1 Thus, our findings are consistent with
a model whereby a distorted estimation of the gain cue u
2 Our results are compatible with
a model whereby a genetic variant in pri-mir-30c-1 leads
3 Our data support
a model whereby a hot L1 source element on Chromosome 17
4 The data support
a model whereby a hydrophobic patch on the ProTx-II surf
5 ased upon our collective results, we propose
a model whereby a newly exported, partially folded inter
6 In summary, we provide
a model whereby a novel interplay between RNA-binding, R
7 Additional biochemical experiments support
a model whereby a single Ca(2+)/CaM bridges the C-termin
8 These data illustrate
a model whereby a single TF alters multi-loop hubs to dr
9 This work supports
a model whereby ABA-triggered stomatal closure requires
10 We believe our results support
a model whereby acidic pH, high ionic strength, or expos
11 We propose
a model whereby activated Fgfr signals through Ras-MAPK
12 These data support
a model whereby activating MLLT1 mutations early in rena
13 Our findings lead to
a model whereby activation of midplastid-localized ARC3
14 Together, these results support
a model whereby afadin determines lumen placement by dir
15 ferative "transit-amplifying" cells supports
a model whereby alpha-cells expand by self-renewal and n
16 preferred substrate for dFic, thus endorsing
a model whereby AMPylation regulates the function of BiP
17 Together our data support
a model whereby antiviral RNAi competes with endogenous
18 Our results support
a model whereby AnxA2 limits the availability of TRPA1 c
19 The results support
a model whereby apo-AcnB directly interacts with the pgd
20 Together, these data support
a model whereby arginine methylation modulates dynamic a
21 Our data support
a model whereby ASB2 contributes to hematopoietic differ
22 Our results support
a model whereby Ascl1 integrates inputs from both stimul
23 Therefore, our data are consistent with
a model whereby augmented and or prolonged presentation
24 These data support
a model whereby autoreactive plasma cells (at least cert
25 On the basis of these findings, we propose
a model whereby B cells contribute to arthritis in mice,
26 We propose
a model whereby B. anthracis LCPs promote attachment of
27 These data support
a model whereby Banf1 is crucial to reset oxidative-stre
28 Taken together, these data suggest
a model whereby bile salts or other detergents destabili
29 Our results are consistent with
a model whereby binding of HIV-1 Gag to phosphoinositide
30 We propose
a model whereby binding of synaptotagmin-1 to the SNARE
31 We propose
a model whereby BRCA1 impacts on 53BP1 to allow access o
32 These data suggest
a model, whereby BRCA1 is present on defined promoters a
33 c, and in vitro findings are consistent with
a model whereby brief exposure of Cdon mutant embryos to
34 in both proteins, which is in agreement with
a model whereby Bs3 evolved from an ancestral YUC gene.
35 Our combined results support
a model whereby BTLA on CD4(+) T cells and additional in
36 Our results support
a model whereby calcium-dependent TSLP release by kerati
37 These findings support
a model whereby calcium-induced modification of PICK1 st
38 Together, our data strongly support
a model whereby CASK recruits FRMD7 to the plasma membra
39 Therefore, our overall results reveal
a model whereby CD1d-mediated Ag presentation is negativ
40 These results support
a model whereby CDK-catalyzed phosphorylation of Sae2 ac
41 These results are consistent with
a model whereby CDKN2A loss affects a range of different
42 Furthermore, they are consistent with
a model whereby Cdo serves as a multifunctional corecept
43 We present
a model whereby CELF1 regulates APA site selection follo
44 We thus propose
a model whereby cell surface ubiquitination precedes end
45 These findings align with
a model whereby cells respond similarly to an equivalent
46 We propose
a model whereby centrosome-associated SYS-1 degradation
47 These data support
a model whereby centrosome-MT interactions during interp
48 We present
a model whereby centrosomes prevent inappropriate beta-c
49 a interaction with Borealin, which suggested
a model whereby CHMP4C inhibits abscission upon phosphor
50 ditioned fear expression, these data suggest
a model whereby chronic EtOH-driven neuroadaptations in
51 Our data support
a model whereby clinically or host-relevant signals medi
52 f specific cerebellar subregions, supporting
a model whereby compensatory morphological changes suppo
53 This trend is consistent with
a model whereby conduction occurs by two different mecha
54 en COQ9 and the hydroxylase COQ7, motivating
a model whereby COQ9 presents intermediates directly to
55 These findings support
a model whereby cotranscriptional recruitment of Rnf20 a
56 ic repeat (CRISPR) subtypes have resulted in
a model whereby CRISPRs function as a defense system aga
57 Our data support
a model whereby CTXphi and TCP bind in a tip-to-tip orie
58 These findings suggest
a model whereby cytokine deficiency leads to oncogenic i
59 Based on these data, we propose
a model whereby cytoplasmic aggregation of alpha-synucle
60 The results support
a model whereby Dab2 acts as a multifunctional adaptor i
61 Taken together, we present
a model whereby Dbp2 functions to cotranscriptionally mo
62 We propose
a model whereby de novo J insertion is mediated by JBP2.
