ライフサイエンスコーパス: adenomatous polyp

1 ell that expands clonally to give rise to an adenomatous polyp.

2 urveillance to patients with newly diagnosed adenomatous polyps.

3 at screening and more aggressive removal of adenomatous polyps.

4 fter cardiac transplantation on the risk for adenomatous polyps.

5 id not correlate with an increased burden of adenomatous polyps.

6 al cancer relies in part on the detection of adenomatous polyps.

7 ctal cancer in the families of patients with adenomatous polyps.

8 30 primary colorectal adenocarcinomas and 24 adenomatous polyps.

9 scopic findings, and of these, 256 cases had adenomatous polyps.

10 mong 1,769 subjects, 520 (29%) had 1 or more adenomatous polyps.

11 early diagnosis and endoscopic resection of adenomatous polyps.

12 elated death globally, typically arises from adenomatous polyps.

13 tory was not associated with risk and age of adenomatous polyps.

14 as a non-invasive assay to diagnose CRC and adenomatous polyps.

15 olonoscopies that identified hyperplastic or adenomatous polyps.

16 ignificantly altered in AN participants with adenomatous polyps.

17 cer was prevented by colonoscopic removal of adenomatous polyps.

18 system for differentiating hyperplastic from adenomatous polyps.

19 cially in the context of hyperplastic and/or adenomatous polyps.

20 APC gene and individual risk of metachronous adenomatous polyps.

21 idence of colorectal cancer in patients with adenomatous polyps.

22 antly more biallelic carriers had coexisting adenomatous polyps.

23 d women aged 50-80 y with recently diagnosed adenomatous polyps.

24 ribute to the development and advancement of adenomatous polyps.

25 pression of haptoglobin in colon cancers and adenomatous polyps.

26 logically proven synchronous or metachronous adenomatous polyps.

27 tions correlated with the presence of >or=20 adenomatous polyps.

28 the incidence of disease through removal of adenomatous polyps.

29 well established for the precursor lesions, adenomatous polyps.

30 and vegetable consumption and first incident adenomatous polyps.

31 4 serrated adenomas, 3 admixed hyperplastic-adenomatous polyps, 10 tubular adenomas, and 6 carcinoma

32 e endoscopists had equivalent miss rates for adenomatous polyps (20% vs. 21%, respectively; P = 0.91)

33 rrated adenomas, 33% of admixed hyperplastic-adenomatous polyps, 30% of tubular adenomas, and 33% of

34 arcinomas (n=41), hyperplastic polyps (n=8), adenomatous polyps (=35), and adenocarcinomas (n=27), us

35 o underwent colonoscopy, 34.6% had 1 or more adenomatous polyps, 4.7% had 1 or more advanced adenomat

36 Of the 69 adenomatous polyps, 46 of the 51 that were 6 mm or more

37 TS polymorphisms in a case control study of adenomatous polyps (510 cases and 604 polyp-free control

38 es of 0.03, 0.03, and 0.04 for patients with adenomatous polyps 6 mm or larger, 6-9 mm, and 10 mm or

39 rgo altered expression in the APC(min) mouse adenomatous polyp, a precursor of cancer, as well as in

40 y of aspirin and statin in reducing advanced adenomatous polyp (AAP) risk in Black patients.

41 The primary outcome measure was advanced adenomatous polyps (AAPs).

42 ctal carcinomas are believed to originate as adenomatous polyps (adenomas), and the identification an

43 persons who have first-degree relatives with adenomatous polyps (adenomas).

44 characteristics of these tests for detecting adenomatous polyps, advanced adenomas based on size, or

45 iduals aged 45-49 years were found to harbor adenomatous polyps, aligning with trends observed in Wes

46 In adenomatous polyps, although DNMT1 expression coincided

47 Age over 60 years old, adenomatous polyp and heavy alcohol consumption may affe

48 Colonic epithelial cells in adenomatous polyps and adenocarcinomas had a decreased i

49 X-2) is aberrantly expressed in premalignant adenomatous polyps and colorectal carcinomas and is asso

50 are aberrantly overexpressed in premalignant adenomatous polyps and colorectal carcinomas of humans.

51 op consensus guidelines for the detection of adenomatous polyps and CRC in asymptomatic average-risk

52 bial communities that drive the detection of adenomatous polyps and CRC in rectal mucus.

53 Ashkenazi Jewish patients with a history of adenomatous polyps and in 1 (5%) of 20 Ashkenazi Jewish

54 Sulindac causes regression of precancerous adenomatous polyps and inhibits the growth of cultured c

55 ion of CBS in human biopsies of precancerous adenomatous polyps and show that forced upregulation of

56 primary chemoprevention trial who developed adenomatous polyps and then had eradication of polyps af

57 nomatous polyps, 4.7% had 1 or more advanced adenomatous polyps, and 5.7% had 1 or more serrated poly

58 sa from 13 healthy subjects, 9 patients with adenomatous polyps, and 9 with cancer.

59 present study of intact human colon crypts, adenomatous polyps, and adenocarcinomas focuses on subce

60 o assess the associations between NSAID use, adenomatous polyps, and apoptosis.

