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1 energy deplete (food restricted) or replete (ad libitum fed).
2 operated animals who were either pair-fed or ad libitum-fed.
3 mpared with saline controls in both the AGRP ad libitum fed (21 +/- 8% of saline control; P < 0.01) a
4 in the brain frontal cortex of 12-month-old ad libitum fed, 26-month-old ad libitum fed, and 26-mont
5 month-old ad libitum fed (6AL), 26-month-old ad libitum fed (26AL), and 26-month-old calorie-restrict
6 predominately Type II fiber), of 6-month-old ad libitum fed (6AL), 26-month-old ad libitum fed (26AL)
9 vely low dose of cocaine (7.0mg/kg, i.p.) in ad libitum fed (AL) and FR rats and take several brain r
11 motor activity did not differ between FR and ad libitum fed (AL) rats, while vertical activity was gr
13 n skin collagen could predict longevities in ad libitum-fed (AL) and caloric restricted (CR) mice.
14 mpared with saline controls in both the AGRP ad libitum fed and AGRP pair-fed groups (3.5 +/- 0.3 [sa
15 ment 2, these compounds were administered to ad libitum fed and food-restricted rats whose LHSS behav
17 ves to heat stress compared to thermoneutral ad libitum fed and thermoneutral feed-restricted counter
18 nd the adjacent cortical areas of 7 Control (ad libitum)-fed and 6 CR male rhesus macaques using immu
19 mRNA and protein expression were measured in ad libitum-fed and calorie-restricted rats at ages 2, 6,
23 nonobese model, we also treated prediabetic, ad libitum-fed and pair-fed Lean-huIAPP transgenic males
24 of 12-month-old ad libitum fed, 26-month-old ad libitum fed, and 26-month-old calorie-restricted (CR)
26 tly increased food intake and body weight in ad libitum fed animals compared with saline-treated cont
27 time showed a delayed adaptation compared to ad libitum fed animals, in terms of the similarity in 24
28 in the programmed feeding model, but not in ad libitum-fed animals, supports the concept that the pr
32 administered ROSI increases AgRP and NPY in ad libitum-fed animals; (4) whether intraperitoneally ad
33 ody temperature was significantly reduced in ad-libitum-fed ATF4 BKO mice, which correlated with Fgf2
34 y acids to fatty acids and ketones, and that ad libitum-fed carbohydrate-restricted diets lead to app
37 nsive rats (n=25) were assigned to either an ad libitum fed control group (AL) or food restricted gro
38 as occurring in 40% ER rats in comparison to ad libitum fed control rats or 40% ER rats that were ene
39 arcinomas were evaluated from rats that were ad libitum fed (control), 40% ER, or 40% ER but energy r
40 of age-related methylation drift compared to ad libitum-fed controls such that their blood methylatio
45 ve groups: ad libitum-fed sedentary control, ad libitum-fed exercise (AL+Exe), exercise but pair-fed
50 onsumed approximately 30% more calories than ad libitum-fed mice at 27 degrees C, but there was no di
53 ocyte-specific p53 ablation in sham-operated ad libitum-fed mice impaired glucose homeostasis, increa
55 in GST-II-positive hepatocytes, 24-month-old ad libitum-fed mice were introduced to 40% diet restrict
60 was 1-1.5% new cells per day, whereas obese ad libitum-fed obob mice exhibited markedly higher fract
65 l as NK cell activity, in the splenocytes of ad libitum-fed, pair-fed, and ethanol-fed Sprague Dawley
66 zed the Fischer 344 rat model of aging under ad libitum-fed (rapid aging) and calorie-restricted (slo
67 c injections of either glucose or insulin in ad libitum fed rats also resulted in an increase in ACh
68 changes in apolipoprotein B-lipoproteins in ad libitum fed rats and mice maintained in a 12-h photop
69 energy metabolism and pituitary function in ad libitum fed rats and rats administered AGRP and then
70 insulin treatment) elevation of threshold in ad libitum fed rats and, more transiently, reversed the
71 eriment 3, chronic i.c.v. leptin infusion in ad libitum fed rats decreased food intake and body weigh
76 imicking of these fasting-induced effects in ad libitum-fed rats after GLP-1 receptor antagonism sugg
77 we infused saline or leptin for 7 days into ad libitum-fed rats and compared these with saline-infus
78 ted by our finding that the drug response in ad libitum-fed rats and the deprivation response are exp
82 s: food-deprived rats given standard chow or ad libitum-fed rats given a palatable chocolate shake.
88 ssigned to one of the following five groups: ad libitum-fed sedentary control, ad libitum-fed exercis
89 ll intestine and colon than in the fasted or ad libitum-fed states, demonstrating that post-fast refe
90 (3.5 +/- 0.3 [saline] vs. 2.7 +/- 0.4 [AGRP ad libitum fed] vs. 2.1 +/- 0.2 ng/ml [AGRP pair-fed]; P