63 We present
a model whereby defects in Suv3p result in accumulation
64 stem and neural progenitor cells, suggesting
a model whereby developmentally regulated transcription
65 Based on these results, we propose
a model whereby Dex- and Mstn-mediated atrophic signals
66 Our data suggest
a model whereby dFMRP is neuroprotective by remodeling T
67 We propose
a model whereby DGAT1 serves as a cellular hub for HCV c
68 We propose
a model whereby DIP-alpha and Dpr10 on opposing synaptic
69 We propose
a model whereby direct inputs of Nanog and Hoxa1 on shar
70 We propose
a model whereby distinct activity states of Rap1 modulat
71 l distinctions from Lgr5(+) ISCs and support
a model whereby distinct ISC populations facilitate home
72 These results give rise to
a model whereby distinct MT-bound and actin-nucleating p
73 We propose
a model whereby dMCRS2 promotes gene transcription by fa
74 We propose
a model whereby DNA shape features of stimulation-depend
75 These data suggest
a model whereby DOT1L-dependent lysine 79 of histone H3
76 for OCRL1 in neural development, and support
a model whereby dysregulation of phosphoinositide metabo
77 coupled with recent structural data, suggest
a model whereby early phosphorylations promote initiatio
78 We propose
a model whereby eEF1A binds to Rho1p-GDP on the vacuolar
79 Overall, our results support
a model whereby EFA6 recruits endophilin on flat areas o
80 Our results support
a model whereby emergent metabolites signal a beneficial
81 Collectively, these data support
a model whereby emerin facilitates repressive chromatin
82 Together, these data support
a model whereby engagement of leukocyte PECAM induces it
83 We propose
a model whereby enhancer binding by Sall4 and other plur
84 These findings support
a model whereby estradiol acts by activation of PI3K/Akt
85 These findings support
a model whereby estradiol acts via the IGF-1 receptor to
86 This work supports
a model whereby expression of a viral protein signals su
87 Our results support
a model whereby FAK-mediated FA remodeling may occur thr
88 tions, and buffering experiments all support
a model whereby fast Nav channel-mediated dSpikes (Na-dS
89 Our data suggest
a model whereby force acting on integrin-talin complexes
90 Our data point to
a model whereby FOX transcription factors control gene e
91 These results strongly support
a model whereby FOXM1 is specifically recruited to chrom
92 ished FoxO1 as a "pioneer" factor, suggested
a model whereby FoxO1 chromatin remodeling at regulatory
93 In light of our findings, we posit
a model whereby functional nucleolytic activity is not t
94 Therefore, these data support
a model whereby geminin promotes the neuronal precursor
95 Our findings support
a model whereby genetic determinants of smoking increase
96 We propose
a model whereby genetic violations to regulatory constra
97 Our results support
a model whereby Gpd1 may be imported as a monomer or a d
98 We propose
a model whereby Gqalpha signaling differentially regulat
99 We propose
a model whereby group I PAKs act downstream of Rac to or
100 These data suggest
a model whereby guidance signaling systematically shapes
101 Our findings are consistent with
a model whereby h3/acidic calponin controls fibroblast m
102 Taken together, our results support
a model whereby hAgo2:miR-122 complexes alter the struct
103 The new evidence proposes
a model whereby HMGA2 directly induces multiple transcri
104 These data suggest
a model whereby HTLV-1 infection augments the number of
105 These results support
a model whereby IFNG uses cis-regulatory elements with c
106 These results support
a model whereby IL-22RA1 enhances host-microbiota mutual
107 ain (CD127) and endocytosis, consistent with
a model whereby IL-7 is internalized via receptor intera
108 Together, our data support
a model whereby impaired mitochondrial [Fe-S] cluster bi
109 The data support
a model whereby inactivation of specific regulatory elem
110 We propose
a model whereby increased affinity for lipid particles,
111 We propose
