ライフサイエンスコーパス: Xenopus oocyte

1 the influx of carbon dioxide (CO(2)) into a Xenopus oocyte.

2 at a desired distance from the membrane of a Xenopus oocyte.

3 ally with GORK and inhibits GORK activity in Xenopus oocytes.

4 d TREK-1 and TREK-2 subunits, coexpressed in Xenopus oocytes.

5 ne for fluorophore labeling, as expressed in Xenopus oocytes.

6 ast two different ways and expressed them in Xenopus oocytes.

7 lters the gating of human ClC-1 expressed in Xenopus oocytes.

8 -electrode voltage clamp after expression in Xenopus oocytes.

9 imulate NBCe1-A, heterologously expressed in Xenopus oocytes.

10 93 and 82%, respectively, when expressed in Xenopus oocytes.

11 an alpha1beta3gamma2L receptors expressed in Xenopus oocytes.

12 the mitochondria, and p38/JNK activation in Xenopus oocytes.

13 rdly rectifying potassium (GIRK) channels in Xenopus oocytes.

14 ities of GIRK1/2 and Gbetagamma expressed in Xenopus oocytes.

15 abditis elegans homomeric ACR-20 receptor in Xenopus oocytes.

16 ompounds on recombinant 5-HT3Rs expressed in Xenopus oocytes.

17 analysis of glutamate receptor responses in Xenopus oocytes.

18 nt channels were heterologously expressed in Xenopus oocytes.

19 ue proteins between nucleus and cytoplasm of Xenopus oocytes.

20 nd the mutants were functionally examined in Xenopus oocytes.

21 ng both human (h) and mouse (m) subunits, in Xenopus oocytes.

22 tivities of human AQP1 channels expressed in Xenopus oocytes.

23 tructs with the alpha5 subunit, expressed in Xenopus oocytes.

24 g progesterone-induced meiotic maturation of Xenopus oocytes.

25 g of somatic nuclei after transplantation to Xenopus oocytes.

26 ngth of S4 from engineered VSDs expressed in Xenopus oocytes.

27 r-mediated ionic currents in SERT-expressing Xenopus oocytes.

28 ivity of MCT1/4, heterologously expressed in Xenopus oocytes.

29 otassium ionic channels (Kv1.3) expressed in Xenopus oocytes.

30 and in ouabain-resistant pumps expressed in Xenopus oocytes.

31 r (FRET) and recording of Cav1.2 currents in Xenopus oocytes.

32 ponses to injections of Abeta oligomers into Xenopus oocytes.

33 variants, DmNav9-1, DmNav22, and DmNav26, in Xenopus oocytes.

34 nt potentiation of NMDA receptor currents in Xenopus oocytes.

35 he human cardiac Na(V) channel, Na(V)1.5, in Xenopus oocytes.

36 ed transport of RNA to the vegetal cortex in Xenopus oocytes.

37 a gliclazide with KATP channels expressed in Xenopus oocytes.

38 not impact on sugar transport as assayed in Xenopus oocytes.

39 various insect sodium channels expressed in Xenopus oocytes.

40 ant alpha1beta2gamma2 GABA(A)Rs expressed in Xenopus oocytes.

41 2;4 functional properties were reassessed in Xenopus oocytes.

42 essed in yeast (Saccharomyces cerevisiae) or Xenopus oocytes.

43 d following expression of SLC2A1 variants in Xenopus oocytes.

44 or for the TCS in maternal mRNAs in immature Xenopus oocytes.

45 the thermal sensitivity of CFTR channels in Xenopus oocytes.

46 nt populations of alpha3beta4alpha5 nAChR in Xenopus oocytes.

47 ents when it was heterologously expressed in Xenopus oocytes.

48 Slo2.1 channels heterologously expressed in Xenopus oocytes.

49 est at metaphases I and II in Drosophila and Xenopus oocytes.

50 nd pharmacology in ion channels expressed in Xenopus oocytes.

51 orters permeable to Ca(2+) when expressed in Xenopus oocytes.

52 wild-type AQP1 and the mutants expressed in Xenopus oocytes.

