ライフサイエンスコーパス: acetaldehyde

1 organic compounds, VOCs (e.g., methanol and acetaldehyde).

2 uld be reproduced by histamine and imidazole acetaldehyde.

3 1) surface is intrinsically selective toward acetaldehyde.

4 ological conditions, preventing a buildup of acetaldehyde.

5 c agent recruited another ALDH to metabolize acetaldehyde.

6 s of nitrogen oxides (NOx), formaldehyde, or acetaldehyde.

7 5, usually interpreted as solely coming from acetaldehyde.

8 s, particularly nonaromatic amino acids, and acetaldehyde.

9 choline to produce trimethylamine (TMA) and acetaldehyde.

10 2-knockdown human keratinocytes treated with acetaldehyde.

11 ing concentrations of free and sulfite-bound acetaldehyde.

12 tly decreased with the addition of exogenous acetaldehyde.

13 molecule that enables ALDH3A1 to metabolize acetaldehyde.

14 es is detailed using the example of indole-3-acetaldehyde.

15 o our great surprise) restricts diffusion of acetaldehyde.

16 or acetone but a net source for methanol and acetaldehyde.

17 ting it to ammonia, inorganic phosphate, and acetaldehyde.

18 ic effects of ethanol-an exogenous source of acetaldehyde.

19 cleavage of isethionate to form sulfite and acetaldehyde.

20 this product by the reaction of ethanol and acetaldehyde.

21 d us to describe reaction of epicatechin and acetaldehyde.

22 poised toward the production of ethanol from acetaldehyde.

23 y of the Julia-Kocienski olefination between acetaldehyde (1) and ethyl 1-phenyl-1H-tetrazol-5-yl sul

24 the wine content in 1-propanol, isobutanol, acetaldehyde, 1,1-diethoxiethane and ethyl lactate.

25 Intra-RVLM acetaldehyde (2 mug), the main metabolic product of etha

26 n of other highly reactive compounds such as acetaldehyde (20-320 Gg y(-1)), propene (50-170 Gg y(-1)

27 d VOCs such as methanol (5.39 pptv/ppbv CO), acetaldehyde (3.93 pptv/ppbv CO), acetone (3.59 pptv/ppb

28 sual AAAD enzyme products including indole-3-acetaldehyde, 4-hydroxyphenylacetaldehyde, and phenyleth

29 Monochloramine reacts with acetaldehyde, a common ozone and free chlorine disinfect

30 te the C-C coupling mechanism between CO and acetaldehyde, a reactive intermediate in both CO(2)RR an

31 ate, and rapid analysis of ethanol (Eth) and acetaldehyde (AA) in a wide variety of beverages and foo

32 ans carry an inactive ALDH2 gene and exhibit acetaldehyde accumulation after alcohol consumption.

33 as alpha-aryl-alpha,alpha-difluoroketones, -acetaldehydes, -acetates, and acetic acids, and difluoro

34 The concentrations of seven VOCs-acetaldehyde, acetone, acetic acid, hexanoic acid, hydro

35 The concentrations of acetaldehyde, acetone, acetic acid, hexanoic acid, hydro

36 s was applied for a combination of six VOCs (acetaldehyde, acetone, acetic acid, hexanoic acid, hydro

37 etermined over selected spectral regions for acetaldehyde, acetonitrile, ethanol, water, methanol, am

38 Formaldehyde, acetaldehyde, acrolein, and several other carbonyls were

39 2, CO, total particulate mass, formaldehyde, acetaldehyde, acrolein, and several polycyclic aromatic

40 Acetaldehyde acts as a bridging compound to form modifie

41 methionine and proline oxidation as well as acetaldehyde adduct formation on lysine or histidine res

42 nant OSE adducts termed MAA (malondialdehyde-acetaldehyde-adducts), which are found on apoptotic cell

43 On the fully oxidized surface, acetaldehyde adsorbs weakly through its carbonyl O inter

44 Asians and results in accumulation of toxic acetaldehyde after consumption of ethanol.

45 Hansenula sp. which quantitatively produces acetaldehyde after reaction for 120 min at 40 degrees C

46 Acetaldehyde-alcohol dehydrogenase (AdhE) enzymes are a

47 n of a Clostridium thermocellum bifunctional acetaldehyde/alcohol dehydrogenase.

