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1 oinflammatory actions are antagonized by PAF acetylhydrolase.
2 paraoxonase 1 and platelet activating factor acetylhydrolase.
3 ogical actions, and it is inactivated by PAF acetylhydrolase.
4 terase from Streptomyces scabies and a brain acetylhydrolase.
5  of intracellular platelet-activating factor acetylhydrolase.
6 hydrolyzed and inactivated by the enzyme PAF-acetylhydrolase.
7 vating factor (PAF) and is also known as PAF acetylhydrolase.
8  residues of PAF, as a candidate for aspirin acetylhydrolase.
9 t after pretreatment of the extract with PAF acetylhydrolase.
10 )), also known as platelet-activating factor acetylhydrolase.
11 nit of the enzyme platelet-activating factor acetylhydrolase.
12 te of degradation, which is catalyzed by PAF acetylhydrolase.
13 hydrolyzed and inactivated by the enzyme PAF acetylhydrolase.
14 tive cells now contained an abundance of PAF acetylhydrolase.
15 -treated animals contained no detectable PAF acetylhydrolase.
16  alpha2 subunit of platelet activator factor acetylhydrolase 1B (PAF-AH1B) for the binding of Lis1, w
17                                          PAF acetylhydrolase 1b (PAFAH1B) hydrolyzes PAF and is compo
18 enin (CTNNA2) and platelet-activating factor acetylhydrolase 1b regulatory subunit 1 (PAFAH1B1, also
19                   Platelet-activating factor acetylhydrolase 1B1 (LIS1), a critical mediator of neuro
20 on of PL-specific platelet-activating factor acetylhydrolase 2 (PAFAH2) markedly reduced EC dysfuncti
21 s of type I platelet-activating factor (PAF) acetylhydrolase, a phospholipase A(2) with selectivity f
22                                   Plasma PAF-acetylhydrolase activity increased 2-fold by 24 h follow
23 rates that plasma platelet-activating factor acetylhydrolase activity increases in men and women with
24                Miwa et al. reported that PAF acetylhydrolase activity is absent in 4% of the Japanese
25 ing that baseline platelet-activating factor acetylhydrolase activity levels are related to the sever
26 al antibody (MAb), 2B11, neutralized the PAF acetylhydrolase activity of SsE.
27    Measurement of platelet-activating factor acetylhydrolase activity represents a complementary appr
28 O-alkyl-2-acetyl-sn-glycero-3-phosphocholine acetylhydrolase activity was not observed.
29 oteins, increased platelet-activating factor acetylhydrolase activity, and secretory phospholipase A(
30  PAF levels and a concurrent decrease in PAF acetylhydrolase activity.
31  was observed for platelet activating factor acetylhydrolase alpha (Paf-AHalpha), and, in mammalian c
32                                   Plasma PAF acetylhydrolase, an enzyme that hydrolyzes PAF and struc
33  of a brain platelet-activating factor (PAF) acetylhydrolase, an enzyme that inactivates PAF by hydro
34                                          PAF acetylhydrolase, an oxidized phospholipid phospholipase
35      Enzymes associated with HDL such as PAF acetylhydrolase and paraoxonase can participate in the e
36 t determining whether SsE(M28) is also a PAF acetylhydrolase and participates in innate immune evasio
37  receptor 2/CD21, platelet-activating factor acetylhydrolase, and oxidized low-density lipoprotein re
38               The platelet-activating factor acetylhydrolases are enzymes that were initially charact
39 ot associate with LDL), the mutant mouse PAF acetylhydrolase associated with lipoproteins.
