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1                                              Wolff often used the human proximal femur as an example
2                                              Wolff rearrangements to 1,8-naphthyleneketenes (15a-d) a
3                                              Wolff's "law" of the functional adaptation of bone is ro
4                                              Wolff-Kishner conditions (KOH/ethylene glycol, 200 degre
5                                              Wolff-Parkinson-White is associated with high ARI disper
6 nm it results in efficient (phi(254) = 0.34) Wolff rearrangement, while irradiation with 355 nm light
7 ext of ventricular tachycardia (VT) (n = 9), Wolff-Parkinson-White (WPW) syndrome (n = 2), atrial fib
8 on of a ketene intermediate resulting from a Wolff rearrangement of the carbenoid, with a rhodium per
9                                      Here, a Wolff metallocarbene (Pt(II)-C-C(O)-OEt) is reported as
10 rovided for the use of TBSH derivatives in a Wolff-Kishner-type reduction protocol that proceeds at l
11                     The key steps involved a Wolff rearrangement, followed by a stereoselective dihyd
12 c management of asymptomatic patients with a Wolff-Parkinson-White (WPW) pattern is controversial.
13 ve consecutive patients aged<18 years with a Wolff-Parkinson-White pattern and persistent preexcitati
14 ophy and electrophysiological abnormalities: Wolff-Parkinson-White syndrome (WPW) and progressive deg
15 about the long-term natural history of adult Wolff-Parkinson-White syndrome (WPW) patients in regard
16 nm light provided diphenylacetic acid amide (Wolff rearrangement).
17 nm light provided diphenylacetic acid amide (Wolff rearrangement).
18 erstanding of the risk of sudden death among Wolff-Parkinson-White patients were published.
19 egrees C results in decarbonylation of 1 and Wolff rearrangement to fulven-6-one (13) either concerte
20 re ventricular complex (0.786 vs 0.800), and Wolff-Parkinson-White (0.800 vs 0.842).
21 ar arrhythmia, ventricular fibrillation, and Wolff-Parkinson-White.
22 ophy also present with skeletal myopathy and Wolff-Parkinson-White (WPW) syndrome; mutations in the g
23 te that leads to elimination of nitrogen and Wolff rearrangement is one of the highest singlet excite
24 fluorescence-plus-Giemsa method of Perry and Wolff to produce harlequin endoreduplicated chromosomes
25 ructurally normal hearts affected by SVT and Wolff-Parkinson-White syndrome and determine causality o
26  This enabled detection of such phenomena as Wolff-Parkinson-White syndrome, QRS aberrancy, and multi
27 e for re-entrant cardiac arrhythmias such as Wolff-Parkinson-White syndrome.
28 n proceeds via metal-free microwave-assisted Wolff rearrangement of the diazo reagent followed by Sta
29 d developed is based on a microwave-assisted Wolff rearrangement/Staudinger [2 + 2] cycloaddition seq
30  the evaluated patients with an asymptomatic Wolff-Parkinson-White preexcitation persisting at peak e
31  study of either symptomatic or asymptomatic Wolff-Parkinson-White patients referred to our Arrhythmo
32 ar tachycardia in patients with asymptomatic Wolff-Parkinson-White ECG pattern.
33 profile analyses reveal that the barrierless Wolff rearrangement proceeds via an out-of-plane carbene
34  LH, Biro FM, Valentin-Blasini L, Blount BC, Wolff MS, for the Breast Cancer and Environment Research
35 2,3-dihydropyridin-2-ylidene 26, followed by Wolff-type ring contraction to 1-azafulvenallene 15.
36 thyloxirene to diacetylcarbene and thence by Wolff rearrangement to acetylmethylketene.
37 athology of many diseases, including cancer, Wolff-Parkinson-White syndrome, neurodegenerative disord
38 ow report a novel mutation in PRKAG2 causing Wolff-Parkinson-White syndrome and conduction system dis
39 ntricular preexcitation, which characterizes Wolff-Parkinson-White syndrome, is caused by the presenc
40                    Inspired by the classical Wolff-Kishner reduction converting carbonyl groups in al
41 e diazodicarbonyl moiety, results in a clean Wolff rearrangement.
42    Thermal decomposition of 1 leads to clean Wolff rearrangement, while heating of 2 causes quantitat
43 cy than the ketone analogue due to competing Wolff rearrangement (WR) in the excited state of the dia
44               One pathway involves concerted Wolff rearrangement and nitrogen extrusion, most likely
45                In order to directly consider Wolff's observations, measurements were also made on two
46 ic study of organophotoredox cycloadditions, Wolff rearrangements, and azobenzene isomerization react
47 edox design, on the basis of dehydrogenation/Wolff-Kishner (WK) reduction, to simultaneously tackle t
48  a-diazoketones all undergo a very efficient Wolff rearrangement.
