ライフサイエンスコーパス: Yersinia

1 spiratory electron acceptor tetrathionate by Yersinia.

2 pe III secretion system (T3SS) of pathogenic Yersinia.

3 data and analysis tools for the analysis of Yersinia.

4 iaTree for constructing phylogenetic tree of Yersinia.

5 the function of the pathogenic machinery in Yersinia.

6 lling the host colonization and virulence of Yersinia.

7 c E. coli, Salmonella, Shigella, Vibrio, and Yersinia.

8 initially low-affinity antibacterial (e.g., Yersinia) Abs cross-reactive with TSHR, eventually leadi

9 Yersinia adhesin A (YadA) belongs to a class of bacteria

10 avirulent strains of Gram-negative bacteria, Yersinia and Klebsiella, and less so by their wild-type

11 compartment showed extreme susceptibility to Yersinia and were deficient in monocyte and neutrophil-d

12 Using Yersinia as a surrogate host, we demonstrated that many

13 Yersinia bacteria cause a range of human diseases, inclu

14 Pathogenic Yersinia bacteria cause a range of human diseases.

15 Pathogenic Yersinia bacteria inject effector proteins termed Yops,

16 crystal structure of the Anbu particle from Yersinia bercovieri (YbAnbu).

17 ronic inflammation, which persist long after Yersinia clearence.

18 ehavior has been reported for Salmonella and Yersinia, consistent with selection of social behavior t

19 tion system, whereas the delivery of GBP2 to Yersinia-containing vacuoles (YCVs) requires hypersecret

20 nded DNA (dsDNA) to a type I-F CRISPR system yersinia (Csy) surveillance complex leads to large quate

21 regulation have come from studies of several Yersinia effectors, which are injected into phagocytes a

22 of pyrin regulation and its interaction with Yersinia effectors.

23 During infection, Yersinia employs two virulence factors, YopE and YopH, t

24 tably Aeromonas sp. (23.8%) by FilmArray and Yersinia enterocolitica (48.1%) by the Luminex assay.

25 li (n = 14, 8%), norovirus (n = 14, 8%), and Yersinia enterocolitica (n = 7, 4%).

26 a coli (n=14, 8%), norovirus (n=14, 8%), and Yersinia enterocolitica (n=7, 4%).

27 hogens Yersinia pseudotuberculosis (Ypt) and Yersinia enterocolitica (Ye), as well as the causative a

28 ed defective clearance of the ileal pathogen Yersinia enterocolitica and an elevated inflammatory cyt

29 Escherichia coli, Yersinia enterocolitica and Salmonella enterica serovar

30 pe stress response required for virulence in Yersinia enterocolitica and Salmonella enterica.

31 rrelation spectroscopy, we show that in live Yersinia enterocolitica bacteria these soluble proteins

32 Yersinia enterocolitica biovar 1B employs two type three

33 Yersinia enterocolitica causes a severe enteric infectio

34 Infection with Yersinia enterocolitica causes acute diarrhea in early c

35 The enteropathogenic bacterium Yersinia enterocolitica deactivates TLR-induced signalin

36 e III secretion system of the human pathogen Yersinia enterocolitica enabled efficient identification

37 t an aberrant acute inflammatory response to Yersinia enterocolitica infection leads to long-lasting

38 Yersinia enterocolitica is an enteropathogenic bacterium

39 Yersinia enterocolitica is typically considered an extra

40 gainst siderophilic extracellular pathogens (Yersinia enterocolitica O9) by controlling non-transferr

41 tween mucosa and joints in a murine model of Yersinia enterocolitica O:3-induced reactive arthritis (

42 conjugation-ready tetrasaccharide of O-PS of Yersinia enterocolitica O:50 strain 3229 and the trisacc

43 chaperone in the physiology and virulence of Yersinia enterocolitica serotype O:3.

44 nit from the lipopolysaccharide O-antigen of Yersinia enterocolitica serovars O:5/O:5,27.

