ライフサイエンスコーパス: WT

1 WT and fat-1 mice were chronically fed EtOH, and ileum R

2 WT and FATP2-null (Fatp2 (-/-)) mice (5 weeks) were main

3 WT and Nod2(-/-) mice were treated with the carcinogen d

4 WT and Y17H viruses had similar environmental stability

5 WT ClC-5 had a 2Cl(-)/H(+) exchange ratio at a V(h) of +

6 WT HNF-1beta binds to two evolutionarily conserved sites

7 WT mice pre-administered with rTSLP or vehicle, TSLPR(-/

8 WT neuropathic animals showed signs of spontaneous pain

9 WT SAMHD1 in differentiated U937 cells significantly inh

10 cles in lobule V of CB (1) -KO as in CB (1) -WT, but their distribution decreased drastically at 300

11 KO remained unchangeable relative to CB (1) -WT.

12 lect distinct differentiation stages of H3.3 WT and H3.3 MUT stromal cells and add to H3.3-G34W-assoc

13 Using cells expressing a WT or S838A/T841A mutant RB fragment, we present evidenc

14 d, Rieske-type [2Fe-2S](1+) center in AbCntA-WT to the mono-nuclear, non-heme iron center through the

15 ging CCL2 with a lower efficiency than ACKR2-WT.

16 signaling by dimerizing with and activating WT C-Raf proto-oncogene, Ser/Thr kinase (CRAF).

17 cient deposits in both the Prnp180Q/196Q and WT mice, underscoring the pivotal role of the GPI-anchor

18 Both R67Q(+/-) (n=8) and WT myocytes (n=9) demonstrated typical n-shaped I(K1) IV

19 FES uptake was compared between Y537S-ER and WT-ER tumors.

20 no difference between the knob-into-hole and WT IgG4 molecules in E. coli.

21 man cancer cells that harbor mutant KRAS and WT p53 (p53), KRAS contributes to the maintenance of low

22 ar dominance plasticity between NF1 mice and WT littermates disappear.

23 ies with (11)C-labeled 1 in both HD mice and WT nonhuman primates (NHPs) demonstrated that the right-

24 rian hamsters infected with Hyper prions and WT mice infected with Rocky Mountain Laboratories prions

25 nse (ABR) thresholds between mutant rats and WT controls.

26 Interestingly, the phenotype of KO->WT and WT->KO mice did not differ from that of WT->WT mice.

27 ion and mediated apoptosis as efficiently as WT BAX.

28 In vivo studies using syngeneic SR-B1 WT (SR-B1(+/+)) and SR-B1 KO (SR-B1(-/-)) prostate cance

29 ipts, while other clones expressed both BCOR-WT and BCOR-mut transcripts.

30 ones expressed either solely the normal BCOR-WT or BCOR-mut transcripts, while other clones expressed

31 e subcloned SCAP-O and separated SCAP-O(BCOR-WT) and SCAP-O(BCOR-mut) as verified by sequencing.

32 sulin to bind with IR did not differ between WT and CD36-deficient hepatocytes.

33 um Ca(2+) content were not different between WT (n=18) and R67Q(+/-) (n=16) myocytes.

34 ene expression is markedly different between WT mice and Het-Met mice.

35 erences in trabecular bone formation between WT and Col6alpha2-KO mice based on the mineral appositio

36 a similar early metabolic response for both WT-ER and Y537S-ER tumors.

37 es are rapidly induced by blue light in both WT and the ppi2 mutant.

38 l transcriptomics and immunostaining of both WT and DKO ERCs confirmed their DR-cell phenotype.

39 r plexiform and inner nuclear layers of both WT and hWtEPOR 8-week-old mice.

40 set compared to the onset of those caused by WT EBV, although the tumors occurred at a similar rate.

41 e size, the latter phenotype complemented by WT mRNA but not by SSBP1 mutant transcripts.

42 homas at rates similar to their induction by WT EBV but with delayed onset.

