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1  slope of CR acquisition was recorded in the abducens nerve.
2  nerve in DRS is secondary to absence of the abducens nerve.
3 hat include misrouting of the oculomotor and abducens nerves.
4       Neural discharge was recorded from the abducens nerve after a single shock unconditioned stimul
5  random sinusoidal cycles, we stimulated the abducens nerve and observed the resultant eye movements.
6 ysfunction of the oculomotor, trochlear, and abducens nerves and/or the muscles that they innervate.
7 on of motor columns, loss of the phrenic and abducens nerves, and intercostal nerve pathfinding defec
8                                      Stalled abducens nerve bundles did not reach the orbit, resultin
9 l correlate of this response recorded in the abducens nerve can be conditioned entirely in vitro usin
10                                          The abducens nerve (CN6) was traced anteriorly from the deep
11                Intraorbital and intracranial abducens nerves (CN6) were small to absent, particularly
12  motor nerves were typically small, with the abducens nerve (cranial nerve [CN]6) often nondetectable
13 , we demonstrate that selectively disrupting abducens nerve development is sufficient to cause second
14 d that in vitro classical conditioning of an abducens nerve eye-blink response is generated by NMDA r
15 o imaging of Chn1KI/KI mice revealed stalled abducens nerve growth and selective trochlear and first
16      Postmortem studies of DRS have reported abducens nerve hypoplasia and aberrant innervation of th
17 with subarachnoid CN3 hypoplasia, occasional abducens nerve hypoplasia, and subclinical ON hypoplasia
18 ted individuals demonstrated small or absent abducens nerves in all four, small oculomotor nerve in o
19 ontine abducens nuclear defects, rather than abducens nerve involvement.
20 is linked to Duane syndrome, a defect in the abducens nerve/lateral rectus muscle connection.
21 rded from pairs of oculomotor, trochlear, or abducens nerves of an in vitro turtle brainstem preparat
22 anifestations of IIH such as papilledema and abducens nerve palsy are well recognized, but less commo
23 imaging of patients with isolated unilateral abducens nerve palsy without other ocular motility disor
24  for human eye movement dysfunctions such as abducens nerve palsy.
25 Cranial nerve assessment revealed mild right abducens nerve palsy; neurologic examination was otherwi
26                                          The abducens nerve phenotype ranges from complete absence, t
27 e-reversed" responses were poorly defined in abducens nerve recordings.
28                                A conditioned abducens nerve response is generated in in vitro brainst
29  model of the classically conditioned turtle abducens nerve response, we investigated the effect of c
30 t the primary cause of DRS is failure of the abducens nerve to fully innervate the lateral rectus mus
31 erized by a failure of cranial nerve VI (the abducens nerve) to develop normally, resulting in restri
32 mice did not have DRS, and embryos displayed abducens nerve wandering distinct from the Chn1KI/KI phe
33 f left oculomotor, right trochlear and right abducens nerves were approximately aligned with leftward
34 nce of Listing's law, we microstimulated the abducens nerve with the eye at different initial vertica