コーパス検索結果 (1語後でソート)
通し番号をクリックするとPubMedの該当ページを表示します
1 ersus established cardiac risk factors after acute myocardial infarction.
2 l deficiency of hepcidin and challenged with acute myocardial infarction.
3 y DNR affected hospital quality measures for acute myocardial infarction.
4 beta inhibition has beneficial effects after acute myocardial infarction.
5 o locally regulate cardiac homeostasis after acute myocardial infarction.
6 incident congestive heart failure (CHF) and acute myocardial infarction.
7 urs in patients with cardiogenic shock after acute myocardial infarction.
8 utic hypothermia for cardiogenic shock after acute myocardial infarction.
9 patients with cardiogenic shock complicating acute myocardial infarction.
10 tablished among a sample of 60 patients with acute myocardial infarction.
11 omen are at highest risk after discharge for acute myocardial infarction.
12 ing 30-day risk-standardized mortality after acute myocardial infarction.
13 e as potential immunomodulatory treatment in acute myocardial infarction.
14 ctive study of patients hospitalized with an acute myocardial infarction.
15 or hospital admission of those who died from acute myocardial infarction.
16 ality indicators (QIs) for the management of acute myocardial infarction.
17 nd mitochondrial function in swine models of acute myocardial infarction.
18 ntribute to reducing the mortality burden of acute myocardial infarction.
19 ould mitigate the risk of plaque rupture and acute myocardial infarction.
20 about deaths and hospital admissions due to acute myocardial infarction.
21 cular events, particularly heart failure and acute myocardial infarction.
22 ably quantify AAR and final IS in reperfused acute myocardial infarction.
23 d cell deficiency of IDO and challenged with acute myocardial infarction.
24 hock (CS) as the most severe complication of acute myocardial infarction.
25 ins (cTns) are the cornerstone of diagnosing acute myocardial infarction.
26 between Staphylococcus aureus infection and acute myocardial infarction.
27 phylactic reaction, acute kidney injury, and acute myocardial infarction.
28 subclinical hypothyroidism in patients with acute myocardial infarction.
29 ucially involved in cardiac remodeling after acute myocardial infarction.
30 ath within 1 year of hospitalization with an acute myocardial infarction.
31 patients with cardiogenic shock complicating acute myocardial infarction.
32 r heart failure, 1.76 (95% CI 1.51-2.05) for acute myocardial infarction, 1.78 (95% CI 1.53-2.07) for
33 ogenic shock (CS), 31% had CS not related to acute myocardial infarction, 11% had mixed shock, and 22
34 here were sustained decreases in PAC use for acute myocardial infarction (20.0 PACs placed per 1000 a
35 additional criteria required for spontaneous acute myocardial infarction (280/397, 71%) versus those
37 1995 to 2015, including 14 423 patients with acute myocardial infarction (59% STEMI) admitted to card
38 ation of Risk Factors in Older Patients With Acute Myocardial Infarction)-a prospective observational
39 was present in individuals with a history of acute myocardial infarction, a disease state linked to p
40 ardiac troponin for accelerated diagnosis of acute myocardial infarction: a systematic review and met
41 in 28 days of being in hospital follow a non-acute myocardial infarction admission as follow an acute
43 ance imaging, adjusted for age, sex, type of acute myocardial infarction, affected coronary artery te
44 onclusions In cardiogenic shock complicating acute myocardial infarction, all-cause mortality is simi
45 nts; median 10.7 y follow-up; 648 deaths) or acute myocardial infarction (AMI) (3670 patients; median
46 s investigating the effect of MSC therapy in acute myocardial infarction (AMI) and chronic ischemic c
47 dence of major cardiovascular events such as acute myocardial infarction (AMI) and congestive heart f
48 ffects of PM(2.5) on the risks of developing acute myocardial infarction (AMI) and dying from cardiov
49 ociated with reduced 30-day readmissions for acute myocardial infarction (AMI) and heart failure (HF)
51 esidential green spaces and the incidence of acute myocardial infarction (AMI) and heart failure (HF)
52 tal defect (VSD) is a lethal complication of acute myocardial infarction (AMI) and is often associate
53 isk-standardized mortality rates (RSMRs) for acute myocardial infarction (AMI) are calculated for Med
54 missions among older adults hospitalized for acute myocardial infarction (AMI) are costly and difficu
56 k-adjusted 30-day home time in patients with acute myocardial infarction (AMI) as a hospital-level pe
58 care units (CICUs) have primarily focused on acute myocardial infarction (AMI) complicated by cardiog
59 Despite advances in treatment, mortality in acute myocardial infarction (AMI) complicated by cardiog
61 ic approach for the care of patients with an acute myocardial infarction (AMI) during the COVID-19 pa
62 ity cardiac troponin-T (hs-cTnT) in a 1-hour acute myocardial infarction (AMI) exclusion algorithm.