43 BALB/c WT and TSLP receptor-deficient (TSLPR(-/-) ) mice were c

44 Here, we compared WT and GluA3 KO mice of both sexes and identified severa

45 od pressure compared with TG mice containing WT intron 2.

46 ses expressing either wild-type CryAB (CryAB(WT)) or CryAB(R120G).

47 Ube2v1 promoted aggregate formation in CryAB(WT) and CryAB(R120G)-expressing neonatal rat ventricular

48 uman wild-type alphaA-transgene in CRYalphaA(WT) mice.

49 Compared to the lenses of CRYalphaA(WT), the lenses of CRYalphaA(N101D) mice exhibited: (A)

50 tein in CRYalphaA(N101D) mice than CRYalphaA(WT) mice occurs, which causes intracellular ionic imbala

51 about 7-months of age relative to CRYalphaA(WT) mice.

52 activities of the LEKTI domain 4 (D4) and D6 WT and AD-associated mutants on the enzyme activities of

53 lls was established to assess the role of D6 WT and D386N on triggering inflammation via the inductio

54 a mRNA expression to a similar extent as did WT SAMHD1, suggesting that SAMHD1-mediated inhibition of

55 ds with specific cellular potency for either WT or mutant SHP2.

56 gene expression was rescued by re-expressing WT but not ChREBP that lacks hydroxylated prolines in Ch

57 monocytic U937 cell lines stably expressing WT SAMHD1 or mutated variants defective in dNTPase activ

58 d 35% (23-49; 19 of 54 patients) in the EZH2(WT cohort.) Median duration of response was 10.9 months

59 cohort and 13.0 months (5.6-NE) in the EZH2(WT) cohort; median progression-free survival was 13.8 mo

60 tatus: mutant (EZH2(mut)) or wild-type (EZH2(WT)).

61 scribed associations, many cases of familial WT remain unexplained.

62 nd chronic leptin treatment of chow diet-fed WT mice decreased MTP expression in the intestine, incre

63 ontrast, ex vivo cardiac function in HFD-fed WT mice dropped ~ 50% relative to low fat diet (LFD) fed

64 Ensemble analyses of surrounding groups for WT and mutant KSIs provided insights into the forces and

65 ishing a genetic diagnosis is imperative for WT families so that individuals harboring a predisposing

66 ore these mechanisms, we engineered V5-FOXL2(WT)- and V5-FOXL2(C134W)-inducible isogenic cell lines a

67 Newborn astrocytes were derived from WT, Tnfalpha(-/-), Il1alpha(-/-)/Il1beta(-/-), and Tlr2(

68 rrow-derived mast cells (BMMCs) and ECs from WT and Fn14(-/-) or TWEAK(-/-) mice were studied.

69 hyperexcitability of trigeminal neurons from WT female mice.

70 ltures from TRAP tdTomato mice, but not from WT mice.

71 d in the ischemic hemispheres and pCECs from WT but not in miR-34a(-/-) mice following stroke reperfu

72 s spectrometry (GC-MS) analyses of sera from WT and Pax5+/- mice demonstrated the presence of a genot

73 yed differentiation compared with those from WT muscles.

74 Furthermore, WT mice treated with P2X4 allosteric agonist ivermectin

75 reduced bioactivity compared with WT GH (GH-WT) because of its decreased ability to bind and activat

76 Interestingly, the phenotype of KO->WT and WT->KO mice did not differ from that of WT->WT mi

77 CC1 null mutation augmented IRI-OLT (CC1-KO->WT) by enhancing ROS expression and HMGB1 translocation

78 induction, and HMGB1 translocation (CC1-KO->WT), whereas ASK1 silencing (siRNA) promoted cytoprotect

79 WT->KO mice did not differ from that of WT->WT mice.

80 abilization compared with reconstituted HDL (WT apoA-I, 1.92 +/- 0.04; 5-OHTrp(72) apoA-I, 2.35 +/- 0

81 CH(4) m(-2) hr(-1) ) due to prolonged higher WT and more optimal methanogenesis conditions.

82 diomyocytes from mice trans-expressing human WT or p.P888L SAP97 and in Chinese hamster ovary (CHO)-t

83 In WT eggs, zinc was detected in cortically-localized vesic

84 Ab-mediated depletion of IL-1alpha in WT mice prevented infection-induced oviduct damage, furt

85 enol, max outward I(K1) increased by ~20% in WT but decreased by ~24% in R67Q(+/-) (P<0.01).

