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1 Waardenburg syndrome (WS) is a clinically and geneticall
2 Waardenburg syndrome (WS) is a hereditary disorder that
3 Waardenburg syndrome manifests hypopigmentation of the i
4 Waardenburg syndrome type 1 is caused by mutations in PA
5 Waardenburg syndrome type 2 (WS2) is a dominantly inheri
6 Waardenburg syndrome typically manifests with congenital
7 Waardenburg syndromes are characterized by pigmentation
8 Waardenburg-Shah syndrome combines the reduced enteric n
9 ges in the PAX3 gene in patients with type 1 Waardenburg syndrome (WS1) including three in the paired
13 ons responsible for Hirschsprung disease and Waardenburg syndrome have been identified, and some pati
15 0% of which were syndromic (mainly Usher and Waardenburg syndromes) and 50.0% were nonsyndromic (main
17 tion with hypopigmentation are classified as Waardenburg syndrome type 4 (Waardenburg-Shah, WS4).
19 Waardenburg syndrome, Type IV, also known as Waardenburg-Hirschsprung disease, characterized by pigme
21 ultiple phenotypic disease) deafness such as Waardenburg syndrome and Usher 1B syndrome, little is kn
23 (microphthalmia transcription factor) cause Waardenburg syndrome type 2 (WS2A) in humans, an autosom
24 In humans, mutations in the PAX3 gene cause Waardenburg syndrome, whereas a chromosomal translocatio
27 Rare variants in PAX3 are known to cause Waardenburg syndrome, which involves deafness, pigmentar
30 to the molecular mechanisms by which certain Waardenburg syndrome-associated missense mutations could
31 the human melanocyte and deafness condition Waardenburg syndrome is sometimes caused by haploinsuffi
34 genes have been identified as causative for Waardenburg syndrome; however, the genetic bases for all
39 nstrate that PAX3, which is often mutated in Waardenburg syndrome, is required for normal enteric gan
40 e that the dominant melanophore phenotype in Waardenburg syndrome IV individuals with SOX10 mutations
49 ns a model for two human neurocristopathies, Waardenburg-Shah syndrome and Hirschsprung's disease.
50 X10 is mutated in the human neurocristopathy Waardenburg-Shah syndrome (WS4), which is characterized
51 gests a novel mechanism for the aetiology of Waardenburg-Shah syndrome in which affected neural crest
52 Because mutations in MITF are causative of Waardenburg syndrome type 2 (WS2), we screened KITLG in
53 sociated with the pigmentary deficiencies of Waardenburg syndrome (WS) and is modeled in Sox10 haploi
56 utations in SOX10 result in a severe form of Waardenburg syndrome, Type IV, also known as Waardenburg
63 or the diagnosis of the associated syndrome (Waardenburg (SOX10 and MITF), Kallmann (CHD7 and SOX10),
64 the occurrence of neurocristopathies in the Waardenburg-Shah syndrome type IV (WS-IV), for which the
67 gastrointestinal motility disorders, whereas Waardenburg syndrome consists of defective melanocyte fu
72 , mutations in this gene are associated with Waardenburg-Shah syndrome, a disorder characterized by p
75 utations have been reported in patients with Waardenburg-Hirschsprung syndrome and its murine model,
76 0 gene have been identified in patients with Waardenburg-Hirschsprung syndrome that is characterized
77 cordance of a pair of monozygotic twins with Waardenburg syndrome (WS) and previous reports of simila