ライフサイエンスコーパス: abdominal pressure

1 a prominent incontinence caused by increased abdominal pressure.

2 renal, and cardiac failure due to increased abdominal pressure.

3 rred in transpulmonary pressure due to intra-abdominal pressure.

4 o continence during rapid increases in intra-abdominal pressure.

5 s in spine anatomy, or external increases in abdominal pressure.

6 lux, which may be related to increased intra-abdominal pressure.

7 an dysfunction resulting from elevated intra-abdominal pressure.

8 stabilization by indirectly increasing intra-abdominal pressure.

9 phincteric closure during increases in intra-abdominal pressure.

10 ity, was filled with water to increase intra-abdominal pressure.

11 se in pleural pressure with increasing intra-abdominal pressure.

12 easured 30 mins after each increase in intra-abdominal pressure.

13 Above intra-abdominal pressure 5 mm Hg, plateau airway pressure incr

14 Addition of sustained continuous negative abdominal pressure (-5 cm H2O) to the positive end-expir

15 cle activity associated with increased intra-abdominal pressure and anal or vaginal EMG probes to obt

16 ge was inserted in order to reduce the intra-abdominal pressure and enable appropriate ventilation.

17 ated with obesity, possibly due to increased abdominal pressure and gastroesophageal reflux disease,

18 ntilation in the presence of increased intra-abdominal pressure and in calculating true transpulmonar

19 d cardiac output 5 mins after each new intra-abdominal pressure and positive end-expiratory pressure

20 casionally difficult, resulting in increased abdominal pressure and possible vascular compromise.

21 are necessary to decompress increased intra-abdominal pressure and prevent or treat persistent and r

22 nimize the risk of developing elevated intra-abdominal pressure and to aggressively treat intra-abdom

23 ure is the gold standard for measuring intra-abdominal pressure, and several nonsurgical methods can

24 evated intracranial pressure, elevated intra-abdominal pressure, and therapeutic hypothermia after ve

25 Increased intra-abdominal pressure appears to produce this effect by aug

26 ction, a negative intrathoracic and positive abdominal pressure are generated [i.e., transdiaphragmat

27 l as minimization of intrathoracic and intra-abdominal pressure as clinically possible.

28 Indeed, increased intra-abdominal pressure, as an extreme marker of abdominal co

29 The mean intra-abdominal pressure at admission day was an independent p

30 The intra-abdominal pressure at baseline was 4 +/- 3 mm Hg in intr

31 as significantly reduced by increasing intra-abdominal pressure at both positive end-expiratory press

32 injured lungs, sustained continuous negative abdominal pressure augmented lung recruitment and oxygen

33 Four levels of intra-abdominal pressure (baseline, 12, 18, and 22 mm Hg) were

34 degrees of matching the corresponding intra-abdominal pressure: baseline positive end-expiratory pre

35 ominal pressure compared with baseline intra-abdominal pressure) but did not change cardiac output (p

36 We investigated the effect of increasing abdominal pressure by waist belt on reflux in patients w

37 e, urinary incontinence during elevations in abdominal pressure, can be divided into 2 systems: a sph

38 Spontaneous respiration (abdominal pressure capsule) and ventilator inflations (a

39 Acutely increased intra-abdominal pressure causes a significant increase in intr

40 g volume (+119% [p < .001] at 22 mm Hg intra-abdominal pressure compared with 5 cm H2O positive end-e

41 g volume (+233% [p < .001] at 22 mm Hg intra-abdominal pressure compared with 5 cm H2O positive end-e

42 % [p < .05], respectively, at 22 mm Hg intra-abdominal pressure compared with baseline intra-abdomina

43 Using an ex vivo model replicating the intra-abdominal pressure conditions of the bladder, the intra-

44 High intra-abdominal pressure could enhance the penetration of chem

45 s muscle) activity that would increase intra-abdominal pressure during EAS contraction, and (4) reinf

46 and acute renal failure with regard to intra-abdominal pressure dynamics, preload limitation, and aft

47 Subjects with a mean intra-abdominal pressure equal to or greater than 12 mm Hg wer

48 otomy negated all effects of increased intra-abdominal pressure except the decreased cardiac index (1

49 -abdominal hypertension, defined by an intra-abdominal pressure greater than or equal to 12 mm Hg (bl

50 01), and intra-abdominal hypertension (intra-abdominal pressure >/= 12 mm Hg) occurred in 19.9%, 20.3

51 to the ICU was associated with higher intra-abdominal pressure, higher plasma C reactive protein con

52 Elevated intra-abdominal pressure (IAP) is a frequent cause of morbidit

53 Elevated intra-abdominal pressure (IAP) is associated with ICP elevatio

54 ently, the gold standard for measuring intra-abdominal pressure (IAP) is via the bladder.

