1 A screen for 1-TbAd mutants, complementation studies, an
2 We carried out
a screen for a potential link between triglyceride mobil
3 We performed
a screen for aberrant nodulation phenotypes using the Lo
4 In
a screen for abnormal dendrite development, we identifie
5 A screen for abnormal root architecture responses to hig
6 ene, ema (endosomal maturation defective) in
a screen for abnormal synaptic overgrowth and defective
7 We report here the isolation of SPPL3 in
a screen for activators of NFAT, a transcription factor
8 A screen for additional Dnf1 mutants capable of replacin
9 In
a screen for additional L. pneumophila proteins that ass
10 A screen for adjuvant activity of microbial products rev
11 A screen for adult ADHD was included in a probability su
12 A screen for agents to re-activate suppressed occludin g
13 A screen for alternative proinflammatory factors of F. t
14 misincorporation at serine codons, following
a screen for amino acid activation in ATP/PP(i) exchange
15 In
a screen for antibiotic resistance determinants in Mycob
16 In
a screen for antiprion systems curing [PSI+] without pro
17 A screen for antiproliferative factors repressed after l
18 These features permit
a screen for antivirals for emerging viruses and select
19 The pbs3-1 mutant, identified in
a screen for Arabidopsis (Arabidopsis thaliana) mutants
20 A screen for Arabidopsis (Arabidopsis thaliana) mutants
21 We applied mutagenomics to
a screen for Arabidopsis (Arabidopsis thaliana) mutants
22 ive1 (cad1/pcs1) mutants, was recovered from
a screen for Arabidopsis mutants with reduced resistance
23 We identified SUO in
a screen for Arabidopsis mutations that increase the acc
24 We identified MYB98 in
a screen for Arabidopsis thaliana genes expressed in the
25 ion of new cryptochrome1 (cry1) alleles from
a screen for Arabidopsis thaliana genomes uncoupled muta
26 In
a screen for Arabidopsis thaliana mutants affected in th
27 ning for phenotypic mutants was validated in
a screen for attachment to abiotic surfaces.
28 utation of TMEM184b, a protein identified in
a screen for axon degeneration mediators.
29 A screen for axonal cargo mislocalization in Caenorhabdi
30 A screen for axonal localization identifies a specific s
31 rhabditis elegans bre-1 gene was isolated in
a screen for Bacillus thuringiensis toxin-resistant (bre
32 al infections underwent standard culture and
a screen for bacteria by amplification and sequencing of
33 e mitochondrial ATP synthase beta subunit in
a screen for Bcl-x(L)-binding partners, we tested and fo
34 A screen for Bcr-Abl mutants emerging in the presence of
35 leanup, we used mass spectrometry to perform
a screening for both targeted and untargeted biomarkers,
36 A screen for C. elegans mutants with a biofilm absent on
37 In
a screen for Caenorhabditis elegans mutant animals that
38 In
a screen for Caenorhabditis elegans mutants that display
39 or Neurogenic Differentiation 2 (NeuroD2) in
a screen for calcium-regulated transcription factors and
40 In
a screen for candidate genes stimulating cell death in G
41 A screen for candidate mediators of the PPARalpha-driven
42 We conducted
a screen for candidate mutations affecting DSB repair an
43 A screen for candidates revealed suppressor of cytokine
44 ved in postsynaptic exocytosis, we conducted
a screen for candidates that disrupted trafficking of a
45 Tropisetron was identified in
a screen for candidates that increase the ratio of the t
46 We performed
a screen for cell types that have high levels of signali
47 In
a screen for cell-cycle regulators, we identified a Dros
48 In this study,
a screen for CER1 physical interaction partners was perf
49 By conducting
a screen for checkpoint genes in zebrafish, we found tha
50 We conducted
a screen for circadian-relevant neurons in the Drosophil
51 e, applying CLIK analysis retrospectively to
a screen for cisplatin sensitivity allowed us to identif
52 ule inhibitors of Cdks that we identified in
a screen for compounds that activate hPXR) leads to acti
53 A screen for compounds that downregulate Mcl-1 identifie
54 In
a screen for compounds that restore radiosensitivity in
55 In
a screen for conserved substrates of AMPK, we identified
56 A screen for corresponding substrates of the neprilysin
57 In
a screen for dauer regulatory genes that control the act
58 We identified elm1 mutants in
a screen for defects in Swe1p degradation and show that
59 Therefore, in a previous study we initiated
a screen for differential gene expression in the telence
60 A screen for differentially expressed genes in granuloma
61 A screen for differentially expressed miRNAs between hea
62 echanisms enable this precision, we designed
a screen for disrupted sepal size and shape uniformity i
63 In
a screen for dominant enhancers of mutations in the home
64 solate additional proteasome-related mutants
a screen for dominant suppressors of Pros26(1) was carri
65 To study needle assembly, we performed
a screen for dominant-negative yscF alleles that prevent
66 From
a screen for Drosophila mutants with aberrant synaptic d
67 is technique may potentially be exploited as
a screen for drug development and analysis.
