ライフサイエンスコーパス: activating transcription factor

1 osphate response element binding protein and activating transcription factor.

2 me stimulates monocytes through induction of activating transcription factor 1 (ATF-1).

3 sponse element binding protein 1 (CREB1) and activating transcription factor 1 (ATF1) are closely rel

4 rthermore, we found that cdk3 phosphorylates activating transcription factor 1 (ATF1) at serine 63 an

5 sponsive element binding protein (CREB), and activating transcription factor 1 (ATF1) pathway is invo

6 esponsive element binding protein (CREB) and activating transcription factor 1 (ATF1), transcription

7 e found that rs1015164 marks variation in an activating transcription factor 1 binding site that cont

8 ic protein EWS-ATF1 (Ewing's sarcoma protein-activating transcription factor 1), which contains the D

9 The 5' element binds CREB/activating transcription factor 1, and the 3' element is

10 ent of CREB transcription factors (CREB1 and activating transcription factor-1) in this functional re

11 an AMPK (AMP-activated protein kinase)/ATF1 (activating transcription factor-1) pathway that directs

12 nse element-binding protein (CREB) and ATF1 (activating transcription factor-1).

13 3) cAMP-responsive binding protein (P-CREB), activating transcription factor-1, and CREB-induced CRE

14 chemokine promoter association of CREB1 and activating transcription factor-1.

15 rect regulation of transcription mediated by activating transcription factor 2 (ATF-2) and (ii) H(2)O

16 sphorylation of JNK, PKCdelta, p38 MAPK, and activating transcription factor 2 (ATF-2) in RA ST fibro

17 t the attachment of CREB binding protein and activating transcription factor 2 (ATF-2) to the cAMP-re

18 Activating transcription factor 2 (ATF2) belongs to the

19 er showed an increase of Thr(P)-71-Thr(P)-69-activating transcription factor 2 (ATF2) binding in resp

20 Activating transcription factor 2 (ATF2) regulates trans

21 y suppressing expression via binding through activating transcription factor 2 (ATF2) to the cyclic a

22 in transcription factors including c-jun and activating transcription factor 2 (ATF2) upon activation

23 adipose tissue (BAT), p38 phosphorylates the activating transcription factor 2 (ATF2) which then tran

24 -response element binding protein (CREB) and activating transcription factor 2 (ATF2), function as a

25 we identify four transcription factors, JUN, activating transcription factor 2 (ATF2), myocyte-specif

26 y lipopolysaccharides, HDAC3 is recruited to activating transcription factor 2 (ATF2)-bound sites wit

27 am transcription factor substrates c-Jun and activating transcription factor 2 (ATF2).

28 phages, only BPPcysMPEG enhanced p38MAPK and activating transcription factor 2 activation.

29 of interferon regulatory factor 3 (IRF3) and activating transcription factor 2 in DCs.

30 Instead, low-dose LPS induces activating transcription factor 2 through Toll-interacti

31 Transcription factors ATF2 (activating transcription factor 2) and ATF7 (activating

32 c tumors, controlled the expression of ATF2 (activating transcription factor 2).

33 nstream effectors: phosphorylation of c-Jun, activating transcription factor 2, and E twenty-six-like

34 lation of the transcription factors, Jun and activating transcription factor 2, in addition to activa

35 ylation of cAMP response-element binding and activating transcription factor 2, which leads to transa

36 maintenance of chromatin protein 1, but not activating transcription factor 2.

37 The activating transcription factor-2 (ATF-2) mRNA encodes a

38 n transcription factors, including c-Jun and activating transcription factor-2 (ATF-2) upon activatio

39 ow that this is dependent on both ERK1/2 and activating transcription factor-2 (ATF-2).

