ライフサイエンスコーパス: acute HIV infection

1 mian immunodeficiency virus macaque model of acute HIV infection.

2 assays as primary tools for the diagnosis of acute HIV infection.

3 modest effects on overall cell death during acute HIV infection.

4 apy (ART) in adults who initiated ART during acute HIV infection.

5 T in 16 individuals who initiated ART during acute HIV infection.

6 which they decay after ART initiation during acute HIV infection.

7 D14, D-dimer, and HA levels were elevated in acute HIV infection.

8 mune pressure in a cohort of 17 persons with acute HIV infection.

9 ing has not routinely included screening for acute HIV infection.

10 ing of populations with a high prevalence of acute HIV infection.

11 s at mucosal sites are essential targets for acute HIV infection.

12 dies, p24 antigen and RNA typically indicate acute HIV infection.

13 163 had established HIV infection, and 8 had acute HIV infection.

14 analysis and MRS in some individuals during acute HIV infection.

15 infection and poor performance for detecting acute HIV infection.

16 making clinical decisions for patients with acute HIV infection.

17 HIV testing: rapid testing and detection of acute HIV infection.

18 entations from the meeting on the Biology of Acute HIV Infection.

19 macytoid DC levels decline very early during acute HIV infection.

20 t disease, tumor immunotherapy regimens, and acute HIV infection.

21 nterventions that modify the consequences of acute HIV infection.

22 role, we examined plasma from patients with acute HIV infection.

23 s, despite limited sensitivity for detecting acute HIV infections.

24 emergence of drug resistance and to diagnose acute HIV infections.

25 ilable therapeutic options; 9) treatment for acute HIV infection; 10) considerations for antiretrovir

26 Of the 23 acute HIV infections, 16 were detected at sexually trans

27 nce estimates were based on (1) detection of acute HIV infection, (2) documentation of HIV seroconver

28 ial increase in plasma virus associated with acute HIV infection (3, 7, 20, 35, 48).

29 Identification of individuals with acute HIV infection (AHI) and rapid initiation of antire

30 A 128-member specimen pooling scheme for acute HIV infection (AHI) detection was evaluated using

31 Antiretroviral therapy (ART) during acute HIV infection (AHI) interrupts viral dynamics and

32 nitiation of antiretroviral therapy (ART) in acute HIV infection (AHI) is effective at limiting seedi

33 y PrEP use history in people with or without acute HIV infection (AHI).

34 s from participants who initiated ART during acute HIV infection (AHI).

35 prior to initiating therapy in patients with acute HIV infection, although therapy should not be dela

36 icians should be alert to the possibility of acute HIV infection and promptly pursue diagnostic testi

37 d the timing of NK cell perturbations during acute HIV infection and the impact of early ART initiati

38 t cytolytic CD4+ T cells emerge early during acute HIV infection and tightly follow acute viral load

39 hould be preferred to avoid missing cases of acute HIV infection and to decrease the related risks of

40 subjects generated these responses early in acute HIV infection and were able to control HIV replica

41 n (ie, with CD4 cell count >=500/muL or with acute HIV infection) and used Kaplan-Meier plots and pro

42 iral RNA technologies and initiating ART for acute HIV infection appears cost effective.

43 s, these factors indicate that events during acute HIV infection are likely to include distortions in

44 Two women had acute HIV infection at enrollment, 4 seroconverted, and

45 he TDF-FTC group who had had an unrecognized acute HIV infection at enrollment.

46 Ten patients were treated during acute HIV infection before complete Western blot (WB) se

47 racterize the early plasma viral dynamics in acute HIV infection better than it has been possible thu

48 42 women were diagnosed with acute HIV infection between Dec 1, 2012, and June 30, 20

49 l responses are important for the control of acute HIV infection but become progressively dysfunction

50 s may modulate the innate immune response to acute HIV infection by reducing viral replication.

51 e HIV diagnostic yield (both established and acute HIV infections) by 10.4% (95% CI, 8.8%-12.2%) and

52 Difficulties inherent in studying acute HIV infection can be overcome by modeling virus-ho

53 Understanding the dynamics of acute HIV infection can offer valuable insights into the

54 In seronegative acute HIV infection, CD8(+) T cell counts increased in t

55 After acute HIV infection, CD8(+) T cells are able to control

56 context of light chain use in subjects with acute HIV infection, chronic HIV infection, and among HI

57 LBT has higher sensitivity for acute HIV infection compared to RT but LBT is not point

58 Although acute HIV infection contributes disproportionately to on

59 In acute HIV infection, CSF NFL levels are inversely associ

60 the POC test failed to detect 100% (3/3) of acute HIV infections (defined as Fiebig stage 2) and 3.8

61 IV Ag/Ab combination testing detected 82% of acute HIV infections detectable by pooled HIV RNA testin

62 lowing a negative rapid test detected 82% of acute HIV infections detectable by pooled HIV RNA testin

63 Number and proportion with acute HIV infections detected.

64 otophobia were significantly associated with acute HIV infections diagnosed in the ED.