53 or all-mouse alpha6beta4*-nAChR expressed in Xenopus oocytes.

54 f K(o)(+) sensitivity of Kir4.2 expressed in Xenopus oocytes.

55 tutive kinase activity to promote M phase in Xenopus oocytes.

56 ver, SecA-liposomes elicit ionic currents in Xenopus oocytes.

57 tor PMA (phorbol 12-myristate 13-acetate) in Xenopus oocytes.

58 es, and (iv) recombinant NMDARs expressed in Xenopus oocytes.

59 and extending into the vegetal hemisphere of Xenopus oocytes.

60 onal effects of IRBITs on NBCn1 and NBCn2 in Xenopus oocytes.

61 odified with azide-reactive alkyne probes in Xenopus oocytes.

62 ing duct cell line, mirroring the results in Xenopus oocytes.

63 a1beta2gamma2 GABA(A) receptors expressed in Xenopus oocytes.

64 temeric ternary GABAA receptors expressed in Xenopus oocytes.

65 D)-, and (alpha3beta4)2alpha5(398N)-nAChR in Xenopus oocytes.

66 d SLC26A6-mediated Cl(-)-oxalate exchange in Xenopus oocytes.

67 fluence the timescale of RNA localization in Xenopus oocytes.

68 o reduced ENaC activity when co-expressed in Xenopus oocytes.

69 mediates a highly electrogenic transport in Xenopus oocytes.

70 nt channels were heterologously expressed in Xenopus oocytes.

71 ine flux measurements in mutant RNA-injected Xenopus oocytes.

72 (and unmodified controls) were expressed in Xenopus oocytes.

73 isoforms of NBCn1 and NBCn2 as expressed in Xenopus oocytes.

74 active muscle using human ClC-1 expressed in Xenopus oocytes.

75 and 5-HT3A serotonin receptors expressed in Xenopus oocytes.

76 s of embryonic muscle receptors expressed in Xenopus oocytes.

77 ion, functional GABA(A) Rs were expressed in Xenopus oocytes after microinjection with membrane fract

78 In conclusion, using Xenopus oocytes allowed us for the first time, to focus

79 alpha6/alpha3beta2beta3 nAChRs expressed in Xenopus oocytes (alpha6/alpha3 is a subunit chimera that

80 yeast S. cerevisiae, approximately 1000 from Xenopus oocytes and >1050 from human cells.

81 co (odorant receptor co-receptor subunit) in Xenopus oocytes and assayed by two-electrode voltage cla

82 USP8 increased ENaC current in Xenopus oocytes and collecting duct epithelia and enhanc

83 erin channels comprising MEC-4 and MEC-10 in Xenopus oocytes and examined their response to laminar s

84 ium channels (ENaC) in H441 and expressed in Xenopus oocytes and exposed mice in vivo.

85 Experiments in Xenopus oocytes and flies indicate that Hodor is a pH-se

86 Kv1.2 with the hERG channel for function in Xenopus oocytes and for overexpression in Pichia.

87 xt we examined channel surface expression in Xenopus oocytes and HeLa cells using a chemiluminescence

88 Third, electrophysiology in Xenopus oocytes and human embryonic kidney cell line 293

89 PCSK9 reduced ENaC current in Xenopus oocytes and in epithelia.

90 n of these proteins (and mutants thereof) in Xenopus oocytes and in mammalian cell lines.

91 diated increases of intracellular calcium in Xenopus oocytes and in neurons, and the latter is also d

92 cterized 7 dicarboxylic acid transporters in Xenopus oocytes and in Saccharomyces cerevisiae engineer

93 Human CFTR was heterologously expressed in Xenopus oocytes and its activity was electrophysiologica

94 In particular, in both Xenopus oocytes and mammalian Chinese hamster ovary cell

95 dulation, upregulates the Kv7.2/3 current in Xenopus oocytes and mammalian human embryonic kidney HEK

96 We expressed Kv4.2 channels in Xenopus oocytes and measured the onset of low-voltage in

97 high suppression potency in mammalian cells, Xenopus oocytes and mice in vivo, producing PTC repair i