86 sufA mutant cells and is decreased to ~4% in WT E. coli cells.

87 -B1 KO (SR-B1(-/-)) prostate cancer cells in WT and apolipoprotein-AI KO (apoA1-KO) C57BL/6J mice rev

88 ection of Th9/Th17 cells with tumor cells in WT, Rag1-/-, Il9r-/-, and Il17ra-/- mice altered tumor g

89 his blockade had no additive cytotoxicity in WT MEFs, suggesting the cytotoxicity is due to MUTYH int

90 5b prevented subtle gait deficits in WT alphaS mice and the PD-like resting tremor and progre

91 lso enhance commitment to differentiation in WT cells.

92 of Escherichia coli, which had no effect in WT mice.

93 with the isotype-specific inhibitor SK1-I in WT cells induced accumulation of cholesterol and reduced

94 Nox2, all of which were induced by Ang II in WT mice.

95 rotein NDUFB8 was significantly increased in WT mice fed a HFD, but remained unchanged in GCN5L1 cKO

96 on during necrotizing fasciitis infection in WT mice but not in S100a9 (-/-) mice lacking CP.

97 ic effect lowered plasma phosphate levels in WT mice and in rats with CKD due to subtotal nephrectomy

98 Wbp, but clearance of these same microbes in WT mice was restored if active thrombin was administered

99 espite the presence of dofetilide (1 muM) in WT K(v)11.1 cells.

100 ed SNHL by impairing cochlear myelination in WT aged mice.

101 rated that PD increases neuroinflammation in WT mice and disrupts the neuroinflammatory response in 5

102 on-like behavior in Het-Met mice, but not in WT mice.

103 ic bursting by GABA neurons also occurred in WT mice in association with theta activity during attent

104 n contrast, PLP(ECD) induced EAE not only in WT mice, but in B cell-sufficient mice incapable of secr

105 hose with pyruvate and palmitoylcarnitine in WT.

106 ential IL-6-related inflammatory programs in WT versus IRF3-KO mice.

107 The structures of the radical in WT, V172I, and V172C variants have been established by d

108 ially slower, then became more rapid than in WT muscle.

109 gnificantly shorter in p.P888L-SAP97 than in WT-SAP97 mice.

110 ly identified genes CTR9, REST and TRIM28 in WT predisposition.

111 ulon genes are critical for GAS virulence in WT mice, whereas they are dispensable for virulence in S

112 macologically sustained elevation of zinc in WT eggs prior to activation resulted in abnormal chorion

113 cal scales does not increase with increasing WT installed capacity.

114 Chemical complementation of infected WT and CSE(-/-) macrophages using the slow H(2)S release

115 tions rendered it nonfunctional, whereas its WT form could restore neuronal morphology and function i

116 The large respiratory capacity of KNM-WT 15000 is compatible with the relatively stocky, more

117 rax of the juvenile H. erectus skeleton, KNM-WT 15000, from Nariokotome, Kenya, along with its estima

118 comparable to adult (8-week-old) littermate WT mice, hWtEPOR mice had thinner inner and outer plexif

119 erozygous Q175 mice, compared to age-matched WT males.

120 (ESCs) into wild-type (WT) blastocysts (mdx/WT chimera).

121 obal Zip14 knockout (KO) and wild-type mice (WT).