48 Acetaldehyde, an alcohol catabolite detoxified by ALDH2,

49 Acetaldehyde, an I/R product (300 microM), elicited an 8

50 TAML/H2O2 slowly degrades metaldehyde to acetaldehyde and acetic acid.

51 densation between the reaction intermediates acetaldehyde and acetone.

52 Acetaldehyde and acrolein coeluted with other wine compo

53 ily average intake of benzene, formaldehyde, acetaldehyde and acrolein were 39 mug, 32 mug, 2.2 mg an

54 oduce toxic byproducts such as formaldehyde, acetaldehyde and acrolein.

55 f CO2 increased the release of formaldehyde, acetaldehyde and antimony (Sb).

56 lyze the decarboxylation of pyruvate to form acetaldehyde and CO(2) and are well known to play a key

57 is 2-aminothiazole formed from beta-mercapto-acetaldehyde and cyanamide in water at neutral pH.

58 ag phase of formation, with the exception of acetaldehyde and diacetyl formation.

59 At 20 degrees C, acetaldehyde and ethanol increased significantly with th

60 ts; i.e., it could either be hydrogenated to acetaldehyde and ethanol or couple with CO leading to th

61 lBEA proceeds via the reaction of coadsorbed acetaldehyde and ethanol to form crotyl alcohol and wate

62 rather, by concerted reaction of coadsorbed acetaldehyde and ethanol.

63 Exposure of the acini to acetaldehyde and ethyl oleate followed by CCK-8 stimulat

64 Acetaldehyde and ethyl oleate redirected CCK-8-stimulate

65 highly active for ethanol dehydrogenation to acetaldehyde and exhibited low activity for 1,3-butadien

66 eaction of CH(3)MgCl in tetrahydrofuran with acetaldehyde and fluorenone as prototypical reagents.

67 The presence of propionaldehyde, acetaldehyde and formaldehyde were correlated, corrobora

68 lyze the decarboxylation of pyruvate to form acetaldehyde and formate, respectively.

69 icting the alcoholic strength, the methanol, acetaldehyde and fusel alcohols content of grape-derived

70 ater (~0.001-0.03 nmol L(-1) h(-1)), whereas acetaldehyde and glyoxal were photochemically stable.

71 ver, the nature of the DNA damage induced by acetaldehyde and how this is repaired remains a key ques

72 ese findings suggest that alcohol, by way of acetaldehyde and its associated adducts, stimulates hepa

73 ence of ethanol, S. pneumoniae AdhE produced acetaldehyde and NADH, which subsequently led Rex (redox

74 Yeast metabolites such as acetaldehyde and pyruvate participate in the formation o

75 ikely as a result of increased production of acetaldehyde and reactive oxygen species and mitochondri

76 Formaldehyde, acetaldehyde and Sb migration increased with sunlight ex

77 Acetaldehyde and, to a lesser degree, ethyl oleate produ

78 opment of alcoholic pancreatitis, oxidative (acetaldehyde) and nonoxidative metabolites (ethyl palmit

79 1) surface is intrinsically selective toward acetaldehyde, and a strong inverse correlation between c

80 s OVOCs, including acetic acid, formic acid, acetaldehyde, and acetone were observed during photodegr

81 Formaldehyde, acetaldehyde, and butyraldehyde were the most significan

82 including acetate, reactive oxygen species, acetaldehyde, and epigenetic changes, that can induce in

83 ADH-mediated interconversions of acetyl-CoA, acetaldehyde, and ethanol but seemed to be poised toward

84 production was observed for glycolaldehyde, acetaldehyde, and formaldehyde only at elevated temperat

85 s well as increases in emissions of ethanol, acetaldehyde, and formaldehyde.

