ライフサイエンスコーパス: abnormal

1 iminated, and chest imaging findings must be abnormal.

2 mal family lives in the stage from normal to abnormal.

3 ion of both glia and neurons, preceded by an abnormal accumulation of glial LDs.

4 Cellular stress induced by the abnormal accumulation of unfolded or misfolded proteins

5 Of those with a normal CFR, 53% had an abnormal AchFR, and 47% had a normal AchFR; ischemia rat

6 vidence has indicated that cytosolic DNA and abnormal activation of the cytosolic DNA sensing adaptor

7 t ventilated) and mild emphysema (normal CT, abnormal ADC) were negatively correlated with FEV(1) (r

8 the digits but fails to bifurcate because of abnormal adhesion of the periderm.

9 The patient also shows abnormal AEPs presenting only Waves I and II.

10 ovement patterns in scapulae along with some abnormal alignment in the upper quarter, which may contr

11 stream disruption, morphological changes and abnormal allocation of cortical interneurons in vivo The

12 tween Alzheimer's disease (defined as having abnormal amyloid, n = 425) and controls (defined as havi

13 Among 61 MSW, abnormal anal cytology was detected in 12 (20%) and was

14 However, oscillometry was markedly abnormal and significantly different from AR0 (P < 0.05)

15 Forty-nine (65%) anal cytology samples were abnormal, and 38 (51%) of anal samples were positive for

16 Shoaling behavior of mutant adults was abnormal, and they did not display social attraction to

17 ic residues of BACE1 and predicts to inhibit abnormal APP processing and reduce Abeta levels in AD ne

18 nt length and number that was accompanied by abnormal Arp2/3 complex and heterodimeric capping protei

19 and malformed notochord sheath formation and abnormal axial skeleton segmentation due to dysregulated

20 Among patients with abnormal baseline EF, information on follow-up EF was av

21 ex lesions presented more frequently with an abnormal baseline EGD (70% vs. 37%; p = 0.04) and had a

22 nal deficits at neural network level lead to abnormal behavioral learning remains unexplored.

23 of the link between perinatal cytokines and abnormal behaviors in offspring, human epidemiological s

24 varying, functionless behaviours -are common abnormal behaviours that often arise in suboptimal condi

25 mmalian visual system.SIGNIFICANCE STATEMENT Abnormal binocular vision and reduced acuity are hallmar

26 ed to occur in PTOA: tissue inflammation and abnormal biomechanical loading.

27 create weaker attractor basins that promote abnormal, bistable representations under certain conditi

28 information to understand the sources of the abnormal blending behavior.

29 ported grade 3 or higher adverse events were abnormal blood chemistry results (33 [15%] of 221 patien

30 (MRI) is readily used for quantification of abnormal blood flow in adult hearts, however, existing i

31 allow for the investigation of causality of abnormal blood flow in CVD.

32 Abnormal BMP signaling has also been associated with dis

33 at replace hematopoietic cells, resulting in abnormal bone marrow function.

34 The role of abnormal brain iron metabolism in neurodegenerative dise

35 difficulties, spasticity or ataxic gait, and abnormal brain MRI.

36 Complete surgical resection of abnormal brain tissue is the most important predictor of

37 In prototype demonstrations, abnormal breath conditions (apnea, hypopnea, polypnea) a

38 PDE4B deficiency leads to abnormal Ca(2+) handling and PDE4B is decreased in press

39 Abnormal CA3 hyperactivity in aged rats has been propose

40 ulation of calcium flux genes accompanied by abnormal calcium-dependent cerebellar membrane excitabil

41 ypes involves the interaction of genetically abnormal cancer cells with normal stromal cells.

42 iate the effect of chronic kidney disease on abnormal cardiac function and cardiovascular events in t

43 s the association of comorbidity burden with abnormal cardiac structure/function in HFpEF.

44 ogation of the lipidomics signature revealed abnormal cardiolipin remodeling in diabetic kidneys, a c

45 s with hypertension can be accompanied by an abnormal cardiovascular response that includes attenuate

46 levels of IL-8 and TNFalpha, associated with abnormal cell division and cell death, respectively.

47 itioning defects are likely a consequence of abnormal cell shapes and not due to TAN1 absence.

48 cell types and quantifications of normal and abnormal cell types in tumours provide a better predicti

49 Computational prediction revealed that the abnormal cell-cell communication patterns observed durin

50 guard against aberrant hypoxic signaling and abnormal cell-cycle progression.

