ライフサイエンスコーパス: Vibrio cholerae

1 cSiaPQM, a sialic acid TRAP transporter from Vibrio cholerae.

2 ra, a severely dehydrating disease caused by Vibrio cholerae.

3 ce gene expression and human colonization by Vibrio cholerae.

4 ation is critical for the infection cycle of Vibrio cholerae.

5 common pathogens, Staphylococcus aureus and Vibrio cholerae.

6 used by certain strains of serogroup O1/O139 Vibrio cholerae.

7 (PLEs) are bacteriophage satellites found in Vibrio cholerae.

8 , and the opposite occurs for the pathogenic Vibrio cholerae.

9 peG enzyme from the important human pathogen Vibrio cholerae.

10 e (Na(+)-NQR) is the main ion transporter in Vibrio cholerae.

11 spectrometry (MS) for the identification of Vibrio cholerae.

12 a bEBP that controls flagellar synthesis in Vibrio cholerae.

13 s one of the major porins of human pathogen, Vibrio cholerae.

14 l residue of Ogawa O-polysaccharide (OPS) of Vibrio cholerae.

15 nd to control chemotaxis and colonization by Vibrio cholerae.

16 kers involved in relevant diseases caused by Vibrio cholerae.

17 pathway within the cholera-causing microbe, Vibrio cholerae.

18 ring by optimizing natural transformation in Vibrio cholerae.

19 urface of the Gram-negative aquatic pathogen Vibrio cholerae.

20 eptor cluster in Rhodobacter sphaeroides and Vibrio cholerae.

21 nd nucleoside drug selectivity of a CNT from Vibrio cholerae.

22 ated platform for ectopic gene expression in Vibrio cholerae.

23 ics of homologous recombination during NT in Vibrio cholerae.

24 es from Clostridium perfringens (CpNanI) and Vibrio cholerae.

25 n the human pathogens Bacillus anthracis and Vibrio cholerae.

26 nonpandemic forms of the intestinal pathogen Vibrio cholerae A nontoxic fragment of the first 386 aa

27 em mediates the export of >/= 20 proteins in Vibrio cholerae, a human pathogen that is indigenous to

28 rs of evolutionary fitness of pathogens like Vibrio cholerae, a mounting threat to global heath.

29 the most well studied AasS to date, and from Vibrio cholerae, a pathogenic model.

30 In Vibrio cholerae, a type IVa pilus (T4aP) is thought to f

31 rosophila model, Fast et al. identified that Vibrio cholerae acts to inhibit epithelial renewal throu

32 Vibrio cholerae, an etiological agent of cholera, circul

33 gase with and without substrate, full-length Vibrio cholerae and Fusobacterium nucleatum glycine ribo

34 The pathogens Vibrio cholerae and Haemophilus influenzae use tripartit

35 The AB(5) toxins cholera toxin (CT) from Vibrio cholerae and heat-labile enterotoxin (LT) from en

36 icity of known bacterial pathogens including Vibrio cholerae and Neisseria meningitidis.

37 Here, we show that biofilms of the pathogens Vibrio cholerae and Pseudomonas aeruginosa can induce la

38 Typhimurium)] and two extracellular (Vibrio cholerae and Staphylococcus aureus) bacterial pat

39 in vivo competition experiments between T6S+ Vibrio cholerae and T6S-sensitive Escherichia coli.

40 s of the non-invasive Gram-negative pathogen Vibrio cholerae and the invasive pathogen Salmonella ent

41 Within 24 h of exposure, strains of Vibrio cholerae and Vibrio alginolyticus were able to di

42 stis), smallpox (Variola virus) and cholera (Vibrio cholerae) - and for three equally important endem

43 rming units of rotavirus, <1 x 10(-4) CFU of Vibrio cholerae, and <9 x 10(-6) Cryptosporidium oocysts

44 In the cholera pathogen, Vibrio cholerae, and in related species, secondary chrom

45 oS is critical for natural transformation in Vibrio cholerae, and it was previously presumed to exert