63 ctive therapies into the clinical setting of acute myocardial infarction (AMI) for patient benefit ha
64 emergency department patients with possible acute myocardial infarction (AMI) has been shown to effe
65 nts on dialysis revealed that mortality from acute myocardial infarction (AMI) has decreased, whereas
68 In recent decades, the rates of incident acute myocardial infarction (AMI) have declined in the U
71 f sex-based differences in older adults with acute myocardial infarction (AMI) have yielded mixed res
72 iac events (MACE) in patients with suspected acute myocardial infarction (AMI) is an unmet clinical n
76 emergency department discharge diagnosis of acute myocardial infarction (AMI) or stroke using Intern
78 The role of left atrial (LA) performance in acute myocardial infarction (AMI) remains controversial.
80 (HDs) and 14 plasma samples of patients with acute myocardial infarction (AMI) was carried out with s
81 /angiotensin receptor blockers (ACEI/ARB) in acute myocardial infarction (AMI) were largely conducted
83 an quantify the area-at-risk (AAR) following acute myocardial infarction (AMI), and has been used to
85 ry disease (CAD) and its major complication, acute myocardial infarction (AMI), are the leading cause
87 s on individual process of care measures for acute myocardial infarction (AMI), little is known about
88 e), defined as in-hospital, all-cause death, acute myocardial infarction (AMI), or acute ischemic str
89 rity levels on this outcome, specifically on acute myocardial infarction (AMI), remains unexplored.
92 and increasing attention to young women with acute myocardial infarction (AMI), who represent an extr
108 e oxygenation (ECMO) is increasingly used in acute myocardial infarction (AMI); however, there are li
110 heart failure, 0.45 [95% CI, 0.42 to 0.47]; acute myocardial infarction [AMI], 0.37 [CI, 0.35 to 0.4
111 n, hospital admission for unstable angina or acute myocardial infarction [AMI], 30-day readmission af
113 lization files, we identified discharges for acute myocardial infarction and cardiac arrest January 2
115 c cardioplegic arrest is only a surrogate of acute myocardial infarction and confounded by the choice
116 ysis in bone marrow cells from patients with acute myocardial infarction and detected increased sulfa
117 117 foods with coronary heart disease risk (acute myocardial infarction and fatal coronary heart dis
118 l risk factors were at target, with risk for acute myocardial infarction and heart failure hospitaliz
122 n two (1%) patients in the radium-223 group (acute myocardial infarction and interstitial lung diseas
123 ogenic Shock), patients with CS complicating acute myocardial infarction and multivessel coronary art
124 omen and men presenting with CS complicating acute myocardial infarction and multivessel coronary art
126 tient and outpatient PCI, as well as PCI for acute myocardial infarction and nonacute myocardial infa
127 ies were larger, treating more patients with acute myocardial infarction and performing more PCIs tha
129 he expression of hepcidin was elevated after acute myocardial infarction and the specific deletion of
130 arch Investigating Underlying Disparities in Acute Myocardial Infarction) and PREMIER (Prospective Re
131 mplications of atherosclerosis, in ischemic (acute myocardial infarction), and nonischemic injury to
132 ed 706 patients who had multivessel disease, acute myocardial infarction, and cardiogenic shock to on
133 125 231 hospitalizations for heart failure, acute myocardial infarction, and pneumonia, respectively
134 or patients hospitalized with heart failure, acute myocardial infarction, and pneumonia, the three co
137 anned readmissions after hospitalization for acute myocardial infarction are among the leading causes
139 Consequently, early signals of an imminent acute myocardial infarction are likely to be found by pl
141 l infarction diagnosis but went on to die of acute myocardial infarction as the underlying cause of d
143 in-hospital outcomes of patients treated for acute myocardial infarction before and after a hospital
144 h and return to pre-infarct activities after acute myocardial infarction, but the trial lacked statis
145 tween the two, and the overall management of acute myocardial infarction can be reviewed for simplici
146 d data on how sex influences the outcomes of acute myocardial infarction-cardiogenic shock (AMI-CS) i
147 aluating regional disparities in the care of acute myocardial infarction-cardiogenic shock (AMI-CS).