45 er Yersinia species, and differences between Yersinia enterocolitica subsp. enterocolitica and Yersin

46 nia enterocolitica subsp. enterocolitica and Yersinia enterocolitica subsp. palearctica.

47 rophomonas maltophilia, Vibrio cholerae, and Yersinia enterocolitica T2S-expressing plant pathogens i

48 ins, fused to the N-terminal fragment of the Yersinia enterocolitica T3S substrate YopE, are effectiv

49 btype the important human-pathogenic species Yersinia enterocolitica to whole-genome resolution level

50 protection against lethal oral infections by Yersinia enterocolitica WA and Y. pseudotuberculosis PB1

51 We report the MgADP structure of Yersinia enterocolitica YopO in complex with actin, whic

52 , is composed of ~22 copies of SctQ (YscQ in Yersinia enterocolitica), which require the presence of

53 lmonella spp., Vibrio spp., Vibrio cholerae, Yersinia enterocolitica, enteroaggregative E. coli, ente

54 eserved stool specimens for the detection of Yersinia enterocolitica, enterotoxigenic Escherichia col

55 Staphylococcus aureus, Escherichia coli and Yersinia enterocolitica--demonstrated that the zone of i

56 cute enteric infection by the proteobacteria Yersinia enterocolitica.

57 olic flagellin of Salmonella Typhimurium and Yersinia enterocolitica.

58 causes plague, Yersinia pseudotuberculosis, Yersinia enterocolitica.

59 ighly resistant to orogastric infection with Yersinia enterocolitica.

60 t Eschericia coli ClyA and the two component Yersinia enterolytica YaxAB show both undergo conformati

61 toxin YenTc, produced by the insect pathogen Yersinia entomophaga.

62 acilitate the ongoing and future research of Yersinia, especially those generally considered non-path

63 comparative genomics, and investigations of Yersinia-flea interactions have disclosed the important

64 ved two structures of an ASBT homologue from Yersinia frederiksenii (ASBTYf) in a lipid environment,

65 Pathogenic bacteria such as Listeria and Yersinia gain initial entry by binding to host target ce

66 Comparison of Yersinia genomes revealed that DNA encoding YapV or each

67 T) for comparative pathogenomics analysis of Yersinia genomes; (3) YersiniaTree for constructing phyl

68 and analysis platform is needed to hold the Yersinia genomic data and analysis tools for the Yersini

69 ialized platform to hold the rapidly-growing Yersinia genomic data and to provide analysis tools part

70 ce and analysis platform for the analysis of Yersinia genomic data.

71 on of sRNAs to the regulation of the T3SS in Yersinia has been largely unstudied, however.

72 The genus Yersinia has been used as a model system to study pathog

73 in sequencing technologies, many genomes of Yersinia have been sequenced.

74 ociated with urinary E. coli isolates is the Yersinia high pathogenicity island (HPI), which directs

75 The Yersinia high-pathogenicity island (HPI) is common to mu

76 vo, suggesting that cell death promotes anti-Yersinia host defense.

77 and other Enterobacteriaceae expressing the Yersinia HPI also secrete escherichelin, a second metall

78 e escherichelin biosynthetic capacity of the Yersinia HPI while eliminating the Ybt biosynthetic capa

79 fect may relate to the apparent selection of Yersinia HPI-positive E. coli in uncomplicated clinical

80 ses play an important role in promoting anti-Yersinia immune defense.

81 of immune signaling by YopJ to promote anti-Yersinia immune defense.

82 unity." The Gram-negative bacterial pathogen Yersinia inactivates critical proteins of the NF-kappaB

83 Pathogenic Yersinia, including Y. pestis, the agent of plague in hu

84 PK1-dependent cell death, we now reveal that Yersinia-induced apoptosis is critical for host survival

85 In this article, we show that Yersinia-induced apoptosis of human macrophages involves

86 Yersinia-induced apoptosis requires the kinase activity

87 3 and caspase-8 or FADD completely abrogated Yersinia-induced cell death and caspase-1 activation.