86 photochemical precursors of CO(2), ammonia, acetaldehyde, and H(2)O(2) and that reaction byproducts

87 se 2 (ALDH2) is a key enzyme that eliminates acetaldehyde, and impairment of ALDH2 increases the risk

88 TRPV1 by histamine, its metabolite imidazole acetaldehyde, and supernatants from biopsy specimens was

89 e of buffer and air to produce formaldehyde, acetaldehyde, and the aldehydes corresponding to the bre

90 enyl lactone of the southern portion with an acetaldehyde appendage on the cyclobutane of the norther

91 Loss and toxic effects of acetaldehyde are minimized by accelerating its consumpti

92 Formaldehyde and acetaldehyde are the dominant HAP concentration and canc

93 ly proline catalysis, heteroaryl-substituted acetaldehydes are fluorinated and then directly engaged

94 raphy for liquid phase analysis, we identify acetaldehyde as a minor product and key intermediate in

95 se-like protein (Ald2) to form 2-(methylthio)acetaldehyde as an intermediate.

96 ommonly produce ethanol from acetyl-CoA with acetaldehyde as intermediate and play a key role in anae

97 ncluding the sulfur-containing compounds and acetaldehyde, as well as lipid oxidation derived odorant

98 vested early facilitated the accumulation of acetaldehyde (associated with low polyphenols concentrat

99 BRCA2-null cells for the ethanol metabolite, acetaldehyde, associated with widespread chromosomal bre

100 The present study employs exogenous acetaldehyde at relatively low and high treatment concen

101 noxidative decarboxylation of pyruvate, with acetaldehyde being the common intermediate precursor of

102 (EC), PM(2.5) organic carbon, formaldehyde, acetaldehyde, benzene, toluene, ethylbenzene, and xylene

103 thought to retain the volatile intermediate acetaldehyde but allow diffusion of the much larger cofa

104 -Phe substitution increases turnover rate of acetaldehyde but decreases turnover rate of larger aldeh

105 BEA does not occur via aldol condensation of acetaldehyde but, rather, by concerted reaction of coads

106 ted chemical species, dominantly ammonia and acetaldehyde, but also two new species previously not re

107 differentiation, we show here that EtOH and acetaldehyde, but not acetate, increase differentiation-

108 Isethionate is then cleaved to sulfite and acetaldehyde by a previously uncharacterized glycyl radi

109 Furthermore, oxidation of indole-3-acetaldehyde by AOs is likely to represent one route to

110 converted glucose to ethanol via acetate and acetaldehyde, catalyzed by the host-encoded aldehyde fer

111 Ethanol and acetaldehyde caused a rapid and synergistic elevation of

112 which are identified as the enolate form of acetaldehyde (CH(2)CHO ).

113 y that interstellar aldehydes and enols like acetaldehyde (CH3CHO) and vinyl alcohol (C2H3OH) act as

114 (H2O2), ozone (O3), formaldehyde (HCHO), and acetaldehyde (CH3CHO).

115 relative contribution of different organs in acetaldehyde clearance through ALDH2 by using global- (A

116 r organs likely also contributes to systemic acetaldehyde clearance.

117 of these proteins was AdhE, a bi-functional acetaldehyde-CoA dehydrogenase and alcohol dehydrogenase

118 increases in oxygenates such as ethanol and acetaldehyde compared to E0 and E10 fuels.

119 The aims of this study were to measure acetaldehyde concentration in different beverages consum

120 Comparison of initial acetaldehyde concentration with that after enzymatic oxi

121 The model underestimation of acetaldehyde concentrations all year round implies a con

122 sso Sea, whereas no differences were seen in acetaldehyde concentrations among these stations.

123 ements in urban areas may have overestimated acetaldehyde concentrations at times due to this interfe

124 Underestimation of acetaldehyde concentrations is responsible for the bulk

125 Aldh2 (-/-) mice showed markedly higher acetaldehyde concentrations than wild-type (WT) mice aft

126 nexpectedly, to hypothermia, increased blood acetaldehyde concentrations, and enhanced lethality.

127 roacetamide under typical monochloramine and acetaldehyde concentrations.

128 ctors couples the three reactions and drives acetaldehyde consumption.

129 he pH, alcoholic strength, methanol content, acetaldehyde content, ethyl acetate content and higher a

130 tive for the aerobic oxidation of ethanol to acetaldehyde (conversion 100%; yield approximately 95%).

131 rds representing different carbonyl classes, acetaldehyde could be ionized only after labeling and MS

132 t does not involve DNA incisions-instead the acetaldehyde crosslink itself is broken.

133 Thus, acetaldehyde/crotonaldehyde mixtures and 2,4-alkadienals

134 h showed excellent catalytic activity in the acetaldehyde cyclotrimerization reaction.

