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1 scular inflammation, intimal hyperplasia and accelerated atherosclerosis.
2 stemic inflammation, contributes to pathogen-accelerated atherosclerosis.
3 ducible nitric oxide synthase expression and accelerated atherosclerosis.
4  disease concomitantly prevents P gingivalis-accelerated atherosclerosis.
5 eriodontal disease, upregulation of TLRs, or accelerated atherosclerosis.
6 emia are important risk factors for diabetes-accelerated atherosclerosis.
7 reated the gld.apoE(-/-) mouse as a model of accelerated atherosclerosis.
8 iency led to decreased titers of T15/EO6 and accelerated atherosclerosis.
9 and ApoE-/-OPN-/- mice developed less Ang II-accelerated atherosclerosis.
10 olesterolemia and diabetes, is implicated in accelerated atherosclerosis.
11 cle cells and lesion progression in diabetes-accelerated atherosclerosis.
12 rom retinopathy, nephropathy, neuropathy and accelerated atherosclerosis.
13 ivation in premenopausal females, leading to accelerated atherosclerosis.
14     In this sense, transplants suffered from accelerated atherosclerosis.
15  CREBH prevented the progression of diabetes-accelerated atherosclerosis.
16 s the causality of certain CHIP mutations in accelerated atherosclerosis.
17 asma low-density lipoprotein cholesterol and accelerated atherosclerosis.
18 ut the impact of S-nitrosylation in diabetes-accelerated atherosclerosis.
19 erse events in patients with cancer, such as accelerated atherosclerosis.
20  efficient strategy for alleviating diabetes-accelerated atherosclerosis.
21 ng, pulmonary and systemic hypertension, and accelerated atherosclerosis.
22  drive increased cardiovascular risk through accelerated atherosclerosis.
23 ermediate intimal hyperplasia and late-stage accelerated atherosclerosis.
24 istance, and fatty liver disease, as well as accelerated atherosclerosis.
25  associated with reduced NO availability and accelerated atherosclerosis.
26 vasculitis, thromboembolism, and potentially accelerated atherosclerosis.
27 curs at an average age of 14.6 years from an accelerated atherosclerosis.
28 n autoimmunity-associated diseases that have accelerated atherosclerosis.
29 d chronic hyperglycemia, which significantly accelerated atherosclerosis.
30 and angiogenesis in an experimental model of accelerated atherosclerosis.
31 3alpha in the development and progression of accelerated atherosclerosis.
32 ted chemokine receptors in a model of injury-accelerated atherosclerosis.
33 nked to cardiovascular disease, possibly via accelerated atherosclerosis.
34 ury that leads to neointimal hyperplasia and accelerated atherosclerosis.
35  in clinical and basic aspects of SLE-driven accelerated atherosclerosis.
36 tein levels, high blood monocyte counts, and accelerated atherosclerosis.
37  - with implications for lupus nephritis and accelerated atherosclerosis.
38 , these subjects had no clinical evidence of accelerated atherosclerosis.
39 ut detectable malignancy, is associated with accelerated atherosclerosis.
40 ter treatment and include cardiomyopathy and accelerated atherosclerosis.
41 s nephritis, as well as diminish the risk of accelerated atherosclerosis.
42 cular inflammation, intimal hyperplasia, and accelerated atherosclerosis.
43 e these are all independent risk factors for accelerated atherosclerosis.
44 eversed the myeloproliferative disorder, and accelerated atherosclerosis.
45 n of these populations with leukocytosis and accelerated atherosclerosis.
46 tes inflammation, TLR4 hypersensitivity, and accelerated atherosclerosis.
47 crease in whole-body cholesterol burden, and accelerated atherosclerosis.
48 riggered by type I IFNs, which might promote accelerated atherosclerosis.
49 with systemic lupus erythematosus (SLE) have accelerated atherosclerosis.
50 ts with systemic lupus erythematosus exhibit accelerated atherosclerosis, a chronic inflammatory dise
51                                In a model of accelerated atherosclerosis after arterial injury in apo
52 crucial role of monocyte trained immunity in accelerated atherosclerosis after MI, implying that SYK
53 pendently associated with the development of accelerated atherosclerosis among otherwise healthy men
54  Type 2 diabetes mellitus is associated with accelerated atherosclerosis and a high rate of arterial
55 re to test whether ICIs were associated with accelerated atherosclerosis and a higher risk of atheros
56 duced heart disease (RIHD), characterized by accelerated atherosclerosis and adverse tissue remodelin
57  Fas ligand and apolipoprotein E and exhibit accelerated atherosclerosis and aggravated lupus-like fe
58 or leukocyte-derived OPN in mediating Ang II-accelerated atherosclerosis and aneurysm formation.