98 Functional studies of mutant NaPi-IIa in Xenopus oocytes and opossum kidney (OK) cells demonstrat

99 Using 2-microelectrode experiments on Xenopus oocytes and patch-clamp electrophysiology on HEK

100 we expressed homotetrameric HCN2 channels in Xenopus oocytes and performed single-channel experiments

101 esses currents as an undocked hemichannel in Xenopus oocytes and provides a model system to study the

102 ombined electrophysiological measurements in Xenopus oocytes and pulldown experiments to analyze the

103 i function is required for the maturation of Xenopus oocytes and specifically for translational activ

104 ubtle effects are observed when expressed in Xenopus oocytes and studied with electrophysiology, does

105 USERs, expressed in Xenopus oocytes and tested using two-electrode voltage c

106 Using heterologous expression in Xenopus oocytes and the engineered cysteine-less hCNT3 p

107 Channels were heterologously expressed in Xenopus oocytes and the two-microelectrode voltage clamp

108 rected mutation constructs were expressed in Xenopus oocytes and their functionality and pharmacology

109 Binding studies in Xenopus oocytes and transfected HEK-293 cells revealed t

110 ediates translational repression in immature Xenopus oocytes and translational activation in mature o

111 hR subtypes were heterologously expressed in Xenopus oocytes and two-electrode voltage clamp recordin

112 s investigated by heterologous expression in Xenopus oocytes and yeast cells.

113 er NBCe1-A in an excised macropatch from the Xenopus oocyte, and indirectly stimulates NBCe1-B and -C

114 ns activate or repress the targeted mRNAs in Xenopus oocytes, and elicit poly(A) addition or removal.

115 ethods, electrophysiological measurements in Xenopus oocytes, and fluorescent microscopy of mammalian

116 ionally following heterologous expression in Xenopus oocytes, and mediates both inward and outward tr

117 e of human cardiac NKA isozymes expressed in Xenopus oocytes, and of native NKA isozymes in rat ventr

118 oral cortices of control and AD brains, into Xenopus oocytes, and recorded the electrophysiological a

119 dentified, synthesized, cloned, expressed in Xenopus oocytes, and studied by two-electrode voltage cl

120 cies) function of alpha6*-nAChR expressed in Xenopus oocytes, and that nAChR halpha6 subunit residues

121 and alpha1beta3 receptors were expressed in Xenopus oocytes, and the effects of substitutions of sel

122 ium channel, AaNav1-1, from Aedes aegypti in Xenopus oocytes, and the functional examination of nine

123 AQP0 were performed on protein expressed in Xenopus oocytes, and the results may therefore also refl

124 The mutants were expressed in Xenopus oocytes, and the unitary water and urea permeabi

125 These residues were mutated, expressed in Xenopus oocytes, and their functions assessed using elec

126 Homomeric MPTL-1 channels reconstituted in Xenopus oocytes are gated by microM concentrations of be

127 is necessary and sufficient for maintaining Xenopus oocytes arrested in prophase.

128 Here, we used Xenopus oocytes as a simple system to study LRRC8 protei

129 Using the Xenopus oocyte assay, we found an absence of GABA-A rece

130 In Xenopus oocyte assays TaNRT2.5 requires a partner protei

131 Here, using a Xenopus oocyte-based system to express and functionally

132 ith the different beta subunits expressed in Xenopus oocytes (beta1, beta2IR, beta3b, and beta4).

133 Similarly, injection of CFTR-1420-57 into Xenopus oocytes blocked the inhibition of cAMP-stimulate

134 e, in two-electrode voltage clamp studies in Xenopus oocytes, both Ca(2+) and Na(+) illicit 5-HT-indu

135 OS-7 cells, and voltage-clamp fluorimetry in Xenopus oocytes, both heterologously expressing the volt

136 PIAS2b is restricted to the cytoplasm of Xenopus oocytes but relocates to the nucleus immediately

137 cal range activated TRPV4 in Muller glia and Xenopus oocytes, but required phospholipase A(2) (PLA(2)

138 ese data indicate that H2S activates CFTR in Xenopus oocytes by inhibiting phosphodiesterase activity

139 hat increased SGLT2 activity in RNA-injected Xenopus oocytes by two orders of magnitude.