40 erved only 3.1- and 1.9-fold differences for activating transcription factor-2 (ATF2) and signal tran

41 However, activating transcription factor-2 (ATF2) expression was

42 we show that under non-stressed conditions, activating transcription factor-2 (ATF2) in cooperation

43 The crucial functions of activating transcription factor-2 (ATF2) in the developm

44 IFN regulatory factor-1 and upregulation of activating transcription factor-2 and c-Jun, transcripti

45 d activation of transcription factors c-Jun, activating transcription factor-2 and, in addition, NF k

46 IFN-gamma recruited activating transcription factor-2 via p38 to the TNF-alp

47 of p38 mitogen-activated protein kinase and activating transcription factor-2, which were blocked by

48 eam of the JNK and p38 pathways as c-Jun and activating transcription factor-2.

49 ressor of cytokine signaling 3 (SOCS-3), and activating transcription factor 3 (ATF-3), which termina

50 te the induction of the stress/injury marker activating transcription factor 3 (ATF-3).

51 ic stress-induced UPR and cell death through activating transcription factor 3 (ATF3) and C/EBP homol

52 ound that microRNA-494 binds to the 3'UTR of activating transcription factor 3 (ATF3) and decreases i

53 C irradiation, immediate early genes such as activating transcription factor 3 (ATF3) are overexpress

54 ped identify a putative response element for activating transcription factor 3 (ATF3) at -258 to -250

55 Here we report that activating transcription factor 3 (ATF3) bound common mu

56 Activating transcription factor 3 (ATF3) can repress Nrf

57 8 mitogen activated protein kinase (p38MAPK)-activating transcription factor 3 (ATF3) dependent pathw

58 Expression of the activating transcription factor 3 (ATF3) gene is induced

59 Activating transcription factor 3 (ATF3) has been propos

60 Demyelination induced activating transcription factor 3 (ATF3) in DRG neurons.

61 A recent study showed the role of activating transcription factor 3 (ATF3) in SCC developm

62 Activating transcription factor 3 (ATF3) is a basic leuc

63 Activating transcription factor 3 (ATF3) is a common med

64 Activating transcription factor 3 (ATF3) is a common str

65 Activating transcription factor 3 (ATF3) is a key mediat

66 Activating transcription factor 3 (ATF3) is a member of

67 Activating transcription factor 3 (ATF3) is a negative r

68 Activating transcription factor 3 (ATF3) is a prime cand

69 Activating transcription factor 3 (ATF3) is an important

70 Activating transcription factor 3 (Atf3) is rapidly and

71 Activating transcription factor 3 (ATF3) promotes neuron

72 Activating transcription factor 3 (ATF3) responds to div

73 Here we report that activating transcription factor 3 (ATF3), a broad DNA da

74 Previous studies demonstrate that activating transcription factor 3 (ATF3), a common stres

75 Here we report that activating transcription factor 3 (ATF3), a common stres

76 In this report, we demonstrate that activating transcription factor 3 (ATF3), a hub of the c

77 issue of Blood, Boespflug et al report that activating transcription factor 3 (ATF3), a member of th

78 Expression of activating transcription factor 3 (ATF3), a nuclear calc

79 in E2 receptor 2 (EP(2)) and EP(4) to induce activating transcription factor 3 (ATF3), a repressive t

80 Here we report the discovery that activating transcription factor 3 (ATF3), a stress respo

81 We tested the hypothesis that activating transcription factor 3 (ATF3), a stress-induc

82 Activating transcription factor 3 (ATF3), an NMD target

83 with rapid induction of heat shock proteins, activating transcription factor 3 (ATF3), and CHOP.

84 or p75NTR, choline acetyltransferase (ChAT), activating transcription factor 3 (ATF3), and cleaved ca

85 n of dual specificity phosphatase 1 (DUSP1), activating transcription factor 3 (ATF3), and tribbles p

86 72 h after surgery, there is an increase in activating transcription factor 3 (ATF3), the neuropepti

87 expression of the transcriptional regulator activating transcription factor 3 (ATF3), which we show

88 Here, we report that activating transcription factor 3 (ATF3)-a broad stress

89 abeling for the stress and injury-associated activating transcription factor 3 (ATF3).