122 Moreover, WT and mutant SAMHD1 similarly interacted with key prote

123 Conversely, four patients who had MYD88(WT) disease showed no major responses.

124 the electrophysiological effects of native (WT) and p.P888L SAP97, a common polymorphism.

125 these phenotypes were restored to WT or near-WT levels when lon-2 mutation was complemented in cis.

126 The middle and the inner ears of WT and Nhe6 KO mice were not different morphologically.

127 y mediators in the cortex and hippocampus of WT mice, whereas levels were mitigated in IFN-beta(-/-)

128 Although the S1-S2 linker of WT Kv1.5 could be cleaved by extracellularly applied pro

129 amatically increased toward term in lungs of WT fetuses, was markedly reduced in Src-1/-2(d/d) fetal

130 were significantly less fit in the lungs of WT mice than in those of neutropenic mice.

131 Pentamidine-decreased maturation of WT K(v)11.1 levels was rescued by 10 muM dofetilide or 1

132 macrophages was observed in knockout mice of WT-knockout pairs.

133 ia, miR-210 was observed in knockout mice of WT-knockout pairs.

134 Nanoscale dynamic organization of WT and mutant NRas relative to BRAF serves as a regulato

135 We also observed that overexpression of WT MAP3K19 activates both the ERK and JNK pathways in a

136 We found that overexpression of WT SLC20A2 increased phosphate uptake, as expected, but

137 We also solved the crystal structures of WT and N53I CaM in complex with the primary calmodulin-b

138 ry in Speg-KO mice was compared with that of WT mice, leading to the identification of similar abnorm

139 tion potencies ranging from 1.8-fold that of WT SC(1-246) to complete loss of function.

140 and WT->KO mice did not differ from that of WT->WT mice.

141 re not significantly different from those of WT vessels, suggesting that basal K(ATP) channel activit

142 In parallel, adoptive transfer of WT neutrophils into Par4(-/-) mice restored inflammation

143 , which is active in vivo Acute treatment of WT mice with ABD-110207 resulted in elevated FAHFA level

144 A half-active complex combining one WT and one D274A monomer also stalled after one electron

145 e containing either the intron conversion or WT intron 2.

146 had exacerbated renal IR injury whereas P2X4 WT mice treated with a selective P2X4 antagonist (5-BDBD

147 t was reduced by half compared with parental WT Ramos B cells, demonstrating that the CTNNBL1 M466V m

148 Control (PLM(WT)), transgenic (PLM(3SA)), ouabain-treated and hypertr

149 he PIs complexed with wild-type Protease (PR(WT)) and highly-multi-PI-resistance-associated PR(DRV)(R

150 may drive development of drugs that release WT p53 and allow tumor suppression.

151 b can modulate host translation and restrict WT mouse coronavirus infection.

152 plicated similarly to wild-type SFTSV (SFTSV-WT), it showed weaker pathogenic activity than SFTSV-WT.

153 showed weaker pathogenic activity than SFTSV-WT.

154 In contrast, during tonic TLR4 signaling, WT cells did not undergo necroptosis, even when MK2 was

155 We studied WT Cel7A and several variants in which one or two of fou

156 monitored soil temperature and water table (WT) level from June 2017 to January 2019.

157 re likely to form a complex with CaMKII than WT.

158 ns lower number of Atoh-1 positive GCPs than WT.

159 b-infected CSE(-/-) mice survive longer than WT mice, and support reduced pathology and lower bacteri

160 kout (KO) mice exhibited worse outcomes than WT mice in both ICH models and were less responsive to I

161 SNHL animals committed more WME and RME than WT animals, demonstrating that isolated SNHL affected co

162 e vessels were >100-fold less sensitive than WT vessels to pinacidil.

163 AI KO (apoA1-KO) C57BL/6J mice revealed that WT hosts, containing higher levels of total and HDL-chol

164 Deltaaur and DeltasspAB mutants in both the WT and fakA mutant backgrounds, we found that the absenc

165 viral load was consistently detected for the WT antibody.

