ライフサイエンスコーパス: acetylcholine

1 allergic immunity, and the neurotransmitter acetylcholine.

2 generate current following stimulation with acetylcholine.

3 e (ChAT) and are a potential local source of acetylcholine.

4 prostaglandin D(2)) and the neurotransmitter acetylcholine.

5 tion of amyloid-beta (Abeta) and deficits of acetylcholine.

6 sitive to standard orthosteric agonists like acetylcholine.

7 cetylcholine receptor that is insensitive to acetylcholine.

8 nd contribute to increased mucosal levels of acetylcholine.

9 a subset of these are specialized to release acetylcholine.

10 nse to the endothelium-dependent vasodilator acetylcholine.

11 acological vasodilatation with adenosine and acetylcholine.

12 proximately 33% of neurons produce and store acetylcholine.

13 ial hyperresponsiveness was determined using acetylcholine.

14 in a manner that would block the binding of acetylcholine.

15 dy is the first to physiologically probe how acetylcholine, a pervasive neuromodulator in the brain,

16 ontractions evoked by potassium (KCl 60 mM), acetylcholine (ACh 10 muM) or serotonin (5-HT 10 muM).

17 cotinic acetylcholine receptors activated by acetylcholine (ACh) are brief.

18 nated interactions between dopamine (DA) and acetylcholine (ACh) at striatal synapses.

19 We directly demonstrate the secretion of acetylcholine (ACh) from BC upon stimulation with the Ta

20 ine receptors (nAChRs) by quantal release of acetylcholine (ACh) from motoneurons is sufficient to pr

21 tudied as a neurotransmitter, T cell-derived acetylcholine (ACh) has recently been reported to play a

22 layers through fast synaptic transmission of acetylcholine (ACh) in rodent medial prefrontal cortex (

23 ined attention task relies on the release of acetylcholine (ACh) into the cortex from cholinergic neu

24 The endogenous neurotransmitter acetylcholine (ACh) is known to affect the excitatory/in

25 , which coincides with elevation of striatal acetylcholine (ACh) levels.

26 the liver via preganglionic parasympathetic acetylcholine (ACh) neurons in the dorsal motor nucleus

27 Acetylcholine (ACh) plays an essential role in cortical

28 Non-neuronal acetylcholine (Ach) plays important roles in various asp

29 Previous work has shown that M(3) muscarinic acetylcholine (ACh) receptors (M3Rs) expressed by pancre

30 The ability to directly measure acetylcholine (ACh) release is an essential step toward

31 Potentiating the transmission of acetylcholine (ACh) via the cholinergic drug donepezil r

32 heart perfusions with normal or low glucose, acetylcholine (ACh), and/or mecamylamine.

33 The rapid hydrolysis of acetylcholine (ACh), one of the key neurotransmitters in

34 ting receptors for neurotransmitters such as acetylcholine (ACh).

35 ligands are actually modulated by endogenous acetylcholine (ACh).

36 aarterial infusions (3 progressive doses) of acetylcholine (ACh; EDD) and sodium nitroprusside (endot

37 ACR and microvascular function (responses to acetylcholine [ACH] and sodium nitroprusside) was tested

38 Acetylcholine acts as a neurotransmitter/neuromodulator

39 Acetylcholine administration in the early phase post-STE

40 d CD cells, RA/RAR activity was repressed by acetylcholine, albumin, aldosterone, angiotensin II, hig

41 ermeability of alpha7 receptors activated by acetylcholine alone, such efficacious PAMs may have cyto

42 This activity relies on acetylcholine, although the molecular mechanisms for thi

43 within nanocapsules that contain choline and acetylcholine analogues, is reported herein.

44 within nanocapsules that contain choline and acetylcholine analogues; such analogues facilitate the p

45 h sexes have elevated extracellular striatal acetylcholine and D2R-induced paradoxical ChI excitation

46 ations of hypoxanthine, proline, choline and acetylcholine and decreased concentrations of phenylalan

47 triatal activity is dynamically modulated by acetylcholine and dopamine, both of which are essential

48 terneurons and target-specific co-release of acetylcholine and GABA.