140 and brain (alpha4beta2) nAChRs expressed in Xenopus oocytes by using a two-electrode voltage clamp a

141 sis for human DMT1 expressed in RNA-injected Xenopus oocytes by using radiotracer assays and the cont

142 over, the purified alpha7nAChR injected into Xenopus oocytes can be activated by acetylcholine, choli

143 All 3 mutants cloned in Xenopus oocytes caused an aberrant modulation of the mec

144 ation by in vitro and ex vivo analysis using Xenopus oocyte, cell culture, and kidney tissue assays d

145 ously in both an Escherichia coli strain and Xenopus oocyte cells, AtDTX50 was found to facilitate AB

146 displayed robust repair capacity, including Xenopus oocytes, Chlamydomonas, and Stentor coeruleus Al

147 arious heterologous cell expression systems (Xenopus oocytes, CHO cells, and rat atrial cardiomyocyte

148 Expression of AE1-M909T in Xenopus oocytes confirmed preservation of its anion exch

149 Human NBCe1 heterologously expressed in Xenopus oocytes could be activated by adding 1-3 mm HCO3

150 from fluorophore-labeled hSERT expressed in Xenopus oocytes could be robustly detected at four posit

151 studies of PIEZO1 mutant R2488Q expressed in Xenopus oocytes demonstrated changes in ion-channel acti

152 Assay on expression in Xenopus oocytes demonstrated that SlPIP2s protein displa

153 Expression in Xenopus oocytes demonstrates that LGC-35 is a homopentam

154 Here we observe that AQP4-expressing Xenopus oocytes display a reflection coefficient <1 for

155 ort function by expressing these proteins in Xenopus oocytes, Drip, Prip, and Eglp2 show significant

156 se initiation and maintenance in Drosophila, Xenopus oocytes/eggs, and mammalian cells.

157 When PON-2 was co-expressed with ENaC in Xenopus oocytes, ENaC activity was reduced, reflecting a

158 In Xenopus oocytes, ERp29 overexpression increased the func

159 duration current-voltage (I-V) protocol with Xenopus oocytes expressing eGFP-tagged NBCe1-A, our grou

160 Our data demonstrate that in Xenopus oocytes expressing either OAT4 or OATP2B1 efflux

161 cell and single-channel electrophysiology of Xenopus oocytes expressing ENaC isoforms assembled from

162 cholesterol enrichment were also observed in Xenopus oocytes expressing GIRK2 channels, the primary G

163 ked the compounds for rapid activation using Xenopus oocytes expressing human alpha7 nAChR with a two

164 pounds were profiled by electrophysiology in Xenopus oocytes expressing human nicotinic acetylcholine

165 inylation and two-electrode voltage-clamp on Xenopus oocytes expressing NBCe1, we demonstrate that th

166 inhibition of glutamate-evoked currents from Xenopus oocytes expressing recombinant homo- or heterome

167 they inhibited uptake of (14)C-glucose into Xenopus oocytes expressing the human glucose transporter

168 cordance, elevated currents were observed in Xenopus oocytes expressing the Kir3.1/Kir3.4 heteromer t

169 g an endocytosis-defective Fpn mutant (K8R), Xenopus oocytes expressing wild-type or K8R Fpn, and mat

170 Using Xenopus oocyte expression and two-electrode voltage clam

171 In this study we utilized the Xenopus oocyte expression system to shed light on how CF

172 copa monnieri Screening was conducted in the Xenopus oocyte expression system, using quantitative swe

173 lamp technique and alpha4beta2 nAChRs in the Xenopus oocyte expression system, we demonstrate that in

174 degradation in human, HEK293 cells, and in a Xenopus oocyte expression system.

175 Using Xenopus oocyte expression, we investigated whether facil

176 Using the Xenopus oocytes expression system and two microelectrode

177 We observed that, in Xenopus oocytes, expression of Ggamma alone activated ho

178 ading MCM2-7 onto replication origins in the Xenopus oocyte extract system.

