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1 s nucleotide (2'(3')-O-(2,4,6-trinitrophenyl)adenosine 5'-triphosphate).
2 esponse to uridine 5'-triphosphate than with adenosine 5'-triphosphate.
3 ine 5'-triphosphate and alpha,beta-methylene adenosine 5'-triphosphate.
4 hat K501 in the KGAP sequence interacts with adenosine 5'-triphosphate.
5 lly catalyse substrate phosphorylation using adenosine 5'-triphosphate.
6 ulation of the P2X7 purinergic receptor with adenosine 5'-triphosphate.
7 escent analog 2'(3')-O-(2,4,6-trinitrophenyl)adenosine 5'-triphosphate.
8 y on oxidative phosphorylation to synthesize adenosine 5'-triphosphate.
9 f Buk2 complexed with (beta,gamma-methylene) adenosine 5'-triphosphate.
10 or antagonist 2',3'-O-(2,4,6-Trinitrophenyl) adenosine 5'-triphosphate (100 microM) indicating P2X re
11 e 5'-triphosphate and high concentrations of adenosine 5'-triphosphate (1000-5000 microM) caused a si
13 he corresponding ATP derivative 2-methylthio-adenosine-5'-triphosphate (2MeSATP) at 3.1 A resolution.
16 vels of mitochondrial membrane potential and adenosine-5'-triphosphate, activation of caspase 3, and
17 d indices of lung tissue metabolic function (adenosine 5-triphosphate/adenosine 5-diphosphate ratio,
18 der of ATP > 2'- and 3'-O-(4-benzoyl-benzoyl)adenosine 5'-triphosphate alpha,beta-methyleneadenosine
20 alues for BzATP (2',3'-O-(4- benzoylbenzoyl) adenosine 5'-triphosphate), an antagonist of the P2Y(12)
21 gs were prevented by either coinfusion of an adenosine-5'-triphosphate analog or pretransfusion incub
23 tation, which was mimicked by application of adenosine 5'-triphosphate and alpha,beta-methylene adeno
24 ion were potentiated by alpha,beta-methylene adenosine 5'-triphosphate and ARL-67156, an adenosine tr
25 d P2X(2/3) antagonist 2',3'-O-trinitrophenyl-adenosine 5'-triphosphate and by the nonselective P2 ant
27 The P2X7 receptor agonist benzoylbenzoyl-adenosine 5'-triphosphate and high concentrations of ade
28 e generation of the primary energy currency, adenosine 5'-triphosphate and its use in the synthesis o
29 sensitivity and Michaelis constants (Km) for adenosine 5'-triphosphate and substrate; however, the LA
32 o and in vivo to probe the roles of released adenosine-5'-triphosphate and adhesion in responses to (
33 nufacturers by monitoring the degradation of adenosine-5'-triphosphate and myo-inositol-1,2,3,4,5,6-h
34 rs the ability of red blood cells to release adenosine-5'-triphosphate and that impaired adenosine-5'
35 rate analog, Ap(CH2)pp (alpha,beta-methylene adenosine 5'-triphosphate), and cyclic AMP compete for t
36 , 26S proteasomes still hydrolyzed peptides, adenosine 5'-triphosphate, and ubiquitinated substrates
37 nosine-5'-triphosphate release, supplemental adenosine-5'-triphosphate, and antibodies to red blood c
40 2-propylthio-D-beta gamma-dichloromethylene adenosine 5'-triphosphate (AR-C67085), a P2Y12 receptor-
41 6-N,N-diethyl-beta-gamma-dibromomethylene-D-adenosine 5-triphosphate (ARL 67156), reduced the [Ca(2+
42 6-N,N-diethyl-beta,gamma-dibromomethylene-D-adenosine-5'-triphosphate (ARL67156) inhibition of nucle
46 tor fructose-6-phosphate (F6P) and substrate adenosine 5'-triphosphate (ATP) (in the presence and abs
49 The active sites of RFC are fully bound to adenosine 5'-triphosphate (ATP) analogs, which is expect