49 (VGLUT3cKO), we investigated the roles that acetylcholine and glutamate released by cholinergic inte

50 nary dilation response to intracoronary (IC) acetylcholine and IC nitroglycerin.

51 ex, we explore potential functional roles of acetylcholine and its effects on the processing of visua

52 y laser Doppler imaging and iontophoresis of acetylcholine and localized heating.

53 show that the orthosteric binding sites for acetylcholine and nicotine in the two major brain nicoti

54 With the larger acetylcholine and P2X(2) stimulations, there were no reg

55 G protein-coupled receptors that respond to acetylcholine and play important signaling roles in the

56 ters and noncompetitively inhibits nicotinic acetylcholine and serotonin (5-HT) type 3A receptors (5-

57 monstrate millisecond-resolved recordings of acetylcholine and voltage indicators, three-dimensional

58 ve in the presence of the endogenous ligand (acetylcholine), and thus theoretically less likely to ca

59 sequential spatial arrangement of glutamate, acetylcholine, and GABA receptors along the dendrite tha

60 ssed by forearm blood flow (FBF) response to acetylcholine, and nitric oxide synthase (NOS) activity

61 of this neuron type identified that it uses acetylcholine as a neurotransmitter and likely expresses

62 vity of isolated aortas to phenylephrine and acetylcholine, as well as marked acute BP sensitivity to

63 ocking of the methylnicotines to the Lymnaea acetylcholine binding protein crystal structure containi

64 r with their structural homologs such as the acetylcholine-binding protein and ligand-binding domain

65 extracellular domain homology model and two acetylcholine-binding protein homologs.

66 of 3 major pathways in the brain, including acetylcholine biosynthesis, being a key mediator of epig

67 we reported elevated extracellular striatal acetylcholine by in vivo microdialysis and paradoxical e

68 to stimulation by neurotransmitters such as acetylcholine, calcium responses in cells are mediated b

69 alogous to its influence on sensory systems, acetylcholine can act directly on cortical premotor circ

70 AMP/PKA dynamics in response to dopamine and acetylcholine co-stimulation in living flies.

71 may be attributed to differences in regional acetylcholine concentrations.

72 Adenosine and acetylcholine coronary flow reserve were calculated as v

73 studies suggested that increases in central acetylcholine could lead to depressed mood.

74 egulation of a constitutively active form of acetylcholine-dependent K(+) current (I(KACh)), called I

75 ed by donepezil and by l-DOPA, confirming an acetylcholine/dopamine deficit.

76 We now report that increased acetylcholine during liquid growth activates an EGL-30-R

77 of interaction between striatal dopamine and acetylcholine dynamics.

78 olar dilatory response to I/R in vivo and to acetylcholine ex vivo, and enhances leukocyte-endothelia

79 The structural group had lower acetylcholine FBF augmentation than the functional or re

80 ic neurons suggests a homosynaptic source of acetylcholine for the maturation of developing glutamate

81 n patterns of all metabotropic receptors for acetylcholine, GABA, and glutamate, completing a map of

82 of the three fast-acting neurotransmitters (acetylcholine, GABA, or glutamate) for communication.

83 oline (VAChT) and glutamate (VGLUT3) and use acetylcholine/glutamate cotransmission to regulate stria

84 The enteric neurotransmitter acetylcholine governs important intestinal epithelial se

85 c circuits, yet the developmental sources of acetylcholine have not been systematically explored.

86 endothelial-dependent vasomotor response to acetylcholine (ie, endothelial function) was also assess

87 ic oxide-dependent vasorelaxation induced by acetylcholine in aortic rings and reduced NADPH oxidase

88 t developmental origin that robustly release acetylcholine in layer 1.

89 f early discoveries which suggest a role for acetylcholine in the pathophysiology of mood disorders.

