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1 o contribute to the biochemical pathology of X-linked adrenoleukodystrophy.
2 from the inflammatory cerebral phenotypes of X-linked adrenoleukodystrophy.
3 commonly involved in cerebral phenotypes of X-linked adrenoleukodystrophy.
4 mplicates this enzyme in the pathogenesis of X-linked adrenoleukodystrophy.
10 f 4PBA or HU in two distinct disease models, X-linked adrenoleukodystrophy and sickle cell disease.
11 c chondrodysplasia punctata, Refsum disease, X-linked adrenoleukodystrophy, and deficiency of mitocho
12 enomyeloneuropathy is the late-onset form of X-linked adrenoleukodystrophy, and is considered the mos
13 s, pathogenesis, diagnosis and prevention of X-linked adrenoleukodystrophy, and therapies are emergin
14 which accumulate in tissues of patients with X-linked adrenoleukodystrophy, are activated by very lon
15 n and spectroscopy have been well studied in X-linked adrenoleukodystrophy, but no data exist on magn
16 ral adrenoleukodystrophy is a severe form of X-linked adrenoleukodystrophy characterized by white-mat
17 evels were partially restored in transfected X-linked adrenoleukodystrophy fibroblasts regardless of
20 12 patients with childhood onset of cerebral X-linked adrenoleukodystrophy have been followed for 5-1
25 for a scientific commentary on this article.X-linked adrenoleukodystrophy is caused by mutations in
35 of 12 patients with childhood-onset cerebral X-linked adrenoleukodystrophy shows the long-term benefi
36 eview and evaluate the new information about X-linked adrenoleukodystrophy that has been reported in
37 Mutations in the gene encoding ALDP cause X-linked adrenoleukodystrophy; the role of ALDR and PMP7
38 noninflammatory myeloneuropathic variant of X-linked adrenoleukodystrophy, where the disease process
39 The most frequent peroxisomal disorder is X-linked adrenoleukodystrophy, which is caused by mutati
40 t widely used procedure for the diagnosis of X-linked adrenoleukodystrophy (X-ALD) and other peroxiso
41 tive was to study the phenotype evolution of X-linked adrenoleukodystrophy (X-ALD) and the relation b
42 usion tensor imaging (DTI), in children with X-linked adrenoleukodystrophy (X-ALD) before and after h
43 orescence assay, we determined the number of X-linked adrenoleukodystrophy (X-ALD) hemizygotes from t