55 sidered the gold standard for indirect intra-abdominal pressure (IAP) measurements.

56 sought to determine whether changes in intra-abdominal pressure (IAP) with aggressive diuretic or vas

57 be present at relatively low levels of intra-abdominal pressure (IAP).

58 sitive end-expiratory pressure (= half intra-abdominal pressure in cm H2O + 5 cm H2O), and high posit

59 gh positive end-expiratory pressure (= intra-abdominal pressure in cm H2O).

60 in positive end-expiratory pressure or intra-abdominal pressures in both conditions.

61 tudied: a) group 1 (n = 9) animals had intra-abdominal pressure increased to 25 mm Hg above baseline,

62 to intra-abdominal pressure to prevent intra-abdominal pressure-induced end-expiratory lung volume de

63 When compared with normal intra-abdominal pressures, intra-abdominal hypertension increa

64 ive end-expiratory pressure matched to intra-abdominal pressure led to a preservation of end-expirato

65 Continuous negative abdominal pressure may be a potential adjunct to positiv

66 Increased abdominal pressure may have a negative effect on intracr

67 into three subgroups according to the intra-abdominal pressure measurement policy in different years

68 Intra-abdominal pressure measurements in predefined at-risk pa

69 redictor of mortality in patients with intra-abdominal pressure measurements started within the first

70 Whether limiting intra-abdominal pressure measurements to preselected at-risk p

71 Serial intra-abdominal pressure measurements, nonoperative pressure-r

72 At each level of intra-abdominal pressure, moderate positive end-expiratory pre

73 We aimed to clarify whether expanded intra-abdominal pressure monitoring results in an increased de

74 In patients with intra-abdominal pressure monitoring, the mean intra-abdominal

75 , and 1,241 patients (46.0%) underwent intra-abdominal pressure monitoring.

76 eover, larger thicknesses increase the intra-abdominal pressure necessary for childbirth.

77 It is now appreciated that elevations of abdominal pressure occur in a wide variety of critically

78 Application of intra-abdominal pressures of 0 and 20 cm H2O at positive end-e

79 ositive end-expiratory pressure to the intra-abdominal pressure on cardio-respiratory parameters.

80 bdominal pressure monitoring, the mean intra-abdominal pressure on the admission day is an independen

81 The detrimental impact of elevated intra-abdominal pressure, progressing to abdominal compartment

82 ; (4) leak-point pressure, the minimum intra-abdominal pressure required to cause incontinence, which

83 n of transient (15 mins) continuous negative abdominal pressure resulted in comparable and lasting (6

84 , and its resuscitation with increased intra-abdominal pressure results in many untoward hemodynamic

85 this condition, routine measurement of intra-abdominal pressure should be performed in high-risk pati

86 citation, an impedance threshold device, and abdominal pressure (sodium nitroprusside-enhanced cardio

87 why persons with chronically increased intra-abdominal pressure, such as the morbidly obese, suffer f

88 d, blood and protein losses, increased intra-abdominal pressure, systemic hyperthermia, and increased

89 At each level of intra-abdominal pressure, three levels of positive end-expirat

90 (insertional force) and the transmission of abdominal pressure through the zone of apposition (appos

91 Increase of intra-abdominal pressure to 25 mm Hg above baseline caused sig

92 he urethra is compressed during increases in abdominal pressure to maintain urethral closure pressure

93 Expanding the measurement of intra-abdominal pressure to more than 50% of intensive care ad

94 ive end-expiratory pressure matched to intra-abdominal pressure to prevent intra-abdominal pressure-i

95 y opening the abdomen, suggesting that intra-abdominal pressure transmission contributes little to co

96 fter PNT, and after SCI+PNT, suggesting that abdominal pressure transmission contributes to continenc

97 eased linearly by ~ 50% of the applied intra-abdominal pressure value, associated with commensurate c

98 y (n = 2) suggested that continuous negative abdominal pressure was an effective adjunct to positive

99 ined, but not transient, continuous negative abdominal pressure was associated with hemodynamic depre

100 Following baseline measurements, intra-abdominal pressure was increased by incrementally inflat

101 Intra-abdominal pressure was measured a minimum of every 8 hou

102 The intra-abdominal pressure was measured in 31.7%, 55.6%, and 41.

103 The intra-abdominal pressure was significantly higher in extrapulm

104 roposed is that central obesity raises intra-abdominal pressure, which increases pleural pressure and

105 aparoscopic surgery produces increased intra-abdominal pressure, which potentially influences hepatic

106 ography (OEP), (ii) intra-thoracic and intra-abdominal pressures with a balloon catheter in each comp