68 measure gap junction mediated diffusion and
a screen for drugs that affect gap junction communicatio
69 We performed
a screen for drugs that upregulate the somatostatin rece
70 sovan and Neanderthal hominids, we conducted
a screen for endogenized retroviruses, identifying six n
71 In
a screen for endogenous tumor-associated T cell response
72 Here the authors develop
a screen for engineering new split FPs, and report a yel
73 In
a screen for enhancers of tir1-1 auxin resistance, we id
74 is a perinatal lethal mutation uncovered in
a screen for ENU-induced mutations on mouse chromosome 5
75 for a biological outcome, here we report on
a screen for epigenetic chemical probes able to distingu
76 We conducted
a screen for ethyl methanesulfonate-induced suppressors
77 During
a screen for ethylnitrosourea-induced mutations in mice
78 A screen for F/P-dependent cytokines was performed in gr
79 Through
a screen for factors involved in kinetochore integrity i
80 entified an HMX homeodomain protein MLS-2 in
a screen for factors required for M lineage patterning.
81 Herein, we identified leupaxin in
a screen for focal adhesion kinase binding partners in a
82 Using
a screen for freezing sensitivity, we have identified a
83 quired for biological activity, we conducted
a screen for functionally important MYC targets and iden
84 dation for the utility of our interactome as
a screen for functionally important novel V-ATPase-regul
85 From
a screen for functionally relevant ERG interactors, we i
86 A screen for fusion mutants in Chlamydomonas identified
87 Through
a screen for G2/M checkpoint regulators in zebrafish, we
88 In
a screen for gene copy number alterations in mouse mamma
89 In
a screen for gene copy-number changes in mouse mammary t
90 In
a screen for genes activated by Zic1, we identify severa
91 From
a screen for genes defective in caspase activation in th
92 In
a screen for genes directly activated by TGF-beta, we fo
93 nd potential therapeutic targets, we perform
a screen for genes essential for Ebola virus (EBOV) infe
94 During
a screen for genes involved in presynaptic homeostatic s
95 In
a screen for genes involved in Slit-Robo repulsion, we h
96 In
a screen for genes involved with prostate cancer metasta
97 vered unexpectedly in healthy CNS neurons in
a screen for genes regulated by neural activity.
98 In
a screen for genes required for ISC proliferation in res
99 several screens, including MAD2 (MAD2L1) in
a screen for genes required for the spindle assembly che
100 us, biclustering is a natural methodology as
a screen for genes that are functionally related, partic
101 We identified diaphanous in
a screen for genes that are necessary for the normal gro
102 A screen for genes that can ectopically activate a Rad3-
103 A screen for genes that control Drosophila heart develop
104 specific genes for regulation, we performed
a screen for genes that modify FOXO activation of TRAIL,
105 In
a screen for genes that modulate C. elegans insulin-like
106 In
a screen for genes that promote GCR, we identified MPH1,
107 In
a screen for genes that regulate cell lineage determinat
108 In
a screen for genes upregulated by taurine in developing
109 In
a screen for genes whose expression was altered in respo
110 new components of this pathway, we performed
a screen for genes whose loss-of-function debilitated TP
111 A (LCAA) antisense plants were obtained from
a screen for genes whose partial down-regulation results
112 me 1 (sxe1, cyp4d21), has been identified in
a screen for genes with sex-biased expression in the hea
113 d-dependent disease phenotypes, we performed
a screen for genetic modifiers of autoreactivity between
114 We performed
a screen for genetic suppressors of cobra, an Arabidopsi
115 A screen for genetic suppressors of the enhanced seed do
116 1 (ged1), which was previously discovered in
a screen for genomes uncoupled-like mutants and shows th
117 We conducted
a screen for glossy-eye flies that fail to incorporate B
118 A screen for growth on the beta-lactam antibiotic cephal
119 To investigate the basis for these defects,
a screen for growth suppressors was performed.