148 subo syndrome (TTS) generally presents as an acute myocardial infarction characterized by severe left
150 ory support devices are increasingly used in acute myocardial infarction complicated by cardiogenic s
153 and clinical outcomes in 7195 subjects with acute myocardial infarction complicated by reduced left
154 ly administered off-the-shelf early after an acute myocardial infarction, comply with stringent crite
155 r older admitted for five medical diagnoses (acute myocardial infarction, congestive heart failure, s
156 ion is available on how primary and comorbid acute myocardial infarction contribute to the mortality
158 A substantial proportion of patients with acute myocardial infarction develop clinical heart failu
159 x groups) and case-fatality rate for primary acute myocardial infarction diagnosed during the first p
160 counter or during subsequent encounters, and acute myocardial infarction diagnosed only as a comorbid
161 s of admission in people who did not have an acute myocardial infarction diagnosis but went on to die
162 rtality among patients who were admitted for acute myocardial infarction (difference-in-differences e
165 entify all primary and comorbid diagnoses of acute myocardial infarction during hospital stay and the
166 ower risk of death after hospitalization for acute myocardial infarction during the full follow-up pe
167 of-concept of efficacy of IL-4 treatment for acute myocardial infarction, encouraging its further dev
168 ivity Troponin-T Assay for Rapid Rule-Out of Acute Myocardial Infarction) evaluated high-sensitivity
171 th resulting from all causes, fatal/nonfatal acute myocardial infarction, fatal/nonfatal stroke, and
172 emiological trends, and modern management of acute myocardial infarction, focusing on the recent adva
173 zerland) Plus Registry enrolls patients with acute myocardial infarction from 83 hospitals in Switzer
174 term MINOCA (incorporating the definition of acute myocardial infarction from the newly released "Fou
177 decades, the holy grail in the treatment of acute myocardial infarction has been the mitigation of l
179 rd rate ratio for women versus men, 1.10 for acute myocardial infarction; hazard rate ratio, 1.04 for
181 tients who were admitted to the hospital for acute myocardial infarction, heart failure, or pneumonia
182 utcomes with Revascularization and Stents in Acute Myocardial Infarction (HORIZONS-AMI) clinical tria
183 stimate the relative risks of three primary (acute myocardial infarction, hospitalisation for heart f
184 hy, and by primary CR-qualifying event type (acute myocardial infarction hospitalization; coronary ar
186 an angiotensin-converting enzyme inhibitors: acute myocardial infarction (HR 0.84, 95% CI 0.75-0.95),
187 5% confidence interval (CI): 1.02, 1.05) and acute myocardial infarction (HRIQR = 1.05, 95% CI: 1.02,
188 ce of ischemic events in patients with prior acute myocardial infarction in a large phase III clinica
190 haracteristics, treatments, and outcomes for acute myocardial infarction in France between 1995 and 2
191 ury vs heart failure, pneumonia, stroke, and acute myocardial infarction in older adults between 2008
192 t can be used to predict the risk of a fatal acute myocardial infarction in such patients, which can
195 sepsis in the intravenous infusion group and acute myocardial infarction in the fixed-dose combinatio
196 inal multicenter cohort study of people with acute myocardial infarction in the United States, querie
197 %) of 446 744 admissions with a diagnosis of acute myocardial infarction, in the second or later phys
200 Subclinical hypothyroidism in patients with acute myocardial infarction is associated with poor prog
202 engineered fibroblasts into a mouse model of acute myocardial infarction led to improved cardiac func
207 ital treatment and outcomes of patients with acute myocardial infarction (MI) have been described, bu
208 ls of bone marrow cell-based therapies after acute myocardial infarction (MI) have produced mostly ne
210 Recent studies reported an increased risk of acute myocardial infarction (MI) in PPI users vs non-use
212 233 US hospitals within 1 year of the index acute myocardial infarction (MI) of 12365 patients enrol
216 ategies significantly reduce mortality after acute myocardial infarction (MI), a large number of pati
218 chemic cardiovascular diseases, particularly acute myocardial infarction (MI), is one of the leading
219 lmark and the primary therapeutic target for acute myocardial infarction (MI), multiple other mechani
220 ed relative to the primary outcome of death, acute myocardial infarction (MI), or hospitalization for
227 ary intervention in patients presenting with acute myocardial infarction, multivessel disease, and ca
228 ll patients aged 40 to 85 presenting with an acute myocardial infarction (n=181 696; 5.7% dog ownersh
229 30), intracerebral haemorrhage (n=3496), and acute myocardial infarction (n=2958); men who reported d
230 vascular diagnoses such as heart failure and acute myocardial infarction, need frequently arises for
231 primary outcome was a composite of nonfatal acute myocardial infarction, nonfatal stroke, or cardiov
232 so calculated what proportion of deaths from acute myocardial infarction occurred in people who had b
233 Well into the 21st century, we still triage acute myocardial infarction on the basis of the presence
234 alyzed Medicare data on hospitalizations for acute myocardial infarction or cardiac arrest among Medi
235 admitted to marathon-affected hospitals with acute myocardial infarction or cardiac arrest on maratho
236 onist, in patients with ST-segment-elevation acute myocardial infarction or heart failure with reduce
237 ws: principal HF hospitalizations; principal acute myocardial infarction or pneumonia hospitalization
238 n myocardial strain imaging in patients with acute myocardial infarction or stable ischemic heart dis
240 are characteristics of patients suffering an acute myocardial infarction or undergoing cardiovascular
241 Patients with a history of PCI or CABG, acute myocardial infarction, or an indication for concom
242 was the composite of incident heart failure, acute myocardial infarction, or cardiovascular death.