a model whereby increased enzyme flexibility facilitates
112 Together, our data set forth
a model whereby increased secretion of Hsp90 in the cran
113 Together, our results support
a model whereby increased tooth number and an accelerate
114 These results support
a model whereby information about rewards signaled in PF
115 a074Me, suppresses this response, supporting
a model whereby ingested particles disrupt lysosomes and
116 n the antagonistic phase, we instead propose
a model whereby inhibitory coupling via postinhibitory r
117 Our study provides
a model whereby insights into the ancient history of vir
118 We propose
a model whereby insufficient levels of vitamin D, EPA, o
119 Our results support
a model whereby insulin promotes exocytic flux primarily
120 er with previous results, these data support
a model whereby insulin stimulates TUG cleavage to liber
121 We present a hypothesis consistent with
a model whereby intrahepatic CD8 TCM cells, being mainta
122 Together, these experiments support
a model whereby JIP1 coordinates APP transport by switch
123 transcription rate and Sen1 activity support
a model whereby kinetic competition between elongating P
124 This suggests
a model whereby KSHV alters ICN-RBPjkappa gene regulatio
125 PUFA in nuclei of living cells and suggested
a model, whereby L-FABP facilitated VLC-PUFA targeting t
126 These observations support
a model whereby lack of a cognate charged tRNA exposes a
127 We propose
a model whereby LcrH-dependent association of YopD with
128 of PNs as a teaching signal, consistent with
a model whereby learning leads first to reductions in PN
129 Our results support
a model whereby LIG1 fidelity is governed by a high-fide
130 and targeted gene disruption studies support
a model whereby limited cleavage of alpha-mannan on the
131 These findings suggest
a model whereby linked metabolic-epigenetic programs are
132 These data support
a model whereby local geometry constrains the orientatio
133 Our findings are consistent with
a model whereby low, intermediate, and high CD122 signal
134 Our data suggest
a model whereby Lrp4 modulates Wnt/beta-catenin signalin
135 These data support
a model whereby LTA backbone synthesis proceeds in S. au
136 We therefore propose
a model whereby MA trimerization is required to form a l
137 Overall, our results suggest
a model whereby maintaining adequate levels of hepatic o
138 Therefore, the present data support
a model whereby MCL-1 depletion increases 53BP1 and RIF1
139 In this study, we present evidence for
a model whereby melanophores and iridophores descend fro
140 We provide
a model whereby messenger ribonucleoprotein (mRNP) assem
141 We propose
a model whereby minus-end microtubule stabilization medi
142 These results suggest
a model whereby MITD1 coordinates the activity of ESCRT-
143 t for gsk-3 and cdk-2 This analysis supports
a model whereby MPK-1/ERK, GSK-3/GSK3 and CDK-2/CDK2, al
144 We propose
a model whereby Mps1 transphosphorylation results in its
145 We propose
a model whereby mTOR kinase domain phosphorylation modul
146 Our results support
a model whereby MTX inhibits reduction of dihydrobiopter
147 From these results, we propose
a model whereby multi-site cdk-dependent phosphorylation
148 Our data support
a model whereby multiple features of HLA-DR are involved
149 We propose
a model whereby multiple infections per cell lead to red
150 These findings are consistent with
a model whereby multiple NLR inflammasomes attenuate dis
151 Our data support
a model whereby multiple Twist1 activity thresholds cont
152 ther with previous studies, our data support
a model whereby Munc18-1 acts as a template for SNARE co
153 We propose
a model whereby Munc18-1 and Munc13-1 are critical not o
154 Our data lead to
a model whereby myosin VIII links phragmoplast microtubu
155 Our data support
a model whereby nascent NepR(IDR)-PhyR interactions and
156 ms of F. tularensis phagocytosis and support
a model whereby natural IgM binds to surface capsular an
157 nance of the spermatogonial pool and propose
a model whereby negative feedback from mTORC1 to the GDN
