ライフサイエンスコーパス: active oxygen

1 iOO(-)), which can be described as adsorbed "active oxygen".

2 sites relative to Co(3+) and by the shift of active oxygen 2p states.

3 nd productive orientation between heme-bound active oxygen and acceptor carbon bond(s).

4 to NO(2), and also favors the production of active oxygen and the catalyst oxygen storage capacity.

5 ns show how the chemical coordination of the active oxygen atoms is responsible for the negative long

6 ts are from the chemical coordination of the active oxygen atoms.

7 ressing VU-3 calmodulin exhibited a stronger active oxygen burst that occurred more rapidly than in n

8 that, by controlling the environment of such active oxygens (e.g., by means of an epitaxial strain),

9 verall, our study highlights a novel, highly active oxygen evolution catalyst; moreover, it provides

10 enewable energy technology to develop highly active oxygen evolution reaction (OER) catalysts with fa

11 transition-metal (oxy)hydroxides are highly active oxygen-evolution reaction (OER) electrocatalysts

12 A highly active oxygen-evolving photosystem II (PSII) complex was

13 moval of this extension is necessary to form active oxygen-evolving photosystem II centers.

14 trocatalyst for those reactions that involve active oxygen intermediates, such as reduction of oxygen

15 late the properties of both the monoxygenase active-oxygen intermediates and the proton-delivery netw

16 ectroscopy has been used to confirm that the active oxygen of HSO(5-) is nonexchanging (t(1/2) >> 1 h

17 d responses including programmed cell death, active oxygen production and transcription of the pathog

18 reasing the surface density of catalytically active oxygen radical sites on a MoVTeNb oxide (M1 phase

19 arge compensation mechanism to stabilize the active oxygen redox reaction.

20 ogeneous, nanoparticulate Pd(0) serves as an active oxygen reduction electrocatalyst to furnish the h

21 Designing highly active oxygen reduction reaction (ORR) catalysts is cruc

22 of this study was to evaluate the effect of active oxygen-releasing gel as an adjuvant, with and wit

23 (n = 17)-SI followed by local application of active oxygen-releasing gel inside the periodontal pocke

24 The use of adjuvant active oxygen-releasing gel, with or without aPDT, resul

25 a previously unknown role for pyridoxine in active oxygen resistance.

26 These results indicate that active oxygen scavenging combined with TRPA1 inhibition

27 O(3)(001) involving surface peroxides as the active oxygen source is suggested.

28 timing and magnitude with development of the active oxygen species (AOS) burst.

29 Active oxygen species (AOS) generated in response to sti

30 The use of relatively low amounts of active oxygen species (such as hypochlorite), followed b

31 eviously reported that hydrogen peroxide, an active oxygen species and a cellular oxidant, induces c-

32 recently reported that hydrogen peroxide, an active oxygen species and a cellular oxidant, stimulates

33 stant attack from spontaneous hydrolysis and active oxygen species and from other intracellular metab

34 r9, respectively, triggered the synthesis of active oxygen species and MAP kinase activation.

35 Afterward, O(2) can be evolved into active oxygen species and promote the deprotonation and

36 and by ozone treatment is the production of active oxygen species and the accumulation of hydrogen p

37 ctors in addition to ceruloplasmin, possibly active oxygen species and/or lipoxygenases, are essentia

38 The cumulative results suggest that active oxygen species are generated near cell walls of v

39 ls and yeast DNA repair mutants sensitive to active oxygen species are not sensitive to this agent, t

40 , indicating that the peroxide is likely the active oxygen species attacking the aromatic substrates.

41 synthesis and phosphorylation in response to active oxygen species could play a role in anti-oxidativ

42 in is likely to have a role in metabolism of active oxygen species derived from internal body metabol

43 Moreover, by trapping active oxygen species generated in the photocatalytic pr

44 yrin radical cation intermediate that is the active oxygen species in these hydroxylation reactions.

45 his paper two hypotheses are tested: (i) the active oxygen species is similar in energetics for all c

46 ch once released can be readily converted to active oxygen species or to dissolved dioxygen.

47 in vitro or in cultured cells also generates active oxygen species such as superoxide, which can indi

48 ed NrdI, responsible for the formation of an active oxygen species suggested to be either a superoxid

49 interfaces as a new active site can provide active oxygen species to the first C-H cleavage of light

50 rate from heat shock protein induction, ABA, active oxygen species, and salicylic acid pathways are i

51 ed changes in SA levels, the accumulation of active oxygen species, defense gene expression, and the

52 of plant cell death and the induction of an active oxygen species-responsive promoter (AoPR1-GUS) we

53 erate atomic oxygen [O(3P)] or an equivalent active oxygen species.

54 athway that is required for the Avr9-induced active oxygen species.

55 lular toxic species in the medium, including active oxygen species.

56 se observations show that more electrophilic active-oxygen species (i.e., lower-energy LMCT) are more