65 Twenty individuals who had acute HIV infection (Fiebig stages I-IV), with average 1

66 subjects studied included two subjects with acute HIV infection, five subjects with chronic HIV infe

67 the presentations on clinical management of acute HIV infection from the 2009 Acute HIV Infection Me

68 People with acute HIV infection have demonstrated enhanced transmiss

69 Fourteen people with acute HIV infection (HIV antibody negative/NAT positive)

70 ns for the therapeutic use of bnAbs to treat acute HIV infections.IMPORTANCE Currently, there is no b

71 ubunit of TFIIH, and concurrently suppresses acute HIV infection in vitro Here we investigated SP as

72 ng TRAILshort on the outcome of experimental acute HIV infection in vitro.

73 f young women detected during Fiebig stage I acute HIV infection in whom treatment was initiated imme

74 findings suggest the widespread diagnosis of acute HIV infections in a routine testing population is

75 Acute HIV infection induced a rapid depletion of M. tube

76 of HIV-specific CD4 T cell responses during acute HIV infection influences disease progression and w

77 Acute HIV infection is associated with a rapid depletion

78 Acute HIV infection is associated with a vigorous immune

79 of HIV-specific CD4 T cell responses during acute HIV infection is beneficial overall and does not f

80 Acute HIV infection is characterized by massive loss of

81 Acute HIV infection is characterized by rapid viral seed

82 time pooled RNA testing for the detection of acute HIV infection is feasible in resource-limited sett

83 er, the ontogeny of humoral responses during acute HIV infection is poorly defined in infants and cha

84 The literature on acute HIV infection is predominantly small nonrandomized

85 Acute HIV infection lasts approximately 3 weeks and earl

86 eeks and early HIV infection, which includes acute HIV infection, lasts approximately 7 weeks.

87 We conclude that early treatment of acute HIV infection leads to a more narrowly directed CT

88 ing antiretroviral therapy (ART) in Fiebig 1 acute HIV infection limits the size of viral reservoirs

89 The detection of acute HIV infections may need to rely on other testing s

90 size and the relationships to viral load in acute HIV infection, measurements of the latent reservoi

91 agement of acute HIV infection from the 2009 Acute HIV Infection Meeting in Boston, Massachusetts.

92 In an in vitro acute HIV infection model, MTB increased HIV replication

93 e cohorts in KwaZulu-Natal were assessed for acute HIV infection monthly (n = 245) or every 3 months

94 Few instances of typhoid fever (n = 8), acute HIV infection (n = 5), and dengue (n = 2) were enc

95 Here, we demonstrate that while acute HIV infection of human microglia/macrophages resul

96 Here virologic and immunologic aspects of acute HIV infection of humans and SIV infection of Asian

97 ions, we have used an in vitro cell model of acute HIV infection of quiescent, primary CD4 lymphocyte

98 In a longitudinal study, the effect of acute HIV infection on M. tuberculosis-specific Th1 cell

99 CSF NFL levels are not elevated in untreated acute HIV infection or after 6 months of immediately ini

100 ogeneity in transmission, for example due to acute HIV infection or the presence of 'core groups' wit

101 ial advantages and disadvantages of treating acute HIV infection outlined in treatment guidelines, an

102 tion of ART at one of the earliest stages of acute HIV infection possible.

103 sponses dominantly targeting Gag over Env in acute HIV infection remained off antiretroviral therapy

104 on of the vivo inhibitory effect of IL-10 on acute HIV infection suggests that further studies may be

105 For detecting acute HIV infection, the antigen portion had a sensitivi

106 barrier already in the seronegative phase of acute HIV infection, thereby inducing microbial transloc

107 is an important IRF3 regulator that supports acute HIV infection through innate immune suppression.

108 ensively investigated in eight patients with acute HIV infection to define the earliest rates of chan

109 mbination immunoassays in cases of suspected acute HIV infection to ensure a timely and accurate diag

110 ever, the low PPV and 100% failure to detect acute HIV infections underscore the importance of priori

111 Thirty participants who started ART during acute HIV infection underwent CNS assessments across 4 A

112 Thirty participants who started ART during acute HIV infection underwent CNS assessments across fou

113 In a humanized mouse model of acute HIV infection, venetoclax monotherapy significantl

114 ART initiated in early acute HIV infection was associated with normalization of

115 Acute HIV infection was detected in 134 participants wit

116 s diagnosed in 1158 participants (1.33%) and acute HIV infection was diagnosed in 168 participants (0

117 tion of Gag-specific CD4 T cell responses in acute HIV infection was significantly inversely correlat

118 tiating potent antiretroviral therapy during acute HIV infection was similar to estimates from treate

119 rapy in individuals who initiated ART during acute HIV infection was well tolerated but did not signi

120 The antigen portion of the Combo RT (for acute HIV infection) was compared with a Roche Monitor H

121 tudying individuals in the first few days of acute HIV infection, we detected the emergence of a uniq

122 Patients with possible acute HIV infection were followed to confirm seroconvers

123 the epitopes targeted at a high frequency in acute HIV infection were recognized at the same frequenc

124 No acute HIV infections were identified.

125 ent a highly reactive cell population during acute HIV infection, which responds independently from t

126 rd HIV antibody tests to detect persons with acute HIV infection who are viremic but antibody-negativ

127 nt is the identification of individuals with acute HIV infection whose contribution to HIV transmissi

128 tive rapid HIV test result were screened for acute HIV infection with an HIV Ag/Ab combination assay

129 on of activated CD8(+)T cells appears during acute HIV infection with diminished capacity to inhibit

130 d to determine whether ART initiation during acute HIV infection would attenuate changes in these bio