90 Acetylcholine in the striatum plays a pivotal role in su

91 the midbrain, a previously unknown source of acetylcholine in the striatum, is a major contributor to

92 under-the-curve carotid artery dilatation to acetylcholine in young: 345 +/- 16 AU vs. old control (O

93 Acetylcholine-induced currents were measured using whole

94 In addition, impaired acetylcholine-induced endothelium-dependent relaxation c

95 trast, EC PKD2 channels do not contribute to acetylcholine-induced vasodilation, suggesting stimulus-

96 The neurotransmitter acetylcholine influences how male finches perform courts

97 Ornithine, imidazole and atropine (acetylcholine inhibitor) inhibit Mtb growth in AMs.

98 ee energy of binding of the neurotransmitter acetylcholine into opening of its central pore.

99 terminate in the infragranular layers of V1, acetylcholine is delivered to more superficial layers th

100 The neuromodulator acetylcholine is released during locomotion to regulate

101 ing metabolites in Mtb-infected KCs and that acetylcholine is the top-scoring in Mtb-infected AMs.

102 Acetylcholine is well-understood to enhance cortical sen

103 d AChE cannot hydrolyze the neurotransmitter acetylcholine leading to its build-up at the cholinergic

104 Our study reveals that loss of acetylcholine leads to a dopamine imbalance in striatal

105 e effects of G(q/11)-coupled muscarinic M(1) acetylcholine (M(1)R) stimulation on M current and on ne

106 2B receptor and the G(q)-coupled muscarinic acetylcholine M1 receptor inhibited the activity of TRPM

107 fiable nature of birdsong to investigate how acetylcholine modulates the cortical (pallial) premotor

108 hat STDP is under neuromodulatory control by acetylcholine, monoamines, and other signaling molecules

109 ssant effects of scopolamine, a nonselective acetylcholine muscarinic receptor antagonist.

110 eir theoretical rationale, nicotinic alpha-7 acetylcholine (nalpha(7)) receptor agonists, have largel

111 We compared three stimulation methods: acetylcholine (natural stimulus), P2X(2) (chemogenetics)

112 uctures that release neuromodulators such as acetylcholine, often nonsynaptically.

113 guest complexes and into the selectivity for acetylcholine over choline.

114 to be efficient and selective receptors for acetylcholine over choline.

115 concentration of dorsal striatal choline (an acetylcholine precursor) changes during reversal learnin

116 ses of ligand, such as low concentrations of acetylcholine present for just tens of seconds or TNFalp

117 ry, resulting in a paucity of tuft cells and acetylcholine production.

118 target postsynaptic proteins, primarily the acetylcholine receptor (AChR) and inhibit signaling at t

119 cle mutation caused progressive denervation, acetylcholine receptor (AChR) cluster fragmentation, and

120 the level of rapsyn, a protein necessary for acetylcholine receptor (AChR) clustering; and expression

121 t MT disorganization and reduces the size of acetylcholine receptor (AChR) clusters.

122 Direct inhibition of acetylcholine receptor (AChR) function by autoantibodies

123 associated with transformation of the muscle acetylcholine receptor (AChR) into an inhibitory channel

124 The maintenance of a high density of the acetylcholine receptor (AChR) is the hallmark of the neu

125 une disease caused by antibodies against the acetylcholine receptor (AChR), muscle-specific kinase (M

126 he outer leaflet of the plasma membrane, and acetylcholine receptor (AChR), which is a well-character

127 tin, a two-pass transmembrane protein, as an acetylcholine receptor (AChR)-associated protein, and we

128 osteric modulators (PAM) of alpha7 nicotinic acetylcholine receptor (alpha7 nAChR) is described.

129 compound 28, a novel PAM of alpha7 nicotinic acetylcholine receptor (alpha7 nAChR).

