ライフサイエンスコーパス: acute stroke

1 patients (medical patients and patients with acute stroke).

2 figurations of motor network connectivity in acute stroke.

3 d specific features of favorable recovery in acute stroke.

4 either symptomatic carotid stenosis or major acute stroke.

5 ent of the DASH as an upper limb measure for acute stroke.

6 ee trials of aspirin versus control in major acute stroke.

7 glycemia aggravates ischemic brain damage in acute stroke.

8 utcome in patients with hyperglycemia during acute stroke.

9 to intravenous thrombolysis in patients with acute stroke.

10 g pneumonia in patients with dysphagia after acute stroke.

11 ional outcome and mortality in patients with acute stroke.

12 ively affects motor learning and severity of acute stroke.

13 hospital-acquired pneumonia in patients with acute stroke.

14 on (MA) constitutes a key tissue response in acute stroke.

15 ls may be sensitive to perfusion deficits in acute stroke.

16 omy with or without intravenous alteplase in acute stroke.

17 lowing delayed intravenous administration in acute stroke.

18 rain-language relationships in speakers with acute stroke.

19 f the evidence to guide BP management during acute stroke.

20 (COVID-19) on the management and outcomes of acute stroke.

21 ood pressure (BP) and patient outcome during acute stroke.

22 tic information for patients presenting with acute stroke.

23 patients who were admitted with symptoms of acute stroke.

24 clinical decision-making in the treatment of acute stroke.

25 g children and young adults hospitalized for acute stroke.

26 decision-making in the clinical treatment of acute stroke.

27 spitalizations, 16,694 (0.045%) women had an acute stroke.

28 or the purpose of lesion-behavior mapping in acute stroke.

29 ietin (EPO) is a potential new treatment for acute stroke.

30 possible novel mode of metabolic imaging in acute stroke.

31 for platelets, is elevated in patients with acute stroke.

32 th GCS in patients admitted to hospital with acute stroke.

33 the extent of ischemic injury in an event of acute stroke.

34 herapies with and without adjuvants to treat acute stroke.

35 in the setting of recanalization therapy for acute stroke.

36 ompare CT and MRI for emergency diagnosis of acute stroke.

37 (56 of 217; 20-32%) for the diagnosis of any acute stroke.

38 to allow improved treatment of patients with acute stroke.

39 ferred for emergency assessment of suspected acute stroke.

40 ccurate diagnosis of patients with suspected acute stroke.

41 pulmonary embolism, sepsis, myocarditis, and acute stroke.

42 hibition is a promising treatment option for acute stroke.

43 dimensions of recovery and disability after acute stroke.

44 abolism is a hallmark of ischaemic injury in acute stroke.

45 tantially diminished to a level of 38% after acute stroke.

46 euroradiologist evaluation in the setting of acute stroke.

47 e focal hypoattenuation at brain CT in early acute stroke.

48 s presenting to hospital with a diagnosis of acute stroke.

49 he prevalence of associated risk factors for acute stroke.

50 edes ascending blood flow) in the context of acute stroke.

51 ty of thyroid hormone levels in prognosis of acute stroke.

52 stem, and represents a promising therapy for acute stroke.

53 nvestigation of fast high-resolution MRSI in acute stroke.

54 vorable versus poor recovery from neglect in acute stroke.

55 ) to evaluate its measurement performance in acute stroke.

56 nities to effectively intervene in and treat acute strokes.

57 plaques were identified in 20 patients with acute stroke (21 [27%] culprit, 12 [15%] probably culpri

58 ments from six other trials of alteplase for acute stroke (2775 patients).

59 ower-extremity peripheral artery disease and acute stroke (35% and 24%, respectively), whereas most v

60 Of 63 650 patients admitted with acute stroke, 55 838 (88%) had a dysphagia screen, and 2

61 tal-based cohort of patients presenting with acute stroke, acid-suppressive medication use was associ

62 Of 16 202 acute stroke admissions, 19.1% were transferred.

63 study cohort comprised 74,307 patients with acute stroke admitted to 199 hospitals.