88 ticle, we report a key role for TNF/TNFR1 in Yersinia-induced cell death of murine macrophages, which

89 R4/TRIF-independent pathways in the death of Yersinia-infected cells.

90 In turn,Yersinia-infected macrophages respond to translocation o

91 In Yersinia-infected macrophages, YopE or YopT triggers inf

92 he processing of MyD88 was not restricted to Yersinia infection and to proapoptotic Toll-IL-1R domain

93 Nevertheless, Yersinia infection induces inflammatory responses in viv

94 vivo cell death-dependent immune control of Yersinia infection, a physiological model of TAK1/IKK in

95 nt mice displayed enhanced susceptibility to Yersinia infection.

96 e cells, and were attenuated in disseminated Yersinia infection.

97 Yersinia is a Gram-negative bacteria that includes serio

98 The genus Yersinia is a large and diverse bacterial genus consisti

99 accurate and reproducible identification of Yersinia isolates to the species level, a process often

100 ucture of the CdiA-CT/CdiIYkris complex from Yersinia kristensenii ATCC 33638.

101 Enterobacter, Vibrio, Shigella, Salmonella, Yersinia, Mycobacterium and Bacillus-yet are relatively

102 e of NF-kappaB and MAPK signaling imposed by Yersinia on infected cells.

103 ichia, Salmonella, Klebsiella, Shigella, and Yersinia opportunistic pathogens, the structure of GusR

104 Here we show that YmoB, the Yersinia orthologue of TomB, and its single cysteine var

105 The Yersinia outer protein J (YopJ) family of bacterial effe

106 The Yersinia outer protein J (YopJ) family of T3Es is a wide

107 dent upon type III secretion system effector Yersinia outer protein J (YopJ), is minimally affected b

108 de host immune defences through injection of Yersinia outer proteins (Yops) into phagocytic cells.

109 These effectors, termed Yersinia outer proteins (Yops), modulate multiple host s

110 an injectisome and effector proteins, called Yersinia outer proteins (Yops), that modulate activation

111 responses in infected macrophages to promote Yersinia pathogenesis.

112 a) in a 19th century intestinal specimen and Yersinia pestis ("Black Death" plague) in a medieval too

113 d release of Bacillus anthracis (anthrax) or Yersinia pestis (plague) would prompt a public health em

114 ), as well as the causative agent of plague, Yersinia pestis (Yp).

115 Here we studied the interaction between the Yersinia pestis ABC heme importer (HmuUV) and its partne

116 Yersinia pestis adopts a unique life stage in the digest

117 -resistant Staphylococcus aureus, as well as Yersinia pestis and Bacillus anthracis, organisms of bio

118 tant of 20 nM between EGFP-labeled LcrV from Yersinia pestis and its cognate membrane-bound protein Y

119 tant to pigmentation locus-negative (pgm(-)) Yersinia pestis and that this phenotype maps to a 30-cen

120 s both the protective F1 capsular antigen of Yersinia pestis and the LcrV protein required for secret

121 e identification of new virulence factors in Yersinia pestis and understanding their molecular mechan

122 owed differences in virulence genes found in Yersinia pestis and Yersinia pseudotuberculosis compared

123 nt of YopJ-dependent cytotoxicity induced by Yersinia pestis and Yersinia pseudotuberculosis paradoxi

124 ncisella tularensis, Bacillus anthracis, and Yersinia pestis are tier 1 select agents with the potent

125 t historically documented pandemic caused by Yersinia pestis began as the Justinianic Plague in 541 w

126 molecule cyclic diguanylate is essential for Yersinia pestis biofilm formation that is important for

127 The 14th-18th century pandemic of Yersinia pestis caused devastating disease outbreaks in

128 Yersinia pestis causes a rapid, lethal disease referred

129 Yersinia pestis causes bubonic plague, a fulminant disea

130 Yersinia pestis causes bubonic, pneumonic, and septicemi

131 Yersinia pestis causes bubonic, pneumonic, and septicemi

132 Yersinia pestis causes the fatal respiratory disease pne

133 Braun lipoprotein (Lpp) and MsbB attenuated Yersinia pestis CO92 in mouse and rat models of bubonic

134 ficant protection from a lethal challenge of Yersinia pestis CO92.