130 a hypothesized role for the alpha7-nicotinic acetylcholine receptor (alpha7-nAChR) in the pathophysio

131 ed chronic inflammation via alpha7 nicotinic acetylcholine receptor (alpha7nAChR) signaling. However,

132 tor of the antiinflammatory alpha7 nicotinic acetylcholine receptor (alpha7nAChR/CHRNA7) that is also

133 The M2 muscarinic acetylcholine receptor (M2R) is a prototypical GPCR that

134 AuAbs to desmocollin 3 (Dsc3), M3 muscarinic acetylcholine receptor (M3AR), and secretory pathway Ca(

135 teric modulators (PAMs) of the M1 muscarinic acetylcholine receptor (mAChR) achieve exquisite selecti

136 ovel 1,4-dioxane analogues of the muscarinic acetylcholine receptor (mAChR) antagonist 2 was synthesi

137 The M(1) muscarinic acetylcholine receptor (mAChR) plays an important role i

138 M(1) muscarinic acetylcholine receptor (mAChR) positive allosteric modul

139 tic studies further indicate that muscarinic acetylcholine receptor (mAchR)/Galphao signaling produce

140 y labeled dibenzodiazepinone-type muscarinic acetylcholine receptor (MR) antagonists, including dimer

141 ind that SAT1 selectively controls nicotinic acetylcholine receptor (nAChR) biogenesis.

142 pha-conotoxin CIA, a potent muscle nicotinic acetylcholine receptor (nAChR) blocker with a significan

143 The alpha7 nicotinic acetylcholine receptor (nAChR) is a potential drug targe

144 The alpha6beta4 nicotinic acetylcholine receptor (nAChR) is enriched in dorsal roo

145 ctivity of the muscle-type Torpedo nicotinic acetylcholine receptor (nAChR) is highly sensitive to li

146 rovide an improved model to assess nicotinic acetylcholine receptor (nAChR) ligands for treating chro

147 Nicotinic acetylcholine receptor (nAChR) ligands that lack agonist

148 alpha7 (IC(50) 77.1 +/- 0.05 mum) nicotinic acetylcholine receptor (nAChR) subtypes, but the activit

149 interacting with alpha7-containing nicotinic acetylcholine receptor (nAChR) subtypes, leads to subnuc

150 e is known about the role of beta4 nicotinic acetylcholine receptor (nAChR) subunit encoded by this c

151 n CHRNA3, which encodes the alpha3 nicotinic acetylcholine receptor (nAChR) subunit gene, increases r

152 RNA3, the gene encoding the alpha3 nicotinic acetylcholine receptor (nAChR) subunit, increases vulner

153 for the alpha5, alpha3, and beta4 nicotinic acetylcholine receptor (nAChR) subunits, to nicotine add

154 fferent synapse is a alpha9alpha10 nicotinic acetylcholine receptor (nAChR).

155 appear to be related to the alpha7 nicotinic acetylcholine receptor (nAChR).

156 ternal risk alleles for the alpha5 nicotinic acetylcholine receptor (rs16969968).

157 ncing myelination by deleting the muscarinic acetylcholine receptor 1 in oligodendrocyte precursor ce

158 We and others have investigated muscarinic acetylcholine receptor 4 (M4) positive allosteric modula

159 cologic blockade of M-current and muscarinic acetylcholine receptor activation.

160 ed for alpha4beta2 and alpha3beta4 nicotinic acetylcholine receptor affinity and activity.

161 derivatives of the highly potent muscarinic acetylcholine receptor agonist iperoxo.

162 ropilin-1 antibodies as well as by nicotinic acetylcholine receptor antagonists, suggesting that thes

163 -up was unremarkable except for detection of acetylcholine receptor antibodies in the serum (21.30 nm

164 of 24 OMG patients who were seropositive for acetylcholine receptor antibody (AchR Ab) converted to G

165 a gravis of less than 5 years' duration, had acetylcholine receptor antibody titres of 1.00 nmol/L or

166 defects, including increased synaptic area, acetylcholine receptor area and density, and extent of p

167 iR-1010 or its binding site in the nicotinic acetylcholine receptor beta2 (nAcRbeta2) 3'UTR fail to g

168 ibrations in the presence and absence of the acetylcholine receptor blockers d-tubocurarine and alpha

169 steric modulators (PAMs) of alpha7 nicotinic acetylcholine receptor can increase channel activation b

170 gand-gated channels and how mutations in the acetylcholine receptor cause congenital myasthenic syndr

171 ears independent of the Agrin-LRP4-MuSK-DOK7 acetylcholine receptor clustering pathway.