179 f-organization of stabilized microtubules in Xenopus oocyte extracts and find that they can form macr

180 Expressing SjAQP in Xenopus oocytes facilitated the permeation of water, gly

181 cid membrane protein that, when expressed in Xenopus oocytes, functions as an Na-Cl cotransporter wit

182 ation of ATP to hP2X7 receptors expressed in Xenopus oocytes gave rise to a current that had a biphas

183 In Xenopus oocytes, Gem suppresses the activity of P/Q-type

184 ts and concatemeric constructs, expressed in Xenopus oocytes, HEK 293 cells, and cultured hippocampal

185 curately recapitulates Ibasal and Ievoked in Xenopus oocytes, HEK293 cells and hippocampal neurons; c

186 Using ion-selective microelectrodes and Xenopus oocytes, here we studied Cl(-)/H(+) coupling pro

187 ly blocked acetylcholine-induced currents in Xenopus oocytes heterologously expressing human muscle-t

188 Utilizing inside-out patches from Xenopus oocytes heterologously expressing NKA, we observ

189 RNA injection of K(+)-transporter genes into Xenopus oocytes, however, both putative K(+) transporter

190 duced currents in this receptor expressed in Xenopus oocytes (IC50 = 236 nm) and less potently inhibi

191 lters the membrane conductance properties of Xenopus oocytes in a manner consistent with a large non-

192 l, as hyperpolarization of CNGC19-expressing Xenopus oocytes in the presence of both cyclic adenosine

193 membranolytic activity could be measured for Xenopus oocytes, in which CsTx-1 and CT1-long increase i

194 YD7 could translocate to the cell surface of Xenopus oocytes independently of the coexpression of alp

195 Mutation studies in Xenopus oocytes indicate that CAIX, via its intramolecul

196 maging conducted in murine retinal cells and Xenopus oocytes indicated that cell swelling in the phys

197 mistry and functional expression analysis in Xenopus oocytes indicates that the capacity of this H(+)

198 ansfer of mammalian somatic cell nuclei into Xenopus oocytes induces transcriptional reprogramming of

199 We used a voltage-clamp assay on Xenopus oocytes injected with the RNAs that encode the a

200 oximal domain, hERG variant were explored in Xenopus oocytes injected with the same amount of RNA enc

201 When expressed in Xenopus oocytes, IR64a and IR8a formed a functional ion

202 Overexpression of Ano1 in HEK cells or Xenopus oocytes is sufficient to generate Ca(2+)-activat

203 ion-induced translation of mRNAs in maturing Xenopus oocytes is the cytoplasmic polyadenylation eleme

204 ulating translation of reporter mRNAs during Xenopus oocyte maturation.

205 Lysosome acidification also occurs during Xenopus oocyte maturation; thus, a lysosomal switch that

206 Using Xenopus oocyte meiosis as a well-established physiologic

207 Kv1.3 disrupts the channel expression on the Xenopus oocyte membrane, suggesting a potential role as

208 on of human aquaporin 1 (hAQP1) expressed in Xenopus oocyte membranes.

209 s confirmed in the heterologously expressing Xenopus oocyte model.

210 In Xenopus oocytes, mutant TRESK subunits exert a dominant-

211 When co-expressed in Xenopus oocytes, NaDC-1 enhanced Slc26a6 transport activ

212 After injection into Xenopus oocyte nuclei, representative GANP-dependent tra

213 ing factor Ascl1 is injected directly into a Xenopus oocyte nucleus which has been preloaded with a l

214 Functional expression in Xenopus oocytes of concatenated pentameric (alpha7)5-, (

215 hen TRPV4 is heterologously expressed in the Xenopus oocyte or yeast.

