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1 ons; in the case of one patient two separate acrodermatitis chronica atrophicans lesions were culture
2 re isolated in culture from skin biopsies of acrodermatitis chronica atrophicans lesions; in the case
3 cells and macrophages in the skin lesions of acrodermatitis chronica atrophicans patients had very li
4 s with erythema migrans and 27 patients with acrodermatitis chronica atrophicans were analyzed for mR
5 onic skin manifestation of Lyme borreliosis, acrodermatitis chronica atrophicans, and infection by B.
6 restricted pattern of cytokine expression in acrodermatitis chronica atrophicans, including the lack
7                                 In contrast, acrodermatitis chronica atrophicans, the typical cutaneo
8 es collected from patients with well-defined acrodermatitis chronica atrophicans.
9 t, but not the exclusive, etiologic agent of acrodermatitis chronica atrophicans.
10 localized (palmoplantar pustulosis [PPP] and acrodermatitis continua of Hallopeau [ACH]) presentation
11  localized (palmoplantar pustular psoriasis, acrodermatitis continua of Hallopeau) and generalized va
12  psoriasis, palmoplantar pustular psoriasis, acrodermatitis continua of Hallopeau, and acute generali
13  Three patients with PPP also presented with acrodermatitis continua of Hallopeau.
14 h a clinical presentation similar to that of acrodermatitis enteropathica (AE) in humans.
15                                              Acrodermatitis enteropathica (AE) is a rare autosomal re
16                 The rare inherited condition acrodermatitis enteropathica (AE) results from a defect
17 s identified because of its association with acrodermatitis enteropathica (AE), a genetic disorder of
18 strated dramatically by the genetic disorder acrodermatitis enteropathica (AE).
19 fe-threatening autosomal recessive disorder, acrodermatitis enteropathica (AE).
20  homeostasis, a process that is defective in acrodermatitis enteropathica in humans.
21          Ectodomain cleavage is inhibited by acrodermatitis enteropathica mutations near a predicted
22 ecessive genetic disorder of zinc metabolism acrodermatitis enteropathica, but the physiological func
23 porter mutated in the human genetic disorder acrodermatitis enteropathica.
24  for the genetic disorder of zinc metabolism acrodermatitis enteropathica.
25 pellagra, necrolytic migratory erythema, and acrodermatitis enteropathica.
26 CD can result in the zinc-deficiency disease Acrodermatitis enteropathica.
27 f inherited nutritional deficiencies such as acrodermatitis enteropathica.
28 he rare, autosomal recessive genetic disease acrodermatitis enteropathica.
29 nt in humans is the zinc deficiency disease, acrodermatitis enteropathica.
30 s suggest that heterozygous mutations in the acrodermatitis gene Zip4 may be associated with a wider
31  deficiencies causing a periorificial and/or acrodermatitis: zinc deficiency, biotin deficiency, kwas