172 ing organ of vertebrate species, a nicotinic acetylcholine receptor composed only of alpha9 subunits

173 Patients with acetylcholine receptor deficiency can also benefit from

174 Acetylcholine receptor deficiency is the most common for

175 ergic agonists for a cohort of patients with acetylcholine receptor deficiency on anticholinesterase

176 sely arranged myosin heavy chain and reduced acetylcholine receptor expression per immunocytochemical

177 The lipid dependence of the nicotinic acetylcholine receptor from the Torpedo electric organ h

178 ors (PAMs) of the M(1) subtype of muscarinic acetylcholine receptor have emerged as an exciting new a

179 selective activators of the M(4) muscarinic acetylcholine receptor have potential as a novel treatme

180 The alpha7 nicotinic acetylcholine receptor is a homopentameric ion channel f

181 vely apply our model to the human muscarinic acetylcholine receptor M1, finding four experimentally c

182 express somatostatin, VIP, or the muscarinic acetylcholine receptor M2.

183 cally prevented by the alpha4beta2 nicotinic acetylcholine receptor partial agonist and alpha7 recept

184 racemic preparation, is an alpha7 nicotinic acetylcholine receptor positive allosteric modulator (PA

185 his to be the case with the alpha7 nicotinic acetylcholine receptor positive allosteric modulator (PA

186 ith targeted disruption of single muscarinic acetylcholine receptor subtype genes (M(1) to M(5)) and

187 iotracer (11)C-LSN3172176 for the muscarinic acetylcholine receptor subtype M1.

188 egulatory pathway mediated via the nicotinic acetylcholine receptor subunit CHRNA2.

189 anges in alpha4, alpha3, and beta2 nicotinic acetylcholine receptor subunit mRNA levels in the nucleu

190 tor ligand to activate a modified muscarinic acetylcholine receptor that is insensitive to acetylchol

191 econdary Gi/o coupling, using the muscarinic acetylcholine receptor type 2 (M2R) as the primary Gi/o-

192 gand-gated ion channel the Torpedo nicotinic acetylcholine receptor(10,11), the large body of structu

193 The nicotinic acetylcholine receptor, a pentameric ligand-gated ion ch

194 n oligodendrocyte glycoprotein, aquaporin 4, acetylcholine receptor, and muscle-specific kinase) was

195 encodes the alpha3 subunit of the nicotinic acetylcholine receptor, in five affected individuals fro

196 ryoEM structure of sAB bound human nicotinic acetylcholine receptor, this work demonstrates that thes

197 We administered a novel alpha7 nicotinic acetylcholine receptor-negative allosteric modulator, BN

198 f geniculocortical inputs with M2 muscarinic acetylcholine receptor-rich patches in layer 1 (L1) sugg

199 treatment reduced disease severity, lowered acetylcholine receptor-specific Abs, and decreased CD19(

200 Correspondingly, model mice had fewer acetylcholine receptor-stained NMJs detected by fluoresc

201 lic analogue and the alpha9alpha10 nicotinic acetylcholine receptor.

202 gate the association of the alpha5 nicotinic acetylcholine receptor.

203 t activity of NS6740 at the alpha7 nicotinic acetylcholine receptor.

204 uscular junction proteins, in particular the acetylcholine receptor.

205 to target the human alpha9alpha10 nicotinic acetylcholine receptor.

206 Previously it was shown that acetylcholine receptors (AChR) are concentrated in the e

207 ween motoneurons and skeletal muscles, where acetylcholine receptors (AChRs) are concentrated to cont

208 umulation of the neurotransmitter receptors, acetylcholine receptors (AChRs), to the postsynaptic mem

209 ng in adult flies requires type A muscarinic acetylcholine receptors (mAChR-A), particularly in the g

210 mba snakes that primarily bind to muscarinic acetylcholine receptors (MAChRs) and modulate their func

211 Furthermore, inhibition of muscarinic acetylcholine receptors (mAChRs) reduced calcium activit

212 racterized two types (A and B) of muscarinic acetylcholine receptors (mAChRs), which were expressed i

213 Muscarinic acetylcholine receptors (MRs), comprising five subtypes

214 beta2* nicotinic acetylcholine receptors (nAChR) are necessary and suffic

215 vity of classical agonists for the nicotinic acetylcholine receptors (nAChR) has prompted us to ident

216 with very high affinity for brain nicotinic acetylcholine receptors (nAChR).