166 iptomic analysis of developing leaves in the WT and the three mutants we identified differentially ex

167 ed after one electron was transferred in the WT half.

168 e "A" conformation but with ~20% each of the WT conformer and an "O" state in which d(z)(2) Ni(p)(I)

169 ine reduced enzymatic activity to <3% of the WT levels.

170 A comparison of the WT protein and a variant with an impaired MIDAS (but oth

171 fferences compared with the structure of the WT receptor.

172 monecrosis to levels similar to those of the WT strain.

173 , lnk mutants were more susceptible than the WT to P. syringae infection.

174 We observed that the WT and P167S/D240G variant with acylated ceftazidime bot

175 d, autoinhibited conformation similar to the WT enzyme, the interactions between its N-SH2 and protei

176 mitted to naive chickens, in contrast to the WT virus.

177 al cells have longer cilia compared with the WT cells because of decreased Kif19a protein levels in t

178 enuated lysosomal rewiring compared with the WT Mtb in both in vitro and in vivo infections.

179 sma membrane association of beta1-p.C162A to WT levels.

180 oxidase levels in PLD2(-/-) mice compared to WT and PLD1(-/-) mice, confirming a novel role of PLD2 a

181 ation, and pulmonary dysfunction compared to WT animals.

182 notypic change and sEV secretion compared to WT CASMCs, which was associated with reduced lysosome-mu

183 resulted in increased mortality compared to WT Kunjin infection.

184 lood pH and a decrease in pCO(2) compared to WT littermates.

185 Rab11 levels in the Nhe6 KO OC, compared to WT littermates.

186 he active phase (nighttime) when compared to WT mice and treatment during the inactive phase (daytime

187 xhibit depression-like behaviors compared to WT mice in an age dependent manner.

188 l and impaired neurotransmission compared to WT mice, while nCLCa-only mice had increased synaptic ve

189 d significant alveolar bone loss compared to WT mice.

190 lt Ext challenged Esr1(-/-) mice compared to WT mice.

191 Compared to WT, fat-1 mice exhibited a markedly plastic transcriptom

192 nt (Src-1/-2(d/d) ) fetal lungs, compared to WT.

193 ed and reduced fusion efficiency compared to WT.

194 xpression of endothelial markers compared to WT.

195 tions were higher in mutant mice compared to WT.

196 in the kidney of KS-tg/OVE mice compared to WT/OVE mice, suggesting a disturbed balance between the

197 ulture results, we found that in contrast to WT mice, diabetic 4E-BP1/2-deficient mice did not exhibi

198 Administration of recombinant HMGB1 to WT, but not Ripk3(-/-) mice, recapitulates ALI-induced p

199 function of most of the tested FH mutants to WT FH levels on a human HAP-1 cell line and on sheep ery

200 ly increased in Atp7b (-/-) mice relative to WT controls.

201 cant delay in the decay of force relative to WT muscle while the return of myosin heads to an ordered

202 both bmr12 and bmr12 35S::SbF5H relative to WT or 35S::SbF5H.

203 in concentrations were decreased relative to WT or 35S::SbF5H.

204 Relative to WT, DeltaFosB expression was increased in D1+ neurons in

205 ecific CD8(+) and CD4(+) T-cell responses to WT ECTV.

206 All these phenotypes were restored to WT or near-WT levels when lon-2 mutation was complemente

207 ty and superprocessivity behavior similar to WT KIF1A.

208 Surprisingly, similarly to WT, infection with W105A inhibited IFN/ISG expression de

209 showed that ATP6v1g1 binds more strongly to WT UBQLN2 than to ALS/FTD mutant UBQLN2 proteins.

210 but not when it is normal or restored toward WT.

211 b treatment inhibited growth of mutant TP53, WT PTEN LN-229 tumors, and sensitized LN-229 tumors to T

212 by competition experiments in isogenic TP53-WT and TP53-null (TP53(-/-)) cell lines.