90 and increased the mRNA and protein levels of activating transcription factor 3 (ATF3).

91 or-activated receptor-gamma (PPARgamma), and activating transcription factor 3 (ATF3).

92 caspase 3 (apoptosis) and nuclear-localized activating transcription factor 3 (regeneration) in SIV

93 ssociated with nerve injury and cell stress, activating transcription factor 3 and growth-associated

94 d to activation of the transcription factors activating transcription factor 3 and protooncogene c-fo

95 Activating transcription factor 3 attenuates chemokine a

96 xpression, and nerve damage was evaluated by activating transcription factor 3 expression.

97 apoptosis in prostate cancer cells by ATF3 (activating transcription factor 3) expression.

98 f the transcription factors c-Jun and ATF-3 (activating transcription factor 3), known regulators of

99 ansection models, the upregulation of c-Jun, Activating transcription factor 3, Heat shock protein 27

100 neurons colocalized with nuclear-accumulated activating transcription factor 3, showing active regene

101 ugh interaction with transcription repressor activating transcription factor 3.

102 e induction of another transcription factor, activating transcription factor 3.

103 and response to danger signals triggered by activating transcription factor 3.

104 Importantly, transcripts for the activating transcription factor-3 (Atf3) and mitochondri

105 er transcripts, including neuropeptide Y and activating transcription factor-3, are upregulated norma

106 Several ER stress/UPR genes, including BiP, activating transcription factor 4 (ATF-4), ATF-6, and a

107 mammalian stress response pathway regulator activating transcription factor 4 (ATF-4).

108 trends toward increasing downstream signals, activating transcription factor 4 (ATF4) and ATF6 indica

109 h preferential translation of mRNAs, such as activating transcription factor 4 (ATF4) and C/EBP-homol

110 UPR was activated, as the expression of both activating transcription factor 4 (ATF4) and CHOP (DDIT3

111 he integrated stress response (ISR) effector activating transcription factor 4 (Atf4) and induction o

112 rotein synthesis but enhances translation of activating transcription factor 4 (ATF4) and is a crucia

113 vating two associated transcription factors: activating transcription factor 4 (ATF4) and nuclear fac

114 te, results from siRNA-mediated knockdown of activating transcription factor 4 (ATF4) and overexpress

115 e brain, as indicated by decreased levels of activating transcription factor 4 (ATF4) and phosphoryla

116 issue of Blood, Huang et al have identified activating transcription factor 4 (ATF4) as a novel regu

117 In silico analysis identified activating transcription factor 4 (ATF4) as a potential

118 ypes of mitochondrial stressors, we identify activating transcription factor 4 (ATF4) as the main reg

119 of FGF21 transcription was conferred by two activating transcription factor 4 (ATF4) binding sites i

120 godendrocytes during EAE are not mediated by activating transcription factor 4 (ATF4) but are accompa

121 sphorylation of eIF2alpha, which upregulates activating transcription factor 4 (ATF4) expression.

122 culum stress pathway, linked functionally to activating transcription factor 4 (ATF4) following treat

123 ed protein response (UPR) and an increase in activating transcription factor 4 (ATF4) has been previo

124 ve was to investigate the protective role of activating transcription factor 4 (ATF4) in controlling

125 We evaluate a potential role of activating transcription factor 4 (Atf4) in invertebrate

126 te skeletal muscle atrophy via expression of activating transcription factor 4 (ATF4) in skeletal mus

127 Here we reveal an essential role for activating transcription factor 4 (ATF4) in survival fol

128 d the signaling pathway of ER stress-induced activating transcription factor 4 (ATF4) in the regulati

129 Activating transcription factor 4 (ATF4) is a critical t

130 Activating transcription factor 4 (Atf4) is a leucine-zi

131 Activating transcription factor 4 (ATF4) is a mediator o

132 Activating transcription factor 4 (ATF4) is a transcript

133 Activating transcription factor 4 (ATF4) is a transcript

134 Activating transcription factor 4 (ATF4) is an osteoblas

135 ently that HRI also activates the eIF2alphaP-activating transcription factor 4 (ATF4) ISR in primary