135 Among these, the bifunctional alcohol/acetaldehyde dehydrogenase (ADH1), highly homologous to

136 ox 2 (a protein coded by CUX2), Glu504Lys of acetaldehyde dehydrogenase 2 (a protein encoded by ALDH2

137 ator LasR and redox-regulated activities for acetaldehyde dehydrogenase ExaC, arginine deiminase ArcA

138 Deletion of ALD6 coding for acetaldehyde dehydrogenase not only prevented acetate ac

139 s syringae strain PtoDC3000 uses an indole-3-acetaldehyde dehydrogenase to synthesize the phytohormon

140 Interestingly, eutE (which encodes acetaldehyde dehydrogenase) was required for UPEC strain

141 ter has two important functions: detoxifying acetaldehyde derived from dietary ethanol [11] and detox

142 Notably, levels of acetaldehyde-derived DNA damage represented by N(2)-ethy

143 preventive role of oesophageal ALDH2 against acetaldehyde-derived DNA damage.

144 were somewhat more efficient than ozone for acetaldehyde destruction, ozone was more efficient for a

145 The combined inactivation of acetaldehyde detoxification and the FA pathway induces m

146 coni anaemia pathway-mediated DNA repair and acetaldehyde detoxification.

147 he formation of the major flavour compounds (acetaldehyde, diacetyl, acetoin, and 2-butanone) followe

148 At 30 degrees C, acetaldehyde did not increase but ethanol increased rapi

149 etaldehyde direction), increased rapidly but acetaldehyde did not rise because of its oxidation to ac

150 ethyl phenylacetate and vanillin for MPX and acetaldehyde diethyl acetal, isobutyl acetate, ethyl iso

151 t 30 degrees C, the ADH activity (ethanol to acetaldehyde direction), increased rapidly but acetaldeh

152 While the coupling between CO and acetaldehyde does occur when the CORR is conducted with

153 Carbonyls such as formaldehyde and acetaldehyde dominated VOC emissions, making up approxim

154 phosphate and acetaldehyde or (2-methylthio)acetaldehyde during both aerobic and anaerobic growth.

155 Acetaldehyde emissions exhibited sharp increases with hi

156 y for the gas-phase SN2 reaction between the acetaldehyde enolate anion and methyl fluoride, for both

157 nthetic equivalent of the alpha-arylation of acetaldehyde enolate.

158 l, glyoxal, glycolaldehyde, ethylene glycol, acetaldehyde, ethane, and methanol).

159 tory quotient 1.5 (DCA-RQ 1.5) increased the acetaldehyde, ethanol and ethyl acetate concentration, r

160 he highest amounts of anaerobic metabolites (acetaldehyde, ethanol and ethyl acetate), regardless of

161 n for the study were ethyl acetate, acetone, acetaldehyde, ethanol, ethylene glycol, dimethylsilanedi

162 harboring putative genes for a bifunctional acetaldehyde/ethanol dehydrogenase (Aad), serine/threoni

163 cid and all of the major volatiles excepting acetaldehyde, ethyl acetate and acetoine, whereas the ap

164 ction of seed tannins, exhibited the highest acetaldehyde, ethyl acetate and C6-compounds levels, and

165 octanoate, butyrolactone, isoamyl alcohols, acetaldehyde, ethyl acetate, 2,3-butanediol, acetoin and

166 higher amounts, with increased citronellol, acetaldehyde, ethyl acetate, dicarboxylic acids esters,

167 nation of six toxic compounds (formaldehyde, acetaldehyde, ethyl carbamate, furan, furfural and acrol

168 The ethanol metabolites acetaldehyde, ethyl palmitate, and ethyl oleate perturb

169 The ethanol metabolites acetaldehyde, ethyl palmitate, and ethyl oleate reduced

170 Two central enzymes convert ethanolamine to acetaldehyde (EutBC) and then to acetyl-CoA (EutE).

171 a crucial role in stabilizing and activating acetaldehyde for coupling reactions.