52 he compounds are competitive with respect to adenosine 5'-triphosphate (ATP) and bind in the kinase A
53 D required Mg2+ or Mn2+ and preactivation by adenosine 5'-triphosphate (ATP) and was inhibited by kno
55 hatase (ATPase) domain residues required for adenosine 5'-triphosphate (ATP) binding and membrane att
58 smaller using SPR detection compared to the adenosine 5'-triphosphate (ATP) bioluminescence assay in
60 o adenine, adenosine monophosphate, and then adenosine 5'-triphosphate (ATP) by 5'-methylthio-adenosi
61 mational change coupled to the hydrolysis of adenosine 5'-triphosphate (ATP) by a mechanism that rema
62 mitochondrial adenosine 5'-diphosphate (ADP)/adenosine 5'-triphosphate (ATP) carrier imports ADP into
64 No major changes in membrane potential or adenosine 5'-triphosphate (ATP) concentration result fro
66 ent conclusive evidence that the presence of adenosine 5'-triphosphate (ATP) facilitates non-equilibr
67 In contrast to UDP, activation by UTP or adenosine 5'-triphosphate (ATP) greatly increased Ca(m),
69 Activation of a purinergic P2 receptor by adenosine 5'-triphosphate (ATP) has previously been show
70 r the basis of stimulation of P-glycoprotein adenosine 5'-triphosphate (ATP) hydrolysis by multiple s
72 P-binding cassette (ABC) transporters couple adenosine 5'-triphosphate (ATP) hydrolysis to substrate
73 h an unfolded protein) increased proteasomal adenosine 5'-triphosphate (ATP) hydrolysis, the step whi
74 w that CtIP also dramatically stimulates the adenosine 5'-triphosphate (ATP) hydrolysis-driven motor
77 tection of inorganic phosphate released from adenosine 5'-triphosphate (ATP) in the glutamine synthet
78 only a slight effect, while the addition of adenosine 5'-triphosphate (ATP) increased the amplitude
79 level of delivery is largely temperature and adenosine 5'-triphosphate (ATP) independent, and the mem
84 in a steady-state model to predict cellular adenosine 5'-triphosphate (ATP) levels, which also depen
87 forward to backward steps and the number of adenosine 5'-triphosphate (ATP) molecules hydrolyzed per
95 one-related small molecules, with comparable adenosine 5'-triphosphate (ATP) rescue activity to idebe
96 y diverse set of inhibitors that bind to the adenosine 5'-triphosphate (ATP) site of type II topoisom
98 on cryo-microscopy structure of the F(1)F(o)-adenosine 5'-triphosphate (ATP) synthase from A. baumann
100 activity of the electron transport chain and adenosine 5'-triphosphate (ATP) synthase with RNA interf
101 -energy phosphate content and limitations in adenosine 5'-triphosphate (ATP) synthesis rate occur dur
102 Here, we show an efficient manner to build adenosine 5'-triphosphate (ATP) synthesizing hybrid mult
104 al NE cells protect the airways by releasing adenosine 5'-triphosphate (ATP) to activate purinorecept
105 of this study was to test the equivalency of adenosine 5'-triphosphate (ATP) to adenosine in their ab
106 protein kinase fold transfer phosphate from adenosine 5'-triphosphate (ATP) to substrates in a proce
109 eotide phosphate (NADPH)), energy coenzymes (adenosine 5'-triphosphate (ATP), adenosine 5'-diphosphat
110 f nucleobase/nucleotide (adenine, adenosine, adenosine 5'-triphosphate (ATP), adenosine 5'-monophosph
111 se preparation is competitively inhibited by adenosine 5'-triphosphate (ATP), an observation that ind