217 new group of inhibitors for muscle nicotinic acetylcholine receptors (nAChRs) and some neuronal nAChR

218 Nicotinic acetylcholine receptors (nAChRs) are crucial for communi

219 btx) and prevent its inhibition of nicotinic acetylcholine receptors (nAChRs) as a lead for the devel

220 Here, we show that activation of nicotinic acetylcholine receptors (nAChRs) by quantal release of a

221 Many peptide ligands of nicotinic acetylcholine receptors (nAChRs) contain a large number

222 gically distinguishing alpha3beta2 nicotinic acetylcholine receptors (nAChRs) from closely related su

223 The two major nicotinic acetylcholine receptors (nAChRs) in the brain are the al

224 Antagonism of nicotinic acetylcholine receptors (nAChRs) in the medial habenula

225 nduced upregulation of alpha4beta2 nicotinic acetylcholine receptors (nAChRs) is associated with chan

226 Nicotinic acetylcholine receptors (nAChRs) mediate and modulate sy

227 Nicotinic acetylcholine receptors (nAChRs) modulate synaptic activ

228 neurons acting through presynaptic nicotinic acetylcholine receptors (nAChRs) on PFn terminals.

229 ping drugs that act at alpha4beta2 nicotinic acetylcholine receptors (nAChRs) to treat alcohol use di

230 the highest density in alpha4beta2 nicotinic acetylcholine receptors (nAChRs) within the whole cortex

231 A d-peptide ligand of the nicotine acetylcholine receptors (nAChRs), termed (D)CDX, enables

232 ear synapse involves alpha9alpha10 nicotinic acetylcholine receptors (nAChRs), which assemble in hair

233 vation of alpha9alpha10-containing nicotinic acetylcholine receptors (nAChRs).

234 vity for neuronal over muscle-type nicotinic acetylcholine receptors (nAChRs).

235 g GABA(A) receptors (GABA(A)R) and nicotinic acetylcholine receptors (nAChRs).

236 ant-derived alkaloid that binds to nicotinic acetylcholine receptors (nAChRs).

237 The currents of alpha7 nicotinic acetylcholine receptors activated by acetylcholine (ACh)

238 These effects require alpha7 nicotinic acetylcholine receptors and are mediated through the nit

239 ers displayed increased distinct clusters of acetylcholine receptors and axon terminals exhibited num

240 Mediated by nicotinic acetylcholine receptors and choline transporters, such n

241 the epithalamus, densely expresses nicotinic acetylcholine receptors and is critical for nicotine int

242 Muscarinic acetylcholine receptors are G protein-coupled receptors

243 Muscarinic acetylcholine receptors are G-protein-coupled metabotrop

244 Thus, autoantibodies against M(3) acetylcholine receptors cause acute postganglionic choli

245 yramidal cells express specialized nicotinic acetylcholine receptors containing the alpha5 subunit en

246 Muscarinic acetylcholine receptors have been implicated as potentia

247 out the physiological function of muscarinic acetylcholine receptors in learning in adult flies.

248 the developmental switch from fetal to adult acetylcholine receptors in muscle (AChRs) and the functi

249 This strongly suggests a role of muscarinic acetylcholine receptors in the generalized sensitization

250 nd nicotine in the two major brain nicotinic acetylcholine receptors interact differently with the py

251 se results represent the first evidence that acetylcholine receptors negatively modulate muscle spind

252 ract may primarily target muscarinic subtype acetylcholine receptors that regulate memory processes.