172 ycles for nonmethane organic gases, ethanol, acetaldehyde, formaldehyde, acetone, nitrous oxide, nitr

173 nature; the same trend of increased ethanol, acetaldehyde, formaldehyde, and CH4 emissions and decrea

174 For example, E85 resulted in high acetaldehyde, formaldehyde, ethanol, ethene, and acetyle

175 , total hydrocarbons (THC), methane, ethene, acetaldehyde, formaldehyde, ethanol, N2O, and NH3 from a

176 nalysis was applied for the combined VOCs of acetaldehyde, formaldehyde, hydrogen sulphide, and methy

177 the fuel, the tailpipe emissions of ethanol, acetaldehyde, formaldehyde, methane, and ammonia increas

178 The GSH reduces browning and acetaldehyde formation for up to 12months.

179 ddition of EDTA to samples prevented de novo acetaldehyde formation from ethanol oxidation.

180 In a relevant photooxidative system acetaldehyde formation was significantly reduced after 6

181 ed the oxygen incorporation mechanism in the acetaldehyde formation.

182 Reaction products of (-)-epicatechin with acetaldehyde formed in model solution were selected for

183 a suite of ring-cleavage products, including acetaldehyde, formic acid, 6-, 7-, or 8-carbon oxoenals

184 We show that acetaldehyde forms at low steady-state concentrations, a

185 onal enzyme DmpFG channels its intermediate, acetaldehyde, from one active site to the next using a b

186 Acetaldehyde (GC-FID) and pyruvic acid (Y15 enzymatic au

187 Acetaldehyde generated by ADH in both liver and Schwann

188 rst, oxidative dehydrogenation of ethanol to acetaldehyde generates an aldehyde-containing stream act

189 ncluding nicotine, nicotyrine, formaldehyde, acetaldehyde, glycidol, acrolein, acetol, and diacetyl.

190 photochemical production and degradation of acetaldehyde, glyoxal, and methylglyoxal along with spat

191 .0-7.1, 1.4-4.8, and 0.25-2.8 nmol L(-1) for acetaldehyde, glyoxal, and methylglyoxal, respectively.

192 0.06-0.2, and 0.02-0.07 nmol L(-1) h(-1) for acetaldehyde, glyoxal, and methylglyoxal, respectively.

193 Furthermore, acetaldehyde had the greatest odor activity value of up

194 Although acetaldehyde has been implicated in the painful alcoholi

195 idium-mediated dehydrogenation of ethanol to acetaldehyde has led to the development of an ethanol-to

196 the following 7 VOCs, acetone, formaldehyde, acetaldehyde, hexanoic acid, hydrogen sulphide, hydrogen

197 often treated with oxygen in order to yield acetaldehyde, however this approach can lead to unintend

198 r billion (ppb) or 8 ppb gas-phase MG and/or acetaldehyde in an aerosol reaction chamber for up to 5

199 asymmetric direct crossed-aldol reaction of acetaldehyde in aqueous media using brine.

200 s and consumer guidance may be necessary for acetaldehyde in beverages.

201 Ethanol is metabolized into acetaldehyde in most tissues.

202 e ERK-dependent pressor effect of ethanol or acetaldehyde in normotensive rats.

203 CO attacks the carbonyl carbon of acetaldehyde in the coupling, and the carbon in CO ends

204 to 53% of the estimated total production of acetaldehyde in the surface mixed layer; a similar estim

205 nn cells revealed that channel activation by acetaldehyde in these cells initiates a NADPH oxidase-1-

206 To temporarily increase metabolism of acetaldehyde in vivo, we describe an approach in which a

207 At 10 degrees C, acetaldehyde increased rapidly and then declined, while

208 Ethanol and acetaldehyde induced a rapid and synergistic increase in

209 In vitro experiments revealed that acetaldehyde induced ALDH2 production in both mouse and

210 ofiles of fermentative compounds (especially acetaldehyde) induced by changes in the polyphenolic con

211 Linoleic acid, but not ethanol or acetaldehyde, induced ALOX15 expression in Hepa-1c1c7 ce

212 TA or Ca(2+)-free medium blocked ethanol and acetaldehyde-induced barrier dysfunction and tight junct

213 eine and cyclosporine A, blocked ethanol and acetaldehyde-induced barrier dysfunction and tight junct

214 -specific activator, Alda-1, Alda-89 reduced acetaldehyde-induced behavioral impairment by causing a

215 However, the repair of acetaldehyde-induced crosslinks results in increased mut

216 Cells are protected against acetaldehyde-induced damage by DNA crosslink repair, whi

217 Here we generate acetaldehyde-induced DNA interstrand crosslinks and dete

218 Moreover, P2X7R silencing prevented ATP- and acetaldehyde-induced renin release.

219 aV1.3 channels, by shRNA blocked ethanol and acetaldehyde-induced tight junction disruption and barri

220 n, whereas aldehyde dehydrogenase attenuated acetaldehyde-induced tight junction disruption.