46 terobactin hydrolysis products by C. jejuni, Vibrio cholerae, and other bacteria with homologous peri

47 ion types of lipid A from Escherichia coli, Vibrio cholerae, and Pseudomonas aeruginosa using an Orb

48 pathogens including Neisseria meningitidis, Vibrio cholerae, and Salmonella enterica harbor these pr

49 egulatory circuit was recently identified in Vibrio cholerae, and the H-NOX protein has been spectros

50 scherichia coli, Pseudomonas aeruginosa, and Vibrio cholerae, and the plant pathogen Xanthomonas camp

51 as aeruginosa, Stenotrophomonas maltophilia, Vibrio cholerae, and Yersinia enterocolitica T2S-express

52 tems of enterotoxigenic Escherichia coli and Vibrio cholerae are among the simplest of Type IV pilus

53 terium diphtheriae, Salmonella enterica, and Vibrio cholerae, are infected with lysogenic bacteriopha

54 enomic framework for identifying VAPs, using Vibrio cholerae as a model.

55 Here, using Vibrio cholerae as our model organism, we show that duri

56 Here, using Vibrio cholerae as our model system, we combine mechanic

57 Here we demonstrate that the Vibrio cholerae autoinducer (S)-3-hydroxytridecan-4-one,

58 s, including other diarrheagenic E. coli and Vibrio cholerae bacteria, suggesting that this mucin-deg

59 The toxigenic bacterium Vibrio cholerae belonging to the O1 and O139 serogroups

60 secreted by the type II secretion system in Vibrio cholerae, belongs to this subfamily.

61 ble live single-cell resolution imaging of a Vibrio cholerae biofilm as it develops from one single f

62 Vibrio cholerae biofilms are hyperinfectious, and biofil

63 microscopy to image all individual cells in Vibrio cholerae biofilms at different stages of developm

64 ructural switch controls the architecture of Vibrio cholerae biofilms by mediating the interactions b

65 Here, by monitoring all individual cells in Vibrio cholerae biofilms during exposure to antibiotics

66 issue, here we present a study of developing Vibrio cholerae biofilms grown on agar substrates in whi

67 d by B. subtilis using the equivalent of the Vibrio cholerae biosynthetic pathway, (2) exogenous nors

68 Current pandemic O1 Vibrio cholerae biotype El Tor is resistant to polymyxin

69 ANR homologs of ETEC and Vibrio cholerae bound to AggR as well as to other member

70 DncV is associated with hyperinfectivity of Vibrio cholerae but has not been found in many bacteria,

71 zation by the major human diarrheal pathogen Vibrio cholerae by degrading the bile salt taurocholate

72 rotein (anti-OmpW) in sensitive detection of Vibrio cholerae by developing an immunosensor based on S

73 tool specimens were collected and tested for Vibrio cholerae by microbiological culture.

74 FlrC, controls motility and colonization of Vibrio cholerae by regulating the transcription of class

75 nt enteric pathogens Salmonella enterica and Vibrio cholerae by repressing AraC-type transcriptional

76 enterica, enterotoxigenic Escherichia coli, Vibrio cholerae, Campylobacter jejuni, norovirus) in coh

77 Vibrio cholerae can enter a viable but non-culturable (V

78 ortant environmentally transmitted pathogen, Vibrio cholerae, can modulate the evolutionary trajector

79 Virulent strains of the bacterial pathogen Vibrio cholerae cause the diarrheal disease cholera by r

80 Vibrio cholerae causes cholera and is the leading cause

81 Vibrio cholerae causes human infection through ingestion

82 Vibrio cholerae causes the human disease cholera by prod

83 Mutations of Y241(Vc) (to A/F/H/S) in the Vibrio cholerae cbb3 eliminate catalytic activity, but a

84 erified bacterial human pathogens, including Vibrio cholerae (cholera) in a 19th century intestinal s

85 RctB, the initiator of replication of Vibrio cholerae chromosome 2 (chr2), binds to the origin

86 lems arising from the circularity of the two Vibrio cholerae chromosomes, chrI and chrII, are resolve

87 ANR homologs of Vibrio cholerae, Citrobacter rodentium, Salmonella enter

88 nships between globally circulating pandemic Vibrio cholerae clones and local bacterial populations.