216 Expression of AQP1 with CAII in Xenopus oocytes or mammalian cells increased water flux

217 Expression of GFP-OsPIP1;3 alone in Xenopus oocytes or rice protoplasts showed OsPIP1;3 misl

218 that reconstitution of NMDA-gated current in Xenopus oocytes, or C. elegans muscle cells, depends on

219 electrode voltage-clamp electrophysiology in Xenopus oocytes, oxytocin was found to completely block

220 Surface expression of PLM in Xenopus oocytes requires coexpression with the Na(+)/K(+

221 Wounded cells such as Xenopus oocytes respond to damage by assembly and closur

222 Coinjection of Rab14 in Xenopus oocytes results in a decrease of UT-A1 urea tran

223 d that coexpression of ASIC2b with ASIC1a in Xenopus oocytes results in novel proton-gated currents w

224 Heterologous expression of both genes in Xenopus oocytes revealed a strong conservation of charac

225 Electrophysiological characterization in Xenopus oocytes revealed that activity exclusively resid

226 orter in vivo However, functional studies in Xenopus oocytes revealed that MCT12 transports creatine

227 a1beta1epsilondelta AChRs (epsilon-AChRs) in Xenopus oocytes revealed that PEA selectively affected t

228 lectrode voltage clamp (TEVC) of transfected Xenopus oocytes revealed that the M2 S31N channel is ess

229 Electrophysiological characterization in Xenopus oocytes revealed that these derivatives differ i

230 For nAChRs expressed in Xenopus oocytes, S- and R-mTFD-MPAB inhibited ACh-induce

231 Although successful in Xenopus oocytes, single subunit counting, manually count

232 C remodeling and glycogen uptake in maturing Xenopus oocytes, suggesting that these processes are evo

233 ecule Na(+) /HCO3(-) cotransport activity in Xenopus oocytes, suggesting that they are suitable candi

234 Electrophysiological experiments in Xenopus oocytes support this notion and suggest that con

235 tics of each rescue protein were tested in a Xenopus oocyte swelling assay.

236 Here, we show in a heterologous Xenopus oocyte system as well as in Arabidopsis thaliana

237 Using the heterologous Xenopus oocyte system combined with molecular dynamics s

238 ode voltage clamp technique using a standard Xenopus oocyte system.

239 luding TREK-1, TREK-2, TRAAK; NaV1.5) in the Xenopus oocyte system.

240 We performed patch clamp studies in Xenopus oocytes that co-expressed BK channel-forming (cb

241 rrents of the Shaker Kv channel expressed in Xenopus oocytes that F184 not only interacts directly wi

242 Here, we investigated in Xenopus oocytes the impact of RasGAP and its fragments o

243 Similar to M phase progression in maturing Xenopus oocytes, the destruction of CPEB during the mamm

244 In Xenopus oocytes, the interaction of AtPP2CA with "phosph

245 In Xenopus oocytes, the IRBITs substantially increase the a

246 m at alpha6beta2,3delta GABAARs expressed in Xenopus oocytes, the pronounced agonism exhibited by the

247 tionality for Si transport when expressed in Xenopus oocytes, thus confirming the genetic capability

248 t responses in CquiOR136*CquiOrco-expressing Xenopus oocytes, thus suggesting a possible link between

249 describe how to use the technique for cells (Xenopus oocyte), tissues (Xenopus epithelium and rat cor

250 kinases and because Nav1.7 had been shown in Xenopus oocytes to be affected by protein kinases C and

251 ity of mosquito sodium channels expressed in Xenopus oocytes to both type I and type II pyrethroids.

252 y) renal cell lines and electrophysiology on Xenopus oocytes to characterize the mutant transporters

253 s and heterologous expression of channels in Xenopus oocytes to characterize the structural basis for

254 ecule imaging and stepwise photobleaching in Xenopus oocytes to directly determine the subunit stoich

255 The hDAT constructs were expressed in Xenopus oocytes to investigate if heightened membrane po

256 activation of BgNa(v) channels expressed in Xenopus oocytes to more negative membrane potentials but

257 bunits and/or concatamers were injected into Xenopus oocytes to obtain receptors of defined subunit c

258 two-electrode voltage clamp measurements in Xenopus oocytes together with targeted mutagenesis to in

259 We show that, when expressed in Xenopus oocytes, TRPV4 with the L596P or W733R mutation

260 Functional studies using a Xenopus oocyte two-microelectrode voltage clamp system r

261 ned the outward currents of TRPV4-expressing Xenopus oocyte upon depolarizations as well as phenotype

262 idea, we recorded NaV1.5 gating currents in Xenopus oocytes using a cut-open voltage-clamp with extr

263 M2-V27A mutant ion channels were measured in Xenopus oocytes using two-electrode voltage clamp (TEV)

264 mutant forms of the channel were measured in Xenopus oocytes using two-electrode voltage clamp assays

265 1 cells in a fluorescence-based assay and in Xenopus oocytes using two-electrode voltage clamp electr

266 d-type and chimeric Kv channels expressed in Xenopus oocytes, using the voltage-clamp technique.