64 Consecutive patients with acute stroke admitted to a stroke unit between April 200

65 Infection is a major complication of acute stroke and causes increased mortality and morbidit

66 ent may contribute to preventing deaths from acute stroke and could be implemented even in settings w

67 ving the functional outcome in patients with acute stroke and hyperglycemia.

68 impact and complicate the treatments used in acute stroke and in secondary prevention.

69 Raised blood pressure is common after acute stroke and is associated with an adverse prognosis

70 course to advance thrombolytic treatment for acute stroke and promises to improve outcomes in acute s

71 to investigate the incidence and outcomes of acute stroke and transient ischemic attack during pregna

72 tissue characterization in diseases such as acute stroke and tumor.

73 Sixteen patients with acute stroke and two with head trauma who had undergone

74 ere admitted to hospital within 1 week of an acute stroke and who were immobile were enrolled from 64

75 acute myocardial infarction, heart failure, acute stroke, and dialysis) were identified and meta-ana

76 tes: well, acute myocardial infarction (MI), acute stroke, and history of MI or stroke.

77 Although patients with acute stroke are routinely evaluated for potential treat

78 Therapeutic options for acute stroke are still limited.

79 Up to 50% of patients with acute stroke are taking antihypertensive drugs on hospit

80 atients (mean age = 63.4 +/- 9.0 years) with acute strokes attributed to high-grade (>/=70%) intracra

81 ional, single-arm study of real-world MT for acute stroke because of anterior-circulation large-vesse

82 these hypotheses by evaluating patients with acute stroke before reorganization of structure-function

83 ss following mechanical thrombectomy (MT) in acute stroke but is undetermined whether the two scores

84 Patients without clinical evidence of acute stroke but with magnetic resonance imaging evidenc

85 gh blood pressure is a prognostic factor for acute stroke, but blood pressure variability might also

86 ve been implicated in the pathophysiology of acute stroke, but the role of mitochondrial DNA (mtDNA)

87 ifficult in some cases because patients with acute stroke can present with atypical or uncommon sympt

88 (Carotid Plaque Imaging in Acute Stroke [CAPIAS]; NCT01284933).

89 A centralized model for acute stroke care across an entire metropolitan city app

90 and magnitude of variation in the quality of acute stroke care across the entire week.

91 actors, and equity of access to high-quality acute stroke care and rehabilitation will probably reduc

92 tors and, thus, the likely effect of optimum acute stroke care and secondary prevention in reducing t

93 vention that can have an immediate impact on acute stroke care in a region.

94 ently being used to give alteplase and guide acute stroke care in eight rural community hospitals in

95 Studies examining the impact of organised acute stroke care interventions on survival in subgroups

96 f a range of evidence-based interventions of acute stroke care on one year survival post-stroke and d

97 osite measure of hospital-level adherence to acute stroke care performance metrics, stroke volume, an

98 etwork is an effective way to extend quality acute stroke care to remote hospitals and to improve pat

99 ratio (OR) for receiving five indicators of acute stroke care was 0.81 (95% CI 0.72 to 0.92).

100 2010 a new multiple hub-and-spoke model for acute stroke care was implemented across the whole of Lo

101 ke, with the aim of ensuring access to rapid acute stroke care.

102 ed adjusted odds ratios for 13 indicators of acute stroke-care quality by fitting multilevel multivar

103 In the workup of acute stroke, carotid MPRAGE-positive signal was associa

104 Acute stroke causes complex, pathological, and systemic

105 In patients with acute stroke, certain findings accurately increase or de

106 urtosis (MK) was sensitive to hyperacute and acute stroke changes, and exhibited different contrast t

107 lights a potential complementary role in the acute stroke clinical decision-making process.

108 h similarities and points of divergence with acute stroke clinical trial design.

109 spheres in 7 patients with diabetes after an acute stroke compared with 12 stroke patients without di

110 172 people with acute stroke completed the DASH.