51 esulted in further growth rate decreases and abnormal cells.

52 ted the potential of NBD-Bu for detection of abnormal cellular metabolic activity.

53 Manifesting in abnormal cerebral perfusion and cerebrospinal fluid conv

54 The incidence of abnormal cerebrospinal fluid (CSF) flow dynamics in chil

55 Abnormal changes of neuronal Tau protein, such as phosph

56 to have diabetes, respiratory rate >22/min, abnormal chest imaging findings, O2 saturation lower tha

57 and its homologs, bin2-3 bil1 bil2, displays abnormal chloroplast development, whereas the gain-of-fu

58 c in WT eggs prior to activation resulted in abnormal chorion elevation similar to that observed in z

59 A maize chromosome variant called abnormal chromosome 10 (Ab10) converts knobs on chromoso

60 deletion induces centromere instability and abnormal chromosome segregation.

61 treat neurological diseases characterized by abnormal circuit excitability.

62 ophrenia were enriched for those involved in abnormal CNS synaptic transmission (PFDR = 0.02) and ant

63 Abnormal control of blood vessel morphogenesis and matur

64 er, make it challenging to accurately detect abnormal copy number events in a cost-effective manner i

65 Patients showed abnormal cortical thinning of temporal cortices and thic

66 major depressive disorder propose a role for abnormal cortico-limbic connectivity.

67 increased cell mobility in early embryos and abnormal craniofacial morphology in later embryos.

68 Abnormal cytokine secretion is often regarded as an earl

69 roblasts with cytoplasmic bridges indicating abnormal cytokinesis and abscission.

70 Among 299 MSM, abnormal cytology was detected in 161 (54%) MSM and was

71 Women with abnormal cytology were referred to colposcopy, biopsy, a

72 9v vaccine was high and was associated with abnormal cytology.

73 hin hours in adults singing while exposed to abnormal (delayed) auditory feedback.

74 ability deficits, cognitive dysfunction, and abnormal dendritic morphogenesis.

75 ant beta-actin mRNA localization resulted in abnormal dendritic protrusions and growth cone dynamics.

76 impaired functional synaptic plasticity and abnormal dendritic spine morphology, but little is known

77 early afterdepolarizations (EADs), which are abnormal depolarizations during the repolarization phase

78 Fibrosis is the abnormal deposition of extracellular matrix, which can l

79 revealed no scleral architectural changes or abnormal deposits.

80 eurodevelopmental disorders that result from abnormal development of the cerebral cortex in utero.

81 ac manifestations in PEX, involving signs of abnormal diastolic and systolic right ventricular functi

82 es such as abnormal social investigation and abnormal discrimination/associative learning, unlike the

83 lts in defective cell function caused by the abnormal distribution of genetic information.

84 nt after 5 days because of an adverse event "abnormal drug taste." The most common adverse events wer

85 continued treatment after 3 days because of "abnormal drug taste." The most commonly reported adverse

86 Gene expression rhythms rapidly become abnormal during critical illness.

87 ts that usually develop normally, results in abnormal ear and lateral line phenotypes.

88 ssociated with incident HF, HF subtypes, and abnormal echocardiographic measures in the absence of cl

89 eceptors, we examined the hypothesis that an abnormal ECM-integrin receptor axis contributes to BM me

90 tability of test results (both 24-2 and 10-2 abnormal, either abnormal, or both normal) was achieved

91 ratio [HR]: 2.91; 95% CI: 1.82 to 4.65) and abnormal electrocardiogram (ECG) (HR: 4.02; 95% CI: 2.51

92 In clinical practice, an abnormal electrocardiogram with normal or non-diagnostic

93 found that GJA1M213L/M213L mice had severely abnormal electrocardiograms despite preserved contractil

94 ssenger cars; they are considered part of an abnormal emission control strategy.

95 orm it beneficial effects on the ACE-induced abnormal emotional behaviors by "calming down" the whole

96 ribed de novo HLHS mutations associated with abnormal endocardial gene and fibronectin regulation.

97 ial studies that patients with PSC harbor an abnormal enteric microbiome.

