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1 ge of the arginine catabolic mobile element (ACME).
2 y island, arginine catabolic mobile element (ACME).
3 us of the arginine catabolic mobile element (ACME).
4 M) and automatic confocal microextensometry (ACME).
5 olates tested, regardless of the presence of ACME.
6 mecA, and arginine catabolic mobile element [ACME]).
9 300) differing in the presence or absence of ACME and a USA300 wild-type/ACME deletion mutant pair we
10 data underscore the functional modularity of ACME and its contribution to the success of USA300 CA-MR
11 the presence or absence of the SCCmec motif, ACME and the unique signature pattern for the prophage i
12 IVa, the arginine catabolic mobile element (ACME), and a specific mutation in capsular polysaccharid
13 ec types, arginine catabolic mobile element (ACME), and PVL-carrying prophage, PhiSa2 or PhiSa2-like
14 L) genes, arginine catabolic mobile element (ACME), and staphylococcal cassette chromosome mec typing
15 PVL), the arginine catabolic mobile element (ACME), and the staphylococcal cassette chromosome mec (S
16 A or VRSA isolates that carried PVL genes or ACME, and most strains (69.8%) were staphylococcal casse
17 tive for the presence of SCCmec type IV, the ACME, and the PVL toxin gene and matched the t008 or t12
18 ulent when evaluated prospectively, and PVL, ACME, and type IV SCCmec are associated with these absce
19 isolates, isolates positive for USA300, PVL, ACME, and type IV SCCmec were significantly associated w
24 ce or absence of ACME and a USA300 wild-type/ACME deletion mutant pair were analyzed for in vitro exp
25 ved) mutants, suggesting that the native and ACME-derived ADIs are compensatory in S. epidermidis.
26 ltaarcA2 (arginine catabolic mobile element [ACME]-derived) mutants, suggesting that the native and A
28 uring infection; however, the acquisition of ACME encoded speG allows USA-300 clones to circumvent po
30 n together, these findings indicate that the ACME-encoded CopB and CopL proteins are additional facto
31 -specific arginine catabolic mobile element (ACME) encoding a Spm/Spd N-acetyltransferase that is nec
32 se data are consistent with a model in which ACME enhances growth and survival of USA300, allowing fo
33 8, with the average causal mediation effect (ACME) explaining ~30% of the total effect (ACME P = 0.00
34 2) BDS is obtained from its acetone solvate, AcMe@G(2) BDS, by single-crystal-to-single-crystal (SC-S
36 c mutants containing deletions of SCCmec and ACME in a USA300 clinical isolate to determine the role
39 on of the arginine catabolic mobile element (ACME) in North American isolates and a novel copper and
40 dentified arginine catabolic mobile element (ACME) in the USA300 genome has been advocated as one pos
46 d that the infection burden significantly (P(ACME) < .05) mediated the association of maternal asthma
47 ants among ACME-positive isolates than among ACME-negative isolates, but there was no significant dif
49 is resulted in declining diversity up to the acme of extinction near the Daptocephalus-Lystrosaurus d
50 s around the world, and (3) co-occur with an acme of prasinophyte algae 'disaster taxa' also dominant
53 with the arginine catabolic mobile element (ACME), Panton-Valentine leukocidin (PVL), and other toxi
55 lpha-hemolysin in culture supernatants among ACME-positive isolates than among ACME-negative isolates
57 ect competitive fitness, whereas deletion of ACME significantly attenuated the pathogenicity or fitne
59 us aureus arginine-catabolic mobile element (ACME) that we hypothesized function in Cu homeostasis.
60 ed to as arrayed cellular microenvironments (ACMEs), that allows for a high-throughput examination of
63 E in S epidermidis, suggesting not only that ACME transfers into USA300 from S epidermidis, but also
67 nt an automated confocal micro-extensometer (ACME), which greatly expands the scope of existing metho
68 a major climatic cooling, which reached its acme with the Heinrich 4 event, forcing the human popula