59 elucidate mechanisms of uPA/uPAR/plasminogen-accelerated atherosclerosis and aneurysm formation.
60 s and implicate specific pathways in uPA/Plg-accelerated atherosclerosis and aneurysmal disease.
61                              To test whether accelerated atherosclerosis and aortic aneurysms were du
62                                          uPA-accelerated atherosclerosis and aortic dilation are larg
63 nsulin-resistant states are characterized by accelerated atherosclerosis and are associated with incr
64 by strain and by diet group, for features of accelerated atherosclerosis and autoimmunity.
65                                          The accelerated atherosclerosis and cardiovascular disease i
66 ersus hyperlipidemia as culprits in diabetes-accelerated atherosclerosis and cardiovascular disease,
67 eNOS) double knockout (DKO) mice demonstrate accelerated atherosclerosis and develop abdominal aortic
68 traordinarily high levels of cholesterol and accelerated atherosclerosis and die prematurely of myoca
69                               In particular, accelerated atherosclerosis and dilated cardiomyopathy,
70 ong youth were identified that predispose to accelerated atherosclerosis and early cardiovascular dis
71  II moderate-risk conditions associated with accelerated atherosclerosis and early CVD and (2) positi
72 s, Abca1(-/-)Abcg1(-/-) BM recipients showed accelerated atherosclerosis and extensive infiltration o
73 ough these changes may increase the risk for accelerated atherosclerosis and fatal myocardial infarct
74 proves, new recognized complications such as accelerated atherosclerosis and hypertension emerge as m
75 purpose was to investigate the mechanisms of accelerated atherosclerosis and identified vascular auto
76                                  Severe HHcy accelerated atherosclerosis and inflammatory monocyte/ma
77 dyslipoproteinemia, hyperglycemia, and hence accelerated atherosclerosis and microvascular disease in
78                                              Accelerated atherosclerosis and neutrophil extracellular
79 hronic cardiovascular complications, such as accelerated atherosclerosis and non-myocarditis-related
80                                 Although the accelerated atherosclerosis and premature aging of the c
81 tosus (SLE) is independently associated with accelerated atherosclerosis and premature arterial stiff
82 est that these children might be at risk for accelerated atherosclerosis and premature cardiovascular
83                                         HHcy accelerated atherosclerosis and promoted Ly6C(high) infl
84                  These results indicate that accelerated atherosclerosis and renal inflammation in SL
85 d chronic inflammation and its consequences (accelerated atherosclerosis and second cancer).
86  immunodeficiency virus (HIV; PLWH) may have accelerated atherosclerosis and shorter TL than the gene
87      Persons living with HIV (PLWH) may have accelerated atherosclerosis and shorter TL than the gene
88                              Diabetes causes accelerated atherosclerosis and subsequent cardiovascula
89 LNK function, and Lnk-deficient mice display accelerated atherosclerosis and thrombosis.
90 ncy virus (HIV) infection is associated with accelerated atherosclerosis and vasculopathy, although t
91 include pulmonary and systemic inflammation, accelerated atherosclerosis, and altered cardiac autonom
92  arrhythmias, venous thromboembolic disease, accelerated atherosclerosis, and atherosclerosis-related
93 ar dysfunction, myocarditis, cardiomyopathy, accelerated atherosclerosis, and coronary vasospasm.
