戻る
「早戻しボタン」を押すと検索画面に戻ります。 [閉じる]

コーパス検索結果 (１語後でソート)

通し番号をクリックするとPubMedの該当ページを表示します
1 tween total sulfate content and the level of activatory activity.
2 hat, during activation by wild-type CRP, the activatory and inhibitory determinants of AR3 balance ea
3 hosphatases with large ectodomains that have activatory and inhibitory effects on TCR triggering.
4 in with novel combinations of affinities for activatory and inhibitory Fc receptors.
5 hat Dok-3/Grb2 modulates the balance between activatory and inhibitory Lyn functions with the aim to
6 lobulin-like receptors encode MHC-I-specific activatory and inhibitory receptors, implying that MHC-I
7 hogenesis demonstrates how subtle changes in activatory and inhibitory signals can alter tooth shape,
8 age ligands, facilitating the integration of activatory and inhibitory signals from multiple receptor
9 ms, including NtrC (where phosphorylation is activatory), and indicate that recognition properties, w
10 osite conformational changes upon binding of activatory (Cdc42, Eps8) or inhibitory (14-3-3) inputs.
11 ependent promoters by making three different activatory contacts with different surfaces of holo RNA
12 ession, signaling, and surface expression of activatory/costimulatory receptors.
13 nitiation assays were used to show that this activatory determinant accelerates the rate of isomerisa
14 de that these residues in AR3 of CRP form an activatory determinant for Class II transcription activa
15 lts provide a structural explanation for the activatory effect of this mutation and provide a greater
16 ngage with CD14(+) monocytes in a reciprocal activatory fashion, thereby amplifying the inflammatory
17 (ADCC) which is mediated in part through the activatory Fc receptor, FcgammaRIIIA.
18 bitory FcgammaRIIB in 3/23 activity, whereas activatory FcgammaR (FcgammaRI, -III, and -IV) was dispe
19 ), has been shown to augment the activity of activatory FcgammaR and promote mAb immunotherapy.
20 cytidylic acid or were given subcutaneously, activatory FcgammaR could also provide cross-linking.
21                                Intriguingly, activatory FcgammaR provided effective cross-linking of
22                        In transfected cells, activatory FcgammaRI, FcgammaRIIa, and FcgammaRIIIa augm
23                                    Using the activatory Gq-coupled human M3 muscarinic receptor (hM3D
24 y ligands of Siglecs to antibodies targeting activatory immune receptors can suppress receptor-mediat
25 al kinase and that other death receptors and activatory immune receptors were co-ordinately upregulat
26                                       A high activatory/inhibitory FcgammaR binding ratio is critical
27 ants like STINGa, which can improve FcgammaR activatory:inhibitory (A:I) ratios on TAM, are appealing
28 lopment and following neuronal injury, while activatory JAK1 mutations are linked to leukemia.
29                              The ligands for activatory KIRs are unknown.
30                          We analyzed whether activatory KIRs carried by the donor influenced the subs
31 NKp30 that upon interaction with its cognate activatory ligand B7-H6 induces rapid production of type
32                             In contrast, the activatory ligands for KIR3DS1 remain obscure.
33 nition of tumor cells expressing the NK cell-activatory ligands H60 or CD137L by mouse natural killer
34 might function in inhibitory or, potentially activatory manners.
35 d by the combination of an inhibitory and an activatory mechanism.
36 controlled by transcription factors that use activatory mechanisms similar to CRP.
37 ity of MT1-MMP from its enzymatic toward its activatory mode, suggesting a tight mechanosensitive reg
38 , which signals via the immunoreceptor-based activatory motif (ITAM)-containing FcRgamma chain.
39 s that contain immunoreceptor tyrosine-based activatory motifs to mediate NK direct lysis of tumor ce
40 ne the structural changes that arise from an activatory mutation (S205L) located in the cytoplasmic d
41 act as ligands for cells expressing a common activatory natural killer-cell receptor (NKG2D).
42 ween HLA-C and KIR genes suggests a shift to activatory NK cell function.
43                                  FcRs may be activatory or inhibitory and regulate a variety of immun
44 ng proteins when phosphorylated, transducing activatory or inhibitory signals.
45 ectively, our findings identify a novel auto-activatory pathway that is essential for optimal NK cell
46    dSTRIPAK depletion leads to increased Hpo activatory phosphorylation and repression of Yki target
47                      This was unexpected, as activatory phosphorylation mediated by MEK is thought to
48                                Moreover, the activatory phosphorylation of Src family kinases was con
49 ations (inhibitory monoubiquitination versus activatory phosphorylation), and kinetic/allosteric prop
50  This astrocyte-specific regulation involves activatory protein-1 (AP-1) because overexpression of do
51 Fc receptor (FcR) gamma-chain complex, a key activatory receptor for collagen on platelet surface mem
52                It is the only known platelet activatory receptor that participates in both of these a
53 n epithelia, and is the major ligand for the activatory receptor, NKG2D, both attributes relevant to
54 erleukin (IL)-10, IL-13, IL-17 secretion and activatory receptors (NKG2D, NKp46i and NKp44) have been
55       FcgammaRIIbT(232) is unable to inhibit activatory receptors because it is excluded from sphingo
56                                Antibodies to activatory receptors typically activate immune cells by
57           Emerging evidence suggests that NK-activatory receptors use KARAP/DAP12, CD3zeta, and Fceps
58 causing autophosphorylation on the known MEK activatory residues, S218 and S222.
59                    Although oxLDL stimulates activatory signaling, it is unclear how these events dri
60           beta-Arrestin-1 and -2 can mediate activatory signals by 7TMRs.
61                       In T cells, SAP relays activatory signals from the T-cell receptor but in its a
62 on in U2OS cells that is, in part, caused by activatory signals mediated by beta-arrestin-1.
63  of cellular proteins including c-Src at the activatory site, and this effect is blocked when the 5'-
64 hange that stabilizes the high-affinity PIP2 activatory site.
65 ell priming if provided appropriately phased activatory stimuli, an observation that could be factore
66 lls can change with the nature and timing of activatory stimuli.
67 n both the inhibitory 'slender retainer' and activatory 'stumpy inducer' arms of the QS control pathw
68                   They autophosphorylate the activatory TEY motif and also other residues, including