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1 microbiome can be useful as a biotherapy for acne vulgaris.
2 uman skin and can promote the common disease acne vulgaris.
3 all relevant studies of isotretinoin use in acne vulgaris.
4 lighted as a dominant etiological factor for acne vulgaris.
5 be a well-tolerated, effective treatment for acne vulgaris.
6 tion is necessary for treating patients with acne vulgaris.
7 re centrally involved in the pathogenesis of acne vulgaris.
8 d be developed as an effective treatment for acne vulgaris.
9 ising therapeutic agent for the treatment of acne vulgaris.
10 l factor contributing to the pathogenesis of acne vulgaris.
11 s frequently prescribed for the treatment of acne vulgaris.
12 s association with the common teenage malady acne vulgaris.
13 ass antibiotic approved for the treatment of acne vulgaris.
14 ernative treatment for antibiotic therapy of acne vulgaris.
15 volved in the progression of inflammation in acne vulgaris.
16 m acnes is also associated with inflammatory acne vulgaris.
17 contribute to the common human skin disease acne vulgaris.
18 ore than 45 million people are affected with acne vulgaris.
19 nsins may be involved in the pathogenesis of acne vulgaris.
20 velopment of new modalities of treatment for acne vulgaris.
21 en hypothesized to contribute to the disease acne vulgaris.
22 n the overgrowth of C. acnes at the onset of acne vulgaris.
23 ntact, and seborrheic dermatitis), 0.29% for acne vulgaris, 0.19% for psoriasis, 0.19% for urticaria,
25 in and general medical conditions, including acne vulgaris (558 [51.0%]), acne conglobata (503 [45.9%
28 oneedles for cosmetic applications including acne vulgaris, acne scars, skin rejuvenation and hair gr
32 search was conducted using the search terms acne vulgaris and rosacea in the following databases: ME
34 plus red light is an effective treatment of acne vulgaris, associated with significant side-effects.
39 cently developed to treat moderate to severe acne vulgaris by directly delivering the combination of
41 of the factors that initiate and exacerbate acne vulgaris continues to increase, so does the diversi
44 vulinic acid was tested for the treatment of acne vulgaris, in an open-label prospective human study.
54 upregulation of beta-defensin expression in acne vulgaris lesions compared to controls suggests that
55 as in perilesional and intralesional skin of acne vulgaris lesions such as comedones, papules, and pu
58 analyzed resulting data: eczema, psoriasis, acne vulgaris, pruritus, alopecia areata, decubitus ulce
59 ed in the development of inflammation during acne vulgaris remain unclear, microbial agents might pla
62 a critical component in the pathogenesis of acne vulgaris, stimulating the production of various inf
63 tifunctional antimicrobial therapy agent for acne vulgaris treatment; FFAs may provide direct antibac