1 ells (RBCs) because of polymerization of the abnormal hemoglobin.

2 verely impaired beta 99 mutants of the human abnormal hemoglobins.

3 ss from sickle erythrocytes concentrates the abnormal hemoglobin and promotes sickling.

4 ible for recidivism, chronic kidney disease, abnormal hemoglobin, and low ejection fraction evaluated

5 kling hemoglobin, whose carriers express the abnormal hemoglobin at 40% to 50%, with a very similar c

6 emia, sickle anemia, thrombosis, stroke, and abnormal hemoglobin forms, this current finding suggests

7                                        Other abnormal hemoglobins, glucose-6-phosphate dehydrogenase

8                   The production of HbS - an abnormal hemoglobin (Hb) - in sickle cell disease (SCD)

9 her has sickle cell trait, his mother has no abnormal hemoglobin (Hb).

10 bin gene that results in the formation of an abnormal hemoglobin [HbS (alpha2betaS2)].

11 l disease is caused by polymerization of the abnormal hemoglobin S upon deoxygenation in the tissues

12 ease are related mainly to the production of abnormal hemoglobin that leads to organ ischemia and inc

13    This phenomenon is due to the presence of abnormal hemoglobin that polymerizes upon deoxygenation.

14 hrocyte protoporphyrin, with the addition of abnormal hemoglobin to classify iron deficiency anemia.