136 Activating transcription factor 4 (ATF4) mRNA and protei

137 and selectively enhances the translation of activating transcription factor 4 (ATF4) mRNA to induce

138 and selectively enhances the translation of activating transcription factor 4 (ATF4) mRNA to induce

139 eIF4E), resulting in enhanced translation of activating transcription factor 4 (ATF4) mRNA.

140 In contrast, obese mice lacking hepatic activating transcription factor 4 (Atf4) showed an exagg

141 a heme-regulated inhibitor kinase/eIF2alpha/activating transcription factor 4 (ATF4) signaling modul

142 protein kinase RNA-like ER kinase (PERK) or activating transcription factor 4 (ATF4) significantly r

143 istration of AP20187 significantly increased activating transcription factor 4 (ATF4) translation and

144 zed enzymes are transcriptionally induced by activating transcription factor 4 (Atf4) via C/ebp-Atf-R

145 expression of the bZip transcription factor activating transcription factor 4 (ATF4) was induced by

146 eIF2alpha phosphorylation induces activating transcription factor 4 (ATF4), a basic leucin

147 Here we show that Activating Transcription Factor 4 (ATF4), a DISC1 bindin

148 ancer-derived cells stimulated expression of activating transcription factor 4 (ATF4), a master trans

149 Here, we have shown that activating transcription factor 4 (ATF4), a master trans

150 regulation of triglyceride metabolism by the activating transcription factor 4 (ATF4), a member of th

151 An important effector of the ISR is activating transcription factor 4 (ATF4), a transcriptio

152 P response element binding protein 2 (CREB2)/activating transcription factor 4 (ATF4), a transcriptio

153 ulated by arsenite, in a manner dependent on activating transcription factor 4 (ATF4), an important m

154 X-box-binding protein-1 mRNA, expression of activating transcription factor 4 (ATF4), and cleavage o

155 master regulator of amino acid homeostasis, activating transcription factor 4 (ATF4), is dysfunction

156 f the main transcriptional effectors of UPR, activating transcription factor 4 (ATF4), is essential f

157 iation, which changes DISC1 interaction with activating transcription factor 4 (ATF4), modifies gene

158 otic conditions, increased the production of activating transcription factor 4 (ATF4), the transcript

159 PERK and its downstream factor, activating transcription factor 4 (ATF4), were crucial f

160 Increased expression of activating transcription factor 4 (Atf4), which indicate

161 ISC1 interacts with the transcription factor Activating Transcription Factor 4 (ATF4), which is invol

162 sion and purine synthesis were stimulated by activating transcription factor 4 (ATF4), which was acti

163 s on betaFurKO islets revealed activation of activating transcription factor 4 (ATF4), which was medi

164 nts that the histone demethylase JMJD3 is an activating transcription factor 4 (ATF4)-dependent targe

165 34 expression by other stresses that involve activating transcription factor 4 (ATF4).

166 ion initiation factor 2alpha (eIF2alpha) and activating transcription factor 4 (ATF4).

167 ins that impact neuronal function, including activating transcription factor 4 (ATF4).

168 nt-binding protein 1 (CREB) and another with activating transcription factor 4 (ATF4).

169 anslation of selected mRNAs such as that for activating transcription factor 4 (ATF4).

170 PERK- and ISR-dependent upregulation of the Activating Transcription Factor 4 (ATF4).

171 bility, in part through direct regulation of activating transcription factor 4 (Atf4).

172 e in aged muscle are positively regulated by activating transcription factor 4 (ATF4).

173 selected stress-related transcripts, such as activating transcription factor 4 (ATF4).

