戻る
「早戻しボタン」を押すと検索画面に戻ります。 [閉じる]

コーパス検索結果 (１語後でソート)

通し番号をクリックするとPubMedの該当ページを表示します
1  of completion of radiotherapy (the early or acute period).
2 s who were followed clinically into the post-acute period.
3 ntial morbidity in individuals surviving the acute period.
4 N-gamma, MCP, IP-10, and Spike IgG) over the acute period.
5 atigue, and respiratory symptoms in the post-acute period.
6 imens were serially collected throughout the acute period.
7                                       In the acute period, 57%, 80%, 46%, and 43% of patients were wi
8 cantly higher in the olanzapine group in the acute period (74% v 52%; P < .001), delayed period (74%
9 s in the olanzapine group achieved CR in the acute period (78% v 59%; P = .001), delayed period (74%
10 otential of NP-based TBI theranostics in the acute period after injury.
11  tested the role of neuroinflammation in the acute period after injury.
12                           US findings in the acute period after IRE are dynamic and evolve.
13 obtain polysomnographic recordings during an acute period after life-threatening experiences and inju
14  that PD-1 expression was upregulated in the acute period after stroke.
15  C5a-C5aR axis thus appears injurious in the acute period but serves a protective and/or reparative r
16    Many stroke survivors with aphasia in the acute period experience spontaneous recovery within the
17  debridement should be avoided in the early, acute period (first 2 weeks), as it has been associated
18 salivary EVs as a potential biomarker in the acute period following head injury to identify injury se
19 87 337) sought medical attention in the post-acute period for one or more new or persistent clinical
20 line period (pre-TB) and particularly in the acute period: incident rate ratios were US, 3.5 (95% con
21 nd on the contralateral SI cortex during the acute period (&lt; or =11 h) after collagenase-induced ICH.
22 mptomatic necrotic collections in the early, acute period (&lt;2 weeks), and in those with WON who are t
23 e last decades for patients who survived the acute period of AMI.
24 g is reversible at several stages within the acute period of brain injury, and that these key factors
25  (CPE) in tissue culture characterized by an acute period of cell death and viral DNA accumulation.
26  a gradual decrease in amplitude during this acute period of ICH.
27 n a wide variety of bodily fluids during the acute period of illness but that the risk of transmissio
28 cy virus type 1 (HIV-1) treatment during the acute period of infection can significantly limit the se
29                                   During the acute period of infection there was a close correlation
30 fused bnAbs and treatment failure during the acute period of infection.
31 r promoting rebound sociability following an acute period of isolation.
32 fused bnAbs and the treatment failure in the acute period of SHIV infection and may have important im
33 itory effects were largely attenuated in the acute period of SHIV infection.
34  (180,000 vs. 44,000 ng/mL), during the most acute period of the infection (6-8 days after onset).
35 ional epigenetic modification in response to acute periods of starvation.
36                       Pupil damage predicted acute-period OHT (P = .004).
37 ptive immune responses in the acute and post-acute period requires in-depth mechanistic analyses to i
38     Surprisingly, VEEG recordings during the acute period showed that a substantial fraction of anima
39                                       In the acute period, significantly more patients in the RMG gro
40                                          The acute period was defined as 3 months before/after TB dia