135 Yersinia pestis continues to cause sporadic cases and ou

136 ts the most severe form of disease caused by Yersinia pestis due to its ease of transmission, rapid p

137 Yersinia pestis has caused at least three human plague p

138 ere, we report the oldest direct evidence of Yersinia pestis identified by ancient DNA in human teeth

139 tudies (influenza A in lesser snow geese and Yersinia pestis in coyotes), we argue that with careful

140 ated with iron(III)-yersiniabactin import in Yersinia pestis In this study, we compared the impact of

141 responses of C57BL/6 and SEG MPs exposed to Yersinia pestis in vitro were examined.

142 n lymph nodes (LNs), or buboes, characterize Yersinia pestis infection, yet how they form and functio

143 Yersinia pestis is a Gram-negative bacterium that is the

144 Yersinia pestis is a tier 1 agent due to its contagious

145 Yersinia pestis is an arthropod-borne bacterial pathogen

146 Bubonic plague results when Yersinia pestis is deposited in the skin via the bite of

147 This frenzied inflammatory response to Yersinia pestis is poorly understood.

148 Yersinia pestis is the causative agent of bubonic and pn

149 Yersinia pestis is the causative agent of bubonic, pneum

150 Yersinia pestis is the causative agent of plague.

151 The bacteria Yersinia pestis is the etiological agent of plague and h

152 The plague bacillus Yersinia pestis is unique among the pathogenic Enterobac

153 e is the deadliest form of disease caused by Yersinia pestis Key to the progression of infection is t

154 We find the origins of the Yersinia pestis lineage to be at least two times older t

155 A Yersinia pestis mutant synthesizing an adjuvant form of

156 Using the Yersinia pestis outer transmembrane beta-barrel Ail as a

157 n are broadly used in many fields, including Yersinia pestis pathogenesis.

158 through membrane remodeling is a hallmark of Yersinia pestis pathogenesis.

159 multiPhATE by annotating two newly sequenced Yersinia pestis phage genomes.

160 Yersinia pestis remains endemic in Africa, Asia, and the

161 Early after inhalation, Yersinia pestis replicates to high numbers in the airway

162 Inhalation of the bacterium Yersinia pestis results in primary pneumonic plague.

163 roinflammatory responses through TLRs by the Yersinia pestis T3S needle protein, YscF, the Salmonella

164 lethal human disease caused by the bacterium Yersinia pestis This study demonstrated that the Y. pest

165 mal system for interrogating such couplings: Yersinia pestis transmission exerts intense selective pr

166 Mutant pyrin interacts less avidly with Yersinia pestis virulence factor YopM than with wild-typ

167 .3, raised to the LcrV virulence factor from Yersinia pestis were characterised for their Fab affinit

168 cessfully produce disease, the causal agent (Yersinia pestis) must rapidly sense and respond to rapid

169 a and Salmonella enterica serovar Typhi, and Yersinia pestis), and 3 protozoa (Leishmania spp., Plasm

170 ymerization of a single protein, e.g., YscF (Yersinia pestis), PscF (Pseudomonas aeruginosa), PrgI (S

171 orical interest - pre-modern bubonic plague (Yersinia pestis), smallpox (Variola virus) and cholera (

172 n, the serine/threonine kinase YopO (YpkA in Yersinia pestis), uses monomeric actin as bait to recrui

173 trophic European Black Death/bubonic plague (Yersinia pestis).