174 on general control nonderepressible 2 kinase-activating transcription factor 4 (GCN2-ATF4) pathway ac

175 d early activation of the PKR-like ER kinase/activating transcription factor 4 (PERK/ATF4) ER stress

176 ed to persistent eif2-alpha phosphorylation (activating transcription factor 4 [ATF-4], C/EBP homolog

177 down of inositol-requiring enzyme 1alpha and activating transcription factor 4 abrogates autophagosom

178 initiation factor 2alpha phosphorylation and activating transcription factor 4 and C/EBP homologous p

179 factor 2alpha (eiF2alpha) and expression of activating transcription factor 4 and tribbles 3 were el

180 eens and by implication of the ISR-inducible activating transcription factor 4 in the process.

181 osphorylated eIF2alpha (p-eIF2alpha) but not activating transcription factor 4 or C/EBP homologous pr

182 ugh arsenite treatment and is independent of activating transcription factor 4 signaling and protein

183 Since ATF4 (activating transcription factor 4) and CCAAT/enhancer-bi

184 ulator homolog 1) was regulated by the ATF4 (activating transcription factor 4) arm of the unfolded p

185 f genes enriched for components of the ATF4 (activating transcription factor 4) arm of the unfolded p

186 ATF4 (activating transcription factor 4) is an osteoblast-enri

187 The transcription factor ATF4 (activating transcription factor 4) is induced by multipl

188 response genes (C/EBP homologous protein and activating transcription factor 4), accumulation of cera

189 on by amino acid deprivation did not require activating transcription factor 4, a critical component

190 alpha, inositol requiring kinase 1alpha, and activating transcription factor 4, all of which are feat

191 such as the pseudokinase tribbles homolog 3, activating transcription factor 4, and transcription fac

192 er binding of the transcriptional activators activating transcription factor 4, C/EBPalpha, Runx1, an

193 luding glucose-regulated protein-78 (GRP78), activating transcription factor 4, growth arrest and DNA

194 rylated eukaryotic initiation factor-2alpha, activating transcription factor 4, inositol requiring ki

195 iation factor 2 (eIF2alpha) phosphorylation, activating transcription factor-4 (ATF4) induction, and

196 eir different redox states and expression of Activating Transcription Factor-4 (ATF4).

197 e proteasome inhibitor bortezomib, levels of activating transcription factor-4 increase dramatically

198 ryotic translation initiation factor 2-alpha/activating transcription factor-4 signaling pathway.

199 UPR induction, particularly up-regulation of activating transcription factor-4, CHOP (C/EBP homologou

200 en and nitrogen species and regulated by the activating-transcription factor-4.

201 Nucleophosmin (NPM1/B23) and the activating transcription factor 5 (ATF5) are both known

202 The expression of activating transcription factor 5 (ATF5) correlates nega

203 th the heat shock protein 70 (HSP70) and the activating transcription factor 5 (ATF5) have been shown

204 Activating transcription factor 5 (ATF5) is a member of

205 Activating transcription factor 5 (ATF5) is a stress-res

206 h mouse and human BCR-ABL-transformed cells, activating transcription factor 5 (ATF5), a prosurvival

207 As a result, seven transcription factors-activating transcription factor 5 (ATF5), early growth r

208 phagocytic cells revealed activation of the activating transcription factor 5 (ATF5)-mediated stress

209 r eif2alpha causing selective translation of activating transcription factor 5 (ATF5).

210 glia, require the transcription factor ATF5 (activating transcription factor 5) for survival.

211 Activating transcription factor-5 (ATF5) is highly expre

212 Activating transcription factor 6 (ATF6) and protein kin

213 vidence highlights a protective role for the activating transcription factor 6 (ATF6) arm of the UPR

214 nositol-requiring protein-1 (IRE1) alpha and activating transcription factor 6 (ATF6) arms of the UPR

215 We recently identified activating transcription factor 6 (ATF6) as a genetic ca

216 Activation of the activating transcription factor 6 (ATF6) branch of the U

217 ections were stained with antibodies against activating transcription factor 6 (ATF6) or activated ca

218 th ER resident kinase (PERK) activation, and activating transcription factor 6 (ATF6) splicing.

