

270 ower recovery from the principal peak of the VEP response that was impacted by MECP2 mutation type.

271 plitudes for the P1 and N1 components of the VEP that were specific to Arabic numerals and to dot con

272 lay and instead increased the latency of the VEP waveform.

273 ssessment explain variation in the P1 of the VEP.

274 ency delays on the mfVEP test but not on the VEP test, presumably due to the mfVEP's ability to detec

275 Our tools, in particular the VEP, have been improved significantly through integratio

276 eat kinase using PFE360 failed to rescue the VEP delay and instead increased the latency of the VEP w

277 Unlike the VEP results, the mfVEP revealed a significant increase i

278 We sought to determine whether the VEP may be responsible for defibrillation failure by cre

279 The authors tested whether the VEP responses were asymmetrical because of abnormal eye

280 concentration (HbO) was correlated with the VEP amplitude during the checks and HD presentations.

281 The VEPs did not have sufficient power to reliably distingui

282 The VEPs obtained in our two volunteers with implants had a

283 The VEPs recorded during surface eye stimulation are similar

284 The VEPs were compared at sub- and supra-threshold stimulati

285 The VEPs were evoked by checkerboard reversal stimulation be

286 The VEPs were recorded with a 15' and 60' reversing checkerb

287 source of the asymmetrical amplitudes of the VEPs, and the visual cortex is at least one source respo

288 Their VEP responses showed a marked delay to increments but on

289 These VEP results are in general agreement with recent psychop

290 This VEP technique provides a rapid estimate of Vernier acuit

291 ty is a sensitive measure of amblyopia, this VEP test may be useful in the future to identify amblyop

292 Short-duration transient VEP objectively identified decreased visual function and

293 ficant delay in the latency of the transient VEPs from the affected side of the SC in late stages of

294 and 38 control eyes (19 subjects) underwent VEP, mfVEP, and visual field testing.

295 n to Ensembl Variant Effect Predictor (VEP), VEP-G2P was used to filter both disease-associated and c

296 15 control children underwent swept vernier VEP acuity testing accompanied by a swept motion control

297 o investigate the specificity of the vernier VEP as a measure of positional acuity, evaluating the po

298 The vernier VEP paradigm, when applied in the manner described, can

299 es may be useful for identifying genes where VEP predictions are likely to be more or less reliable.

300 Whereas VEP amplitude and static visual functions recovered, VEP