89 In Vibrio cholerae, ClpV binds the N terminus of TssC withi

90 Vibrio cholerae colonizes intestinal epithelial cells (I

91 Vibrio cholerae colonizes the human terminal ileum to ca

92 Populations of the bacterium Vibrio cholerae consist of dozens of distinct lineages,

93 Vibrio cholerae contains three chemotaxis clusters (I, I

94 The diarrheal pathogen Vibrio cholerae contains three gene clusters that encode

95 The Vibrio cholerae Cpx system was previously found to respo

96 Vibrio cholerae CqsA/S synthesizes and detects (S)-3-hyd

97 l receptor-destroying enzyme (RDE) (II) from Vibrio cholerae culture fluid specifically modulates IgE

98 Vibrio cholerae cytolysin (VCC) is a potent membrane-dam

99 Vibrio cholerae cytolysin (VCC) is a toxin secreted by t

100 rm of the related, but inactive, lectin from Vibrio cholerae cytolysin.

101 Vibrio cholerae cytolysin/hemolysin (VCC) is an amphipat

102 Like many bacteria, Vibrio cholerae deploys a harpoon-like type VI secretion

103 We recently incorporated a Vibrio cholerae diguanylate cyclase into an adenovirus v

104 nstrate detection of DNA coils formed from a Vibrio cholerae DNA target at picomolar concentrations u

105 Detection of a Vibrio cholerae DNA-sequence using an optomagnetic read-

106 -polymerase beta-like superfamily (including Vibrio cholerae DncV), a minimal version of the CRISPR p

107 Vibrio genus, including the enteric pathogen Vibrio cholerae, encode only a single PG amidase, AmiB.

108 The waterborne agent of cholera, Vibrio cholerae, encounters phosphate limitation in both

109 Biofilm formation by Vibrio cholerae facilitates environmental persistence, a

110 Here we report the structure of Vibrio cholerae FadR (VcFadR) alone, bound to DNA, and i

111 to identify the undermethylated sites in the Vibrio cholerae genome for the two DNA methyltransferase

112 igh-throughput sequencing to reconstruct the Vibrio cholerae genome from the preserved intestine of a

113 brio vulnificus, Vibrio parahaemolyticus and Vibrio cholerae, grow in warm, low-salinity waters, and

114 nstability of the Fe(II) form suggested that Vibrio cholerae H-NOX may act as a sensor of the redox s

115 RNAP-LuxR interaction domain is conserved in Vibrio cholerae HapR and is required for activation of t

116 The bipartite genome of Vibrio cholerae harbors sporadic and conserved MGEs that

117 Vibrio cholerae hazards were poorly associated with plat

118 We focus here on Caulobacter crescentus, Vibrio cholerae, Helicobacter pylori, and Campylobacter

119 phenotypes, and, in the major human pathogen Vibrio cholerae, Hfq inactivation has been linked to red

120 We investigated the roles of the Vibrio cholerae high-molecular-weight bifunctional penic

121 f NaDC3 on the basis of the structure of the Vibrio cholerae homolog vcINDY.

122 We also present two structures of the Vibrio cholerae IMPDH in complex with IMP/NAD(+) and XMP

123 Growth of the cholera bacterium Vibrio cholerae in a biofilm community contributes to bo

124 the MshE N-terminal domain (MshEN1-145) from Vibrio cholerae in complex with c-di-GMP at a 1.37 A res

125 The structures of SpeG from Vibrio cholerae in complexes with polyamines and cofacto

126 py to explore biofilms of the human pathogen Vibrio cholerae in conditions mimicking its marine habit

127 robability of achieving zero transmission of Vibrio cholerae in Haiti with current methods of control