174 Plague is initiated by Yersinia pestis, a highly virulent bacterial pathogen.

175 he consequence of a singular introduction of Yersinia pestis, after which the disease established its

176 rofloxacin resistance in Bacillus anthracis, Yersinia pestis, and Francisella tularensis.

177 een host-pathogen interactions of B. mallei, Yersinia pestis, and Salmonella enterica.

178 plague, the most severe form of infection by Yersinia pestis, are needed, as past US Food and Drug Ad

179 include the bacteria Francisella tularensis, Yersinia pestis, Burkholderia mallei, and Brucella speci

180 a multifunctional outer membrane protein of Yersinia pestis, confers cell binding, Yop delivery and

181 The second plague pandemic, caused by Yersinia pestis, devastated Europe and the nearby region

182 -six distinct strains of Bacillus anthracis, Yersinia pestis, Francisella tularensis, Burkholderia ma

183 lytic domains from four pathogenic bacteria: Yersinia pestis, Francisella tularensis, Burkholderia ps

184 Plague, caused by the bacterium Yersinia pestis, has killed millions in historic pandemi

185 ns cause host cell death upon infection, and Yersinia pestis, infamous for its role in large pandemic

186 ns, particularly the N terminus of YscF from Yersinia pestis, influences host immune responses.

187 Here we show that in Yersinia pestis, irp2, a gene encoding the synthetase (H

188 Pneumonic plague, caused by Yersinia pestis, is a rapidly progressing contagious dis

189 lague, caused by the Gram-negative bacterium Yersinia pestis, is favored by a robust early innate imm

190 onment where the agent of flea-borne plague, Yersinia pestis, must replicate to produce a transmissib

191 ng disease throughout human history, such as Yersinia pestis, Mycobacterium tuberculosis, and Mycobac

192 biothreat agents such as Bacillus anthracis, Yersinia pestis, or Burkholderia pseudomallei Convention

193 r gene products are functional receptors for Yersinia pestis, the agent of plague, as shown by overex

194 f pyrin by YopM is required for virulence of Yersinia pestis, the agent of plague.

195 The arthropod-borne transmission route of Yersinia pestis, the bacterial agent of plague, is a rec

196 Yersinia pestis, the bacterium that causes plague, is a

197 Yersinia pestis, the causative agent of plague, binds ho

198 Yersinia pestis, the causative agent of plague, expresse

199 hat distinguish DNA amplicons generated from Yersinia pestis, the causative agent of plague, from the

200 For transmission to new hosts, Yersinia pestis, the causative agent of plague, replicat

201 nd F1-V, a candidate recombinant antigen for Yersinia pestis, the causative agent of plague.

202 m (T3SS) is essential in the pathogenesis of Yersinia pestis, the causative agent of plague.

203 f essential gene prediction in the bacterium Yersinia pestis, the causative agent of plague.

204 In Yersinia pestis, the deadly agent that causes plague, th

205 Yersinia pestis, the etiologic agent of plague, is a bac

206 t the discovery and genome reconstruction of Yersinia pestis, the etiological agent of plague, in Neo

207 The causative agent of plague, Yersinia pestis, uses a type III secretion system to sel

208 that includes serious pathogens such as the Yersinia pestis, which causes plague, Yersinia pseudotub

209 ncient plague strains are basal to all known Yersinia pestis.

210 major surface protein of the deadly pathogen Yersinia pestis.

211 genome sequences, of which the majority are Yersinia pestis.

212 ue is a deadly respiratory disease caused by Yersinia pestis.

213 ement for Hfq in the closely related species Yersinia pestis.

214 were challenged with inhaled lethal doses of Yersinia pestis.

215 including CDC category A/B pathogens such as Yersinia pestis.