120 A screen for HAI1-interacting proteins identified HAI1-I
121 ew derivatives, which were then submitted to
a screening for HDAC inhibition as well as to a prelimin
122 A screen for HDAC6 inhibitors identified acyl derivative
123 A screen for hepatitis C virus (HCV) NS3 helicase inhibi
124 In
a screen for hypermethylated CpG islands in cancer, bidi
125 A screen for imprinted genes on mouse Chromosome 7 recen
126 hod is of high throughput, and it can act as
a screen for in vitro cancer stem cell response to drugs
127 A screen for increased longevity in Caenorhabditis elega
128 A screen for inhibitors of the protein binding domains o
129 To test this idea, we performed
a screen for inhibitors selective for Plasmodium Kinesin
130 In
a screen for inhibitors that block the association of TR
131 A screen for interdomain interactions by solution-state
132 We conducted
a screen for ISGs that inhibit HIV-1 virion production a
133 C. uda was identified in
a screening for its ethanologenic potential from AFEX-CS
134 In
a screen for kinases that promote survival of KRAS-depen
135 proteins, Ceg9, was previously identified in
a screen for L. pneumophila IDTS that manipulate secreto
136 A screen for LATS2-interacting proteins in liver-derived
137 ld protein IQGAP1 was recently identified in
a screen for LBRC-interacting proteins.
138 During
a screen for liver defects from a zebrafish insertional
139 Using mariner mutagenesis and
a screen for loss of cytotoxicity, a genetic locus encod
140 re the utility of this method by undertaking
a screen for magnetic cells in the pigeon.
141 In
a screen for mediators of PhoPQ-regulated OM remodeling
142 Using
a screen for members of the Wnt pathway, we demonstrate
143 We conducted
a screen for microbiome metabolites that would function
144 In
a screen for microRNAs (miRNAs) that modulate the DNA da
145 e amygdala from these mice was collected and
a screen for microRNAs that were recruited to the RNA-in
146 We conducted
a screen for miRNA function in primary T cells and ident
147 In
a screen for miRNAs regulated by myocardin-related trans
148 Here we report
a screen for miRNAs that affect the Wingless (Wg) pathwa
149 Using
a screen for mitophagy-deficient mutants, we found that
150 A screen for MMS-sensitive mutants identified a novel tr
151 Through
a screen for modified transport to the vacuole (mtv) mut
152 In this work, we performed
a screen for modifiers of dendritic growth in hippocampa
153 A screen for modifiers of Dpp adult phenotypes led to th
154 From
a screen for modifiers of Notch signaling in Drosophila
155 The turnip (tnp) mutant was identified in
a screen for modifiers of POLARIS expression, a gene req
156 In
a screen for modifiers of the oocyte DV patterning defec
157 We identified sqd in
a screen for modifiers of the Protein Kinase A (PKA) oog
158 A screen for modifiers of the six cycB phenotypes identi
159 r mutants with spindle formation defects and
a screen for molecules that antagonized EB1 function.
160 In
a screen for molecules that participate with XIAP in reg
161 cmpy was identified in
a screen for motoneuron-expressed genes and encodes a si
162 A screen for mRNAs whose polyadenylation is altered by G
163 ne containing mltG was initially isolated in
a screen for multicopy plasmids generating a lethal phen
164 We performed
a screen for multicopy suppressors of arp2-7 and identif
165 In
a screen for mutagenesis-defective mutants, we isolated
166 ation to the length of the night, we devised
a screen for mutant Arabidopsis thaliana plants in which
167 In
a screen for mutants affected in photosynthesis, we iden
168 In
a screen for mutants affecting gonad formation we identi
169 Snx4/Atg24 proteins have been identified in
a screen for mutants defective in a type of selective ma
170 The rev6-1 allele was isolated in
a screen for mutants deficient for UV-induced reversion
171 We have identified the new PAK gene max-2 in
a screen for mutants disrupted in UNC-6/netrin-mediated
172 Dam methylase mutants were recovered in
a screen for mutants sensitive to UV irradiation or mild
173 In
a screen for mutants showing altered photomorphogenesis
174 A screen for mutants that affect this process identified
175 In
a screen for mutants that are defective in transitioning
176 omes 1 (brt1) mutant was first identified in
a screen for mutants that exhibit a reduced epidermal fl
177 A screen for mutants that mimic the hira-1 mutant phenot
178 In
a screen for mutants that no longer require vernalizatio
179 A screen for mutants that poorly expressed a comEA-lucif
180 In
a screen for mutants that suppress the expression of the
181 In
a screen for mutants with abnormal mitochondrial morphol
182 iated protein (CLASP) Peg1 was identified in
a screen for mutants with spindle formation defects and
183 Here we used
a screen for mutations affecting endodermal organ morpho
184 Five new alleles of dig-1 were isolated in
a screen for mutations affecting the morphology or funct
185 In
a screen for mutations modifying clv1 mutants, we have i
186 els of sHsp chaperone activity, we performed
a screen for mutations of Synechocystis Hsp16.6 that red
187 In
a screen for mutations protecting C. elegans from hypoxi
188 in short days6 (esd6) mutant was isolated in
a screen for mutations that accelerate flowering time.