98 ven with its immunomodulatory actions, while abnormal epithelial repair responses may contribute to r

99 for liver development in infants exposed to abnormal estrogen levels or estrogenic compounds during

100 in the grin1 double-mutant larvae, including abnormal evoked responses to light and acoustic stimuli,

101 Abnormal neonatal outcome was defined as abnormal exam and/or abnormal imaging.

102 ctive value, positive predictive value of an abnormal examination, and positive predictive value of b

103 Fhf2 knockout (Fhf2(KO)) mice predisposes to abnormal excitability at the tissue level are not well d

104 Abnormal excitatory and inhibitory synapse assembly and

105 esial temporal lobes, with development of an abnormal excitatory fronto-temporal projection in the le

106 tates, however, Fmr1(-/y) circuits exhibited abnormal experience-dependent plasticity as they did not

107 ents have recurrent bacterial infections and abnormal expression of CD56 by monocytes.

108 res the expression of T-bet and corrects the abnormal expression of immature NK genes.

109 d E10.5 showed that Fkbp8-/- embryos have an abnormal expression profile within tissues harvested at

110 ng of the molecular mechanisms that underlie abnormal eye development and impaired vision.

111 normalcy and positioned themselves as living abnormal family lives in the stage from normal to abnorm

112 deeper insight into etiologic mechanisms of abnormal fetal growth outcomes.

113 EF is the activation and proliferation of an abnormal fibroblast phenotype that is resistant to apopt

114 These agents must be designed to target the abnormal fibroblasts with high specificity because many

115 In patients with an abnormal field with either test, the overlap varied from

116 Catheters are the second most common abnormal finding on radiographs.

117 Equal numbers of children with abnormal findings (29 of 45, 64%) and normal findings (5

118 owed benign findings in 95 (64.6%) women and abnormal findings in 11 (7.5%) women; one or both ovarie

119 isease within the surrounding mesentery, and abnormal findings in adjacent structures.

120 Electronic medical records were reviewed for abnormal findings on ultra-widefield fluorescein angiogr

121 on initial or follow-up examination but had abnormal findings on UWFFA or OCT.

122 Quantitative sensory testing showed abnormal findings.

123 barriers to diagnostic colonoscopy following abnormal FIT in an academic healthcare setting.

124 al FIT result, and a majority learned of the abnormal FIT result by direct communication with provide

125 Fifty-one were aware of the abnormal FIT result, and a majority learned of the abnor

126 rming volatile compounds that transmitted an abnormal flavor which tasters identified as rancid.

127 re Pten((fl/fl)) Kras(G12D/+)(G12D mice) had abnormal follicle structures and developed low-grade ser

128 ed whether GSK3beta inhibition reverses both abnormal gamma oscillations and behavioral deficits with

129 down, but not GSK3A knockdown, also reversed abnormal gamma oscillations and behavioral deficits.

130 We thus explored the abnormal gastric retention induced by PDE4 inhibition in

131 In humans, abnormal gastric retention of food is known as gastropar

132 une function, visceral hypersensitivity, and abnormal gastrointestinal motility.

133 rst cohort included eyes with mild glaucoma (abnormal glaucoma hemifield test results, pattern standa

134 Abnormal glucose in combination with WBC levels of >=10

135 Therefore, abnormal glucose metabolism, cognitive impairment and wi

136 n patients with CUD would be associated with abnormal glutamatergic metabolites in the putamen.

137 data support our hypothesis and suggest that abnormal glutamine and glutamate transporter activity is

138 A major hallmark of LD is the presence of abnormal glycogen aggregates in neurons and other tissue

139 e Kallmann syndrome (KS) is characterized by abnormal GnRH-1ns migration, we examined whole-exome seq

140 Conventional impedance standards for abnormal GOR frequency are weakly sensitive for the dete

141 ptogenetic inhibition of the STN exacerbated abnormal GPe activity, and (6) exaggerated beta band act

142 authors to preemptively identify and remove abnormal grafts.

143 Abnormal HA accumulation within blood vessel walls is as

144 acroplaxome during spermatid elongation and abnormal head morphologies in mature spermatozoa.

145 l disease is caused by polymerization of the abnormal hemoglobin S upon deoxygenation in the tissues

146 tment-related deaths in three (3%) patients (abnormal hepatic function, hepatic cirrhosis, and pneumo

147 n the United States are discarded because of abnormal histology.

148 In particular, abnormal hypermetabolism in the brain, as evaluated by(1

149 educed level of activated Rac in muscle, and abnormal IACs at muscle cell boundaries.

150 outcome was defined as abnormal exam and/or abnormal imaging.