267 Expression of THIK-1 channels in Xenopus oocytes was used to compare wild-type channels w

268 n coexpression of tandem PIP2-PIP1 dimers in Xenopus oocytes, we can address, for the first time to o

269 g studies in both sperm and voltage clamp of Xenopus oocytes, we define a molecular mechanism for GM1

270 Using heterologous expression in Xenopus oocytes, we demonstrate that NAM is a soluble ag

271 When expressed in Xenopus oocytes, we find that the R510H and Q913R-mutant

272 Using single molecule optical imaging in Xenopus oocytes, we found that MEC-4 forms homotrimers a

273 I(4,5)P(2)-sensitive KCNQ2/KCNQ3 channels in Xenopus oocytes, we identified four positions (A242C, R2

274 es ENaC functional and surface expression in Xenopus oocytes, we investigated the mechanism by which

275 Upon coexpression of ASIC1a and ASIC2a in Xenopus oocytes, we observed the formation of heteromers

276 A isoform IV was heterologously expressed in Xenopus oocytes, we observed, by measuring H(+) at the o

277 In Xenopus oocytes, we show that a genetically encoded vers

278 In HeLa cells and Xenopus oocytes, we show that Cx43-G8V, Cx43-A44V and Cx

279 le membranes from selected ALS patients into Xenopus oocytes, we show that PEA reduces the desensitiz

280 notropic glutamate receptor AMPA subunits in Xenopus oocytes, we show that this effect is through dir

281 Xenopus oocytes were injected with RNA encoding 5-HT(3)A

282 Functional Nav-LBT channels expressed in Xenopus oocytes were voltage-clamped, and distinct LRET

283 K(2P) channels, expressed heterologously in Xenopus oocytes, were measured by two-electrode voltage

284 d CNGC18 resulted in activation of CNGC18 in Xenopus oocytes where expression of CNGC18 alone did not

285 MEC evoked inward current in SERT-expressing Xenopus oocytes, whereas 4-MePPP was inactive in this re

286 mma2S GABAA receptors (GABAARs) expressed in Xenopus oocytes, whereas it displayed highly diverse fun

287 The cloned acHCN gene was expressed in Xenopus oocytes, which displayed a hyperpolarization-ind

288 work affect ion channels in the membrane of Xenopus oocytes, which shows the possibility to access a

289 We use injected Xenopus oocyte with two-electrode voltage clamp techniqu

290 specific, inhibiting KcsA-Shaker channels in Xenopus oocytes with a Ki of 0.5 nM whereas Shaker, Kv1.

291 he channel, mouse ENaCs were co-expressed in Xenopus oocytes with each of the 23 mouse DHHCs.

292 We expressed (alpha4beta2)2 concatamers in Xenopus oocytes with free accessory subunits to obtain d

293 method for CRISPR-mediated genome editing in Xenopus oocytes with homology-directed repair (HDR) that

294 edo (alphabetagammadelta) nAChR expressed in Xenopus oocytes with IC50 values of approximately 1 muM.

295 urally altered TaALMT1 proteins expressed in Xenopus oocytes with phylogenic analyses of the ALMT fam

296 siological measurements in HEK-293 cells and Xenopus oocytes with pulldown experiments, we analyzed t

297 ge clamp electrophysiology, we found that in Xenopus oocytes with RACK1 overexpression Pkd2L1 channel

298 and screened this library by coexpression in Xenopus oocytes with TRPA1.

299 nd segregation of Rho and Cdc42 zones during Xenopus oocyte wound repair and the role played by Abr,

300 In RNA-injected Xenopus oocytes, ZIP8-mediated (55)Fe(2+) transport was