128 he energetics of drug extrusion by NorM from Vibrio cholerae in proteoliposomes in which purified Nor

129 e, Yan et al. show that matrix production in Vibrio cholerae increases the osmotic pressure within th

130 one of the porins of Gram-negative bacteria Vibrio cholerae, induces TLR1/2-MyD88-NF-kappaB-dependen

131 ical agent of the diarrheal disease cholera, Vibrio cholerae, infected by ICP1, a phage ubiquitous in

132 The innate immune response to Vibrio cholerae infection is poorly understood, but this

133 in people recovering from food poisoning and Vibrio cholerae infections.

134 Vibrio cholerae inhabits aquatic environments and coloni

135 Vibrio cholerae interacts with many organisms in the env

136 richia coli, Mycobacterium tuberculosis, and Vibrio cholerae is a dimer.

137 Vibrio cholerae is a Gram-negative bacterial pathogen th

138 Vibrio cholerae is a Gram-negative bacterium that persis

139 Vibrio cholerae is a Gram-negative human pathogen and th

140 Vibrio cholerae is a Gram-negative pathogen that can use

141 Vibrio cholerae is a Gram-negative, facultative anaerobi

142 The bacterium Vibrio cholerae is a natural inhabitant of aquatic ecosy

143 Vibrio cholerae is a natural inhabitant of aquatic envir

144 Vibrio cholerae is a natural resident of the aquatic env

145 person, although it is well-recognized that Vibrio cholerae is also capable of growth and long-term

146 Vibrio cholerae is an aquatic microbe that can be divide

147 Vibrio cholerae is an intestinal pathogen that causes th

148 Cell, Alavi et al. report that infection by Vibrio cholerae is blocked by gut microbiome-mediated hy

149 Vibrio cholerae is lethal to the model host Drosophila m

150 Vibrio cholerae is responsible for the diarrheal disease

151 Vibrio cholerae is the bacterium that causes the diarrhe

152 Vibrio cholerae is the causative agent of cholera, a pot

153 Vibrio cholerae is the causative agent of the acute diar

154 Vibrio cholerae is the causative agent of the severe dia

155 Vibrio cholerae is the causative bacteria of the diarrhe

156 Vibrio cholerae is ubiquitous in aquatic environments, w

157 bial resistance by sequencing the genomes of Vibrio cholerae isolates from the epidemic in Yemen and

158 ied in the pathogen and model biofilm-former Vibrio cholerae It is unknown how spatial trajectories o

159 ce mechanism in which the bacterial pathogen Vibrio cholerae jettisons outer membrane proteins and li

160 In contrast, we report here that Vibrio cholerae lacking DacA-1, a PBP5 homologue, displa

161 The Vibrio cholerae MARTXVc toxin delivers three effector do

162 Very little is known about Vibrio cholerae Mlc.

163 ing infection, the human intestinal pathogen Vibrio cholerae must overcome noxious compounds that dam

164 that a successful enteric pathogen, such as Vibrio cholerae, must circumvent.

165 h the core regions in a crystal structure of Vibrio cholerae Na(+)-dicarboxylate transporter VcINDY,

166 21.9; icddr,b AF 16.6%; CI: 14.4, 19.4) and Vibrio cholerae (Nationwide AF 10.2%, CI: 9.1, 11.3; icd

167 An occurrence of Vibrio cholerae non-O1/O139 gastroenteritis in the U.S.

168 From 2000 to 2012, Vibrio cholerae O1 and Shigella species isolates from ur

169 r advance.PXVX0200, based on live attenuated Vibrio cholerae O1 classical Inaba vaccine strain CVD 10

170 phage VP5, one of the typing phages for the Vibrio cholerae O1 El Tor biotype.

171 Vibrio cholerae O1 El Tor strains have been responsible

172 Vibrio cholerae O1 is a major cause of acute watery diar

173 We used genomic data from 1070 Vibrio cholerae O1 isolates, across 45 African countries

174 odulates biofilm development and motility in Vibrio cholerae O1 of the classical biotype.