216 lysaccharide (LPS) for the enteric pathogens Yersinia pseudotuberculosis (Ypt) and Yersinia enterocol

217 infection sites, we established a system for Yersinia pseudotuberculosis (Yptb) growth in microfluidi

218 elated food- and waterborne enteric pathogen Yersinia pseudotuberculosis A combination of population

219 secreted in a type III-dependent manner from Yersinia pseudotuberculosis and also secreted from C. tr

220 Enteric pathogens such as Yersinia pseudotuberculosis and enteropathogenic Escheri

221 structures, we mapped the RNA structurome of Yersinia pseudotuberculosis at three different temperatu

222 and core metabolism in the enteric pathogen Yersinia pseudotuberculosis by integrated transcriptome

223 virulence genes found in Yersinia pestis and Yersinia pseudotuberculosis compared to other Yersinia s

224 lymph nodes and associated lymphatics after Yersinia pseudotuberculosis infection and clearance.

225 esponses within SLOs during gastrointestinal Yersinia pseudotuberculosis infection to limit pathogen

226 vated with lipopolysaccharide (LPS) prior to Yersinia pseudotuberculosis infection, caspase-1 is acti

227 In a mouse model of Yersinia pseudotuberculosis infection, we show that at l

228 ensal microbiota or animal susceptibility to Yersinia pseudotuberculosis infection.

229 We demonstrate that, in addition to MyD88, Yersinia pseudotuberculosis inhibits TRIF signaling thro

230 Yersinia pseudotuberculosis is a foodborne pathogenic ba

231 Yersinia pseudotuberculosis is a Gram-negative enteropat

232 Here, we constructed a Yersinia pseudotuberculosis mutant strain with arabinose

233 ordingly, caspase-1-dependent clearance of a Yersinia pseudotuberculosis mutant was enhanced in BCAP-

234 cytotoxicity induced by Yersinia pestis and Yersinia pseudotuberculosis paradoxically leads to decre

235 n this study, a novel recombinant attenuated Yersinia pseudotuberculosis PB1+ strain (chi10069) engin

236 itating biofilm on Caenorhabditis elegans by Yersinia pseudotuberculosis represents a tractable model

237 have suggested that rfaH may be required for Yersinia pseudotuberculosis resistance to antimicrobial

238 We report that oral infection with Yersinia pseudotuberculosis results in the development o

239 ium evolved from an ancestral enteroinvasive Yersinia pseudotuberculosis strain by gene loss and acqu

240 PS)-primed murine macrophages with DeltayopM Yersinia pseudotuberculosis strains expressing wild-type

241 itor identified by in vitro screening, using Yersinia pseudotuberculosis Using a mouse model of P. ae

242 protein YtfE contributes to the survival of Yersinia pseudotuberculosis within the spleen following

243 iI complexes from Escherichia coli EC869 and Yersinia pseudotuberculosis YPIII to explore the evoluti

244 nal peptide generated by auto-proteolysis of Yersinia pseudotuberculosis YscU, is secreted by the T3S

245 acterial pathogen that evolved recently from Yersinia pseudotuberculosis, an enteric pathogen transmi

246 Following clearance of Yersinia pseudotuberculosis, sustained inflammation and

247 Yersinia pseudotuberculosis, the closely related food-an

248 as the Yersinia pestis, which causes plague, Yersinia pseudotuberculosis, Yersinia enterocolitica.

249 Using this tool for Yersinia pseudotuberculosis-infected lymphatic tissues,

250 Salmonella enterica serovar Typhimurium and Yersinia pseudotuberculosis.

251 uring primary infection of C57BL/6 mice with Yersinia pseudotuberculosis.

252 of the T3SS in the gastrointestinal pathogen Yersinia pseudotuberculosis.

253 haracterize modified peptide-cytidylate from Yersinia pseudotuberculosis.

254 s cell death mediates immune defense against Yersinia remain poorly defined.

255 eature of T3SA, as an effectorless strain of Yersinia remains confined to phagosomes.