189 A screen for mutations that affect habenular laterality
190 In
a screen for mutations that affect the biosynthesis of r
191 A screen for mutations that affect the recruitment of th
192 Unexpectedly,
a screen for mutations that affected the activity of the
193 essential for organ abscission, we conducted
a screen for mutations that alter floral organ shedding
194 separate set of substitutions, identified in
a screen for mutations that alter the emission of a fluo
195 In
a screen for mutations that bypassed the critical requir
196 ed a dominant mutation in wapl (wapl(AG)) in
a screen for mutations that counteract silencing mediate
197 serine/threonine kinase PAR-1 (MARK/Kin1) in
a screen for mutations that disrupt border cell migratio
198 In
a screen for mutations that disrupted brain laterality,
199 A screen for mutations that enhance ROP1-overexpression-
200 rabidopsis thaliana mutants were isolated by
a screen for mutations that impair cold-induced transcri
201 A screen for mutations that interact with CDC34(tm) unco
202 In
a screen for mutations that modify wg loss-of-function p
203 Through
a screen for mutations that restore organ separation in
204 Further,
a screen for mutations that suppress the effects of the
205 A screen for mutations that suppress this precocious phe
206 seele was identified in
a screen for mutations that, when homozygous in ovarian
207 A screen for N-terminal peptide sequences that can bind
208 A screen for NAB-regulated genes identified several (inc
209 Following
a screen for neuromuscular mouse mutants, we identified
210 In
a screen for neuronal Ca(2+) sensors that respond to cha
211 In
a screen for neurons that enable Drosophila melanogaster
212 From
a screen for new memory mutants, we identified alleles o
213 ed arginine and glutamate rich 1 (ARGLU1) in
a screen for new modulators of glucocorticoid signaling
214 uable tool that holds broad applicability in
a screen for non-ATP site binders.
215 Using a transposon library and
a screen for nonencapsulated mutants, we identified the
216 62 and MDV3100, two compounds optimized from
a screen for nonsteroidal antiandrogens that retain acti
217 tional co-activator mastermind, we conducted
a screen for Notch signal modifiers using the Exelixis c
218 In
a screen for novel candidate genes involved in human T(r
219 A screen for novel colonization factors by use of TnphoA
220 A screen for novel inhibitors of eCB hydrolysis identifi
221 he mouse, mhyp (mosaic hypopigmentation), in
a screen for novel proviral integration sites in a multi
222 From
a screen for novel targets of bun repression we have ide
223 ators of migration could be obtained through
a screen for overly adhesive mutants.
224 We performed
a screen for P2Y1 receptor-mediated receptor tyrosine ki
225 on GOLD36, an EMS mutant identified through
a screen for partial displacement of the Golgi marker, S
226 mutants, frd4-1 and frd4-2, were isolated in
a screen for plants that do not induce Fe(III) chelate r
227 fluorescence5 (ref5-1) mutant, identified in
a screen for plants with defects in soluble phenylpropan
228 From
a screen for plastid-to-nucleus signaling mutants in Ara
229 In
a screen for potential downstream effectors of spheroid
230 Using
a screen for potential interactions between telomere rep
231 A screen for potential regulators of the CLDN7 gene duri
232 In
a screening for potential monoamine oxidase A and B inhi
233 2+) and integrin-binding protein-1 (CIB1) in
a screen for previously unknown regulators of cardiomyoc
234 In
a screen for proteins co-precipitating with CHP1 in quie
235 In
a screen for proteins relevant to this IFN-stimulatory D
236 We identified RBP2 in
a screen for proteins that bind to such pRB variants.