94 osis and aneurysm formation, vasculitis, and accelerated atherosclerosis, and might be caused directl
95 ently develop tendon and tuberous xanthomas, accelerated atherosclerosis, and premature coronary arte
96 a and develop tendon and tuberous xanthomas, accelerated atherosclerosis, and premature coronary arte
97 , and develop tendon and tuberous xanthomas, accelerated atherosclerosis, and premature coronary arte
98 s and develop tendon and tuberous xanthomas, accelerated atherosclerosis, and premature coronary arte
99 lations between increased Plg activation and accelerated atherosclerosis are reported in several huma
100 cute thrombosis, neointimal hyperplasia, and accelerated atherosclerosis are the 3 mechanisms that le
101 ession of genes and proteins associated with accelerated atherosclerosis as compared to subjects with
102                                In a model of accelerated atherosclerosis associated with diabetes in
103                   Aged WT arteries developed accelerated atherosclerosis associated with enhanced TNF
104 pensive anti-inflammatory drug, prevents the accelerated atherosclerosis associated with TET2-mutant
105 herefore, be a potential method for treating accelerated atherosclerosis associated with transplantat
106            Antibody deficiency significantly accelerated atherosclerosis at both the aortic root and
107 the endothelial cells caused dysfunction and accelerated atherosclerosis because of loss of insulin-s
108     In conclusion, SNO-GNAI2 drives diabetes-accelerated atherosclerosis by coupling with CXCR5 and a
109 metformin abated the progression of diabetes-accelerated atherosclerosis by inhibiting mitochondrial
110 lial function and protected against diabetes-accelerated atherosclerosis by preventing GNAI2 S-nitros
111 , including hypertriglyceridemia and severe, accelerated atherosclerosis can be reproducibly induced
112  correlations linking diabetes mellitus with accelerated atherosclerosis, cardiomyopathy, and increas
113                             A unique form of accelerated atherosclerosis, CAV remains the leading cau
114 ivation in a model of angiotensin II (AngII)-accelerated atherosclerosis, characterized by increased
115 ed with Ldlr-/-Npc1-/- macrophages exhibited accelerated atherosclerosis despite lower serum choleste
116 onal haematopoiesis (CH) are associated with accelerated atherosclerosis development in mice and a hi
117                         Colchicine prevented accelerated atherosclerosis development in the mouse mod
118 N-acetylcysteine or ezetimibe, inhibited the accelerated atherosclerosis development.
119 littermate controls, uPA-transgenic mice had accelerated atherosclerosis, dilated aortic roots, occlu
120 ase (ESRD), DM is associated with blindness, accelerated atherosclerosis, dyslipidemia, cardio- and c
121    We found that CXCL16-/-/LDLR-/- mice have accelerated atherosclerosis, enhanced macrophage recruit
122 s in people with HIV and could contribute to accelerated atherosclerosis, especially of coronary lesi
123 ies examining mechanisms underlying diabetes-accelerated atherosclerosis have been limited by the lac
124 lammatory high-density lipoprotein (HDL) and accelerated atherosclerosis have been reported in patien
125 re to cholesterol as a strong determinant of accelerated atherosclerosis, highlighting the importance
126 ibute to the development of hypertension and accelerated atherosclerosis, highlighting vitamin D repl
127  this results in elevated levels of TRLs and accelerated atherosclerosis in a mouse model of T1DM.
128 c effect, arterial thrombosis was related to accelerated atherosclerosis in animal models; however, c
129             C. pneumoniae infection markedly accelerated atherosclerosis in ApoE-deficient mice that
130 nts associated with Porphyromonas gingivalis-accelerated atherosclerosis in apolipoprotein E knockout
131                                              Accelerated atherosclerosis in APS patients was found as
132 egnancy outcomes, thrombotic conditions, and accelerated atherosclerosis in autoimmune disease such a
133 herapeutic implications for dyslipidemia and accelerated atherosclerosis in diabetes and age-related
134 that promote an inflammatory environment and accelerated atherosclerosis in diabetes are poorly under
135 r receptor is involved in the development of accelerated atherosclerosis in diabetes, and identify th
136 aortic smooth muscle cells may contribute to accelerated atherosclerosis in diabetes.
137 id-selective deletion of ACSL1 also prevents accelerated atherosclerosis in diabetic mice without aff
138 les, macrophage foam cell formation, and the accelerated atherosclerosis in diabetic mice.
139 ed vascular hyperpermeability and suppressed accelerated atherosclerosis in diabetic rodents.
140  glucose conditions provides a mechanism for accelerated atherosclerosis in diabetics.
141             We found that an alternating HFD accelerated atherosclerosis in Ldlr(-/-) and Apoe(-/-) m
142 als with anti-inflammatory therapies and the accelerated atherosclerosis in many autoimmune diseases
143 of coronary heart disease in humans and with accelerated atherosclerosis in mice.
144 , abnormal glucose and lipid metabolism, and accelerated atherosclerosis in nontransplant patients.
145                                              Accelerated atherosclerosis in patients with diabetes is
146 t an important role for endogenous Ang II in accelerated atherosclerosis in renal dysfunction and off
147 present the first small animal model showing accelerated atherosclerosis in response to hyperglycemia
148 ation and autoimmunity in the development of accelerated atherosclerosis in SLE.
