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1        A total of 48/66 (72.7%) cases had an abnormal karyotype.
2 uble antisense transfected cells with highly abnormal karyotype.
3  both accounted for 76% of all cases with an abnormal karyotype.
4 ally delayed, three died, and two others had abnormal karyotype.
5  found in 36 of 158 successive patients with abnormal karyotypes.
6 ors by Southern blot analysis, and three had abnormal karyotypes.
7 tes were similar in patients with or without abnormal karyotypes.
8 cheduled tetraploid cells can acquire highly abnormal karyotypes.
9                           In 14 cancers with abnormal karyotypes, 65% (123 of 188) of the chromosomal
10 L cases including 29 normal karyotype AML, 8 abnormal karyotype AML and 8 AML without karyotype infor
11  shared between the normal karyotype AML and abnormal karyotype AML.
12 nd 4) established from these colonies had an abnormal karyotype and altered morphology, but were not
13 lls, and a strong correlation exists between abnormal karyotype and tumorigenicity.
14                    Mayo Clinic patients with abnormal karyotypes and FISH-detected IgH translocation
15 fore and after spontaneous transformation to abnormal karyotypes and in correlation to cancer cells.
16 with poorer outcomes included age >60 years, abnormal karyotype, and terminal deoxynucleotidyltransfe
17 rom 2 large cohorts of patients with MM with abnormal karyotype, and then validated it in 2 independe
18 S was determined independent of blast count, abnormal karyotypes, and concurrent variants, including
19 The engrafted human cells exhibited the same abnormal karyotype as primary cells in a portion of the
20 ologies was completely reliable to detect an abnormal karyotype, but the best protocol for an interpr
21      We obtained data for the details of all abnormal karyotypes by reason for referral and assessed
22                                          The abnormal karyotype correlated with shorter time to first
23 alyzed 1,054 adult patients with MDS with an abnormal karyotype from the Spanish Registry of MDS.
24                   We propose that cells with abnormal karyotypes generate a signal for their own elim
25 tively high frequency of cells with the same abnormal karyotype (>5-10%; presumably of clonal origin)
26 ics had CNA, whereas 40% of patients with an abnormal karyotype had additional CNA detected by SNP ar
27     However, most human solid tumors have an abnormal karyotype implying that gain and loss of chromo
28       Metaphase cytogenetics (MC) detects an abnormal karyotype in only half of patients with AML, ho
29    Overall, 103 of 169 (61%) patients had an abnormal karyotype, including 31 with del(6q), 29 with 1
30  (82.4%) had a normal and 426 (17.6%) had an abnormal karyotype, including 329 patients (13.6%) with
31 udies have shown that pluripotent cells with abnormal karyotypes may grow faster, differentiate less
32 ts, 29 (48%) had secondary AML, 30 (50%) had abnormal karyotypes (monosomy 5 and/or 7 in 15 [25%]), a
33                    Conversion from normal to abnormal karyotype occurred at a constant rate after ini
34                              Evolution to an abnormal karyotype occurred in 16 (19%), most within 6 m
35 s), 69 (13%) trios had a clinically relevant abnormal karyotype or chromosomal microarray finding, 51
36 er International Prognostic Score (P = .01), abnormal karyotype (P = .05), the presence of excess bla
37 linical parameters linked to poor prognosis, abnormal karyotype (P =.002) and high serum beta(2)-micr
38 isease, incomplete blood count recovery, and abnormal karyotype pre-HCT, MRD-positive HCT was associa
39 0 and one in 40 samples having an undetected abnormal karyotype, respectively.
40 erations at 12q14-15 and in one case with an abnormal karyotype that included double minute chromosom
41                In multivariable analysis, an abnormal karyotype, the presence of FLT3-internal tandem
42            Given the potential of cells with abnormal karyotypes to become cancerous, do pathways tha
43 ary cells, but the specific contributions of abnormal karyotypes to cancer, a disease characterized b
44 ndem duplication in 447 AML patients with an abnormal karyotype treated in Medical Research Council (
45                                           An abnormal karyotype was found in 21.5% and 35.7% of cases
46                                              Abnormal karyotypes were detected in 37% of our patients
47             Meiotic errors lead to uniformly abnormal karyotypes, while mitotic errors lead to chromo
48        Because the odds are very low that an abnormal karyotype will surpass the viability of a norma