111 ur methods are applied to the analysis of an acute stroke dataset collected from multiple hospitals,

112 hospital mortality among pregnant women with acute stroke decreased (5.5% in 2007 vs. 2.7% in 2015; p

113 ustrate the essential role of reperfusion in acute stroke, delineating aspects of arterial revascular

114 The rates of acute stroke did not change (42.8 per 100,000 hospitaliz

115 telet Transfusion Versus Standard Care After Acute Stroke Due to Spontaneous Cerebral Hemorrhage Asso

116 Acute stroke due to supratentorial intracerebral haemorr

117 ty and efficacy of endovascular treatment of acute strokes due to vertebrobasilar artery occlusion.

118 s deployed when the dispatchers suspected an acute stroke during emergency calls.

119 d approaches to prevention and management of acute stroke during pregnancy and puerperium are warrant

120 Acute stroke during pregnancy and puerperium was associa

121 Acute stroke during pregnancy or within 6 weeks of child

122 is one of the main causes of mortality after acute stroke, early dysphagia assessment may contribute

123 e stroke; the use of supplementary oxygen in acute stroke; early or first aid use of aspirin for ches

124 CS use were available for 1971 patients with acute stroke enrolled into ENOS from February 2003 to Ap

125 euroimaging is an essential component of the acute stroke evaluation.

126 Hyperglycemia is common in patients with acute stroke, even in those without preexisting diabetes

127 icant effect on any outcome in patients with acute stroke except for an increase in major bleeding ev

128 Most patients with acute stroke exhibited some degree of preventability.

129 ovider capabilities and address shortages of acute stroke expertise nationwide.

130 robust evidence that screening patients with acute stroke for dysphagia reduces the risk of stroke-as

131 nosed SAP in 1088 patients who had dysphagic acute stroke from 37 UK stroke units between 21 April 20

132 ents was conducted within 3 months following acute stroke from July 9, 2003, to October 1, 2007.

133 ardial infarction, congestive heart failure, acute stroke, gastrointestinal hemorrhage, hip fracture,

134 The slow progress in thrombolysis for acute stroke has been multifactorial.

135 lthough the inflammatory nature of M-MPhi in acute stroke has been well documented, their role during

136 Reperfusion therapy for acute stroke has evolved from the initial use of intrave

137 from 1995 through 2012 were used to analyze acute stroke hospitalization rates among adults aged 18

138 Trends in acute stroke hospitalization rates by stroke type, age,

139 The aim of this study was to determine acute stroke hospitalization rates for children and youn

140 Acute stroke hospitalizations were identified by the pri

141 on are associated with worse outcomes during acute stroke; however, the optimal hemodynamic parameter

142 Amongst 40 patients with moderate to severe acute stroke imaged up to 26 h after onset, lactate conc

143 Acute stroke impairments often result in poor long-term

144 While we have assessed 8 key acute stroke impairments, this study is limited by a foc

145 trends over time in the prevalence of these acute stroke impairments.

146 long-term mortality and cause of death after acute stroke in adults aged 18 through 50 years and to c

147 he safety and efficacy of thrombolysis after acute stroke in children have not been established.

148 ctive cohort study of patients admitted with acute stroke in England and Wales.

149 n is still controversial in the treatment of acute stroke in hypertensive patients.

150 e stroke and promises to improve outcomes in acute stroke in the near future.

151 to optimise the treatment and prevention of acute stroke in these much older people will increasingl

152 arker models to assist with the diagnosis of acute stroke in those with mild symptoms.

153 The role of carotid artery stenting in acute stroke, including its use in the intracranial circ

154 M), reduced infarct size by 25% following an acute stroke induced by MCA occlusion for 90 min.

155 as it may yield new therapeutic targets for acute stroke injury and other neurological diseases invo

156 systems of care to provide timely access to acute stroke intervention to patients in the United Stat

157 Hyperglycaemia after acute stroke is a common finding that has been associate

158 Acute stroke is a serious concern in emergency departmen

159 lateral circulation in patients suspected of acute stroke is afforded by a combination of PCT and CTA

160 using tissue plasminogen activator (tPA) in acute stroke is associated with increased risks of cereb

161 resonance imaging (MRI) for the diagnosis of acute stroke is increasing, this method has not proved m

162 Whether the outcome of acute stroke is influenced by Ag-specific activation of

163 trategy could dramatically transform the way acute stroke is managed in the United States.