158 Together, the results of this study support
a model whereby neural organization at key stages of dev
159 These results suggest
a model whereby Neuralized1-dependent ubiquitination fac
160 it?" decisions; out of this work has emerged
a model whereby neurons accumulate information about the
161 Our data support
a model whereby NLS-RecA-Gal4 DNA filaments bind to comp
162 We propose
a model whereby nuclear pore complexes either compete wi
163 Our results are consistent with
a model whereby OA neuronal signaling increases after co
164 Collectively, these data support
a model whereby OA/Gpnmb acts as a negative regulator of
165 We, therefore, propose
a model whereby ORF57 interacts directly with PYM to enh
166 We propose
a model whereby OSA treatment may affect both Abeta rele
167 removed from the Rac1 interface, supporting
a model whereby P-Rex1 binding to PIP3 and/or Gbetagamma
168 Collectively, these data favor
a model whereby P. graminicola coopts the plant BR pathw
169 These observations give rise to
a model whereby papillomavirus oncoproteins, including B
170 Our observations lead to
a model whereby paxillin contributes to talin and vincul
171 These data support
a model whereby PDE10A trafficking and localization can
172 We propose
a model whereby persistence of long-term memory results
173 tly, genetic and molecular evidence supports
a model whereby PHF8 regulates zebrafish neuronal cell s
174 phatidylinositol, inhibits TRPV1, supporting
a model whereby phosphoinositide turnover contributes to
175 Our data support
a model whereby phosphorylation increases structural fle
176 Our data suggest
a model whereby PIP5Pase controls PI(3,4,5)P3 binding by
177 Our data support
a model whereby postnatal loss of hair forming ability i
178 These results suggest
a model whereby posttermination ribosomes/ribosomal subu
179 These results indicate
a model whereby Pou3f4 in the otic mesenchyme establishe
180 These data are consistent with
a model whereby PP M cells are the primary route by whic
181 We propose
a model whereby PRC1 acts in concert with specific lncRN
182 Our data suggest
a model whereby,
prior to hair initiation, proximally lo
183 We describe
a model whereby promoter demethylation requires the co-i
184 We propose
a model whereby PTIP stabilizes the Pax5 DNA interaction
185 Our data support
a model whereby purine motifs towards the 3' end of the
186 These results are consistent with
a model whereby PV and HRV disrupt nucleo-cytoplasmic tr
187 Our data support
a model whereby QscR polypeptides fold properly in the a
188 n of Spag with both Hsp70 and Hsp90 suggests
a model whereby R2TP would accompany clients from Hsp70
189 Our data support
a model whereby Rab13 can mediate its effects on cell pr
190 We propose
a model whereby RAPGEF5 activates the nuclear GTPases, R
191 The studies also support
a model whereby receptor expression is limited by cell d
192 These findings support
a model whereby repeat expansions elicit cellular stress
193 These results suggest
a model whereby retinoic acid signaling regulates Lhx8 a
194 These results support
a model whereby RPA, best known for its role in DNA repl
195 The results are consistent with
a model whereby RTNLB1 and RTNLB2 regulate the transport
196 We propose
a model whereby Sar1 dimers assemble into ordered arrays
197 They also support
a model whereby selection for the ability to mount a bro
198 We present
a model whereby '
selfish' positive selection acting on a
199 Mechanistically, we propose
a model whereby SETX attenuates the activity of RNA poly
200 We therefore propose
a model whereby ShcD competes with neurotrophic receptor
201 o the 5' or 3' ends of P4R2 RNAs, suggesting
a model whereby siRNAs are generated from either end of
202 Together, these findings favor
a model whereby SLR1 acts as a positive regulator of hem
203 These data suggest
a model whereby SM, a viral protein, recruits and co-opt
204 Our findings suggest
a model whereby small perturbations in a self-reinforcin
205 This study supports