61 agreed that most colon cancers develop from adenomatous polyps, and it is this fact on which screeni

62 be prevented by the detection and removal of adenomatous polyps, and survival is significantly better

63 RCs with corresponding normal mucosa, and 52 adenomatous polyps (APs).

64 Siblings and parents of patients with adenomatous polyps are at increased risk for colorectal

65 Adenomatous polyps are by definition dysplastic and path

66 Findings from six studies of MTHFR C677T and adenomatous polyps are inconsistent.

67 Adenomatous polyps are precursors to colorectal cancer (

68 Adenomatous polyps are the most common neoplastic findin

69 ugs, designed to decrease the growth rate of adenomatous polyps, are very efficient at lowering colon

70 nguinity, who developed 10 carcinomas and 35 adenomatous polyps at age 23 and duodenal adenocarcinoma

71 Detection rates of adenomatous polyps at initial colonoscopy was higher in

72 ive value both for colorectal cancer and for adenomatous polyps at least 1 cm in diameter.

73 ) for detecting retrospectively identifiable adenomatous polyps at least 1 cm in size.

74 f genetic risk score (GRS) with detection of adenomatous polyps at screening colonoscopy.

75 y layers of the intestinal wall, cancer, and adenomatous polyps based on the REIMS fingerprint of eac

76 ccurred in 30% of adenocarcinomas and 34% of adenomatous polyps but not in hyperplastic polyps or nor

77 s elevated in Ashkenazi Jewish patients with adenomatous polyps, but not hyperplastic polyps.The I130

78 e migration of epithelial cells within human adenomatous polyps by determining the distribution of pr

79 esota Cancer Prevention Research Unit (CPRU) adenomatous polyp case-control study.

80 With a case-control design involving adenomatous polyp cases (n = 564), colonoscopy-negative

81 unoreactive cells was strikingly reversed in adenomatous polyps compared with normal mucosa.

82 ssion analysis, age over 60 years old, male, adenomatous polyp, current smoking and heavy alcohol con

83 Given the high rate of adenomatous polyp detection on colonoscopy, the amount o

84 Positive adenomatous polyp detection rate in patient's age > 40 y

85 Participants with large-bowel adenomatous polyps diagnosed in the past 6 mo were rando

86 ial appearances, eight had carcinoma, 24 had adenomatous polyps, five had hyperplasia, 11 had fibroid

87 he aim of this study was to evaluate colonic adenomatous polyps for microsatellite instability to det

88 errated adenomas, and 3 admixed hyperplastic-adenomatous polyps from 13 patients with hyperplastic po

89 s from the training set and 102 ICVs and 138 adenomatous polyps from the test set could be segmented

90 A total of 386 ICVs and 67 adenomatous polyps from the training set and 102 ICVs an

91 Adenomatous polyps had surface "grooves" or "sulci." Sen

92 sor lesions to colorectal cancer, colorectal adenomatous polyps have been studied to enhance knowledg

93 ate endoscopic prediction of hyperplastic or adenomatous polyp histology could reduce procedural time

94 of digital chromoendoscopy for prediction of adenomatous polyp histology was 90.1 %.

95 in 7 (6%), hyperplastic polyps in 4 (3.5%), adenomatous polyps in 2(2%), history of ulcerative colit

96 nic milieu as reflected by the high rates of adenomatous polyps in AN participants.

97 ction of IL10 and have increased colitis and adenomatous polyps in chemical and genetic models of col

98 , including folate, reduce the recurrence of adenomatous polyps in humans, the effect is small.

99 his referral center and higher prevalence of adenomatous polyps in men in accordance with western rep

100 Yet, adenomatous polyps in noncolitic colons are usually remo

101 asurement test for the diagnosis of CRCs and adenomatous polyps in plasma and stool samples in an Ira

102 GRS was significantly associated with adenomatous polyps in subjects undergoing screening colo

103 ted disease characterized by the presence of adenomatous polyps in the colon and rectum, with inevita

104 f energy from fat) diet on the recurrence of adenomatous polyps in the large bowel.

105 n-3 PUFAs are associated with lower risk of adenomatous polyps in women, and the association may be

106 s of human colorectal carcinoma (CRC) and in adenomatous polyps, indicating its suppression occurs ea

107 stores, and iron intake as risk factors for adenomatous polyps, intermediate markers for colorectal

108 urveillance colonoscopy for individuals with adenomatous polyps is based on adenoma histology, size,

109 er, compared with patients with a history of adenomatous polyps, is unknown.

110 Colorectal neoplasms (adenomatous polyps) missed at OC before VC results were

111 hat whereas Apc(Min)(/+) mice developed many adenomatous polyps, mostly in the small intestine, by 3

112 00), in those APC-negative patients with <20 adenomatous polyps (n = 26), or in those with CRC who we

113 Treg cells that expand in adenomatous polyps no longer produce IL-10 and instead s

114 Six patients (19%) had adenomatous polyps; no carcinomas were detected.

115 with a first-time diagnosis of at least one adenomatous polyp of the distal colon or rectum and 502

116 classified as having no evidence of disease, adenomatous polyps of less than 10 mm, of 10 mm or more,

117 It has been suggested that controls with adenomatous polyps of the colon and rectum should be exc

118 nosuppression does not increase the risk for adenomatous polyps of the colon.