175 ess of humans caused by toxigenic strains of Vibrio cholerae O1 or O139.

176 rapid, sensitive and selective detection of Vibrio cholerae O1 which converts the antibody-antigen b

177 dministration of the cholera vaccine (killed Vibrio cholerae O1 whole cells and recombinant cholera t

178 ulted in lower vibriocidal responses against Vibrio cholerae O1, and there was a positive relationshi

179 ed two novel non-toxigenic (ctxA/B-negative) Vibrio cholerae O1.

180 spected cases who tested positive to PCR for Vibrio cholerae O1.

181 em, which is restricted to EI Tor biotype of Vibrio cholerae O1.

182 haride fragment of the O-specific antigen of Vibrio cholerae O139 were synthesized by applying 1 + 1,

183 end in the O-specific polysaccharide of both Vibrio cholerae O22 and O139.

184 n acute, watery diarrhoeal disease caused by Vibrio cholerae of the O1 or O139 serogroups.

185 Toxigenic Vibrio cholerae of the O139 serogroup have been responsi

186 ntrol of human pathogens such as Salmonella, Vibrio cholerae or enterohaemorrhagic Escherichia coli.

187 to an antigen of interest were purified from Vibrio cholerae or Escherichia coli and used for immuniz

188 Anti-OMV IgG renders Vibrio cholerae organisms immotile, thus they pass throu

189 ShyA, the primary EP of the cholera pathogen Vibrio cholerae Our data suggest that ShyA assumes two d

190 ay use intercellular communication to stymie Vibrio cholerae pathogenesis, indicating how the microbi

191 Here, we show that the Vibrio cholerae pathogenicity factor DncV is a prokaryot

192 One such satellite in Vibrio cholerae, phage-inducible chromosomal island-like

193 Among 845 patients, 11% (90) were Vibrio cholerae positive; among these 90 patients, analy

194 Vibrio cholerae possesses multiple quorum-sensing (QS) s

195 a bacterial DASS family member, VcINDY from Vibrio cholerae, predict an elevator-like transport mech

196 Pathogenic Vibrio cholerae produces a cholera toxin which is the ca

197 e mutualist Vibrio fischeri and the pathogen Vibrio cholerae promotes release of a potent bacteria-de

198 st identify ancestral forms still present in Vibrio cholerae, Pseudomonas aeruginosa, Shewanella onei

199 onas aeruginosa (a tit-for-tat species) with Vibrio cholerae (random-firing), revealing that P. aerug

200 Vibrio cholerae remains a major global health threat, di

201 Pathogenic Vibrio cholerae remains a major human health concern.

202 The acute, voluminous diarrhoea caused by Vibrio cholerae represents a dramatic example of enterop

203 Vibrio cholerae, responsible for acute gastroenteritis s

204 Analyses of intraintestinal Vibrio cholerae revealed infection-stage and region-spec

205 Vibrio cholerae's CarRS two-component regulatory system

206 factors are known to modulate expression of Vibrio cholerae's principal virulence factors.

207 aracterized core DNA MTases, like those from Vibrio cholerae, Salmonella enterica, Clostridioides dif

208 non-protein coding RNA (npcRNA) sequences of Vibrio cholerae, Salmonella sp. and Shigella sp., which

209 Recent studies have linked Vibrio cholerae secreted toxins to inflammasome activati

210 The human intestinal pathogen Vibrio cholerae secretes a pore-forming toxin, V.cholera

211 tic bacterium and human intestinal pathogen, Vibrio cholerae, senses and responds to a variety of env

212 demonstrated with the detection of toxigenic Vibrio cholerae serogroups O1 and O139, which are associ

213 ~25,000 RNase E-dependent cleavage sites in Vibrio cholerae, several of which resulted in the accumu

214 lytic bacteriophage and the etiologic agent Vibrio cholerae share commonalities across bacterial tax

215 -resolution structure of a native contracted Vibrio cholerae sheath determined by cryo-electron micro