112 (dTMP-PCP), thymidine 5'-diphosphate (dTDP), adenosine 5'-triphosphate (ATP), and adenosine 5'-O-(3-t
113 posed of stabilized microtubules, depletant, adenosine 5'-triphosphate (ATP), and clusters of kinesin
114 diated by a purine neurotransmitter, such as adenosine 5'-triphosphate (ATP), binding to P2Y1 recepto
117 glycolysis is an inefficient way to generate adenosine 5'-triphosphate (ATP), however, and the advant
118 nase (PPDK) catalyzes the interconversion of adenosine 5'-triphosphate (ATP), orthophosphate (P(i)),
122 ance regulator (CFTR) is a membrane-spanning adenosine 5'-triphosphate (ATP)-binding cassette (ABC) t
123 a membrane protein complex, the heteromeric adenosine 5'-triphosphate (ATP)-binding cassette transpo
124 loit currently noncatalytic cysteines in the adenosine 5'-triphosphate (ATP)-binding site via electro
125 e found to maximize occupancy of the active, adenosine 5'-triphosphate (ATP)-bound mtHsp70 at the cha
126 esults in either the high-DNA affinity form (Adenosine 5'-triphosphate (ATP)-bound) or the more inact
128 AK), a primary enzyme in cell metabolism and adenosine 5'-triphosphate (ATP)-consuming processes, pla
129 surement of inorganic pyrophosphate (PPi) in adenosine 5'-triphosphate (ATP)-contaminated samples.
132 hether it is possible to transform a natural adenosine 5'-triphosphate (ATP)-dependent enzyme into a
133 ression of synaptic strength results from an adenosine 5'-triphosphate (ATP)-derived and A1 adenosine
135 owing peripheral nerve injury, extracellular adenosine 5'-triphosphate (ATP)-mediated purinergic sign
136 rected mutagenesis supports a model in which adenosine 5'-triphosphate (ATP)-powered movements of the
140 enhanced their activity after application of adenosine 5'-triphosphate (ATP, 1 mM) or alpha,beta-meth
143 th timing and force) and immediately measure adenosine 5(')-triphosphate (ATP) release and calcium mo
144 atalyzing process is that PKA can hydrolysis adenosine-5'-triphosphate (ATP) and ALP can hydrolysis p
145 in situ multiple fluorescence monitoring of adenosine-5'-triphosphate (ATP) and guanosine-5'-triphos
146 xocytosis of LBs, following stimulation with adenosine-5'-triphosphate (ATP) and phorbol 12-myristate
147 of phosphate containing metabolites such as adenosine-5'-triphosphate (ATP) and pyrophosphate (PPi).
148 Phosphate-containing metabolites such as adenosine-5'-triphosphate (ATP) and pyrophosphate play a
149 he insertion of aptamer sequences (e.g., the adenosine-5'-triphosphate (ATP) aptamer) or ion-binding
151 e diatom, Skeletonema costatum, in utilizing adenosine-5'-triphosphate (ATP) based on incubation expe
156 We have shown that red blood cell (RBC) adenosine-5'-triphosphate (ATP) is better maintained and
159 ytes can be termed as energy inefficient for adenosine-5'-triphosphate (ATP) production but energy ef
160 leading to reductions in lactate production, adenosine-5'-triphosphate (ATP) production, and reduced
161 stimulation induces the release of cellular adenosine-5'-triphosphate (ATP) that regulates T-cell ac
162 were found to selectively adsorb adenine and adenosine-5'-triphosphate (ATP), as compared to other nu
164 that associates with the MTase and catalyses Adenosine-5'-triphosphate (ATP)-dependent DNA translocat
166 substrate complexes allowed the detection of adenosine-5'-triphosphate (ATP, detection limit 10 muM).
