ライフサイエンスコーパス: acute-phase reactant

1 CRP (C-reactive protein) is a prototypical acute phase reactant.

2 r of the pentraxin family of proteins and an acute phase reactant.

3 s a unique serum pentraxin and the prototype acute phase reactant.

4 remia, consistent with hepcidin's role as an acute-phase reactant.

5 nate immune recognition molecule and classic acute-phase reactant.

6 monstrating that human hepcidin is a type II acute-phase reactant.

7 pain, HAQ, patient's global assessment, and acute-phase reactant.

8 ssment Questionnaire (HAQ), and levels of an acute-phase reactant.

9 oncentration and glycosylation state of main acute phase reactants.

10 erum levels of proinflammatory cytokines and acute phase reactants.

11 uring each period and measured for levels of acute-phase reactants.

12 signs and symptoms of RA and normalized the acute-phase reactants.

13 erum levels of proinflammatory cytokines and acute-phase reactants.

14 ng cell and by producing cytokines and other acute-phase reactants.

15 us MMP-2 inhibitors such as TIMPs and/or the acute phase reactant alpha-2-macroglobulin.

16 r factor (NF)-kappaB or transcription of the acute phase reactant alpha1-acid glycoprotein (AGP), wou

17 Chronic elevation of systemic levels of acute phase reactants and inflammatory cytokines found i

18 r (PPAR) signalling and lipid metabolism, of acute phase reactants and of two adipocytokines, leptin

19 tory investigation revealed the elevation of acute phase reactants and strongly positive cytoplasmic

20 relationship of C-reactive protein (CRP), an acute-phase reactant and marker of systemic inflammation

21 ean concentrations of C-reactive protein, an acute phase reactant, and tissue plasminogen activator (

22 g syndrome (electrolytes and phosphate), and acute phase reactants, and recorded the nutritional ther

23 s with some findings such as fever, elevated acute phase reactants, and/or comorbidities could requir

24 ssion of GST isozymes, positive and negative acute-phase reactants, and antioxidant enzymes were dete

25 d a much greater lymphocytosis, elevation in acute-phase reactants, and fall in serum iron and albumi

26 eft hindpaw volumes, circulating leukocytes, acute-phase reactants, and urinary collagen crosslinks t

27 lipopolysaccharide binding protein (LBP), an acute phase reactant; and plasma levels of bactericidal-

28 ; persistence for mean of 2 months; elevated acute phase reactants; and laboratory (usually serologic

29 Associations between maternal Cu and acute-phase reactants (APRs) and infection status were a

30 of inflammation and vascular insufficiency, acute-phase reactants (APRs), and imaging including conv

31 cell count (WBC) and altered levels of other acute phase reactants are cardinal signs of inflammation

32 return when glucocorticoids are reduced, and acute phase reactants are increased less during flares i

33 arkers of inflammation such as cytokines and acute phase reactants are reliably elevated in a subset

34 Increases in acute phase reactants are typical of polymyalgia rheumat

35 Acute-phase reactants are, by far, the strongest determi

36 adipose tissue to the systemic elevation of acute phase reactants associated with chronic hyperglyce

37 Serum amyloid A (SAA) proteins are acute-phase reactant associated with high-density lipopr

38 te that adipose tissue expresses a number of acute phase reactants at high levels, including serum am

39 an indicator of functional ID that is not an acute-phase reactant, but challenges in its interpretati

40 of human neutrophils, it was found that the acute phase reactant C-reactive protein (CRP) activates

41 The human acute-phase reactant C-reactive protein (CRP) also prote

42 The acute-phase reactant C-reactive protein (CRP) is an impo

43 d high-density lipoprotein), serum levels of acute-phase reactants (C-reactive protein), erythrocyte

44 Serum levels of the acute-phase reactant, C-reactive protein (CRP), increase

45 Serum concentrations of the classic acute-phase reactants, C-reactive protein and serum amyl

46 est that LBP levels, which vary as LBP is an acute phase reactant, could be relevant to initiating in

47 Furthermore, levels of the acute-phase reactants ESR, hsCRP, and SAA were lower at

48 ammatory cytokine TNF-alpha and the positive acute phase reactant fibrinogen are also elevated in the

49 A small cluster included acute-phase reactant genes (SAA1, SAA2, and SAA2-SAA4).

50 serum protein that is >90% homologous to the acute-phase reactant haptoglobin (Hp).

51 Ferritin, an acute-phase reactant, has been found to be elevated in m

52 Because IL-22 induces the expression of acute phase reactants, IL-22RA2 may play an important ro

53 C-reactive protein (CRP) is the classic acute phase reactant in humans, with serum levels elevat

54 ChoP is the target of an acute phase reactant in serum, C-reactive protein (CRP),

55 hat is one of the most dramatically changing acute phase reactants in the circulation.