216 sted alongside highly diverse members of the Vibrio cholerae species in Argentina, and we contrast th

217 Here, we show that the initiator protein of Vibrio cholerae specific to chromosome 2 (Chr2) also has

218 mG is known to be involved in remodeling the Vibrio cholerae surface, but its specific role was not c

219 am-negative rod and human diarrheal pathogen Vibrio cholerae synthesizes a VPS exopolysaccharide-depe

220 tion X-ray structures of VcINDY, a DASS from Vibrio cholerae that catalyses the co-transport of Na(+)

221 ree PL genosensor for sensitive detection of Vibrio cholerae that is based on a DNA hybridization str

222 Vibrio cholerae the causative agent of cholera epidemics

223 In pathogenic vibrios, including Vibrio cholerae, the accumulation of autoinducers trigge

224 Vibrio cholerae, the bacterium responsible for the fatal

225 sequence-specific detection of the bacterium Vibrio cholerae, the causative agent of acute diarrheal

226 controls virulence and biofilm formation in Vibrio cholerae, the causative agent of cholera disease.

227 Like many other pathogens, Vibrio cholerae, the causative agent of cholera, can mod

228 Vibrio cholerae, the causative agent of cholera, has res

229 Vibrio cholerae, the causative agent of cholera, remains

230 By directly applying a reporter strain of Vibrio cholerae, the causative agent of cholera, to a th

231 We show that Vibrio cholerae, the causative agent of cholera, use the

232 For example, Vibrio cholerae, the causative agent of cholera, utilize

233 nc transport systems in the enteric pathogen Vibrio cholerae, the causative agent of cholera.

234 chanism of horizontal gene transfer (HGT) in Vibrio cholerae, the causative agent of cholera.

235 creases both the survival and infectivity of Vibrio cholerae, the causative agent of cholera.

236 igated citrate utilization and regulation in Vibrio cholerae, the causative agent of cholera.

237 Vibrio cholerae, the causative agent of the acute diarrh

238 Like other Gram-negative pathogens, Vibrio cholerae, the causative agent of the diarrheal di

239 Toxigenic Vibrio cholerae, the causative agent of the disease chol

240 Vibrio cholerae, the causative agent of the human diarrh

241 The human pathogen Vibrio cholerae, the causative agent of the severe diarr

242 Vibrio cholerae, the causative agent of the severe diarr

243 Vibrio cholerae, the causative agent of the severe diarr

244 ce, as documented in the intestinal pathogen Vibrio cholerae, the causative agent of the severe diarr

245 kinase/response regulator pair that enables Vibrio cholerae, the cholera pathogen, to survive exposu

246 In El Tor biotype strains of toxigenic Vibrio cholerae, the CTXvarphi prophage often resides ad

247 Vibrio cholerae, the etiological agent of cholera, has e

248 The genome of Vibrio cholerae, the etiological agent of cholera, is an

249 Vibrio cholerae, the etiological agent of cholera, is kn

250 e environmental survival and transmission of Vibrio cholerae, the facultative human pathogen responsi

251 We considered the formate channel from Vibrio cholerae, the hydrosulphide channel from Clostrid

252 negative bacteria Haemophilus influenzae and Vibrio cholerae, the master regulator Sxy/TfoX controls

253 In the pathogen Vibrio cholerae, the quorum-sensing autoinducer 3,5-dime

254 In Vibrio cholerae, the transport system requires three pro

255 Shigella spp and enteroinvasive E coli, and Vibrio cholerae-the strength of association with diarrho

256 In Vibrio cholerae, there are 13 distinct PTS transporters.

257 In Vibrio cholerae, three chromosomal clusters each encode

258 Here, we explore the cobamide specificity in Vibrio cholerae through examination of three natural var

259 the toxin-linked cryptic satellite phage of Vibrio cholerae, TLCPhi, which integrates into and excis