167 Extracellular triphosphate nucleotides (adenosine 5'-triphosphate [ATP], uridine 5'-triphosphate
168 n addition to previously published assays of adenosine 5'-triphosphate binding and hydrolysis, measur
169 nd type II and III inhibitors at or near the adenosine 5'-triphosphate binding sites are well defined
171 scular mimicry and concomitantly express the adenosine 5'-triphosphate-binding cassette transporter b
174 nd the acceptor (2'(or 3')-O-(trinitrophenyl)adenosine 5'-triphosphate) binds reversibly to the activ
175 quencher for boron dipyrromethene-conjugated adenosine 5'-triphosphate (BODIPY-ATP) that is highly fl
176 the P2X(7) receptor agonist, (benzoylbenzoyl)adenosine 5' triphosphate (BzATP) or the alpha(1D)-AR ag
177 h the P2X7 receptor agonist, (benzoylbenzoyl)adenosine 5' triphosphate (BzATP), cholinergic agonist c
178 riphosphate) and 2',3'-O-(4-benzoyl-benzoyl) adenosine 5'-triphosphate (BzATP), but not by adenosine.
180 elective agonist, 2'-3'-O-(4-benzoylbenzoyl)-adenosine 5'-triphosphate (BzATP: 100 microM), triggers
181 receptor agonist 2'3'-O-(4-benzoyl-benzoyl)-adenosine 5'-triphosphate (BzATP; 10 microM) caused a ra
182 y addition of 2'- and 3'-O-(4-benzoylbenzoyl)adenosine 5'-triphosphate (BzATP; 30 microm) resulted in
183 de (LPS)-primed and 2'(3')-O-(benzoylbenzoyl)adenosine-5'-triphosphate (BzATP)-induced IL-1B release
184 of ATP or 2'- (or-3'-) O-(4-benzoylbenzoyl) adenosine 5'-triphosphate (BzBzATP) activated an inward
185 oquinolin-7,9-dione (1) was identified as an adenosine 5'-triphosphate competitive inhibitor of lck b
186 tazone decreased oxidative stress, increased adenosine 5'-triphosphate concentration, and exerted neu
187 % flow, no changes in function were seen and adenosine 5'-triphosphate concentrations decreased durin
188 t regulate mitochondrial membrane potential, adenosine 5'-triphosphate contents, and reactive oxygen
190 The naturally occurring nucleotide 2-deoxy-adenosine 5'-triphosphate (dATP) can be used by cardiac
191 with controls reflecting higher than normal adenosine 5'-triphosphate degradation in the malignant h
193 assess the roles of the RSC, ISW1, and CHD1 adenosine 5'-triphosphate-dependent chromatin remodelers
194 work, we report the structural mechanism for adenosine 5'-triphosphate-dependent chromatin remodeling
196 mark them for destruction by a multisubunit, adenosine 5'-triphosphate-dependent protease called the
197 The 26S proteasome is a large intracellular adenosine 5'-triphosphate-dependent protease that identi
198 (CTE) of simian retrovirus was identified as adenosine 5'-triphosphate-dependent RNA helicase A.
