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1 ar neurodegeneration and progressive ataxia (abnormal gait).
2  restriction in jaw and tongue movements and abnormal gait.
3 ation findings of frontal release signs, and abnormal gait.
4  often leading to lower limb deformities and abnormal gait.
5 viving mice display hindlimb weakness and an abnormal gait.
6                    Mice lacking RGS6 exhibit abnormal gait and ataxia characterized by impaired rotar
7 ypotension, and arrhythmias, as well as with abnormal gait and balance, physical frailty, sensory imp
8 ; nausea and vomiting (75%), headache (67%), abnormal gait and coordination (60%), and papilloedema (
9 nd vomiting (19%) for central brain tumours; abnormal gait and coordination (78%), cranial nerve pals
10 ociated with a motoric (posture instability, abnormal gait and decreased locomotion) and partial sens
11                Homozygous mutant mice had an abnormal gait and difficulties in maintaining balance.
12 models with spontaneous Kcnn2 mutations show abnormal gait and locomotor activity, tremor and memory
13  severe motor impairment, resting tremor and abnormal gait and posture, phenotypes reminiscent of Par
14 ological disorders that are characterized by abnormal gait and spasticity of the lower limbs.
15                               Therefore, the abnormal gait and torsion of the limb caused by the hemi
16 mutant mouse line nur5 display tremor and an abnormal gait, as well as Purkinje cell degeneration and
17                  However, tremor, ataxia and abnormal gait become apparent in older mice.
18 ss of coordination, tremors, hypokinesis and abnormal gait, before dying prematurely.
19 igigait analysis shows these mice develop an abnormal gait by 12 months of age.
20  gene cause skeletal defects, hyperactivity, abnormal gait, deafness, white belly spotting, and hypop
21 re-defined syndrome consisting of hypotonia, abnormal gait, developmental delay/intellectual disabili
22 a suggest that pain is the primary driver of abnormal gait in models of inflammatory but not nerve in
23  motor coordination and locomotion including abnormal gait, inability to run or swim, impaired rotaro
24 neurodevelopmental disorder characterized by abnormal gait, intellectual disabilities, and seizures,
25 seizures, frequent smiling and laughter, and abnormal gait, is one of the best examples of human dise
26 nstrated decreased body size, grip weakness, abnormal gait, joint laxity, and early-onset osteoarthri
27                    Clinical features include abnormal gait, muscle weakness and wasting, and generali
28 aluated with video motion-tracking confirmed abnormal gait patterns in the hindlimbs.
29                Of non-Alzheimer's dementias, abnormal gait predicted the development of vascular deme
30 mice exhibited impaired long-term memory and abnormal gait, recapitulating a subset of clinical featu
31                          Homozygotes have an abnormal gait, show abnormal posturing when suspended by
32                           Among the types of abnormal gait, unsteady gait predicted vascular dementia
33  defect results in the limb being held in an abnormal gait, which causes problems in the locomotion o