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1 rders and the non-prescription medicines for acid reflux.
2        Three patients exclusively had weakly acid reflux.
3 PIs even in the absence of abnormal rates of acid reflux.
4 vation that arise from early-life esophageal acid reflux.
5 agus, size of hiatus hernia, and severity of acid reflux.
6  of nausea were accompanied by an episode of acid reflux.
7 ral diaphragm, esophageal common cavity, and acid reflux.
8 s were dysphagia (70% and 86%) and heartburn/acid reflux (55% and 49%), and common physician-reported
9               Esophagitis, whether caused by acid reflux, allergic responses, graft-versus-host disea
10 She has past medical history significant for acid reflux and Clostridium difficile colitis.
11                             Gastroesophageal acid reflux and human papillomavirus infection were rule
12 ad persistently pathological intraesophageal acid reflux and no change in villin or PCNA expression.
13                             It is clear that acid reflux as identified by a conventional pH electrode
14                             Gastroesophageal acid reflux at each of the pH sensors extending 5.5 cm p
15                                      Besides acid reflux, bile acid reflux may also play an important
16                Symptoms are more common with acid reflux but are also produced by nonacid reflux.
17  and acid taste were more commonly linked to acid reflux but were also produced by nonacid reflux.
18                                      Chronic acid reflux by itself is not likely to play a role in re
19  Ambulatory impedance testing underestimates acid reflux compared to esophageal acid exposure by disc
20 rospective study of patients with pathologic acid reflux confirmed by esophageal pH testing undergoin
21 ce, which lack squamous epithelia, may model acid-reflux damage.
22                                Percentage of acid reflux decreased (from 45% to 3%, P = 0.02) and non
23 tal hernia, and frequency of reflux, but not acid reflux, differed between CRs and ICRs.
24               The in vitro data suggest that acid reflux enhances the indirect alloresponse to proces
25 rolonged period of mucosal contact with each acid reflux episode, particularly in the recumbent posit
26  episodes (except the 30 mg dose) and weakly acid reflux episodes (all doses) significantly, relative
27  dose-dependently reduced the mean number of acid reflux episodes (except the 30 mg dose) and weakly
28 d more studies as abnormal than MII-detected acid reflux episodes [42 vs 34 % (p < 0.01)].
29 centage of time with a pH < 4, the number of acid reflux episodes and the percentage of proximal esop
30 ophagus, we found belt compression increased acid reflux following a meal.
31                     The second step involves acid reflux (HCl or HNO(3)) to remove the residual growt
32 IGN, SETTING, AND PARTICIPANTS: The Study of Acid Reflux in Children With Asthma, a randomized, maske
33 gm inhibition, esophageal common cavity, and acid reflux in normal subjects.
34                                              Acid reflux-induced oesophagitis and the multilayered ep
35 hageal reflux disease (GERD), which leads to acid reflux into the esophagus, is a common gastrointest
36  (EGJ) is a high-pressure zone that prevents acid reflux into the oesophagus.
37 d GERD; 4) in 50% of the patients with GERD, acid refluxed into the proximal esophagus.
38                                              Acid reflux is directly related to lower levels of LESP.
39                  The mechanisms whereby bile acid reflux may accelerate the progression from Barrett'
40                       The mechanisms whereby acid reflux may accelerate the progression from BE to EA
41                    Besides acid reflux, bile acid reflux may also play an important role in the progr
42                                              Acid reflux may be a contributing factor in the pathogen
43                                              Acid reflux may contribute to the progression from Barre
44                          While patients with acid reflux may have a few eosinophils, patients with EE
45  less sensitive than pH-testing in detecting acid reflux off therapy as a result of discounting durat
46 tinal issues (OR, 2.5; 95% CI, 1.8-3.2), and acid reflux (OR, 2.3; 95% CI, 1.5-3.6).
47                     It is possible that bile acid reflux present in patients with BE may increase rea
48 ur eyes to the trafficking of much more than acid reflux through the esophageal lumen.
49                                     Abnormal acid reflux was found to be the cause of intractable nau