260 Programmable transposition of Vibrio cholerae Tn6677 in Escherichia coli requires CRIS

261 res of a TniQ-Cascade complex encoded by the Vibrio cholerae Tn6677 transposon using cryo-electron mi

262 Introduction of Vibrio cholerae to Haiti during the deployment of United

263 Additionally, C10-AMS stopped the ability of Vibrio cholerae to recycle fatty acids from media and su

264 We use the facultative human pathogen Vibrio cholerae to show that VacJ/Yrb is silenced early

265 Type VI secretion is critical for Vibrio cholerae to successfully combat phagocytic eukary

266 architecture to detect the B subunit of the Vibrio cholerae toxin at improved sensitivity (100 pg/ml

267 The specific machine we analyse is the Vibrio cholerae toxin-coregulated pilus machine (TCPM).

268 ToxR activates expression of T3SS2 resembles Vibrio cholerae ToxR regulation of distinct virulence el

269 Two of the primary virulence regulators of Vibrio cholerae, ToxR and TcpP, function together with c

270 Comprehensive quantification of Vibrio cholerae tRNAs revealed that the abundance of som

271 We describe a Vibrio cholerae Type 3 secretion system effector VopE th

272 t & Microbe, Suzuki et al. (2014) describe a Vibrio cholerae Type-III-secreted effector that targets

273 The human pathogen Vibrio cholerae typically exists as a curved rod, but st

274 e previously showed that naturally competent Vibrio cholerae use their type VI secretion system (T6SS

275 Some predatory species, such as Vibrio cholerae, use their T6SS in an untargeted fashion

276 Vibrio cholerae uses a multiprotein transcriptional regu

277 Vibrio cholerae uses a quorum-sensing (QS) system compos

278 Here, we utilize Vibrio cholerae (V. cholerae) as a proof-of-concept for

279 ion (LAMP) targeting the waterborne pathogen Vibrio cholerae (V. cholerae).

280 tal structure characterization of DHBPS from Vibrio cholerae (vDHBPS) with a competitive inhibitor 4-

281 In Vibrio cholerae, VgrG3 has a hydrolase extension domain

282 bacteria including Plesiomonas shigelloides, Vibrio cholerae, Vibrio fischeri, Shewanella putrefacien

283 eats-in-toxin) family toxins are produced by Vibrio cholerae, Vibrio vulnificus, Aeromonas hydrophila

284 athogens, including life-threatening spp. of Vibrio cholerae, Vibrio vulnificus, and Aeromonas hydrop

285 The major Vibrio cholerae virulence gene transcription activator,

286 Vibrio cholerae was identified in 525 stool specimens, a

287 idual bacterial species Aeromonas veronii or Vibrio cholerae was sufficient to block locomotor hypera

288 ce (CRISPRi) knockdown in the human pathogen Vibrio cholerae We demonstrate that CRISPRi knockdown of

289 However, in Vibrio cholerae, we demonstrate that amidase activity al

290 mathematical modelling and experiments with Vibrio cholerae, we show how killing adjacent competitor

291 In Vibrio cholerae, we uncover that this requirement is due

292 y to identify 90 proteins present in OMVs of Vibrio cholerae when grown under conditions that activat

293 This includes Vibrio cholerae, where indole was shown to regulate biof

294 the phenomenon in a bacterial suspension of Vibrio cholerae, where the fluorescent protein (mKO; mon

295 Using experiments with Vibrio cholerae, which secretes extracellular enzymes to

296 previously reported Tn7-like transposon from Vibrio cholerae, which uses a Type I-F CRISPR-Cas system

297 cteria (Escherichia coli, Salmonella spp and Vibrio cholerae), with 8 strains of each bacterium, and

298 d kinetic and structural characterization of Vibrio cholerae WT ApbE and mutants of the conserved res

299 or adenovirus 40/41, norovirus, rotavirus A, Vibrio cholerae, Yersinia enterocolitica, Entamoeba hist

300 shigelloides, Salmonella spp., Vibrio spp., Vibrio cholerae, Yersinia enterocolitica, enteroaggregat