ライフサイエンスコーパス: abdominal abscess

1 diagnostic imaging of suspected acute intra-abdominal abscess.

2 omplications, including anastomotic leak and abdominal abscess.

3 ammation did LA significantly increase intra-abdominal abscesses.

4 h their ability to induce experimental intra-abdominal abscesses.

5 virulence factors in the formation of intra-abdominal abscesses.

6 e both able to induce the formation of intra-abdominal abscesses.

7 anaerobe most frequently isolated from intra-abdominal abscesses.

8 uded three anastomotic leaks and three intra-abdominal abscesses.

9 s an established method for the treatment of abdominal abscesses.

10 sis, intra-abdominal fluid collection, intra-abdominal abscess (10% vs 25%, P = 0.027), severe (>/=gr

11 was 10.3%, wound dehiscence 4.4%, and intra-abdominal abscess 5.8%, and did not significantly differ

12 The third complication was an intra-abdominal abscess after a TV appendectomy.

13 he enteric anastomosis site, including intra-abdominal abscess and enterocutaneous fistula formation,

14 Intra-abdominal abscess and higher blood loss decreased the ch

15 l symptoms and signs of peritonitis or intra-abdominal abscess and isolation of Candida species eithe

16 postoperative complications including intra-abdominal abscess and pancreatic fistula (all P < 0.02).

17 CE 6: In patients with IBD who have an intra-abdominal abscess and/or phlegmonous inflammation that l

18 of penetrating complications (such as intra-abdominal abscesses and fistulae), although more than ha

19 ponse necessary for the development of intra-abdominal abscesses and underscore the role of IL-17 in

20 hole, diffuse fibrinopurulent exudate, intra-abdominal abscess, and extraluminal fecalith were indepe

21 Pneumonia, intra-abdominal abscess, and sepsis were the most common outco

22 M101 contributes to persistence within intra-abdominal abscesses, at least in part through activation

23 .93; 95% CI, 0.38-2.32; P = .88) or on intra-abdominal abscess development (OR, 0.89; 95% CI, 0.34-2.

24 postoperative hemorrhage (P = 0.174), intra-abdominal abscess formation (P = 0.199), biliary leakage

25 Intra-abdominal abscess formation was more common following la

26 ative pyrexia, ileus, wound infection, intra-abdominal abscess formation, operative time, and postope

27 secondary outcome was the incidence of intra-abdominal abscess formation.

28 irrigation (NI) reduces postoperative intra-abdominal abscess (IAA) in children with perforated appe

29 al [UI] 34.8-36.6) of 10 175 patients, intra-abdominal abscess in 1619 (18.3%, 17.5-19.1) of 8830 pat

30 ment included CT-guided drainage of an intra-abdominal abscess in 23 patients, and total parenteral n

31 citis (PA) is the most common cause of intra-abdominal abscess in children and is associated with hig

32 There were no intra-abdominal abscess in either groups.

33 and in vivo they were attenuated in an intra-abdominal abscess infection model.

34 ssary for maximal virulence in a mouse intra-abdominal abscess model.

35 90 days included wound infections and intra-abdominal abscess (n = 75) and failure to thrive (n = 38

36 e, percutaneous drainage of associated intra-abdominal abscess or colon resection).

37 sary to repair the perforation to prevent an abdominal abscess or sepsis.

38 There were no intra-abdominal abscesses or other major complications associa

39 recurrence (n = 66, 12%) or because of intra-abdominal-abscess or fistula (n = 93, 16%).

40 nic procedures, OR = 1.5), presence of intra-abdominal abscess (OR = 5.0) or enteric fistula (OR = 4.

41 4.90; 95% CI, 1.41-17.06; P = .01) and intra-abdominal abscess (OR, 7.46; 95% CI, 1.65-33.66; P = .00

42 e IAI (peritonitis, biliary tract infection, abdominal abscess, or enteritis) with those who did not

43 omy closure (6.5%), trauma (2.7%), and intra-abdominal abscess/other peritonitis (1.9%).

44 erminal ileal chronic perforation with intra-abdominal abscess, peritoneal space to rectal and vagina

45 olitis, gastric obstruction, lung infection, abdominal abscess, post-surgical atrial fibrillation, an

46 With CPG-directed care, intra-abdominal abscess rate decreased from 0.24 to 0.10 (adju

47 OS (12): mortality, bowel obstruction, intra-abdominal abscess, recurrent appendicitis, complicated a

48 ts were postoperative wound infection, intra-abdominal abscess, reoperation, length of hospital stay,

49 ery and postoperative wound infection, intra-abdominal abscess, reoperation, or readmission.

50 s were fatal), 6 had viral infections, 2 had abdominal abscesses requiring surgery, one had arm cellu

51 olated organism from clinical cases of intra-abdominal abscesses, suggesting its potential to induce

52 gastrointestinal tract [12; colitis (seven), abdominal abscess (three), and diverticulitis and esopha

53 d collections, one pseudocyst, and one intra-abdominal abscess; two donors underwent reoperation.

54 ic antibiotics, and the development of intra-abdominal abscess was significantly lower in patients re

55 The risk of abdominal abscesses was higher for laparoscopic surgery

56 atients who had an anastomotic leak or intra-abdominal abscess were included in the infection group (

57 diseased terminal ileum longer than 40 cm or abdominal abscesses were excluded.

58 cteroides fragilis is the formation of intra-abdominal abscesses, which are induced by the capsular p

59 CT-guided drainage of abdominal abscesses with small- and very small-bore drai

60 the effectiveness and safety of drainage of abdominal abscesses with small-bore (6F and 9F) drains.

61 rtality and postoperative morbidities (intra-abdominal abscess, wound dehiscence, and intestinal stri