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5 s in this activity and include the conserved actin-related protein 2/3 (Arp2/3) complex as well as a
6 nitiated by nucleation/branching through the actin-related protein 2/3 (Arp2/3) complex downstream of
7 tt-Aldrich syndrome protein (N-WASP) and the actin-related protein 2/3 (Arp2/3) complex have emerged
8 ve amounts of F-actin, free barbed ends, and actin-related protein 2/3 (Arp2/3) complex in lamellipod
9 lular component of polarized growth, and the actin-related protein 2/3 (ARP2/3) complex is an importa
13 ion of N-WASP, which binds and activates the actin-related protein 2/3 (Arp2/3) complex, dramatically
14 mation by enhancing actin nucleation via the actin-related protein 2/3 (Arp2/3) complex, is activated
15 lescence of BCR microclusters depends on the actin-related protein 2/3 (Arp2/3) complex, which nuclea
16 of other actin-binding proteins, notably the actin-related protein 2/3 (Arp2/3) complex, which nuclea
17 hanges were accompanied by activation of the actin-related protein 2/3 (Arp2/3) complex, which stimul
18 rolin homologous protein 1 (WAVE1) regulates actin-related protein 2/3 (Arp2/3) complex-mediated acti
23 ing imposed by elevated viscosity induces an actin-related protein 2/3 (ARP2/3)-complex-dependent den
24 BAR domain-containing protein that inhibits actin-related protein 2/3 (Arp2/3)-dependent actin polym
27 nd that cancer cell motility mediated by the actin-related protein 2/3 complex (Arp2/3) is required f
28 ively regulates cortical localization of the actin-related protein 2/3 complex (Arp2/3), its regulato
29 , which induces actin polymerization through actin-related protein 2/3 complex (Arp2/3), to address t
30 tor of the dendritic spine cytoskeleton, the actin-related protein 2/3 complex (Arp2/3), we report he
31 ished cell junctions causes actin-driven and actin-related protein 2/3 complex (ARP2/3)-controlled la
33 WASp result in unregulated activation of the actin-related protein 2/3 complex and increased actin po
34 is an F-actin binding protein that activates actin-related protein 2/3 complex and is localized withi
35 kott-Aldrich syndrome protein (WASp) and the actin-related protein 2/3 complex generates forces at mu
36 aned animals, diazepam binding inhibitor and actin-related protein 2/3 complex mRNA levels were suppr
37 rited actin polymerization defects by either actin-related protein 2/3 complex subunit 1B or megakary
38 eins alpha-actinin-1 and alpha-actinin-4 and actin-related protein 2/3 complex subunit 3 (Arp3), are
39 ettsial invasion, including actin filaments, actin-related protein 2/3 complex, phosphatidylinositol-
43 drich syndrome protein (N-WASp) and the Arp (actin-related protein) 2/3 complex, and activation of th
44 cently, formins have been identified as Arp (actin-related protein) 2/3-independent nucleators of act
46 istent with this hypothesis, deletion of the actin-related protein-2/3 (ARP2/3) complex, which regula
47 on of dendritic actin filament arrays by the actin-related protein-2/3 complex is regulated, the in v
51 on macrophage deformability is dependent on actin-related proteins 2/3, indicating that stress hormo
54 t binds actin-filaments and colocalizes with actin-related protein 2 and 3 (Arp2/3) complex in the me
57 CDC42-dependent conformational change of the actin-related proteins 2 and 3 (ARP2/3) actin polymeriza
58 -specific protein 1 (HS1) is an F-actin- and actin-related proteins 2 and 3 (Arp2/3)-binding protein
59 l GTPase dynamin, the cytoskeletal regulator Actin-related protein 2, and the protein kinase Casein k