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1 lymphoid tissue of Waldeyer's ring (tonsils/adenoids).
2 the surface of the nasopharyngeal tonsil or adenoid.
3 center and memory B cells in the tonsils and adenoids.
4 -MALT) such as Peyer's patches, tonsils, and adenoids.
5 diffuse lymphoid cells and lacks tonsils and adenoids.
6 oves only the palatine tonsils and sometimes adenoids.
7 infection in lymphocytes of the tonsils and adenoids.
8 the subepithelial zones of both tonsils and adenoids.
9 tory epithelium of nasopharyngeal tonsils or adenoids.
10 ng 806 RSV F-specific antibodies from paired adenoid and peripheral blood samples from 4 young childr
14 egions along the adenoid, tonsils, and where adenoid and tonsils overlap; and (3) 10 segments at 10%
15 versus 2.5 +/- 1.2 cm(3); p < 0.005) and the adenoid and tonsils were larger (9.9 +/- 3.9 and 9.1 +/-
16 , the upper airway is restricted both by the adenoid and tonsils; however, the soft palate is also la
18 ded T cell clonotypes were found in tonsils, adenoids and blood post-COVID-19, some with CDR3 sequenc
21 ial and intraepithelial lymphocytes of human adenoids and tonsils were characterized and directly com
24 immune and epithelial lineage, while tonsil, adenoid, and blood samples were analyzed using a 31-plex
25 primary epithelial cells derived from human adenoids, and human bronchial epithelium (NHBE cells).
30 s, paired tonsil-adenoid samples, and paired adenoid-blood samples, which may have implications for c
31 G, IgA, and IgM levels in nasal secretions, adenoid cell supernatants, and sera collected from 12 ch
33 r medullary carcinoma (57 patients [22.4%]), adenoid cystic carcinoma (653 patients [48.1%]), and met
34 rior for patients with medullary (91.7%) and adenoid cystic carcinoma (88.4%) compared with patients
35 systemic therapy for recurrent or metastatic adenoid cystic carcinoma (ACC) approved by the US Food a
43 , including squamous cell carcinoma (HNSCC), adenoid cystic carcinoma (ACC), and other salivary and c
44 , including squamous cell carcinoma (HNSCC), adenoid cystic carcinoma (ACC), and other salivary and c
45 inoma types: mucoepidermoid carcinoma (MEC), adenoid cystic carcinoma (ACC), and salivary duct carcin
47 x 4) is one of the most upregulated genes in adenoid cystic carcinoma (ACC), relative to non-neoplast
49 histology-based cohorts (32 patients each): adenoid cystic carcinoma (ACC; cohort 1) and other SGCs
53 ared with 62 (53-72) years for patients with adenoid cystic carcinoma and 63 (52-74) years for patien
55 patients (81%), followed by 1357 (16%) with adenoid cystic carcinoma and only 255 (3%) with medullar
56 7 and p53, and newer nuclear markers MYB for adenoid cystic carcinoma and PLAG1 for pleomorphic adeno
59 arate images of pancreatic, lung, colon, and adenoid cystic carcinoma organoids, which showed excelle
64 noma, tubular and solid/basaloid variants of adenoid cystic carcinoma, and a morphologically heteroge
66 se large B-cell lymphoma (n = 1)], one PR in adenoid cystic carcinoma, and one PR in HR-positive brea
67 on characteristics included: adenocarcinoma, adenoid cystic carcinoma, meningioma, chondrosarcoma and
78 increased frequency of NOTCH1 alterations in adenoid cystic carcinomas compared with previous studies
80 t the 7%-10% of prostate adenocarcinomas and adenoid cystic carcinomas reported to have homozygous de
82 gland neoplasms (11 pleomorphic adenomas, 10 adenoid cystic carcinomas, 5 mucopidermoid carcinomas, a
89 aracterize hTERT-immortalized HFKs and human adenoid epithelial cells (HAKs) for genotypic and phenot
90 4 and HIV-1 RNA were detected in tonsils and adenoids even in asymptomatic seropositive individuals.
91 ange bacterial primers, we demonstrated that adenoids from both diagnostic groups were colonized with
92 Haemophilus influenzae was present in more adenoids from the COM group (P = 0.005), but there was n
93 l airway problems (upper airway collapse and adenoid hypertrophy) and functional airway problems (lar
96 iosis: ICC, 0.27; hypertrophy of tonsils and adenoids: ICC, 0.26; supracondylar fracture of humerus:
97 oropharyngeal tonsils in one, nasopharyngeal adenoids in three, unilateral pharyngeal tonsil and ipsi
98 gue, soft palate, nasopharyngeal airway, and adenoid increased with age and maintained constant propo
101 depth and comprehensive understanding of the adenoid microbial communities and test the 'pathogen res
104 thelium is a major site of viral entry, with adenoid nasal-associated lymphoid tissue (NALT) generati
105 , we detected a high frequency of viruses in adenoids obtained from children without acute respirator
106 e children without OSA do not have increased adenoid or tonsillar volume; reduced upper airway size i
107 in a discrete region adjacent to either the adenoid or tonsils, but rather in a continuous fashion a
109 of the neutralizing antibodies isolated from adenoids originate from a unique population of IgM(+) an
110 Mitogen-triggered T cells from tonsils and adenoids produced both Th1- and Th2-type cytokines, clea
112 rched for in tissues of palatine tonsils and adenoids removed from patients without ARI symptoms.
114 aired nasal-bronchial samples, paired tonsil-adenoid samples, and paired adenoid-blood samples, which
115 sal neutralization was detected in nasal and adenoid samples, mostly from children who had previously
116 o investigate the bacterial community in the adenoid, samples were obtained from 35 children undergoi
117 treatment induced significant reductions in adenoid size and respiratory-related sleep disturbances,
119 luorescent imaging approaches on fresh human adenoid slices to provide static and dynamic information
120 pearman rho >= 0.87, P <= .0003 for all) and adenoid (Spearman rho >= 0.57, P <= .05 for all) neutral
122 ate that soft tissues, including tonsils and adenoid, surrounding the upper airway grow proportionall
123 C adenovirus DNA can be found in tonsil and adenoid T lymphocytes from the majority of pediatric don
125 1-R and LT2-R mRNA was similarly abundant in adenoid tissues, but increased LT1-R and LT2-R protein e
126 t quantities of adenovirus DNA in tonsil and adenoid tissues, infectious virus was rarely present, as
127 ibution and organization of pathogens in the adenoids to determine whether pathogenic bacteria exhibi
130 (1) the total airway; (2) regions along the adenoid, tonsils, and where adenoid and tonsils overlap;