a model whereby SMN deficiency impedes transport and loc
206 The results support
a model whereby SNX3-retromer is a minimally concentrati
207 ine diseases generally, the findings support
a model whereby specific neuronal circuits suffer insult
208 Altogether, the data support
a model whereby stereocilia actin cores are largely stat
209 These data support
a model whereby STIM1 is critical to deactivate a key ne
210 Our results are consistent with
a model whereby Stn2 is required to preserve SV protein
211 te immune signal transduction and then offer
a model whereby structurally distinct proteins can be gr
212 Our results support
a model whereby subcellular anchoring of CaN by AKAP150
213 The results suggest
a model whereby successful biolistic transformation reli
214 These findings suggest
a model whereby SUMO regulates the distribution, oligome
215 These findings suggest
a model whereby sumoylation of Rad1 promotes its disenga
216 We previously described
a model whereby suppression drives a Drosophila grooming
217 Our results support
a model whereby swelling and proinflammatory signals ass
218 We propose
a model whereby switching between monomeric and dimeric
219 These findings support
a model whereby synapsin accumulates at sites of synapti
220 They further suggest
a model whereby synthesis and turnover of PI(3,4)P(2) ar
221 In all, these data support
a model whereby T-BET serves to promote appropriate chro
222 Based on these findings, we propose
a model whereby T4P unipolarity is accomplished by stimu
223 Our data support
a model whereby tau, facilitated by Abeta, transits from
224 We propose
a model whereby TBC1D14 and TRAPPIII regulate a constitu
225 We propose
a model whereby territory size is determined by the numb
226 We propose
a model whereby tethering of MIWI2 to a nascent transpos
227 The data are consistent with
a model whereby the 3'-end of the tRNA remains free to s
228 Together, these experiments support
a model whereby the A(2A)AR can prime JAK-phosphorylated
229 We propose
a model whereby the amide side chain is first required t
230 We present
a model whereby the amorphous nature of the aggregates r
231 Taken together, our results support
a model whereby the amphipathic helix in SM N100 attache
232 Our results support
a model whereby the C-terminal end of the Hendra virus F
233 Our findings support
a model whereby the cell cycle machinery not only contro
234 These observations point to
a model whereby the coiled-coil domain plays a key role
235 Based on these studies, we propose
a model whereby the concerted recruitment of CHMP4B and
236 We propose
a model whereby the CTD of VgrG-5-, propelled by T6SS-5-
237 Our data support
a model whereby the degree of apoptosis induction is reg
238 These data established
a model whereby the development and progression of infla
239 Our data support
a model whereby the disulfide bond and PAS domain of Srr
240 the binocular matching process, and suggest
a model whereby the dominant input instructs the develop
241 We therefore propose
a model whereby the downregulation of the NPM mRNA, medi
242 These results support
a model whereby the flippase activity of ALA4 and ALA5 i
243 These findings suggest
a model whereby the genomic vRNA serves as a switch to r
244 Our results support
a model whereby the high-fidelity replicative DNA polyme
245 These data suggest
a model whereby the Hsp70-Hsp110 chaperone complex antag
246 We propose
a model whereby the instantaneous rates of surface and v
247 We propose
a model whereby the intrinsic stochasticity of gene expr
248 Based on these results, we propose
a model whereby the light- and day length-dependent inte
249 Our data suggest
a model whereby the LISD promotes meiotic spindle assemb
250 We propose
a model whereby the LysM domain ensures septal localizat
251 xovirus F-nAb structures, these data support
a model whereby the membrane-distal region of the F prot
252 in filaments in anaphase, these data support
a model whereby the Myo19 actin-based motor helps to con
253 en together, our results are consistent with