119 racterized by the development of hundreds of adenomatous polyps of the colorectum.

120 mor has only been reported to originate from adenomatous polyps of the gastric mucosa in these clinic

121 In 2003-2005, cases with adenomatous polyps only (n = 639), hyperplastic polyps o

122 (95% confidence interval (CI): 1.4, 2.5) for adenomatous polyps only, 5.0 (95% CI: 3.3, 7.3) for hype

123 The study was based on 236 cases with adenomatous polyps or cancer and 409 controls, all colon

124 No differences in detection of adenomatous polyps or frequency of complications were fo

125 ate with sex, age, size, or dysplasia of the adenomatous polyps or with differentiation and Dukes' st

126 inations, but not for patients with low-risk adenomatous polyps (OR = 1.8; 95% CI, 0.9-3.7).

127 io [OR] = 3.1; 95% CI, 1.7-5.5) or high-risk adenomatous polyps (OR = 3.0; 95% CI, 1.2-8.0), compared

128 use aspirin were also not at reduced risk of adenomatous polyps (OR, 1.59; 95% CI, 0.68-3.73).

129 ie, no prior diagnosis of colorectal cancer, adenomatous polyps, or inflammatory bowel disease; no pe

130 ) carcinomas (P = 0.0001) and 12 of 24 (50%) adenomatous polyps (P = 0.0001).

131 also associated with early age diagnosis of adenomatous polyps, p < 0.001.

132 centrations and the prevalence of colorectal adenomatous polyps (precursors to colorectal cancer) in

133 wice daily and the Prevention of Spontaneous Adenomatous Polyps (PreSAP) trial used 400 mg once daily

134 The Adenomatous Polyp Prevention on Vioxx (APPROVe) study as

135 the hypothesis that colonoscopic removal of adenomatous polyps prevents death from colorectal cancer

136 These results imply that development of adenomatous polyps proceeds through a top-down mechanism

137 ion, proposed mechanisms of development from adenomatous polyps, rationale for screening, and screeni

138 ns were detected by direct sequencing in 738 adenomatous polyps removed at baseline from 639 particip

139 7; 95% confidence interval [CI], 1.64-6.47), adenomatous polyps (RR, 2.18; 95% CI, 1.18-4.61) and hea

140 Eighty percent of adenomatous polyps showed no instability.

141 copy and polypectomy because of a history of adenomatous polyps; standardized incidence ratios were 1

142 ed region in colorectal FAP: allelic loss in adenomatous polyps tended to occur when the germline mut

143 RS was significantly higher in subjects with adenomatous polyps than those without; mean (95% confide

144 sceral adipose tissue (VAT) in subjects with adenomatous polyps, the precursor lesion of colorectal c

145 dergoing complete colonoscopy and removal of adenomatous polyps; they remained in the study for appro

146 can detect cancer early and also can detect adenomatous polyps, thus providing a greater potential f

147 adenocarcinomas, including cases in which an adenomatous polyp was also present, by immunohistochemis

148 The detection rate of adenomatous polyps was 10.8%, 29.0% and 39.7% in subject

149 s 7.57 (95% CI 1.84-31.4) and with high-risk adenomatous polyps was 3.11 (1.86-5.18).

150 The sensitivity of optical colonoscopy for adenomatous polyps was 87.5 percent, 91.5 percent, and 9

151 0% (81.8-99.1), and for cancer and high-risk adenomatous polyps was 90.1% (84.4-94.0); specificity wa

152 The sensitivity of virtual colonoscopy for adenomatous polyps was 93.8 percent for polyps at least

153 The specificity of virtual colonoscopy for adenomatous polyps was 96.0 percent for polyps at least

154 orectal cancer (CRC) patients with normal or adenomatous polyps, we found that both the adenoma and c

155 ios for cancer, and for cancer and high-risk adenomatous polyps were also calculated.

156 Similar values were found when only adenomatous polyps were considered.

157 The proportion of examinations in which adenomatous polyps were detected by barium enema colonos

158 Adenomatous polyps were detected in 16 of 32 AN particip

159 mpared with 38 (8%) of 497 P/P participants; adenomatous polyps were found in 28 (16%) N/N individual

160 National Polyp Study who had newly diagnosed adenomatous polyps were interviewed for information on t

161 Adenomatous polyps were present in 111 of 120 gene carri

162 In the group >3 years posttransplant, adenomatous polyps were present in 16%, hyperplastic pol

163 In the group <3 years posttransplant, adenomatous polyps were present in 25%, hyperplastic pol

164 Neoplastic lesions (mostly adenomatous polyps) were detected in 23.5% of screened s

165 r high grade dysplasia or size > 1 cm or > 3 adenomatous polyps) were found in 98 cases (7 %), low ri

166 ed apoptosis, and suppressed angiogenesis in adenomatous polyps, which reduced both tumor number and