151 he humoral immune response toward generating abnormal immature memory B cells (MB), while impairing p

152 The abnormal immune cells may be the drivers of systemic abn

153 A is not understood but is likely related to abnormal immune responses and environmental exposures.

154 In contrast, automatic inhibition was abnormal/impaired in patients with tic disorders.

155 LFT were abnormal in 6 of them, and a newly detected hepatic lesi

156 ients across the entire cortex, and they are abnormal in mental illness.

157 ults: Neuroendocrine biomarker profiles were abnormal in most patients.

158 urons (cINs) are consistently observed to be abnormal in SCZ postmortem brains.

159 inite ALPS patients had a significantly more abnormal in vitro apoptosis function, with lower annexin

160 ypes of ALPS patients; the combination of an abnormal in vitro apoptosis functional test and elevated

161 The combination of elevated DNTs and an abnormal in vitro apoptosis functional test was the most

162 No PFA animals (0 of 6, 0%) developed abnormal in-life observations, but 1 of 4 RFA animals (2

163 nderstanding these changes and the resulting abnormal interactions of SGCs with sensory neurons could

164 These regions are known to contribute to abnormal interhemispheric information transfer, heteromo

165 tes that these disruptions can be related to abnormal interoception (i.e., the sensing of visceral fe

166 ith the DM group, the DBT1 group had a lower abnormal interpretation rate (AIR) (adjusted odds ratio

167 Furthermore, the abnormal intracellular HA was also observed in periphera

168 at loss of CBX2 in mouse fibroblasts induces abnormal large-scale chromatin structure and chromosome

169 Three of 170 patients (2%) had abnormal left ventricular ejection fraction.

170 Each disorder stems from the abnormal lengthening of a glutamine repeat in a differen

171 More abnormal levels of baseline cerebrospinal fluid phosphor

172 f different neurological diseases related to abnormal levels of dopamine.

173 ide) <100 pg/mL), and those with ECG-LVH and abnormal levels of either biomarker (malignant LVH).

174 e between IL-18 and IL-18BP is dysregulated, abnormal levels of free bioactive IL-18 are detected, su

175 Abnormal levels of N-acetyl compounds, glutamate+glutami

176 e stress episodes before reaching persistent abnormal levels.

177 Although abnormal LFTs are already frequently present upon admiss

178 iver disease, who underwent liver biopsy for abnormal liver biochemistry and/or clinical suspicion of

179 .18-4.48), serious adverse events related to abnormal liver function tests (OR 11.19, 95% CI: 2.09-60

180 Abnormal liver tests occur in most hospitalized patients

181 significant proportion of these patients had abnormal liver tests prehospitalization (AST 25.9%, ALT

182 Abnormal liver tests were commonly observed in hospitali

183 de and in in vivo joint cartilage exposed to abnormal loading.

184 Although Abeta levels become abnormal long before severe cognitive impairments appear

185 cer drugs have been associated with a highly abnormal loss of redox control in TNBC cells.

186 ns have a role in the pathophysiology of the abnormal LTP-like plasticity in PD.

187 Abnormal maternal serum biomarkers (AMSB), identified th

188 Their membranes exhibited numerous large abnormal membrane microdomains (aMMDs), which trap and i

189 eas, including the striatum, which shapes an abnormal memory trace of drug consumption that virtually

190 ethanesulfonate-induced mutant that exhibits abnormal mesocarp cell differentiation could help elucid

191 ling is patients with rare disease for which abnormal metabolic signatures may yield clues into the g

192 subset of limb-innervating motor neurons and abnormal migration of V2a interneurons.

193 Abnormal mitochondria can trigger a series of human dise

194 In turn, this resulted in abnormal Mk proliferation that was reverted using a spec

195 Abnormal MoCA scores (<26) were predictive of CNS diseas

196 In all CHRs, abnormal modular connectome organization at baseline was

197 Abnormal modulation of Aurora B in interphase leads to c

198 For 13 of the 20 symptoms (abnormal mole, breast lump, post-menopausal bleeding, re

199 or the first 4 or 12 weeks of life prevented abnormal molecular and cognitive phenotypes in rT1, demo

200 TTC7A-KO HAP1 cells have abnormal morphology and undergo apoptosis, due to increa

201 or intermittent muscle contractions causing abnormal movements and postures, often occurring in abse

202 s involuntary muscle contractions leading to abnormal movements and postures.