243 ticipants were diagnosed with heart failure, acute myocardial infarction, or cardiovascular death.
244 ncipal discharge diagnosis of heart failure, acute myocardial infarction, or pneumonia and estimated
245 eneficiaries hospitalized for heart failure, acute myocardial infarction, or pneumonia, reductions in
246 -AMI OUTCOME CMR (Stem Cells Mobilization in Acute Myocardial Infarction Outcome Cardiac Magnetic Res
247 for acute nonischemic myocardial injury and acute myocardial infarction, particularly type 2 myocard
248 arch Investigating Underlying Disparities in Acute Myocardial Infarction Patients' Health Status) is
249 arch Investigating Underlying Disparities in Acute Myocardial Infarction Patients' Health Status) reg
250 arch Investigating Underlying Disparities in Acute Myocardial Infarction Patients' Health Status).
251 a rigorously imaging-based selected group of acute myocardial infarction patients, with detailed safe
253 prescribed to frail, older adults following acute myocardial infarction, potentially because of the
254 Prasugrel and Ticagrelor in the Treatment of Acute Myocardial Infarction [PRAGUE-18]; NCT02808767).
255 gene expression profiling at the time of an acute myocardial infarction provides information concern
256 aker/implantable cardioverter defibrillator, acute myocardial infarction, pulmonary embolism, stroke/
257 ncluding heart failure (r = 0.39; P < 0.01), acute myocardial infarction (r = 0.30; P < 0.01), pneumo
260 270 admissions using temporary MCS, 33% had acute myocardial infarction-related cardiogenic shock (C
262 e with right coronary artery disease, recent acute myocardial infarction, renal dysfunction, and pati
263 I, 1.46 to 1.72) and a 40% increased risk of acute myocardial infarction (RR 1.40; 95% CI, 1.23 to 1.
264 c natural killer cell depletion 24 hours pre-acute myocardial infarction significantly improved infar
265 if they are 18 years or older, admitted with acute myocardial infarction (ST-segment-elevation myocar
266 uclear cell (BM-MNC) therapy in ST-elevation acute myocardial infarction (STEMI) has no biological in
267 tios (HR) for the composite outcome of AIDS, acute myocardial infarction, stroke, end-stage renal dis
268 ovascular mortality, coronary heart disease, acute myocardial infarction, stroke, heart failure, and
269 e risk of cardiovascular diseases, including acute myocardial infarction, stroke, peripheral artery d
271 Prasugrel and Ticagrelor in the Treatment of Acute Myocardial Infarction) study did not find any sign
272 During Percutaneous Coronary Intervention in Acute Myocardial Infarction], TASTE [Thrombus Aspiration
273 rction contribute to the mortality burden of acute myocardial infarction, the share of these deaths t
274 64 for mortality among patients admitted for acute myocardial infarction to 2615 for mortality among
275 patients with subclinical hypothyroidism and acute myocardial infarction, treatment with levothyroxin
278 Four strategies for very early rule-out of acute myocardial infarction using high-sensitivity cardi
279 LTS We identified all patients admitted with acute myocardial infarction using the California State I
280 emia, coagulopathy, obesity, major bleeding, acute myocardial infarction, vascular complications, and
281 additional criteria required for spontaneous acute myocardial infarction versus those patients who do
283 s the case-fatality rate of patients in whom acute myocardial infarction was a primary diagnosis.
285 hin the 28 days preceding death, and whether acute myocardial infarction was one of the recorded diag
287 ire administered 30 days after discharge for acute myocardial infarction, wherein higher scores repre
288 ot been clearly established in patients with acute myocardial infarction who are undergoing percutane
289 PCI rates and in mortality for patients with acute myocardial infarction who did not undergo PCI.
290 ne oxygen therapy in patients with suspected acute myocardial infarction who do not have hypoxemia at
291 ranslation into treatments for patients with acute myocardial infarction, who typically are of advanc
292 ents (57 years +/- 12; 78% men) with a first acute myocardial infarction, who were prospectively enro
293 This study recruited 3,959 patients with acute myocardial infarction with a median and minimum fo
295 had multivessel coronary artery disease and acute myocardial infarction with cardiogenic shock, the
296 distinct kinetics for PCSK6 in patients with acute myocardial infarction, with a peak on postinfarcti
297 s been investigated in patients suffering an acute myocardial infarction, with the final aim of salva
299 r Use and Mortality in Hospital Survivors of Acute Myocardial Infarction Without Heart Failure; NCT02
300 ) and by qualifying event type (range: 7.1% [acute myocardial infarction without procedure] to 55.3%