199 We also discuss evidence suggesting that adenosine-5'-triphosphate-dependent chromatin-remodeling
202 n, phospholamban (PLN) is an inhibitor of an adenosine-5'-triphosphate-driven calcium pump, the Ca2+-
204 iphosphate), and 2',3'-O-(4-benzoyl-benzoyl) adenosine 5'-triphosphate each induced cAMP accumulation
205 s were observed in a subpopulation of cells; adenosine 5'-triphosphate failed to elevate Cai in some
206 In addition, 2'(3')-O-(2,4,6-trinitrophenyl)adenosine 5'-triphosphate fluorescence titration of BVR
207 of the adenylation of 4-chlorobenzoate with adenosine 5'-triphosphate followed by acyl transfer from
208 pends in part on the release of antiadhesive adenosine-5'-triphosphate from red blood cells, and stor
209 ping mechanism, likely arising from just one adenosine 5'-triphosphate hydrolysis event being require
212 prokaryotic model Hsp70 DnaK throughout its adenosine-5'-triphosphate hydrolysis (ATPase) cycle usin
213 ke the bacterial flagellar motor (BFM), ATP (adenosine-5'-triphosphate) hydrolysis probably drives bo
215 ion of P2X(7) receptors with (benzoylbenzoyl)adenosine 5'-triphosphate increased [Ca(2+)](i), peroxid
217 al preparation was used to elucidate whether adenosine 5'-triphosphate is released from the mucosa in
219 ed intracellular levels of G6P, lactate, and adenosine-5'-triphosphate, leading to improved functiona
221 se reaction mixture generated from magnesium adenosine 5-triphosphate (MgATP) and 4-CBA in the absenc
222 P2X7 receptor antagonist, periodate oxidized adenosine 5'-triphosphate (o-ATP), substantially inhibit
227 rescence enhancement, we discovered a robust adenosine 5'-triphosphate-powered dsRNA translocation ac
228 NRF2 activity, which enhances mitochondrial adenosine 5'-triphosphate production and cellular resist
229 pathologies and point to improving neuronal adenosine 5'-triphosphate production kinetics as a promi
230 y increasing PGK1 expression boosts neuronal adenosine 5'-triphosphate production kinetics that is su
232 ectum led to pressure-dependent increases in adenosine 5'-triphosphate release from colorectal epithe
233 ood cells, and storage-induced deficiency in adenosine-5'-triphosphate release from transfused red bl
234 on of fresh red blood cells treated with the adenosine-5'-triphosphate release inhibitors glibenclami
236 adenosine-5'-triphosphate and that impaired adenosine-5'-triphosphate release was injurious in vivo,
238 transducer that, upon binding galactose and adenosine 5'-triphosphate, relieves Gal80p repression.
239 Adenosine, after ectoenzymatic breakdown of adenosine 5'-triphosphate, seems to be involved in the l
242 y, which led to the discovery of a drug-like adenosine 5'-triphosphate-site PI3K/mTOR kinase inhibito
243 articularly affected by alpha,beta-methylene adenosine 5'-triphosphate, suggesting a correlation betw
244 ation with the first enzyme in this pathway, adenosine-5'-triphosphate sulfurylase, conferred signifi
247 lysis rate of 2', 3'-O-(2,4,6-trinitrophenyl)adenosine 5'-triphosphate (TNP-ATP) by F1-ATPase require
248 O-(2,4,6-trinitrophenylcyclo-hexadienylidene)adenosine 5'-triphosphate (TNP-ATP) to maltose-binding f
249 eotide analog 2',3'-O-(2,4,6-trinitrophenyl) adenosine 5'-triphosphate (TNP-ATP), which acts as a flu
251 ith AF-353 or 2',3'-O-(2,4,6-Trinitrophenyl)-adenosine-5'-triphosphate (TNP-ATP), structurally distin
253 es the conversion of glucose 1-phosphate and adenosine 5'-triphosphate to ADP-glucose and pyrophospha
254 ted with beta2, cytidine 5'-diphosphate, and adenosine 5'-triphosphate to generate a NH(2)Y(730)(*) i
255 talyzes the transfer of gamma-phosphate from adenosine 5(')-triphosphate to the threonine residue of
256 ropic receptor agonist, alpha,beta-methylene adenosine 5'-triphosphate, to distinguish the ADP-induce
257 ogue TNP-ATP [2'(3')-O-(2,4,6-trinitrophenyl)adenosine 5'-triphosphate] to investigate the two ATP-bi
258 ATP(o) and 2'- and 3'-O-(4-benzoylbenzoyl)adenosine 5'-triphosphate triethylammonium salt (Bz-ATP)
259 of sarcoplasmic reticulum Ca-release-induced adenosine 5'-triphosphate turnover as a potential minima
262 able ATP analog, ADPCP (beta,gamma-methylene adenosine 5'-triphosphate), was able to support apoptoso
263 MantATP [2'(3')-O-(-N-methylanthraniloyl)-adenosine 5'-triphosphate] was employed as a fluorescenc