164 Correct diagnosis of acute stroke is of paramount importance to clinicians to

165 The sole FDA approved treatment for acute stroke is tissue type plasminogen activator (tPA).

166 CAPIAS (Carotid Plaque Imaging in Acute Stroke) is an observational multicenter study that

167 ypoxia is common in the first few days after acute stroke, is frequently intermittent, and is often u

168 MRI detected acute stroke (ischaemic or haemorrhagic), acute ischaemi

169 s (aged >16 years) admitted to hospital with acute stroke (ischaemic or primary intracerebral haemorr

170 Temporal trends in acute stroke (ischemic and hemorrhagic)/transient ischem

171 Acute stroke-lesioned brains fail to generate the LFO si

172 cular CD4(+) T cells in the area surrounding acute stroke lesions, suggesting that IL-21-mediated bra

173 tients developed IPO concomitantly during an acute stroke-like episode.

174 Acute stroke management has been revolutionised by evide

175 In acute stroke management, time is brain.

176 thrombolysis and mechanical thrombectomy in acute stroke management.

177 in neonatal brain than in adult brain after acute stroke may have major implications for the treatme

178 fits of blood pressure-lowering treatment in acute stroke might differ between patients with major is

179 to oxytocin before induction of experimental acute stroke model via oxygen-glucose deprivation-reperf

180 hms to predict ischemic tissue fate based on acute stroke MRI typically utilized data at a single tim

181 After acute stroke, multiparous mice had smaller infarcts, les

182 The most common misses were acute stroke (n = 3), aneurysm (n = 3), vascular occlusi

183 tion]) with cerebrovascular ischemic events (acute stroke, n = 20; subacute stroke, n = 2; chronic st

184 lysis of pregnancy-related hospitalizations, acute stroke occurred in 1 of every 2,222 hospitalizatio

185 Conclusion In acute stroke of the middle cerebral artery, the Alberta

186 Acute stroke or hemorrhage, or delayed radiographic prog

187 on in a subgroup analysis of patients in the Acute Stroke or Transient Ischaemic Attack Treated with

188 d with a ST-elevation myocardial infarction, acute stroke, or trauma; had a cardiac arrest prior to a

189 me and should be considered the main goal in acute stroke patient management.

190 s were validated in an independent cohort of acute stroke patients (n = 101) using model-based predic

191 ine data from National Scottish datasets for acute stroke patients admitted between 2005 and 2011.

192 We enrolled 62 acute stroke patients admitted to our stroke unit during

193 atine tonsils and cervical lymph nodes of 28 acute stroke patients and 17 individuals free of neurolo

194 lial fibrillary astrocytic protein (GFAP) in acute stroke patients and healthy controls and investiga

195 Because an increasing number of acute stroke patients are treated with tPA, it is import

196 for deep vein thrombus (DVT) prophylaxis in acute stroke patients before and after publication of th

197 inally included with a total number of 3,936 acute stroke patients for analysis.

198 Interhospital transfer of acute stroke patients is becoming increasingly important

199 ed a multicenter cohort study of consecutive acute stroke patients scheduled to undergo endovascular

200 ntation performance, we found that of the 18 acute stroke patients tested, only the four patients wit

201 Acute stroke patients were categorized into "acute carot

202 Ninety acute stroke patients who previously received aspirin th

203 ion scans, predict 27-hour infarct volume in acute stroke patients who were treated with reperfusion

204 volving pharyngeal electrical stimulation in acute stroke patients with dysphagia, an individual pati

205 We recruited 413 acute stroke patients, 161 (39%) experienced SAI.

206 In 14 acute stroke patients, volumetric agreement between auto

207 In acute stroke patients, we measured FC in a dorsal attent

208 inical severity and stroke infarct volume in acute stroke patients.