56 C-reactive protein (CRP), the prototypic acute-phase reactant in humans, is synthesized in liver

57 raxin, C-reactive protein (CRP), is a strong acute-phase reactant in man and an opsonin.

58 physiological importance of the prototypical acute-phase reactant in the etiology of the disease is u

59 ced by T cells and induces the production of acute-phase reactants in vitro and in vivo, suggesting i

60 nd diminished insulin sensitivity (increased acute phase reactants) in patients with histologically p

61 The function of this acute-phase reactant includes activation of complement a

62 use cutaneous SSc for 60 months and elevated acute-phase reactants, including those with ILD, receive

63 hypothesis that human fibrinogen (FBG) - an acute phase reactant - inhibits human MMP-2.

64 Elevation of acute phase reactants is common due to the inflammatory

65 ence that ceruloplasmin, a copper-containing acute phase reactant, is secreted by zymosan-activated U

66 Serum amyloid A protein (SAA) is an acute-phase reactant, known to mediate pro-inflammatory

67 rrelation overall between EMP counts and the acute-phase reactant levels in the febrile disease contr

68 Birmingham Vasculitis Activity Score and the acute-phase reactant levels in the patients with systemi

69 nt's and physician's global assessments, and acute-phase reactant levels).

70 All individual ASAS components, acute-phase reactant levels, and spinal mobility measure

71 ut does not completely normalize symptoms or acute-phase reactant levels.

72 esis in HD patients) and plasma levels of an acute phase reactant, lipopolysaccharide binding protein

73 C-reactive protein (CRP), an acute-phase reactant long considered merely an innocent

74 dult liver specific gene, and its role as an acute phase reactant may well be more diverse than previ

75 mor necrosis factor-alpha (TNFalpha) and the acute phase reactant orosomucoid-1 (ORM1).

76 proinflammatory syndrome with alterations in acute-phase reactants, particularly at doses >50 mg/ m2/

77 Etanercept also reduced levels of acute-phase reactants, particularly during asymptomatic

78 evels of C-reactive protein (CRP), a hepatic acute phase reactant, predict risk for coronary heart di

79 onse, C-reactive protein being the classical acute-phase reactant produced in response to tissue dama

80 C-reactive protein (CRP) is an acute phase reactant protein produced primarily by the l

81 Serum amyloid A (SAA) is a highly conserved acute phase reactant protein, and its concentration in s

82 manifested by elevated circulating levels of acute phase reactant proteins and pro-inflammatory cytok

83 Determination of types I and II acute phase reactant proteins, constitutive serum protei

84 2, 5, and 7 and serum glucose, electrolytes, acute phase reactant proteins, tumor necrosis factor alp

85 differential nasal airway response including acute-phase reactants proteins (fibrinogen, haptoglobin

86 serum ferritin is a secreted protein and an acute phase reactant regulated by inflammatory hormones

87 ator may be limited by the lack of available acute-phase reactant results at the time of the clinical

88 ntrations of their most sensitive endogenous acute phase reactants, SAA and serum amyloid P component

89 racterized by the systemic deposition of the acute-phase reactant serum amyloid A (SAA).

90 oid fibrils are derived from the circulating acute-phase reactant serum amyloid A protein (SAA), but

91 of extracellular aggregates derived from the acute-phase reactant serum amyloid A protein.

92 Hepatic induction of the acute-phase reactant serum amyloid A1 resulted in its in

93 tored closely, and persistent high levels of acute phase reactants should raise concerns about amyloi

94 tored closely, and persistent high levels of acute-phase reactants should raise concerns about amyloi

95 The circulating cytokine milieu (IL-6) and acute phase reactants such as SAA may reflect alteration

96 ocument the marked elevations of nonspecific acute-phase reactants, such as C-reactive protein and er

97 om the precursor protein serum amyloid A, an acute phase reactant synthesized excessively in the sett

98 hycardia, decreasing haemoglobin, increasing acute phase reactants tests, and multiorgan failure.

99 saccharide (LPS)-binding protein (LBP) is an acute phase reactant that may play a dual role in vivo,

100 C-reactive protein (CRP) is an acute-phase reactant that is positively associated with

101 C-reactive protein (CRP) is an acute-phase reactant that is positively correlated with

102 C-reactive protein (CRP) is an acute-phase reactant that is positively correlated with

103 A-SAAs are major acute-phase reactants, the in vivo concentrations of whi

104 Since D-dimer and fibrinogen are acute phase reactants, these data support the hypothesis

105 as a minimum, joint counts and levels of an acute-phase reactant to define remission.

106 Acute-phase reactants were elevated with worsening of sy

107 hepatic gene expression, but in one class of acute-phase reactants, which includes fibrinogen, IL-1be

108 We here show that ceruloplasmin (Cp), an acute phase reactant with important functions in iron ho

109 C-reactive protein (CRP) is an acute phase reactant with roles in innate host defense,

110 orrelation of symptoms, signs, and levels of acute-phase reactants with disease activity in approxima