ライフサイエンスコーパス: acid-base balance

1 ketone-body transport is needed to maintain acid-base balance.

2 duced ability of Oxgr1(-/-) mice to maintain acid-base balance.

3 roton flux in tissues that maintain systemic acid-base balance.

4 intravenously, have fewer adverse effects on acid-base balance.

5 no adverse changes in serum electrolytes or acid-base balance.

6 kaline-forming properties mediate the body's acid-base balance.

7 metabolic alkalosis in the regulation of the acid-base balance.

8 maintaining adequate fluid, electrolyte, and acid-base balance.

9 retion of acids and partially restore normal acid-base balance.

10 scades are intimately linked to the cellular acid-base balance.

11 limit gross perturbations of electrolyte and acid-base balance.

12 ssibly because of their beneficial effect on acid-base balance.

13 (+) to regulate ventilation, blood gases and acid-base balance.

14 tion and is involved in Na(+) absorption and acid-base balance.

15 urine output, osmolality, salt excretion, or acid-base balance.

16 nt composition, 4) micronutrient density, 5) acid-base balance, 6) sodium-potassium ratio, and 7) fib

17 tine, and that lack of the exchanger impairs acid-base balance and Na+-fluid volume homeostasis.

18 Hemodynamic resuscitation and maintenance of acid-base balance and tissue oxygen availability were pr

19 Volume resuscitation and maintenance of acid/base balance and tissue oxygen availability were pr

20 ammonia toxicity and aid in osmoregulation, acid-base balance, and excretion have been well document

21 With the addition of acetazolamide, however, acid-base balance as well as ENaC subunit abundance and

22 gas homeostasis and rapidly regulate tissue acid-base balance by driving lung ventilation and CO(2)

23 cleic acid synthesis, and the maintenance of acid-base balance by urinary buffering.

24 o 20 days) at 5050 m; involving: (1) resting acid-base balance (control); (2) following metabolic aci

25 Indeed, alterations in cerebrovascular acid-base balance directly affect cerebral blood flow (C

26 used to examine changes in gas exchange and acid-base balance during exposure to therapeutic hyperca

27 xchange efficiency, arterial oxygenation and acid-base balance during human locomotion.

28 xchange efficiency, arterial oxygenation and acid-base balance during prolonged locomotor exercise, 1

29 thus for optimizing arterial oxygenation and acid-base balance, during prolonged human locomotion.

30 cal homeostasis, including solute clearance, acid-base balance, electrolyte levels, and kidney hormon

31 The kidney maintains electrolyte, water, and acid-base balance, eliminates foreign and waste compound

32 triving to typify compensations for abnormal acid-base balance have reported their findings in terms

33 The topics examined included alteration in acid-base balance, hemodynamics, femoral venous flow, an

34 Atp6i-/- mice exhibit a normal acid-base balance in blood and urine.

35 Dietary contribution to acid-base balance in early life may influence subsequent

36 ism that controls breathing, blood gases and acid-base balance in response to changes in CO(2) /H(+)

37 importance of non-type A ICs in maintaining acid-base balance in response to metabolic challenges or

38 letion of K(ir)5.1 affects the regulation of acid-base balance in salt-sensitive hypertension.

39 precursors may have opposing effects on the acid-base balance in the body and therefore on bone loss

40 blood sample could be used to assess central acid-base balance in the early stage of arrest and cardi

41 The optimal approach to managing acid-base balance is less well defined for patients rece

42 Local cerebral changes of acid-base balance may interfere with neuronal communicat

43 at regulates cell volume, ion transport, and acid-base balance; mice knocked out for ClC-2 are blind

44 Normoxic cells remotely regulate the acid-base balance of cells at the hypoxic core of connex

45 source, calcium intake, weight loss, and the acid/base balance of the diet.

46 Importantly, these mutations also disrupt acid-base balance, particularly causing metabolic acidos

47 uate the severity of in vivo electrolyte and acid-base balance perturbations.

48 a) To determine the relationship of acid-base balance (pH, PCO2) of blood samples from the i

49 ppear healthy and are normal with respect to acid-base balance, plasma electrolyte concentrations, se

50 on long-term NaCl reabsorption, BP, K+, and acid-base balance, processes that all are disrupted in F

51 lated cells, which maintain sodium/water and acid/base balance, respectively, but the epigenetic cont

52 ntifying the three predominant influences on acid-base balance: sodium chloride, albumin, and unmeasu

53 egulate calcium levels and the regulation of acid base balance via regulation of carbon dioxide level

54 , 67 (26%) had a different interpretation of acid base balance when the Fencl method was used compare

55 Alterations in acid-base balance with progressive acclimatization to hi

56 nergy to maintain their calcified shells and acid/base balance with ocean acidification.