256 Yersinia remodels its membrane during its life cycle as

257 inia genomic data and analysis tools for the Yersinia research community.

258 the YersiniaBase, a robust and user-friendly Yersinia resource and analysis platform for the analysis

259 rm is the use of the Csy-type (CRISPR system yersinia) ribonuclease 4 (Csy4) and tRNA processing enzy

260 eromonas salmonicida subsp. salmonicida, and Yersinia ruckeri and a parasitic ciliate Ichthyophthiriu

261 Here we studied Yersinia ruckeri antifeeding prophage 18 (Afp18), the to

262 Yersinia ruckeri causes enteric redmouth disease (ERM) t

263 Yersinia ruckeri produces two surface-exposed adhesins,

264 oyed by many pathogens, including the genera Yersinia, Shigella, Pseudomonas, and Salmonella, to deli

265 Yersinia species cause zoonotic infections, including en

266 Pathogenic Yersinia species deliver Yop effector proteins through a

267 Pathogenic Yersinia species employ several strategies to evade the

268 The YopJ protein of pathogenic Yersinia species inhibits NF-kappaB and MAPK signaling,

269 t contrary to hypotheses that all pathogenic Yersinia species share a recent common pathogenic ancest

270 Pathogenic Yersinia species utilize a type III secretion system to

271 ersinia pseudotuberculosis compared to other Yersinia species, and differences between Yersinia enter

272 tical virulence component for the pathogenic Yersinia species, and the regulation of this system is t

273 Using whole-genome sequencing of all Yersinia species, we delineate the gene complement of th

274 tial virulence factor produced by pathogenic Yersinia species.

275 lavage (BAL) fluid from immunized mice, and Yersinia-specific CD4(+) and CD8(+) T cells producing hi

276 We identified a Yersinia-specific sRNA, Ysr141, carried by the T3SS plas

277 potential to elicit broad protection against Yersinia spp.

278 ulation, while its similarity with yscW from Yersinia spp. argued in favor of a role in machinery ass

279 Vibrio spp., Salmonella spp., Shigella spp., Yersinia spp., Citrobacter spp., enterotoxigenic (ETEC)

280 itors of type III secretion system (T3SS) in Yersinia spp., have an inhibitory effect on chlamydial i

281 However, some non-pestis Yersinia strains and Enterobacteriaceae did elicit signa

282 quence typing-based scheme that can identify Yersinia strains to the species level to a level of reso

283 & Microbe, Chung et al. (2016) show that the Yersinia T3SS effector protein YopM counteracts this rec

284 In Yersinia, the switch to secretion of effector proteins i

285 e sites may be a strategy used by pathogenic Yersinia to prevent inactivation of this important virul

286 which the initial attachment/recognition of Yersinia to/by C. elegans is followed by bacterial growt

287 The Yersinia translocation regulatory protein YopK promotes

288 r findings indicate that lysosomal damage by Yersinia translocon proteins promotes inflammasome activ

289 g vacuoles (YCVs) requires hypersecretion of Yersinia translocon proteins.

290 The Yersinia type III secretion system (T3SS) translocates Y

291 Yersinia up-regulates the gene and expression dose of th

292 In this study, we identify the Yersinia urease enzyme as the responsible oral toxin.

293 ells, thereby limiting cellular detection of Yersinia virulence activity.

294 ckade of NF-kappaB and MAPK signaling by the Yersinia virulence factor YopJ inhibits cytokine product

295 -1 in activated macrophages and in promoting Yersinia virulence.

296 bust effector Yop function, is important for Yersinia virulence.

297 e to host defense peptides, and virulence of Yersinia, we constructed DeltarfaH mutants of Y. pseudot

298 to express the invasin surface receptor from Yersinia, which enables uptake via mammalian host beta1-

299 esigned to interfere with internalization of Yersinia without disturbing endocytosis.

300 he immunogenicity and protective capacity of Yersinia YopB, a conserved type III secretion system pro