40            Plasma platelet-activating factor acetylhydrolase becomes progressively activated as ather
41                                          PAF acetylhydrolase catalyzes the degradation of PAF and rel
42 s of paraoxonase, platelet-activating factor acetylhydrolase, ceruloplasmin, and apoJ in HDL occur du
43 ith a recombinant platelet-activating factor acetylhydrolase completely abolishes the proapoptotic ef
44 ssociation defines the physical state of PAF acetylhydrolase, confers a long half-life, and is a majo
45                              We asked if PAF acetylhydrolase deficiency correlates with the incidence
46          We found that the prevalence of PAF acetylhydrolase deficiency is higher in Japanese asthmat
47                              Recombinant PAF-acetylhydrolase degrades PAF generated by NMDA-receptor
48 lasma samples from subjects deficient in PAF acetylhydrolase do not release F2-isoprostanes from este
49                         In human plasma, PAF acetylhydrolase (EC 3.1.1.47) circulates in a complex wi
50 m, using acetylcholinesterase (acetylcholine acetylhydrolase; EC 3.1.1.7; abbreviated herein AChE) as
51 ulated in oxLDL with a recombinant human PAF acetylhydrolase eliminated the inhibitory effects of oxL
52            We examined the regulation of PAF acetylhydrolase expression and demonstrated the administ
53 pffer cells were established in culture, PAF-acetylhydrolase expression became constitutively activat
54     The up-regulation of the plasma-type PAF acetylhydrolase expression constitutes an important mech
55 B 2170, inhibited by 50% the increase in PAF acetylhydrolase expression in response to LPS.
56               Alterations in plasma-type PAF-acetylhydrolase expression may constitute an important m
57 ssues examined, the greatest increase in PAF acetylhydrolase expression was observed in lung followed
58 uman plasma platelet-activating factor (PAF) acetylhydrolase functions by reducing PAF levels as a ge
59 ng the 5' genomic sequence of the plasma PAF acetylhydrolase gene and further characterized the promo
60 nor with an inactivating mutation in the PAF acetylhydrolase gene did not hydrolyze oxidized phosphol
61        Finally, deletion of the oxaloacetate acetylhydrolase gene in H915-1 eliminated oxalate format
62                     We conclude that the PAF acetylhydrolase gene is a modulating locus for the sever
63       A missense mutation (V279F) in the PAF acetylhydrolase gene results in the complete loss of act
64 ined the regulation of expression of the PAF acetylhydrolase gene.
65 d that both the intracellular and plasma PAF acetylhydrolases have high affinity for esterified F2-is
66 FAH1B2, but not its family member plasma PAF acetylhydrolase, hydrolyzed aspirin, and PAF competitive
67 man gene encoding platelet-activating factor acetylhydrolase IB subunit alpha (Pafah1b1), also called
68 atelet-activating factor metabolizing enzyme acetylhydrolase II blunted alpha-toxin-induced arachidon
69                        The intracellular PAF acetylhydrolase II, which shares homology to the plasma
70 f paraoxonase and platelet-activating factor acetylhydrolase in HDL decreased after infection and rea
71 ing did not reveal detectable amounts of PAF acetylhydrolase in PON1 preparations, although very low
72  LPS resulted in increased expression of PAF acetylhydrolase in several tissues.
73      In addition, this enzyme acted as a PAF-acetylhydrolase in the absence of lipid acceptor molecul
74  tumor cells with platelet-activating factor acetylhydrolase inhibited tumor growth at the site of im
75  or PAFAH1B3, and the competitive type I PAF acetylhydrolase inhibitor NaF reduced erythrocyte hydrol
76 y of plasma platelet-activating factor (PAF) acetylhydrolase is an autosomal recessive syndrome that
77  paraoxonase 1 or platelet-activating factor acetylhydrolase is responsible for the antioxidant activ
78    We conclude that intracellular type I PAF acetylhydrolase is the major aspirin hydrolase of human
79     We show that inherited deficiency of PAF acetylhydrolase is the result of a point mutation in exo
80                         We conclude that PAF acetylhydrolase is the sole phospholipase A(2) of HDL an
81 A2, also known as platelet-activating factor acetylhydrolase, is a new risk factor that may have the
82 in zeta-1 (FEZ1), platelet-activating factor acetylhydrolase, isoform Ib, PAFAH1B1 or lissencephaly 1
83 These data on the molecular basis of the PAF acetylhydrolase-LDL association provide a new level of u
84 otein (apo) B100 in the formation of the PAF acetylhydrolase-LDL complex, we tested the ability of PA
85 important mechanism for elevating plasma PAF-acetylhydrolase levels and an important component in min
86 olipids turnover in vivo and the role of PAF acetylhydrolase/Lp-PLA(2) in this process.