203 ssion levels impact the onset or severity of abnormal movements or neuropathological features in DYT1

204 Abnormal mPRM measurements of small airways disease (nor

205 Forty-three patients had abnormal MRI findings 2-4 weeks after symptom onset (58.

206 Importantly, patients with abnormal MRI findings at onset were more likely to have

207 s, high-penetrance PVs, and any PVs) who had abnormal MSI/IHC testing and/or PREMM(5) score >= 2.5%.

208 osition and phosphorylation state, and found abnormal mTOR expression in both complexes in SZ DLPFC.

209 ter 2 years there was complete resolution of abnormal multifocal reduced diffusion.

210 suggest strategies that counter CKD-induced abnormal muscle protein catabolism.

211 al for development; however, if unregulated, abnormal muscle remodeling can occur, such as in the hea

212 Importantly, the abnormal myeloid progenitors (AMPs), a leukemia-initiati

213 functional changes may promote expansion of abnormal myeloid progenitors in del(5q) MDS, and in rare

214 ) in osteocytes (Dmp1-Gsalpha(KO) mice) have abnormal myelopoiesis, osteopenia, and reduced adipose t

215 nteractions with both partners, resulting in abnormal N protein oligomerization that was further conf

216 essive neurological abnormalities (including abnormal neonatal cry, hypotonia, epilepsy, polyneuropat

217 Abnormal neonatal outcome was defined as abnormal exam a

218 Abnormal neuroanatomic brain networks have been reported

219 Results support multifocal abnormal neuron or glial density, mitochondrial energeti

220 the algebraic connectivity metric) caused by abnormal neuronal firing during a seizure onset.

221 synthesis and actin dynamics can lead to an abnormal neuronal morphology resulting in deficits in le

222 Abnormal neurotransmission is central to schizophrenia (

223 ilable machine learning algorithm that flags abnormal noncontrast CT examinations for ICH was impleme

224 8 A+/-/T+/-/N+/- categories according to the abnormal (+)/normal (-) status of Abeta ((18)F-florbetap

225 Despite the correlation between abnormal nuclear shape and human pathologies, the mechan

226 f lean NAFLD patients who were metabolically abnormal or had elevated alanine aminotransferase (ALT)

227 results (both 24-2 and 10-2 abnormal, either abnormal, or both normal) was achieved in 55% to 70% of

228 thin intercalated discs were revealed by the abnormal organization of desmoplakin, plectin, N-cadheri

229 insonian motor symptoms through reduction of abnormal oscillatory activity in the STN-associated neur

230 e opposite sinus, presence of intramurality, abnormal ostium-and symptoms or evidence of myocardial i

231 dings (65 patients) separated for normal and abnormal outcome at 9-24 months follow-up.

232 Abnormal outcome trajectories were associated with clini

233 Abnormal PAC predicted HFrEF (HFpEF: 2.0 [1.0-4.0, p = 0

234 ed to identify neuroanatomical correlates of abnormal pain perception.

235 pecc1lDeltaC510/DeltaC510 homozygotes showed abnormal palate rugae but did not show cleft palate.

236 e in normal epithelial tissue repair and its abnormal persistence in disease conditions.

237 utrophils showed transcriptional evidence of abnormal phagocytic and cell-survival programs.Conclusio

238 Further, abnormal phalloidin and wheat germ agglutinin (WGA) stai

239 levated, especially during cortical ACT, (2) abnormal phasic suppression of PV GPe neuron activity wa

240 annel blockade as a robust suppressor of the abnormal phenotypes in both models of MYL4 (myosin light

241 Abnormal phosphorylation is thought to precede aggregati

242 This implies that abnormal phosphorylations at certain sites may not affec

243 0.28-0.99], p = 0.048), and the frequency of abnormal PiB SUVrs (OR [95% CI] = 0.10 [0.01-0.90], p =

244 In addition, abnormal positioning of Muller cell bodies and bipolar c

245 eatures included perisylvian polymicrogyria, abnormal posterior clinoid processes and persistent trig

246 genetic and epigenetic changes that underlie abnormal proliferation of hematopoietic stem and progeni

247 t regulation of the immune system leading to abnormal proliferation of skin cells.

248 enic variants in VMA21 in male patients with abnormal protein glycosylation that result in mild chole

249 s in the urine of CKD patients and extracted abnormal protein signals comparing with the healthy cont

250 a better predictor of FA-M/E positivity than abnormal protein.