221 Cu weaken the binding energy of the reduced acetaldehyde intermediate and inhibit its further reduct

222 They convert acetyl-CoA to ethanol via an acetaldehyde intermediate during ethanol fermentation in

223 , we showed that the oxygen in the as-formed acetaldehyde intermediate originates from the reactant C

224 rnal coenzyme B12 and injecting its product, acetaldehyde, into the lumen, where it is degraded by th

225 Acetaldehyde is a highly reactive, DNA-damaging metaboli

226 Acetaldehyde is a naturally-occurring carcinogenic compo

227 Acetaldehyde is an ethanol-derived definite carcinogen t

228 Impaired detoxification of acetaldehyde is common in the Asian population, and is a

229 w steady-state concentrations, and that free acetaldehyde is difficult to detect in alkaline solution

230 monstrating that the improved selectivity to acetaldehyde is due to the electronic effect from Ag inc

231 Our results suggest that acetaldehyde is likely supersaturated in surface seawate

232 Acetaldehyde is produced with a Faradaic efficiency of a

233 2-(Methylthio)acetaldehyde is reduced to 2-(methylthio)ethanol, which

234 me for detoxification the ethanol metabolite acetaldehyde, is recognized as a promising therapeutic t

235 e smaller organic compounds such as acetone, acetaldehyde, isoprene, or cysteamine can be detected in

236 ospheric trace gases, methylglyoxal (MG) and acetaldehyde, known to be surface-active, can enhance ae

237 using a rapid reduction in blood ethanol and acetaldehyde levels after acute ethanol intoxication in

238 Acetaldehyde levels in hepatocyte-specific Aldh2 knockou

239 adjustments were permitted, formaldehyde and acetaldehyde levels were higher respectively for 16/19 a

240 ucrose, and increasing fructose, glucose and acetaldehyde levels, which are potential contributors to

241 -) mice had higher levels of malondialdehyde-acetaldehyde (MAA) adduct and greater hepatic inflammati

242 ompounds (VOCs) such as ethylmercaptan (EM), acetaldehyde (MeCHO) and methyl ethyl ketone (MEK) among

243 function, but it dose-dependently increased acetaldehyde-mediated tight junction disruption and barr

244 m alcohol-treated mice had a greater rate of acetaldehyde metabolism and respiration when treated wit

245 the liver is the major organ responsible for acetaldehyde metabolism, a cumulative effect of ALDH2 fr

246 Total acetaldehyde, Mn, Cu/Fe, blue and red pigments and galli

247 ling of the carbonyl O and the acyl C of two acetaldehyde molecules.

248 Facile acetylation of dimethylamine by acetaldehyde occurs with high selectivity on oxygen-cove

249 ange (IQR) increases in prenatal exposure to acetaldehyde [odds ratio (OR) = 2.30; 95% CI: 1.44, 3.67

250 sion of CO and H2 into methane, ethanol, and acetaldehyde on the Rh (211) and (111) surfaces, chosen

251 igated the synergistic effect of ethanol and acetaldehyde on the tight junction integrity in Caco-2 c

252 ocked this synergistic effect of ethanol and acetaldehyde on tight junction.

253 occur when the CORR is conducted with added acetaldehyde, only a minor fraction (up to 36%) of 1-pro

254 into adenine, dihydroxyacetone phosphate and acetaldehyde or (2-methylthio)acetaldehyde during both a

255 nd ethyl acetate formed from condensation of acetaldehyde or acetic acid with excess ethanol.

256 creased supply of peroxyacetyl radicals from acetaldehyde oxidation, and the lower NO(x) emissions fo

257 isomer 2-phenyl-2-(propan-2-ylidenehydrazono)acetaldehyde oxime (7).