87 eparations, although very low amounts of PAF acetylhydrolase might still account for PON1 phospholipa
88 osure with the production of plasma-type PAF acetylhydrolase mRNA and protein expression specifically
89 ed the tissue distribution of the plasma PAF acetylhydrolase mRNA in humans.
90          Additionally, the expression of PAF acetylhydrolase mRNA increased in circulating leukocytes
91 cells were responsible for the increased PAF-acetylhydrolase mRNA levels.
92 tration had no effect on the increase in PAF-acetylhydrolase mRNA seen at 24 h.
93 onstrated very low levels of plasma-type PAF-acetylhydrolase mRNA transcripts in the livers of saline
94 ing LPS exposure, a 20-fold induction of PAF-acetylhydrolase mRNA was detected.
95            In Kupffer cells, plasma-type PAF-acetylhydrolase mRNA was induced by 12 h, peaked at 24 h
96  barely detectable levels of plasma-type PAF-acetylhydrolase mRNA, when Kupffer cells were establishe
97                                  Oxalacetate acetylhydrolase (OAH), a member of the phosphoenolpyruva
98 different biochemical pathways, oxaloacetate acetylhydrolase (OAH)-catalyzed hydrolytic cleavage of o
99 grates with platelet-activating factor (PAF) acetylhydrolase on KBr density gradients.
100 an also be blocked by co-incubation with PAF acetylhydrolase, or a PAF receptor antagonist.
101 ng whether plasma platelet-activating factor acetylhydrolase, or lipoprotein-associated phospholipase
102 he plasma form of platelet-activating factor acetylhydrolase (PAF AH) in tissue macrophages.
103 ymes paraoxonase, platelet activating factor acetylhydrolase (PAF), and glutathione peroxidase.
104 lved to target PAF by characterizing the PAF acetylhydrolase (PAF-AH) activity and substrate specific
105                   Platelet-activating factor acetylhydrolase (PAF-AH) activity was inversely associat
106                                          PAF acetylhydrolase (PAF-AH) degrades PAF and regulates its
107                   Platelet-activating factor acetylhydrolase (PAF-AH) is a phospholipase A2 that inac
108                                          PAF acetylhydrolase (PAF-AH) is a recently isolated naturall
109        The enzyme platelet-activating factor acetylhydrolase (PAF-AH) rapidly degrades PAF and oxidiz
110 ine hydrolysis or platelet-activating factor acetylhydrolase (PAF-AH) treatment.
111 sma form of platelet-activating factor (PAF) acetylhydrolase (PAF-AH), also known as lipoprotein-asso
112 tivity represents platelet-activating factor acetylhydrolase (PAF-AH), an esterase that co-purifies w
113 ontains secretory platelet-activating factor acetylhydrolase (PAF-AH), the enzyme capable of hydrolyz
114                                          PAF acetylhydrolase (PAF-AH), which is known to hydrolyze po
115 These lipid mediators are inactivated by PAF-acetylhydrolase (PAF-AH).
116 active metabolite lyso-PAF by the enzyme PAF acetylhydrolase (PAF-AH).
117 active lyso-PAF by cellular and secreted PAF-acetylhydrolase (PAF-AH).
118                   Platelet-activating factor acetylhydrolases (PAF-AHs) are a group of enzymes that h
119                   Platelet-activating factor acetylhydrolases (PAF-AHs) play an important role in the
120 ied out by specific intra- and extracellular acetylhydrolases (PAF-AHs), a subfamily of phospholipase
121  enzyme activity (platelet-activating factor acetylhydrolase [PAF-AH] and superoxide dismutase) were
122                  A single enzyme (plasma PAF acetylhydrolase [PAF-AH] or lipoprotein-associated phosp
123 PAF-CPT), and its main catabolic enzyme (PAF acetylhydrolase; PAF-AH), on U937 cells, in cell free an
124    We report that platelet-activating factor acetylhydrolase (PAFAH) Ib, comprised of two phospholipa
125 2) enzyme complex platelet-activating factor acetylhydrolase (PAFAH) Ib, which consists of alpha1, al
126  reduced turnover due to lower levels of PAF acetylhydrolase (PAFAH), the enzyme that catabolizes PAF
127 ng was restored by HDL or HDL-associated PAF acetylhydrolase (PAFAH), which mediates inactivation of
128 inished levels of platelet-activating factor acetylhydrolase (PAFAH).