209 evious model of 30 local hospitals receiving acute stroke patients.

210 y, and the treatment has been used safely in acute stroke patients.

211 of autologous MNCs were safe and feasible in acute stroke patients.

212 to directly quantify collateral perfusion in acute stroke patients.

213 teral perfusion and delayed blood arrival in acute stroke patients.

214 e and manual actions in a large group of sub-acute, stroke patients (n = 740).

215 ecludes many higher-risk (acute ischemic and acute stroke) patients from undergoing MRI and MRI-guide

216 re are no clinically validated biomarkers of acute stroke, previous studies have focused on markers a

217 schemia was defined as posterior circulation Acute Stroke Prognosis Early CT score (pc-ASPECTS) < 8.

218 Children with acute stroke received alteplase infrequently and at time

219 We conclude that in patients with acute stroke receiving MT, success should be redefined a

220 te ischemic stroke patients (1,645 patients; Acute STroke Registry and Analysis of Lausanne registry)

221 ducted using data from the Georgia Coverdell Acute Stroke Registry between September 2005 and Septemb

222 participating in the Paul Coverdell National Acute Stroke Registry.

223 not subjected to stroke indicated that these acute stroke-related changes in vascular function could

224 st that, at least in a subgroup of patients, acute stroke-related headache might be centrally driven.

225 ty of these procedures is usually defined by acute stroke risk, it is also becoming clear that far mo

226 pressure with lisinopril and labetalol after acute stroke seems to be a promising approach to reduce

227 necteplase in larger trials of patients with acute stroke seems warranted.

228 udy of all patients older than 18 years with acute stroke seen in the emergency department or admitte

229 mimics are an important subgroup admitted to acute stroke services and have a distinct demographic an

230 esults The COMPASS study (Comprehensive Post-Acute Stroke Services) was a pragmatic cluster-randomize

231 setting and the symptomatic effects of hyper-acute stroke shaped the form, content and manner of know

232 After acute stroke, short-interval intracortical inhibition (S

233 of vasopressor drugs to treat hypotension in acute stroke should be limited to selective situations.

234 s were randomised less than 48 h after major acute stroke, stratified by severity of baseline neurolo

235 The European Cooperative Acute Stroke Study (ECASS) III demonstrated benefit to e

236 in the Enhanced Control of Hypertension and Acute Stroke Study (ENCHANTED) and the clinical predicto

237 (PH) was defined using European Cooperative Acute Stroke Study criteria.

238 ic in 9 (7%), and mild (European Cooperative Acute Stroke Study grades HI1 or HI2) in all but 1 child

239 anial hemorrhage (sICH, European Cooperative Acute Stroke Study II definition), 3-month mortality, an

240 95% CI = 0.82-1.70 per European Cooperative Acute Stroke Study II) after adjustment for age, stroke

241 s associated with sICH (European Cooperative Acute Stroke Study II) was evaluated, and we developed o

242 l Disorders and Stroke, European Cooperative Acute Stroke Study II, and Safe Implementation of Thromb

243 H was defined using the European Cooperative Acute Stroke Study III.

244 hemorrhage based on the European Cooperative Acute Stroke Study-II definition (any intracranial bleed

245 based, observational study that enrolled 100 acute stroke subjects.

246 bectomy (MT) is beneficial for patients with acute stroke suffering a large-vessel occlusion, althoug

247 ients older than 18 years who presented with acute stroke symptoms at one of four remote spoke sites

248 iage, and emergency treatment of people with acute stroke symptoms.

249 In the brain, cerebral SVD can cause an acute stroke syndrome known as lacunar stroke or more su

250 7 major North American academic centers with acute stroke teams.

