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1 This was due to an increase in the amount of adenine nucleotide translocase.
2 source, ATP depletion, or inhibition of the adenine nucleotide translocase.
3 tage-dependent anion channel (porin) and the adenine nucleotide translocase.
4 of porin, mitochondrial creatine kinase, and adenine nucleotide translocase.
5 hondria is known to be cardioprotective, and adenine nucleotide translocase 1 (ANT1) is a key mediato
7 ondria, to interact with and dephosphorylate adenine nucleotide translocase 1 (ANT1), a central molec
9 nd several mitochondrial proteins, including adenine nucleotide translocase 1 (ANT1), were more oxidi
11 eviously cloned cDNAs derived from the mouse adenine nucleotide translocase-1 and -2 genes (Ant1 and
13 his study sheds light on the central role of adenine nucleotide translocase 2 (ANT2) in the pathogene
14 olve saturated fatty acid stimulation of the adenine nucleotide translocase 2 (ANT2), an inner mitoch
16 us-purified voltage-dependent anion channel, adenine nucleotide translocase and the fusion protein we
17 bility transition pore that is formed by the adenine nucleotide translocase and the voltage-dependent
18 increased oxygen consumption independent of adenine nucleotide translocase and uncoupling proteins,
19 ctivity was insensitive to inhibitors of the adenine nucleotide translocase (ANT) and of the voltage-
20 ease of the apparent molecular weight of the adenine nucleotide translocase (ANT) by up to 1.2 kDa.
21 een mitochondrial creatine kinase (MtCK) and adenine nucleotide translocase (ANT) can play an importa
25 t the MPTP has two molecular components, the adenine nucleotide translocase (ANT) family of proteins
27 n maleimide is known to attack Cys159 of the adenine nucleotide translocase (ANT) in a CAT-sensitive
28 nding puzzle is that in permeabilized cells, adenine nucleotide translocase (ANT) is less accessible
31 oside and bongkrekic acid, inhibitors of the adenine nucleotide translocase (ANT) that lock the trans
33 mely voltage-dependent anion channel (VDAC), adenine nucleotide translocase (ANT), and hexokinase II
34 was attenuated by knockdown or inhibition of adenine nucleotide translocase (ANT), cyclophilin D (Cyp
35 trometry analysis identified this protein as adenine nucleotide translocase (ANT), represented by two
36 or alpha induced RIP-dependent inhibition of adenine nucleotide translocase (ANT)-conducted transport
37 ibits all ADP-ATP-using reactions except the adenine nucleotide translocase (ANT)-mediated mitochondr
42 rebellar glucocorticoid hormone receptor and adenine nucleotide translocase (ANT1) were downregulated
44 ds, these mitochondria also showed a greater adenine nucleotide translocase-catalysed proton conducta
47 d increased proton transport activity of the adenine nucleotide translocase (dependent on fatty acids
48 cle depended on electron transport chain and adenine nucleotide translocase functionality, but it was
49 This study provides evidence for a role of adenine nucleotide translocase in the mechanism underlyi
51 ependent anion channel (outer membrane), the adenine nucleotide translocase (inner membrane) and cycl
52 ge-dependent anion channel (outer membrane), adenine nucleotide translocase (inner membrane) and cycl
53 ation, increased levels of the mitochondrial adenine nucleotide translocase stress-sensitive B (SesB)
54 the voltage-dependent anion channel and the adenine nucleotide translocase were similar in the two m
55 ction of both the F(1)F(O)-ATPase and of the adenine nucleotide translocase, which delivers nucleotid
56 ation/knockdown-induced dysregulation in the adenine nucleotide translocase, which results in a slowe