251 were not studied due to equipment failure or abnormal pulmonary arterial anatomy.

252 s to define the prevalence and correlates of abnormal pulmonary pressure, resistance, and compliance

253 Abnormal pulmonary VR was defined by the slope of increa

254 ], HFrEF: 2.8 [1.4-5.5, p = 0.003]), whereas abnormal PVR was not predictive of either (HFpEF: 0.9 [0

255 Abnormal QT interval responses to heart rate (QT dynamic

256 persensitivity to low luminance contrast and abnormal rapid blood oxygenation level-dependent signal

257 In Parkinson's disease and its models, abnormal rates and patterns of GPe-STN network activity

258 ture (LST) changes in North America are very abnormal recently, but few studies have systematically r

259 ioblastoma multiforme (GBM) cells would have abnormal redox status due to bio-thiols, like glutathion

260 The abnormal regulation of this process, which is connected

261 y characterizes microdamage accumulation and abnormal remodeling mechanisms in the diabetic skeleton,

262 /- 1.5, and the mean HAS-BLED (hypertension, abnormal renal/liver function, stroke, bleeding history

263 inhibition of the PFC-BNST pathway restored abnormal responses to predator odor in alcohol-exposed m

264 However, it is unclear whether there is abnormal reward-linked effective connectivity between th

265 nostic colonoscopy completion in adults with abnormal screening fecal immunochemical test (FIT) resul

266 ring contraction of cyclohexenes exhibits an abnormal selectivity pattern in which a formally concert

267 d cerebellum, as well as the contribution of abnormal sensorimotor processing.

268 Abnormal sensory processing has been observed in autism,

269 The prevalence of adolescents with one abnormal serum lipid component was 43.2% and was signifi

270 h produces relevant mouse phenotypes such as abnormal social investigation and abnormal discriminatio

271 ther diseases or disorders that present with abnormal solid masses in the brain, including cysts and

272 defects including a low number of sperm and abnormal sperm morphology.

273 protein (an ortholog of kindlin-2) prevents abnormal spindle assembly; however, the mechanism remain

274 The abnormal status of DNA 6 mA modification has been report

275 The implications of abnormal structural connectivity for disrupted conscious

276 ing, and in an animal model of FD it rescued abnormal sympathetic target tissue innervation.

277 eta appears early, followed by deposition of abnormal tau aggregates and subsequent neurodegeneration

278 in order to reach a consensus on normal and abnormal tau imaging values that can be universally impl

279 ive diseases associated with accumulation of abnormal tau protein in the brain.

280 nd found that, although this correlates with abnormal TCA fasciculation, it does not induce topograph

281 For the first time multi-level and abnormal telegraph noise can be utilised, which greatly

282 m, with a gradually increasing proportion of abnormal test results remained stable, even if samples f

283 e propose an analytical pipeline to identify abnormal thalamocortical network dynamics in cLBP patien

284 olonization by the DeltassrA strain was also abnormal: the epithelial cells underwent premature swell

285 ve perspective of normalcy in the stage from abnormal to normal.

286 se level, often with normal or only slightly abnormal transaminases.

287 h atherosclerosis, hypercholesterolemia, and abnormal transforming growth factor beta (TGF-beta) sign

288 to HIG administration (AOR, 3.0; P = .002), abnormal ultrasounds (AOR, 59; P = .0002), and diagnosis

289 Lack of HIG and abnormal ultrasounds also predicted symptoms (P = .001).

290 vidence that CAKUTs can arise as a result of abnormal urothelial development.

291 uced FGFR2/ERK signaling apparently leads to abnormal urothelial repair after cyclophosphamide exposu

292 5mug/dL or less is an abnormal value, and 3mug/dL or less is considered to be

293 Abnormal vasculature in tumors leads to poor tissue perf

294 uncharacteristic electrical impulses due to abnormal VGSC functional expression influenced by the la

295 a total of 5,321 patients presented severely abnormal vital signs.

296 dividuals with normal VR (n = 8), those with abnormal VR (n = 21) displayed higher central and pulmon

297 In cohort 2, plasma P-tau217 discriminated abnormal vs normal tau-PET scans (AUC, 0.93 [95% CI, 0.9

298 Specifically, VS responses that were abnormal with respect to predictions from reinforcement

299 All 3 parameters were increasingly abnormal with the development of dark flecks and decreas

300 C1q, and that V1b development would also be abnormal without C1q-mediated synapse elimination.