251 pril are effective antihypertensive drugs in acute stroke that do not increase serious adverse events

252 Overall, the study showed in patients with acute stroke the presence of myelin and neuronal Ags ass

253 rease the effective use of thrombolytics for acute stroke, the expertise of vascular neurologists mus

254 Among nonhypoxic patients with acute stroke, the prophylactic use of low-dose oxygen su

255 hyperthermia and heatstroke; recognition of acute stroke; the use of supplementary oxygen in acute s

256 Using acute stroke therapy as an illustration, we present an e

257 ures to limit risk, are urgent challenges in acute stroke therapy research.

258 e treated with intravenous or intra-arterial acute stroke therapy while these treatments were provide

259 lasminogen activator remains the mainstay of acute stroke therapy within the initial 4.5 hours after

260 to benefit far more stroke patients than any acute stroke therapy, and represents the greatest opport

261 the ischemic core present novel dimension to acute stroke therapy, focused on ischemia and not just t

262 roke onset, which has initiated a new era of acute stroke therapy.

263 allowing more patients to be candidates for acute stroke therapy.

264 rcumvent the currently severe limitations of acute stroke therapy.

265 Among those with acute stroke, there were increases in prevalence of obes

266 ves endeavor to raise public awareness about acute stroke to improve triage for emergency treatment,

267 roved functional outcome among patients with acute stroke treated with stent retrievers.

268 marize what is known about the use of MRI in acute stroke treatment (predominantly thrombolysis), to

269 ogical classification (Trial of Org 10172 in Acute Stroke Treatment [TOAST]).

270 METHODS AND The HASTE (Hurry Acute Stroke Treatment and Evaluation) project was imple

271 While most research thus far has focused on acute stroke treatment and neuroprotection, the exploita

272 Additional strategies to increase acute stroke treatment are needed.

273 Acute stroke treatment has entered a golden age, and man

274 Although the efficacy of acute stroke treatment is time dependent, the use of fix

275 African Americans receive acute stroke treatment less often than non-Hispanic whit

276 ification systems, TOAST (Trial of Org 10172 Acute Stroke Treatment) and causative classification of

277 Among TOAST (Trial of Org 10172 in Acute Stroke Treatment) subtypes, adjusted for age, sex,

278 he assessment of perfusion images as part of acute stroke treatment.

279 n, stroke risk factors, stroke severity, and acute stroke treatment.

280 and response of emergency medical services; acute stroke treatment; subacute stroke treatment and se

281 mic stroke (and TOAST [Trial of Org 10172 in Acute Stroke Treatment] subtypes) (effective sample size

282 e ischemic territory forms the basis of most acute stroke treatments.

283 uring the first 72 hours provided at 8 hyper-acute stroke units (HASUs) compared to the previous mode

284 Some patients admitted to acute stroke units are diagnosed as stroke mimics.

285 ional outcome and mortality in patients with acute stroke using a meta-analysis of the available evid

286 unction (TJ) changes during BBB breakdown in acute stroke, very little is known about the type of alt

287 ypoattenuation, sensitivity for detection of acute stroke was 48% (nonenhanced scans) and 70% (angiog

288 hrough 50 years, 20-year mortality following acute stroke was relatively high compared with expected

289 randomized clinical trial, 8003 adults with acute stroke were enrolled from 136 participating center

290 raphic protocol, 159 patients with suspected acute stroke were evaluated with both brain diffusion te

291 comorbidities among those hospitalized with acute stroke were identified by secondary ICD-9-CM codes

292 17 of whom had a final clinical diagnosis of acute stroke, were assessed.

293 e of these disorders occur immediately after acute stroke, whereas others can develop later, and yet

294 er-based interventional revascularization in acute stroke, which appears to be successful, has shifte

295 Among patients with acute stroke who had last been known to be well 6 to 24

296 The delivery of care to patients with acute stroke will also need to incorporate newly proven

297 Improving outcomes for acute stroke will require patient education to encourage

298 We compared a group of 20 patients with acute stroke with anterior temporal pole damage to a gro

299 o organised stroke unit care for people with acute stroke, with active physiological monitoring and p

300 may be elevated in patients with sepsis and acute stroke without underlying acute coronary syndrome,