213 antagonist, GSK2193874, in elastase-treated WT mice and in AngII-treated ApoE(-/-) mice caused a sig

214 Wilms' tumor (WT) morphologically resembles the embryonic kidney, cons

215 mpacts from current and future wind turbine (WT) deployments necessary to achieve 20% electricity fro

216 We characterized GEC from wild type (WT) and col4alpha5 knockout AS mice, a hereditary disord

217 nt causes larger lesions than the wild type (WT) during murine skin infection.

218 nged LPS challenge as compared to wild type (WT) neutrophils.

219 th and inflammatory phenotypes to wild type (WT) profiles.

220 compared the results to the PMSS1 wild type (WT).

221 in intrinsic excitability between wild-type (WT) and Foxp2(+/R552H) neurons.

222 Wild-type (WT) and FSTL-1 hypomorphic (Hypo) mice were infected wit

223 direct respiration calorimetry in wild-type (WT) and heterozygous R163C (HET) mice over a range of am

224 Ratios of wild-type (WT) and I38T mutant were assessed by digital RT-PCR.

225 rol selective cation transport in wild-type (WT) and mutant (N629D) hERG1 variants.

226 insertion mutants in the lungs of wild-type (WT) and neutropenic mice using transposon sequencing (Tn

227 us cocaine self-administration in wild-type (WT) and Npas2 mutant mice at different times of day.

228 on, cue-induced reinstatement) in wild-type (WT) and Npas2 mutant mice at different times of day.

229 Wild-type (WT) and SXRKO mice were chronically or perinatally expos

230 eased chromosome instability in a wild-type (WT) background, suggesting that such mutants have the po

231 embryonic stem cells (ESCs) into wild-type (WT) blastocysts (mdx/WT chimera).

232 ter release and promotes sleep in wild-type (WT) but not pcdr-1 and slo-1 mutants.

233 distribution in miR-155(-/-) and wild-type (WT) C57BL/6 mice.

234 When compared to wild-type (WT) cardiomyocytes, ARDKO displayed reduced fractional s

235 ed mitochondrial respiration than wild-type (WT) cells.

236 Analyses of wild-type (WT) chromosomes and de novo circular minichromosomes rev

237 LgDel versus wild-type (WT) CNgV transcriptomes differ significantly at E10.5 ju

238 A1, Fpr2/3 knockout (KO) mice and wild-type (WT) controls were infected intranasally with S pneumonia

239 compared to those in age-matched wild-type (WT) controls.

240 alues are increased compared with wild-type (WT) EMB.

241 H7 cells were less sensitive than wild-type (WT) enzyme to degradation evoked by DPTA, suggesting tha

242 7D breast cancer cells expressing wild-type (WT) ER or an activating ESR1 mutation, Y537S-ER, were us

243 e expression of the corresponding wild-type (WT) gene, due to either variations in copy number or tra

244 e were greater in Taz(KD) than in wild-type (WT) hearts, but there were no differences in oxidative p

245 wing reduced values compared with wild-type (WT) IFI, whereas for EMB-3b these values are increased c

246 short-term memory (STM), which in wild-type (WT) is time-of-day (TOD) independent, is decreased acros

247 g familial AD mutations vs. their wild-type (WT) isogenic controls in order to characterize the aberr

248 ort that overexpression of either wild-type (WT) LIN28B or a LIN28B mutant that is unable to inhibit

249 Ghrelin knockout (KO) mice and wild-type (WT) littermates underwent an insulin bolus-induced hypog

250 mpare the catalytic efficiency of wild-type (WT) Methylorubrum extorquens AM1 PqqE to a range of muta

251 Citrobacter rodentium compared to wild-type (WT) mice evidenced by more severe intestinal inflammatio

252 d apoptosis when compared to P2X4 wild-type (WT) mice subjected to renal IR.

253 4 postnatal weeks, SOD1-G93A and wild-type (WT) mice were evaluated in the rotarod test, to be sacri

254 We infected RdRP mice and wild-type (WT) mice with various doses of a pathogenic retrovirus (

255 stimulator of interferon genes in wild-type (WT) mice.