219 tases (OAS) genes, and selectively activated activating transcription factor 6 (ATF6), an unfolded pr

220 ol-requiring 1A/X box binding protein 1, and activating transcription factor 6 (ATF6), and each of th

221 binding protein 1 (XBP1) mRNA, activation of activating transcription factor 6 (ATF6), and protein ki

222 ates that DKK3 added as a cytokine activates activating transcription factor 6 (ATF6), leading to the

223 d accompanied by a reduction in the level of activating transcription factor 6 (ATF6), one of the tra

224 a cells induced the nuclear translocation of activating transcription factor 6 (ATF6), which is part

225 in 78 (GRP 78), and nuclear translocation of activating transcription factor 6 (ATF6).

226 sion of several SKI-1 target genes including activating transcription factor 6 (ATF6).

227 Another UPR pathway activates the activating transcription factor 6 (ATF6).

228 s protein kinase R-like ER kinase (PERK) and activating transcription factor 6 (ATF6).

229 d the unfolded protein response (UPR) sensor activating transcription factor 6 (ATF6).

230 ress indexes in vivo and in vitro, decreased activating transcription factor 6 activation (cleavage,

231 nd X-box-binding protein 1 (XBP1) but not by activating transcription factor 6 alpha (ATF6alpha).

232 ty control genes through an association with activating transcription factor 6 alpha (ATF6alpha, also

233 , activating the transcription factor, ATF6 (activating transcription factor 6 alpha), which induces

234 ), which involves activation of the Ire1 and activating transcription factor 6 branches, but not the

235 tions of spliced X-box binding protein 1 and activating transcription factor 6 levels in affected liv

236 ide, a serine protease inhibitor that blocks activating transcription factor 6 processing.

237 ion primary response gene 88), but not ATF6 (activating transcription factor 6) or XBP1 (X-box-bindin

238 lucose-regulated protein was down-regulated, activating transcription factor 6, and eIF2alpha were ac

239 Direct miR-199a-5p targeting of activating transcription factor 6, p50, and p65 by miR-1

240 GRP78, activating transcription factor 6, p50, and p65 were inc

241 onary circulation involves the activation of activating transcription factor 6, which, via induction

242 found to play a critical role in driving the activating transcription factor 6-mediated arm of this r

243 or of caspases, HtrA serine peptidase 2, and activating transcription factor 6.

244 1alpha, protein kinase R-like ER kinase, and activating transcription factor 6.

245 ER signal kinase 1alpha but did not activate activating transcription factor 6.

246 BP1) is selectively impaired in DN, inducing activating transcription factor-6 (ATF6) and C/EBP homol

247 augmented tunicamycin-induced activation of activating transcription factor-6 (ATF6) and induction o

248 s, protein kinase RNA-like ER kinase (PERK), activating transcription factor-6 (ATF6), inositol requi

249 ER kinase (PERK; official name EIF2AK3), and activating transcription factor-6, control the UPR.

250 nase (PERK), inositol-requiring enzyme-1 and activating transcription factor-6.

251 ozygous mutations in the ATF6 gene (encoding activating transcription factor 6A), a key regulator of

252 ion of the unfolded protein response through activating transcription factor 6alpha (ATF6alpha) in ca

253 Thbs bind the ER lumenal domain of activating transcription factor 6alpha (Atf6alpha) to pr

254 tion factor involved in ER stress signaling, activating transcription factor 6alpha (ATF6alpha), thro

255 lasmic reticulum (ER) stress pathway through activating transcription factor 6alpha (Atf6alpha).

256 tol-requiring enzyme-1alpha (IRE1alpha), and activating transcription factor-6alpha (ATF6alpha)-were

257 tol-requiring protein-1alpha (IRE1alpha) and activating transcription factor-6alphaalpha (ATF6alpha).