258 s conditions were found for formaldehyde and acetaldehyde (p < 0.01).

259 ts, including carbon monoxide, formaldehyde, acetaldehyde, peroxyacetyl nitrate, and ozone.

260 (ALDH2) is the major enzyme that metabolizes acetaldehyde produced from alcohol metabolism.

261 The acetaldehyde produced is oxidized to acetyl-CoA by a deh

262 n melon showed that this gene is involved in acetaldehyde, propanal and pentanal production, while it

263 us direct identification and quantitation of acetaldehyde, pyruvic acid, acetoin, methylglyoxal, and

264 unds mainly responsible for trapping SO2 are acetaldehyde, pyruvic acid, and 2-oxoglutaric acid.

265 8), alcoholic strength (r(2)=97.2; RPD=6.0), acetaldehyde (r(2)=98.2; RPD=7.5) and fusel alcohols (r(

266 Acetaldehyde Reactive Polyphenols (ARPs) may be key elem

267 The acetaldehyde reacts with 2,4-dinitrophenylhydrazine form

268 rylalkylamines and the formation of aromatic acetaldehydes, respectively.

269 s to produce aromatic monoamines or aromatic acetaldehydes, respectively.

270 by double reductive alkylation with melamine acetaldehyde, resulting in a tertiary amine side chain t

271 The present work found that acetaldehyde served as a relatively poor precursor for t

272 The alcohol metabolite, acetaldehyde, significantly decreased TER and reduced ju

273 trate that both PD20 and UM are sensitive to acetaldehyde, supporting a role for FANCD2 in repair of

274 These results demonstrate that ethanol and acetaldehyde synergistically disrupt tight junctions by

275 s thaliana and Petroselinum crispum aromatic acetaldehyde synthases primarily converts the enzymes ac

276 metabolism and respiration when treated with acetaldehyde than control.

277 smoke constituents (such as formaldehyde and acetaldehyde) that may be emitted at concentrations that

278 ring enzyme that catalyses the conversion of acetaldehyde to ethanol during fermentation.

279 D (acetyl-CoA to acetyl phosphate) and EutG (acetaldehyde to ethanol).

280 otein that is required for the conversion of acetaldehyde to ethanol.

281 catalyze the key aldol coupling reaction of acetaldehyde to exclusively yield the C4 coupling produc

282 e on the si face of the aldehyde carbonyl of acetaldehyde to form 4(S)-hydroxy-2-oxopentanoate.

283 ay be attributed to the adsorption of MG and acetaldehyde to the gas-aerosol interface, leading to su

284 n, epicatechin, caffeic acid, coumaric acid, acetaldehyde, total and reduced glutathione.

285 The high acetaldehyde treatment significantly increased polymeric

286 NOX4 promoter was induced in HSC by acetaldehyde treatment, and NOX4 has significantly incre

287 addition of (E)-2-butenyltrimethylsilane to acetaldehyde under electrophilic (BF3, H3O(+)) and nucle

288 ds of ethane and convert it into ethanol and acetaldehyde using nitrous oxide as the terminal oxidant

289 The Faradaic efficiency to acetaldehyde was further enhanced to 70% by increasing t

290 Daily exposure for formaldehyde and acetaldehyde was higher for 17/19 participants when usin

291 Acetaldehyde was present in the highest amount in RP-O (

292 cohols (1-propanol, 2-propanol, acetone, and acetaldehyde) was found in the majority of alcoholic bev

293 , alpha-ketoglutarate, pyruvate, acetoin and acetaldehyde were derivatised with 2,4-dinitrophenylhydr

294 Formaldehyde and acetaldehyde were the most abundant carbonyl compounds i

295 s involved in butyrate synthesis (ethanol or acetaldehyde) were significantly associated with clinica

296 However, formaldehyde and acetaldehyde, which are formed in the air photochemicall

297 the two-electron/two-proton hydrogenation of acetaldehyde, which reverses the EtOH photooxidation rea

298 that planar CuAg electrodes can reduce CO to acetaldehyde with over 50% Faradaic efficiency and over

299 o decline (e.g., benzene) or increase (e.g., acetaldehyde) with ethanol usage.

300 ning intermediate reaction products, such as acetaldehyde, with solvent water and not another mechani