a model whereby the NaStEP and NaSIPP interaction, in in
254 Our findings support
a model whereby the NLRP3 inflammasome, acting as an ext
255 These findings suggest
a model whereby the normal role of SAPAP3 is to inhibit
256 arify the roles of pax8 and sox3 and support
a model whereby the otic placode forms first and induces
257 Our data support
a model whereby the PilY1/minor pilin complex functions
258 A model whereby the protoxin undergoes a conformational
259 These data suggest
a model whereby the RAG signal end complex is stabilized
260 Our combined data support
a model whereby the risk variant augments the BCR and co
261 These findings support
a model whereby the Rs1 protein binds to PS in the retin
262 We propose
a model whereby the S. aureus Deltalcp mutant, defective
263 These findings support
a model whereby the selective removal of Nup FG repeat d
264 We postulate
a model whereby the SGBP-A, SGBP-B, and BoGH9 work toget
265 These data support
a model whereby the SLC16A12 (c.643C>T) mutation causes
266 not detectable in infected cells, we propose
a model whereby the species recruited to assembling VLPs
267 Our data support
a model whereby the trafficking machinery plays an impor
268 This suggests
a model whereby the transcriptional activity of AIB1 is
269 We propose
a model whereby the uncoupling of protein levels of biog
270 These data support
a model whereby these two functionally distinct alloster
271 Based on these observations, we posit
a model whereby these two motors generate forces that at
272 We present
a model whereby this inhibition permits the muscle-deriv
273 In all, this work suggests
a model whereby this self-association stimulates the aut
274 Our observations also support
a model whereby tissue organisation and cell division ar
275 ion in response to LPS treatment, suggesting
a model whereby TLR signaling causes the phosphorylation
276 Our results support
a model whereby TOP1 and TOP2 act in separate pathways t
277 Our genetic and biochemical studies support
a model whereby Top1p recruits Sir2p to the rDNA and cla
278 We propose
a model whereby transcription factors activate lincRNAs
279 However, strong indirect evidence favors
a model whereby transendocytosis of the Notch extracellu
280 These data support
a model whereby TRIM14 acts as a scaffold between TBK1 a
281 These data are consistent with
a model whereby Trp-76 anchors the C terminus of the cyt
282 Overall, our results support
a model whereby TRPV4 differentially regulates cell volu
283 Our data support
a model whereby ubiquitinated Rsp5 adaptors are more act
284 We propose
a model whereby uncleaved alpha2delta subunits maintain
285 These data lead us to propose
a model whereby unique and additive activities of FGFR1
286 Our data support
a model whereby VAI functions as a PKR inhibitor because
287 Importantly, our data support
a model whereby VEGF regulates differentiation through a
288 We propose
a model whereby vhs-mediated destruction of SG mRNA prom
289 Based upon this data, we propose
a model whereby vimentin promotes FAK stabilization thro
290 Overall our data promote
a model whereby vinculin controls the transmission of in
291 Combined, our data support
a model whereby WNV infection of human DCs compromises W
292 Combining these observations, we propose
a model whereby WNV subverts human DC activation to comp
293 The results corroborate
a model whereby X. oryzae pv. oryzae enhances the releas
294 These data are consistent with
a model whereby XRCC4/XLF complexes hold DNA ends togeth
295 he acetylation state of histones and provide
a model whereby yeast spliceosome assembly is tightly co
296 These results suggest
a model whereby Yku70 sumoylation upon DNA association s
297 We propose
a model whereby Yra1 terminates a cycle of mRNP assembly
298 ivity of PP4 during meiotic prophase suggest
a model whereby Zip1-S75 phosphorylation dynamically des
299 We propose
a model whereby zipcoded mRNP and/or tER ligands couple
300 tight junction (TJ) protein ZO-1, leading to
a model whereby ZO-1 acts by sequestering DbpA at the TJ