149 e management of traditional risk factors for accelerated atherosclerosis in systemic lupus erythemato
150                         These mice developed accelerated atherosclerosis in the aortic roots and arch
151  that chronic systemic inflammation promotes accelerated atherosclerosis in these patients, the mecha
152 IgG production, eventually ameliorating HHcy-accelerated atherosclerosis in vivo.
153 advantage of a new porcine model of diabetes-accelerated atherosclerosis, in which diabetic animals a
154 s of cardiovascular events was attributed to accelerated atherosclerosis, inadequate control of blood
155 ts with systemic lupus erythematosus develop accelerated atherosclerosis independent of traditional r
156 es have supported a causal role for Lp(a) in accelerated atherosclerosis, independent of other risk f
157 ndent neutrophil progenitor reprogramming in accelerated atherosclerosis induced by alternating HFD.
158 opportunities to correct dyslipoproteinemia, accelerated atherosclerosis, insulin resistance, and oth
159                             However, whether accelerated atherosclerosis is a cause or a consequence
160                                              Accelerated atherosclerosis is a hallmark of chronic kid
161                                              Accelerated atherosclerosis is a major cause of morbidit
162                                              Accelerated atherosclerosis is an important cause of mor
163                       This predisposition to accelerated atherosclerosis is genetically determined bu
164 ed cardiovascular and pulmonary involvement, accelerated atherosclerosis is of increasing concern in
165 activation and the development of nephritis, accelerated atherosclerosis is, instead, related to comp
166 system disorder causes failure to thrive and accelerated atherosclerosis leading to early death.
167 renal failure and nerve damage, and diabetes-accelerated atherosclerosis leads to increased risk of m
168 y lipoprotein cholesterol, hypertension, and accelerated atherosclerosis) may be responsible for the
169  Patients with APS are at increased risk for accelerated atherosclerosis, myocardial infarction, stro
170 ronic allogeneic stimulation participates to accelerated atherosclerosis observed after transplantati
171 ion of circulating monocytes, leading to the accelerated atherosclerosis observed in diabetics.
172 ic plaques could provide a mechanism for the accelerated atherosclerosis observed in patients with RA
173 atherosclerosis is subclinical, dramatically accelerated atherosclerosis occurs in some pediatric dis
174 ansion, which prevented the myelopoiesis and accelerated atherosclerosis of ApoE(-/-) mice transplant
175 ity by this mechanism may partly explain the accelerated atherosclerosis of diabetes.
176  over disease induction, we demonstrated the accelerated atherosclerosis of mature diabetic Akita mic
177 ent as acute vasospasm, acute thrombosis and accelerated atherosclerosis, of cancer therapies have ga
178               Arteriopathy, sometimes termed accelerated atherosclerosis, often impairs transplants.
179 clude that LDLR-/-; Tg(apoB+/+) mice exhibit accelerated atherosclerosis on a chow diet and thus prov
180 holesterol sterols, which is associated with accelerated atherosclerosis, premature coronary artery d
181 ic lesions and that macrophage-derived 25-HC accelerated atherosclerosis progression, promoting plaqu
182 atients with Kawasaki disease are at risk of accelerated atherosclerosis remains controversial, but t
183 gnized that SVG will develop progressive and accelerated atherosclerosis, resulting in a stenosis, an
184         Hypertension is not required for the accelerated atherosclerosis seen in apoE/eNOS DKO animal
185 lue in elucidating mechanisms underlying the accelerated atherosclerosis seen in human diabetic indiv
186 ls may be an independent risk factor for the accelerated atherosclerosis seen in primary hypothyroidi
187 n-receptor-null mice develop monocytosis and accelerated atherosclerosis, sleep-fragmented mice with
188 that inhibits the neointimal hyperplasia and accelerated atherosclerosis that are at the root of most
189 idence that renal transplant recipients have accelerated atherosclerosis that is manifest by increase
190 ts the neointimal hyperplasia and subsequent accelerated atherosclerosis that lead to human bypass-gr
191 sly shown, using a porcine model of diabetes-accelerated atherosclerosis, that diabetes leads to an i
192 ouse model of disturbed flow that results in accelerated atherosclerosis to identify novel mechanosen
193 1 and 2 diabetes, conditions associated with accelerated atherosclerosis, to inflammation.
194 se and dialysis vintage are characterized by accelerated atherosclerosis, volume overload, and progre
195                   Using mouse models of T1DM-accelerated atherosclerosis, we found that relative insu
196 at normal mice were deficient in a gene that accelerated atherosclerosis with diabetes.
197          Low-allele-burden (LAB) mice showed accelerated atherosclerosis with increased features of p
198  or 10 mg/kg) substantially attenuated AngII-accelerated atherosclerosis without altering blood press

 
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