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1 naling is dispensable for the development of acute myocarditis.
2 ated genetic variants in 8% of patients with acute myocarditis.
3 n a population-based cohort of patients with acute myocarditis.
4 entify a novel microRNA for the diagnosis of acute myocarditis.
5 e (CMR) imaging is commonly used to diagnose acute myocarditis.
6 se Criteria is used to make the diagnosis of acute myocarditis.
7 lar magnetic resonance to detect and monitor acute myocarditis.
8 ng showed high accuracy for the diagnosis of acute myocarditis.
9 rence standard was the clinical evidence for acute myocarditis.
10 enhanced T1 shortening for the diagnosis of acute myocarditis.
11 R approaches in patients suspected of having acute myocarditis.
12 and global myocardium edema in patients with acute myocarditis.
13 s T. cruzi control and protection from fatal acute myocarditis.
14 acute from chronic MI and identification of acute myocarditis.
15 L-1beta, and IL-6 levels in the heart during acute myocarditis.
16 ting that IL-17A plays a minimal role during acute myocarditis.
17 min in cardiac myocytes, results in a lethal acute myocarditis.
18 rdiovirulent phenotype in a murine model for acute myocarditis.
19 ng over 12 months in patients with suspected acute myocarditis.
20 nt myocarditis, and 132 met the criteria for acute myocarditis.
21 lminant myocarditis compared with those with acute myocarditis.
22 ction at six months compared with those with acute myocarditis.
23 f interferon, may determine reovirus-induced acute myocarditis.
24 minant (fractional shortening 19 +/- 4%) and acute myocarditis (17 +/- 7%) had LV systolic dysfunctio
25 ompared with no improvement in patients with acute myocarditis (19 +/- 7%, p < 0.01 for interaction b
26 Eighteen patients with clinical diagnosis of acute myocarditis (25 years [23-38 years]; 78% males) we
27 compared with only 45 percent of those with acute myocarditis (95 percent confidence interval, 30 to
28 ction is one of the most prevalent causes of acute myocarditis, a disease that frequently is identifi
29 nfection is one of the most common causes of acute myocarditis, a serious and sometimes fatal disease
30 Coxsackievirus B3 (CVB3) is a major cause of acute myocarditis, a serious condition that is refractor
33 tis (pAM), and expert consensus criteria for acute myocarditis (AM) and complicated myocarditis (CM).
35 e is observed not only in most patients with acute myocarditis (AM) but also in some oncology patient
39 enhancement (LGE) has not been clarified in acute myocarditis (AM) with preserved left ventricular (
40 ome sequencing of 42 unrelated children with acute myocarditis (AM), some with proven viral causes.
42 mines the incidence and clinical outcomes of acute myocarditis among adults following mRNA vaccinatio
44 +/- 19 [SD]; 46 men) with clinically defined acute myocarditis and 42 healthy controls who underwent
46 receiving alpha-MyHC-pulsed bmDCs developed acute myocarditis and accumulated interferon (IFN)-gamma
50 ackie-B-viruses (CVB) are frequent causes of acute myocarditis and dilated cardiomyopathy, but an eff
51 curacy to discriminate between patients with acute myocarditis and healthy controls was 86% for T2>52
53 among unselected patients hospitalized with acute myocarditis and their first-degree relatives compa
54 ata from ITAMY (ITalian multicenter study on Acute MYocarditis) and evaluated CMR results from 386 pa
60 cted with Brazil strain of T cruzi developed acute myocarditis by day 21 after infection, consisting
62 role of viral RNA synthesis in induction of acute myocarditis by infecting primary cultures of cardi
64 cate that Th1-type immunity protects against acute myocarditis by reducing viral replication and prev
65 acute myocardial infarction associated with acute myocarditis caused by coronavirus 229E in a middle
66 mouse models with opposite susceptibility to acute myocarditis caused by the myotropic Colombiana str
67 lammatory microRNA-155 is upregulated during acute myocarditis, contributes to the adverse inflammato
69 ) had the highest diagnostic performance for acute myocarditis diagnosis (P < .05 against all other s
71 ammation in 2/17 participants diagnosed with acute myocarditis early after COVID-19 vaccination, but
72 ased cohort of 336 consecutive patients with acute myocarditis enrolled in London and Maastricht.
73 performed: first, to identify patients with acute myocarditis for the 3-month period between Februar
75 e-associated myocarditis to 14 patients with acute myocarditis from other causes, we found that patie
77 el microRNA for distinguishing patients with acute myocarditis from those with myocardial infarction
78 /- 0.2 cm) at presentation, while those with acute myocarditis had increased diastolic dimensions (6.
79 s and severe hemodynamic compromise, whereas acute myocarditis has an indistinct presentation, less s
81 t in the human heart long after the signs of acute myocarditis have abated are still not completely u
82 tions regarding athletic participation after acute myocarditis have heightened the importance of earl
83 tients with COVID-19 vaccination- associated acute myocarditis have higher left ventricular ejection
84 ng findings were found to be consistent with acute myocarditis in 7 patients; 4 of whom had preceding
86 consistently and strongly upregulated during acute myocarditis in both humans and susceptible mice.
87 among the most commonly identified causes of acute myocarditis in children and adults and have been i
88 irus B3 (CVB3) is a principal viral cause of acute myocarditis in humans and has been implicated in t
89 erent reovirus reassortant viruses to induce acute myocarditis in mice correlates with cytopathogenic
93 enation for 147 patients with a diagnosis of acute myocarditis in the Extracorporeal Life Support Org
95 dition, we demonstrate that the induction of acute myocarditis involves the engagement of CD43 cytopl
100 omatic and asymptomatic participants without acute myocarditis.Keywords: Myocarditis, Vaccination, CO
103 localized granzyme B activity in hearts with acute myocarditis monitored by fluorescent molecular tom
107 ugh both T2 and T1 mapping reliably detected acute myocarditis, only T2 mapping discriminated between
108 y 2001 and November 2016 with a diagnosis of acute myocarditis (onset of symptoms <1 month) of whom 5
111 ican Heart Association criteria for probable acute myocarditis (pAM), and expert consensus criteria f
112 association of the mRNA-based vaccines with acute myocarditis, predominantly among male adolescents
113 roximately 82% to 95% of adult patients with acute myocarditis present with chest pain, while 19% to
115 Functional Imaging, Feature-Tracking Strain, Acute Myocarditis Supplemental material is available for
116 s were significantly longer in patients with acute myocarditis than in control subjects (1185.3 msec
117 rotozoan parasite Trypanosoma cruzi leads to acute myocarditis that is accompanied by autoimmunity to
119 hile viral RNA synthesis is a determinant of acute myocarditis, this is not due to generation of infe
120 esting should be considered in patients with acute myocarditis to help reassure the majority while im
126 hophysiological mechanisms, and treatment of acute myocarditis were gained during the last years, no
127 bruary 1 and April 30, 2021, 7 patients with acute myocarditis were identified, of which 4 occurred w
129 Group B enteroviruses are common causes of acute myocarditis, which can be a precursor of chronic m
130 y T1 shortening of 70% or greater identified acute myocarditis with 93% sensitivity, 100% specificity
131 ession in plasma obtained from patients with acute myocarditis with the expression in various control
132 n was different between myocardial layers in acute myocarditis, with midmyocardial GCS and subepicard