258 Here we identify the activating transcription factor 7 interacting protein (A

259 activating transcription factor 2) and ATF7 (activating transcription factor 7) are highly homologous

260 miRNA activity, possibly via the downstream Activating Transcription Factor-7 (ATF-7).

261 the downstream 36-bp region containing CREB/activating transcription factor and kappaB6 sites.

262 pathways and the transcription factors ATF (activating transcription factor) and CREB (cyclic AMP re

263 PYD, pyrin N-terminal homology domain; ATF, activating transcription factor; and PTEN, phosphatase a

264 luding the bZip transcription factor atfs-1 (activating transcription factor associated with stress).

265 , we examined the mechanism by which ATFS-1 (activating transcription factor associated with stress-1

266 eport that anisomycin, a potent activator of activating transcription factor (ATF) 2, and c-Jun-NH(2)

267 meric complexes with the AP-1 family members activating transcription factor (ATF) 2, ATF3, and ATF7.

268 tiation factor 2, and increased synthesis of activating transcription factor (ATF) 4 by a translation

269 identified CHOP as an interacting partner of activating transcription factor (ATF) 4 in a yeast two-h

270 anced levels of phosphorylated eIF2alpha and activating transcription factor (ATF) 4, which is essent

271 ern and the role of different members of the activating transcription factor (ATF) family in survival

272 tion factors are dimers of JUN, FOS, MAF and activating transcription factor (ATF) family proteins ch

273 CREB3L1), a transcription factor of the CREB/activating transcription factor (ATF) family, increases

274 -related protein (SMAD)-3 (nt -584 to -581), activating transcription factor (ATF)-2 (nt -571 to -568

275 tion and activation of p38 kinase substrate, activating transcription factor (ATF)-2.

276 equires binding sites for sequence-dependent activating transcription factor (ATF)-adenosine 3',5'-mo

277 Activating transcription factor (ATF)3 regulates the exp

278 f type I IFN gene expression, interacts with activating transcription factor (ATF)4, a key component

279 RAD is increased by the ER stress modulator, activating transcription factor (ATF)6, which can induce

280 expression of the transcriptional repressor activating transcription factor (ATF-3) in a STAT1-depen

281 kinase and PKR-like ER kinase (PERK)-induced activating transcription factor (ATF3) binding to its pr

282 alpha and upregulation of gene expression of activating transcription factors ATF4 and ATF6.

283 homology to the cyclic AMP response element/activating transcription factor binding sites.

284 It binds an activating transcription factor complex in erythroid cel

285 cts of cAMP response element binding protein/activating transcription factor (CREB/ATF) and AP-1 tran

286 gulation of late-stage stress-response genes activating transcription factors (e.g., Atf4) and heat-s

287 (cAMP response element modulator), and atf (activating transcription factor) genes.

288 1) family transcription factor BATF (B cell, activating transcription factor-like) was among the gene

289 tion across the PHO mRNA promoters displaces activating transcription factor Pho7.

290 ression mediated, in part, by members of the activating transcription factor protein (AP1) group.

291 Amongst ROS-activating transcription factors, RelA/p65 induces Greml

292 h defects in IFN regulatory factor 4, B cell-activating transcription factor, retinoic acid-related o

293 Subsequent recruitment of the activating transcription factor Sp1 to the remodeled pro

294 nhibition can increase cAMP and cGMP levels, activating transcription factors such as the cAMP respon

295 ndelian disease, we show that DNA binding of activating transcription factor (TF) determines histone

296 mones regulate many aspects of plant life by activating transcription factors (TFs) that bind sequenc

297 reasing histone deacetylase activity, or (e) activating transcription factors that antagonize chronic

298 at alternating recruitment of repressive and activating transcription factors to shared cis-regulator

299 drial ROS augmented the expression of B cell-activating transcription factor, which may contribute to

300 Ectopic expression of the EMT-activating transcription factor ZEB1 stimulated Golgi co