237 In
a screen for proteins that interact with the FLAGELIN-SE
238 In
a screen for proteins that interact with the Rsp5 C2 dom
239 A screen for proteins that interacted with the AAD of fu
240 A screen for proteins that preferentially bound wild-typ
241 Here, we performed
a screen for putative IKKepsilon substrates using an unb
242 A screen for putative IL-10 elicitors revealed that IL-2
243 In
a screen for PyMT variants that could not activate the A
244 Rad51-G103E was identified in
a screen for Rad51 interaction-deficient mutants and was
245 articipants in these responses, we undertook
a screen for regulators that, when present on a high-cop
246 Here, we report the results of
a screen for repair genes induced in cancer cells treate
247 solated catalase-deficient mutants (cat2) in
a screen for resistance to hydroxyurea-induced cell deat
248 A screen for resistance to the nucleotide analog 6-thiog
249 strain CFT073 (CFT073 fim L-ON), followed by
a screen for restoration of motility in soft agar.
250 Finally,
a screen for retinoid-induced genes identifies metabolic
251 To circumvent this problem, we performed
a screen for revertants and dominant suppressors of the
252 for mitochondrial integrity and function, in
a screen for RNAs that exit the nucleus through NE buddi
253 ture-sensitive allele (rsw12) of the gene in
a screen for root radial swelling mutants.
254 A screen for Saccharomyces cerevisiae temperature-sensit
255 Here, via
a screen for scm-like mutants we describe a new allele o
256 embryonic chicken pancreatic cDNA library in
a screen for secreted factors.
257 We developed
a screen for small molecules that block Wnt secretion.
258 In
a screen for small molecules that protect cells from end
259 The compound Retro-1 was discovered in
a screen for small molecules that reduce the actions of
260 Here we design
a screen for small-molecule inhibitors of beta-catenin's
261 A screen for substrates of AMPK identified mitochondrial
262 In
a screen for substrates of these kinases involved in Sna
263 ypothesis, we have developed and carried out
a screen for such candidate proteins using an in vitro e
264 netically regulated by histone deacetylases;
a screen for suitable histone deacetylase inhibitors ide
265 A screen for supernodulating Medicago truncatula mutants
266 A screen for suppressors of a fission yeast chk1 mutant
267 Through
a screen for suppressors of a secA2 mutant, we identifie
268 r to identify gp2.5 interactions we designed
a screen for suppressors of gp2.5 lacking the C-terminal
269 on intragenic mec-4 mutations identified in
a screen for suppressors of mec-4(d)-induced necrosis, w
270 d regulation of wax production, we performed
a screen for suppressors of the cer7 mutant.
271 The sel-6 gene was previously identified in
a screen for suppressors of the egg-laying defect associ
272 he recovery of alleles of six new genes from
a screen for suppressors of the egg-laying defect associ
273 A screen for suppressors of the highwire synaptic overgr
274 In
a screen for suppressors of the SIN mutant cytokinesis c
275 A screen for suppressors of this low-temperature phenoty
276 A screen for suppressors of transgenic Killer of prune l
277 transmembrane helix 2 of Pdr5 identified in
a screen for suppressors that eliminated Pdr5-mediated c
278 -binding domains that is also highlighted in
a screen for synapsis mutants.
279 In
a screen for synaptic mutants in Drosophila, we identifi
280 Using
a screen for synthetic lethality, we found that the V. c
281 In
a screen for T cell-inhibitory molecules in cHL, we foun
282 em coupled with RNA sequencing analysis, and
a screen for tandem TTGAC cis-elements in the upstream s
283 In
a screen for temperature-sensitive conditional seizure m
284 tivation sensitizes IRS1 to degradation, and
a screen for the responsible E3 ligase identified Fbxo40
285 A screen for the systematic identification of cis-regula
286 A screening for the BsmI SNP may emphasize the importanc
287 We identified AGL61 in
a screen for transcription factor genes expressed in the
288 Here, we describe
a screen for transcription factors (TFs) that regulate t
289 In
a screen for transcription factors regulating the expres
290 brm was identified in
a screen for transcriptional activators of homeotic gene
291 A screen for transcripts robustly induced in cultured ne
292 In
a screen for trichome branching defects, we isolated a m
293 In
a screen for tumour suppressors that cooperate with onco
294 A screen for type III effector proteins from Pst for the
295 In
a screen for ubi4Delta suppressors, a hypomorphic allele
296 In
a screen for unexplained mutation events we identified a
297 We performed
a screen for unidentified actors in the cellulose-respon
298 and its linked gene, vciA, were isolated in
a screen for V. cholerae genes that permitted growth of
299 To that end,
a screen for visual system mutants was performed on 250
300 Fourth,
a screen for yeast mutants that enhance or suppress grow