129 alpha2 subunit of platelet-activating factor acetylhydrolase (Pafah1a2) at the chromosomal breakpoint
130 e beta subunit of platelet-activating factor acetylhydrolase (PAFAH1B1, also known as LIS1) in humans
131 acellular form of platelet-activating factor acetylhydrolase, PAFAH1b2.
132                   Platelet-activating factor acetylhydrolase (PLA2G7) is a potent pro- and anti-infla
133               Our results suggested that PAF acetylhydrolases play key roles in the hydrolysis of F2-
134 LA2), also called platelet-activating factor acetylhydrolase, plays in atherosclerosis.
135      Human plasma platelet activating factor acetylhydrolase (pPAF-AH) is a phospholipase A(2) that s
136 (PAF)-like lipids in L5 by a recombinant PAF acetylhydrolase prevented both FGF-2 downregulation and
137                         The abundance of PAF acetylhydrolase production in the liver lobule likely li
138        There are multiple regions in the PAF acetylhydrolase promoter that contain responsive element
139 A2, also known as platelet-activating factor acetylhydrolase, rapidly cleaves oxidized phosphatidylch
140 A crystal structure is also presented of PAF acetylhydrolase reacted with the organophosphate compoun
141 termination of PAF action by recombinant PAF-acetylhydrolase (rPAF-AH) were investigated.
142 ammatory activities of recombinant human PAF-acetylhydrolase (rPAF-AH), which converts PAF to biologi
143 rats were treated with human recombinant PAF acetylhydrolase (rPAF-AH), which efficiently inactivates
144 LA(2)) and plasma platelet-activating factor acetylhydrolase (rPAF-AH).
145 determine whether platelet-activating factor acetylhydrolase Sse and streptolysin S (SLS) have synerg
146         The platelet-activating factor (PAF) acetylhydrolase SsE produced by GAS is required for inva
147  for SpeB and the platelet-activating factor acetylhydrolase Sse, and a new type of variant that had
148 1 copurifies with platelet-activating factor acetylhydrolase subunits alpha 1 and alpha 2 [12], and w
149 ernalized, and overexpression of PLA2g7 (PAF acetylhydrolase) that specifically hydrolyzes such oxidi
150 phospholipase A2 (platelet-activating factor acetylhydrolase), the expression of which is regulated b
151 ctive metabolite, lysoPAF, by the enzyme PAF acetylhydrolase, the activity of which has shown to corr
152 h can be prevented by co-incubation with PAF acetylhydrolase, the enzyme that catabolizes PAF in the
153 se-LDL complex, we tested the ability of PAF acetylhydrolase to bind to lipoproteins containing trunc
154 1 purification by first depleting HDL of PAF acetylhydrolase to find PON1 purified in this way no lon
155                            Studies using PAF acetylhydrolase transgenic mice indicated that these ani
156 ins within the primary sequence of human PAF acetylhydrolase, tyrosine 205 and residues 115 and 116,
157     When residues 115 and 116 from human PAF acetylhydrolase were introduced into mouse PAF acetylhyd
158 PS) significantly inhibited synthesis of PAF acetylhydrolase, whereas other cytokines, including IFNa
159 etylhydrolase were introduced into mouse PAF acetylhydrolase (which normally does not associate with
160 h lower levels of platelet activating factor acetylhydrolase, which may contribute to the loss of ant
161  is effectively and tightly regulated by PAF acetylhydrolases, which convert PAF back to lysoPAF.
162 B plays a key role in the association of PAF acetylhydrolase with LDL.

 
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