253 losteric modulator (PAM) of alpha7 nicotinic acetylcholine receptors that, like 4BP-TQS and its activ

254 by CRF results in the activation muscarinic acetylcholine receptors type 5, which mediate potentiati

255 The corresponding postsynaptic acetylcholine receptors were stained using Alexa Fluro-5

256 vitro demonstrated that the mutant nicotinic acetylcholine receptors were unable to generate current

257 binding follows hyperbolic (M(4) muscarinic acetylcholine receptors) or nonhyperbolic relationships

258 Specific nicotinic acetylcholine receptors, alpha3, alpha7, beta2, beta4 we

259 site is the alpha6 subunit of the nicotinic acetylcholine receptors, and different mutations in this

260 e was due to autoimmunity against muscarinic acetylcholine receptors, blocking their activation.

261 Lypd6, an endogenous modulator of nicotinic acetylcholine receptors, enhances experience-dependent p

262 y activation of beta(2)-containing nicotinic acetylcholine receptors.

263 ction is mediated by TRPA1 and not nicotinic acetylcholine receptors.

264 ty and was attenuated by blocking muscarinic acetylcholine receptors.

265 where it regulates the function of nicotinic acetylcholine receptors.

266 of the peptide and involved alpha7 nicotinic acetylcholine receptors.

267 mine neuron axons by activation of nicotinic acetylcholine receptors.

268 uld be attributed to inhibition of nicotinic acetylcholine receptors.

269 Prevention of acetylcholine release at the neuromuscular junction caus

270 Blocking acetylcholine uptake and vesicular acetylcholine release by hemicholinium-3 also enhanced s

271 Inhibition of acetylcholine release from midbrain terminals in the str

272 Optical stimulation of acetylcholine release from NAc cholinergic interneurons

273 the effects observed following inhibition of acetylcholine release from striatal cholinergic interneu

274 activity temporally overlaps with US-evoked acetylcholine release from the basal forebrain.

275 tic and anti-kinetic effects of dopamine and acetylcholine release, respectively.

276 he critical period for binocular plasticity, acetylcholine released from the basal forebrain during p

277 onately, enabling caudal cells that are less acetylcholine sensitive to assume control.

278 d-effect transistors (gFETs) for building up Acetylcholine sensors.

279 ith CUD involved in neurotransmission (GABA, acetylcholine, serotonin, and dopamine) and drug addicti

280 Although the importance of acetylcholine signaling in normal bladder function has b

281 shift is a rate-dependent phenomenon whereby acetylcholine slows central pacemaker rate disproportion

282 ception threshold and vascular reactivity to acetylcholine, sodium nitroprusside, and heat.

283 od flow at rest were assessed in response to acetylcholine, sodium nitroprusside, local heating (42 d

284 ongly onto other inhibitory interneurons and acetylcholine sparsely onto layer 1 interneurons and oth

285 han one neurotransmitter, but found that the acetylcholine specific gene ChAT is transcribed in many

286 Acetylcholine stimulated the largest dopamine release (0

287 In airway SM, ACh (acetylcholine) stimulated the binding of S100A4 to the N

288 g to integrate calcium influx resulting from acetylcholine stimulation and G-protein activation resul

289 ly reported for the selective recognition of acetylcholine, they display improved fluorescent propert

290 nsory modalities stimulate motor neurons via acetylcholine, this mechanism enables broad sensory perc

291 oline acetyltransferase (ChAT) and vesicular acetylcholine transporter (VAChT) in specific neural cel

292 Vesicular acetylcholine transporter (VAChT) PET ligands are better

293 ere stained for cholinergic marker vesicular acetylcholine transporter (VAChT).

294 xamined expression of ChAT and the vesicular acetylcholine transporter in the embryonic and early pos

295 Blocking acetylcholine uptake and vesicular acetylcholine release

296 erneurons express vesicular transporters for acetylcholine (VAChT) and glutamate (VGLUT3) and use ace

297 ce, and vasomotor responses to intracoronary acetylcholine (vasospasm provocation) was performed in 1

298 se in cross-sectional area in response to IC acetylcholine was associated with higher hazard of angin

299 ivated by the nerve-derived neurotransmitter acetylcholine, we show that muscarinic receptors 1 and 3

300 eratinocyte-secreted factors, endothelin and acetylcholine, which acted via specific melanocyte recep