49 ular nodal reentrant tachycardia), excluding Wolff-Parkinson-White, comparing digoxin with propranolo
50 N,N-diethyl diazoamides) or almost exclusive Wolff rearrangement to ketenes (in the case of the cycli
51 s shown to be more effective in facilitating Wolff rearrangement than copper(I), although both are mo
52 utation (Arg302Gln) responsible for familial Wolff-Parkinson-White (WPW) syndrome.
53 979 and 1989 before undergoing operation for Wolff-Parkinson-White syndrome.
54 eactions, cyclopropanation, ylide formation, Wolff rearrangement, and cycloaddition reactions.
55  JG, Chen J, Holland NT, Barr DB, Perera FP, Wolff MS.
56 re was more neurology taught under Harold G. Wolff at Cornell University Medical College in New York
57 ine, or tetrahydrofurfuryl alcohol generates Wolff-rearranged, pyrrole ring-contracted azeteoporphyri
58  biochemical properties reminiscent of human Wolff-Parkinson-White syndrome, arising from mutations i
59 uperior alternatives to simple hydrazones in Wolff-Kishner-type reduction reactions, in the Barton vi
60 reatment of the clinical arrhythmias seen in Wolff-Parkinson-White syndrome.
61                           Two-photon induced Wolff rearrangement of a terphenyl diazoketone 1 was ach
62 es, in situ derived from a thermally induced Wolff rearrangement of 2-diazo-1,3-diketones, and N-(5-p
63 onding EP beta-lactams via an intramolecular Wolff rearrangement.
64 idt, Egon Macher, Gerd Steigleder, and Klaus Wolff.
65  rise to clean C-H insertion with only minor Wolff rearrangement to ketenes.
66                            (1) In 6 cases of Wolff-Parkinson-White syndrome, ECM accurately identifie
67 he adjacent phenyl group, and no evidence of Wolff rearrangement.
68  pre-excitation, a characteristic feature of Wolff-Parkinson-White syndrome.
69 d to be responsible for an inherited form of Wolff-Parkinson-White syndrome.
70                            The management of Wolff-Parkinson-White is based on the distinction betwee
71 ent retrospective and prospective studies of Wolff-Parkinson-White syndrome in asymptomatic pediatric
72 inception in the eighteenth century works of Wolff and Goethe, through the mid nineteenth century dis
73 some affected individuals (pre-excitation or Wolff-Parkinson-White syndrome).
74 ich multiple members were affected by SVT or Wolff-Parkinson-White pattern (preexcitation) on ECG ide
75 s, hypothyroidism is related to a persistent Wolff-Chaikoff effect and often has a vague presentation
76 ults in an efficient (Phi(254) = 0.34) photo-Wolff reaction, while at 355 nm, the formation of diazir
77 by a radical-carbanion relay sequence (photo-Wolff-Kishner reaction).
78 er in high ee, a scalable flow photochemical Wolff rearrangement to build the key bicyclo[3.1.1]hepta
79 hanism triggered by thermal or photochemical Wolff rearrangement of a diazo ketone.
80  becomes electron-rich, and the photoinduced Wolff rearrangement produces a highly emissive rhodol dy
81 c abnormalities, particularly preexcitation (Wolff-Parkinson-White syndrome) and atrioventricular con
82  synthetic utility of the thermally promoted Wolff rearrangement has been extended to the interaction
83 ial flutter, atrioventricular nodal reentry, Wolff-Parkinson-White syndrome, and atrial tachycardia.
84  HR: 1.73; 95% CI: 1.07-2.77, respectively), Wolff-Parkinson-White (WPW) syndrome (HR: 2.46; 95% CI:
85       This method is inspired by the Semmler-Wolff reaction, a classic method that exhibits limited s
86 duces ketenes by both concerted and stepwise Wolff rearrangements.
87  in individuals with familial reentrant SVT, Wolff-Parkinson-White ECG pattern, and structurally norm
88                       Among 8575 symptomatic Wolff-Parkinson-White patients with atrioventricular ree
89 f a key cyclobutane intermediate by a tandem Wolff rearrangement/asymmetric ketene addition, (2) a di
90                            Subsequent tandem Wolff rearrangement/lactonization of these alpha-diazo i
91 ntal and computational results indicate that Wolff rearrangement of the diacetylcarbene occurs with a
92 nusual wavelength selectivity indicates that Wolff rearrangement and isomerization originate from dif
93                                          The Wolff rearrangement (WR) is widely used for the synthesi
94                                          The Wolff-Parkinson-White registry was an 8-year prospective
95                                          The Wolff-Parkinson-White syndrome, with a prevalence in Wes
96 the very bulky dye residues was low, and the Wolff rearrangement did not occur.