256 d on a high-fat diet, compared to wild-type (WT) mice.

257 d phagocytic function relative to wild-type (WT) mice.

258 ll NHE isoforms were expressed in wild-type (WT) mouse cochlea.

259 r immune complexes, compared with wild-type (WT) neutrophils.

260 Heterozygous TLR2(+/-) pups from wild-type (WT) or TLR2(-/-) dams were fed either by their biologic

261 aves, and lower grain yields than wild-type (WT) plants.

262 mutants can recruit and activate wild-type (WT) SHP2 in LLPS to promote MAPK activation.

263 d bmr12 stover than 35S::SbF5H or wild-type (WT) stover; S-lignin and total lignin concentrations wer

264 Compared with that of wild-type (WT) strain, the infectivity of the mutant was severely a

265 ction than after infection with a wild-type (WT) strain.

266 enicity in DBA/2 mice compared to wild-type (WT) virus (activation pH, 5.5).

267 ng to red blood cells (RBCs) from wild-type (WT), alpha1,3-galactosyltransferase gene-knockout (GTKO)

268 FMRP regulates leak closure in wild-type (WT), but not FX synapses, by stimulus-dependent ATP synt

269 growth of PIK3CA-mutant, but not wild-type (WT), colorectal cancers.

270 Vaginal challenges of wild-type (WT), Y181C, and Y181V HIV-1 were performed in mice left

271 he overall state of ISGylation in wild-type (WT)-infected cells.

272 Using miR-210 replete (wild-type [WT]) and knockout mice, we tracked blood-borne miR-210 u

273 Kalpha1alpha2lox/lox littermates (wild-type [WT]).

274 k component of mammalian enamel, and, unlike WT enamel, appears to be composed of less organized arra

275 ansfer of CD4(+) T cells from PIV-vaccinated WT mice to naive CD4-deficient (CD4 KO) mice demonstrate

276 RNA-Seq analysis of ASH1L knockout versus WT ATC cell lines revealed that ASH1L is involved in the

277 immune network was similar in IRF3-KO versus WT septic mice, although the tempo of connectivity diffe

278 ays are ~2-fold lower in heart from T2DM vs. WT under all conditions studied.

279 gh fat diet (HFD) feeding for 6 or 18 weeks, WT and AIF1L deficient mice gained weight similarly, sho

280 strate affinity (Km) values to the wildtype (WT) Km value but had a lower turnover number and transfr

281 photoperiod exposure versus their wildtype (WT) littermates.

282 ermline genetic alterations in children with WT.

283 after 10 nmol/L isoproterenol compared with WT (n=7; P<0.05).

284 ) activity (mumol/min/mg) when compared with WT apoA-I and comparable PON1 activation/stabilization c

285 s across several lipid classes compared with WT bacteria.

286 gher dimerization potential as compared with WT BRAF.

287 e presence of zymosan in vitro compared with WT cells, and the effect was also LTB4- and BLT1-depende

288 iposity, and hepatic steatosis compared with WT controls.

289 oxidation in RYR1 mutants when compared with WT controls.

290 H-C53S has reduced bioactivity compared with WT GH (GH-WT) because of its decreased ability to bind a

291 er insensitive to ethanol when compared with WT GlyRs.

292 r conditioned place preference compared with WT littermate controls.

293 Compared with WT mice, Pd1-/- mice exhibited increased baseline pain s

294 As compared with WT mice, we show that the activity of IL-13 is dramatica

295 creased in MECs from TSP1(-/-) compared with WT mice, whereas several extracellular matrix and inflam

296 smaller in MECs from TSP1(-/-) compared with WT mice.

297 e effector-binding site in Ras compared with WT RBD.

298 retina of REDD1-deficient mice compared with WT.

299 510A than when cells were reconstituted with WT IQGAP1.

300 dendritic spine density compared with young WT.