97 rine 2 leads to the loss of nitrogen and the Wolff rearrangement, apparently via a carbene intermedia
98  processes: loss of nitrogen followed by the Wolff rearrangement and isomerization into diazo compoun
99 fies this intermediate as resulting from the Wolff rearrangement of the diazochlorins upon N(2) loss.
100 3 from Family 1 and 8 from Family 2) had the Wolff-Parkinson-White syndrome.
101 ognized as key reactive intermediates in the Wolff rearrangement and in interstellar environments.
102 xo carbene/carbenoid chemistry, that is, the Wolff rearrangement, has not been realized in this conte
103 H insertion product 6, while products of the Wolff rearrangement were not detected in both cases.
104 ions yields ketoester 3a, the product of the Wolff rearrangement, while products produced from the si
105 ures a previously unknown application of the Wolff-Kishner reduction of a nonresonance stabilized or
106 ought to characterize an animal model of the Wolff-Parkinson-White (WPW) syndrome to help elucidate t
107                         The prognosis of the Wolff-Parkinson-White syndrome essentially depends on in
108     Under these conditions, formation of the Wolff-rearranged product is inhibited and the phenyl add
109 as accomplished by a new modification of the Wolff-Semmler rearrangement.
110 y discloses the first examples that show the Wolff rearrangement can be readily realized by alpha-oxo
111 Laser power dependence studies show that the Wolff rearrangement is induced by two-photon absorption
112 Taken together, the results suggest that the Wolff rearrangement is subject to the same kind of nonst
113 calculations, allows us to conclude that the Wolff rearrangement of 1 is a concerted process.
114 e diterpenoid core was carried out under the Wolff rearrangement.
115 , more stereoselective approach utilizes the Wolff rearrangement as the key step.
116 owing that azeteoporphyrin formation via the Wolff rearrangement is dependent upon the structural dis
117      We identified two families in which the Wolff-Parkinson-White syndrome segregated as an autosoma
118 combination of photoredox catalysis with the Wolff-Kishner (WK) reaction allows the difunctionalizati
119 ement in most asymptomatic patients with the Wolff-Parkinson-White ECG pattern.
120 tegy in asymptomatic young patients with the Wolff-Parkinson-White electrocardiographic pattern are s
121 tene-based benzannulation in which a thermal Wolff rearrangement generates a vinylketene which combin
122 ) acetate leads to a remarkably facile "thia-Wolff rearrangement", producing thio-substituted ketenes
123 represents the lower limit to the barrier to Wolff rearrangement of the carbene.
124 in nascent carbenes results in the ultrafast Wolff rearrangement of the hot species.
125 yl-4-(alpha-diazoacyl)-1H-imidazoles undergo Wolff rearrangement followed by the selective 6pai-cycli
126                           The latter undergo Wolff rearrangement followed by the 6pai-cyclization of
127 n-poor core and, upon irradiation, undergoes Wolff rearrangement to give a ring-expanded xanthene cor
128 ischer carbene, which subsequently undergoes Wolff rearrangement and loss of CO.
129 igh yields from amino acids in two steps via Wolff rearrangement.
130  the serendipitous discovery of a vinylogous Wolff rearrangement, which serves to establish the C9 qu
131                               In adults with Wolff-Parkinson-White syndrome, a shortest RR interval <
132  R531G (AMPKgamma2(RG)), are associated with Wolff-Parkinson-White (WPW) syndrome, a cardiac disorder
133 thy children and adults when associated with Wolff-Parkinson-White syndrome.
134 eloped in adults also identify children with Wolff-Parkinson-White syndrome at risk for sudden death.
135 he largest reported to date of children with Wolff-Parkinson-White syndrome having a cardiac arrest,
136 w York, a teaching hospital of Cornell, with Wolff as my Director of Training.
137  for the asymptomatic pediatric patient with Wolff-Parkinson-White syndrome.
138             Fourteen pediatric patients with Wolff-Parkinson-White syndrome and no other congenital d
139                    Symptomatic patients with Wolff-Parkinson-White syndrome generally have a good out
140  of detailed long-term data in patients with Wolff-Parkinson-White syndrome is limited, and no prospe
141 rom pacing or preexcitation in patients with Wolff-Parkinson-White syndrome).
142 ing ventricular arrhythmias in patients with Wolff-Parkinson-White syndrome.
143 luate its outcome in pediatric patients with Wolff-Parkinson-White syndrome.
144  the involvement of benzazirines and without Wolff-type ring contraction to fulvenimines.

 
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