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1 n other cells by releasing a compound called adenosine monophosphate.
2 toxin fails to increase intracellular cyclic adenosine monophosphate.
3 osine, an adenosine surrogate, and of cyclic adenosine monophosphate.
4 xchange protein directly activated by cyclic adenosine monophosphate 1 (EPAC1)-RAP1-dependent model o
7 e nucleotide signaling molecule 3',5'-cyclic adenosine monophosphate (3',5'-cAMP) plays important phy
8 e stimulation of A2a receptors causes cyclic adenosine monophosphate accumulation at the back of cell
9 -chlorophenyl hydrazone (CCCP) treatment and adenosine monophosphate activated protein kinase (AMPK)
10 s of adiponectin receptor-2, inactivation of adenosine monophosphate activated protein kinase (AMPK),
11 th DENV activates the metabolic regulator 5' adenosine-monophosphate activated kinase (AMPK), and tha
13 UDCA activates SMILE gene expression through adenosine monophosphate-activated kinase phosphorylation
14 d kinase) enzyme, which belongs to the AMPK (adenosine monophosphate-activated kinase) family, was es
15 promoter activity and gene expression in an adenosine monophosphate-activated kinase-dependent manne
21 tion while activating "stress" signals of 5' adenosine monophosphate-activated protein kinase (AMPK)
22 in high-altitude populations is one for the adenosine monophosphate-activated protein kinase (AMPK)
27 that regulates metabolism and growth through adenosine monophosphate-activated protein kinase (AMPK)
28 hat this response does not require canonical adenosine monophosphate-activated protein kinase (AMPK)
29 at the antidiabetes drug metformin (MET), an adenosine monophosphate-activated protein kinase (AMPK)
33 A targeted screen identified a role for 5' adenosine monophosphate-activated protein kinase (AMPK)
34 ses involved in autophagy induction such as; Adenosine monophosphate-activated protein kinase (AMPK)
36 fatty acid beta-oxidation, and activating 5'adenosine monophosphate-activated protein kinase (AMPK)
38 The increase in the ROS level activated 5' adenosine monophosphate-activated protein kinase (AMPK),
39 antagonized the catalytic alpha1 subunit of adenosine monophosphate-activated protein kinase (AMPK),
40 nvolved in energy stress response, including adenosine monophosphate-activated protein kinase (AMPK),
41 f rapamycin (mTORC1) to the energy sensor 5'-adenosine monophosphate-activated protein kinase (AMPK),
42 mechanistic target of rapamycin (mTOR), and adenosine monophosphate-activated protein kinase (AMPK)-
43 et of the peptide via modulation of upstream adenosine monophosphate-activated protein kinase (AMPK)-
44 competent up to p-Akt activation; however, p-adenosine monophosphate-activated protein kinase (p-AMPK
45 f heme oxygenase-1 (HO-1) and phosphorylated adenosine monophosphate-activated protein kinase (pAMPK)
48 e induction of trained immunity, whereas the adenosine monophosphate-activated protein kinase activat
49 enosine triphosphate-citrate lyase inhibitor/adenosine monophosphate-activated protein kinase activat
50 kinases 1/2, phosphatase and tensin homolog, adenosine monophosphate-activated protein kinase alpha,
51 of the metabolic modulators p38-alpha and 5' adenosine monophosphate-activated protein kinase alpha.
52 hich controls IEB permeability by inhibiting adenosine monophosphate-activated protein kinase and inc
53 regulates IEB permeability by inhibiting an adenosine monophosphate-activated protein kinase and inc
54 (PTEN) induces activation of the phospho-5' adenosine monophosphate-activated protein kinase and pho
55 adou et al. report that the metabolic sensor adenosine monophosphate-activated protein kinase influen
56 hate-activated protein kinase, total protein adenosine monophosphate-activated protein kinase levels,
58 d association with nitric oxide synthase and adenosine monophosphate-activated protein kinase pathway
59 use adenosine triphosphate-citrate lyase and adenosine monophosphate-activated protein kinase play ce
61 creased hepatocyte apoptosis, independent of adenosine monophosphate-activated protein kinase signali
62 rectly targets the 3' untranslated region of adenosine monophosphate-activated protein kinase subunit
64 -dependent phosphorylation of distinct AMPK (adenosine monophosphate-activated protein kinase) family
65 cAMP-response element binding, p38 MAPK and adenosine monophosphate-activated protein kinase) in way
66 f ATM (ataxia telangiectasia mutated)/PRKAA (adenosine monophosphate-activated protein kinase) signal
67 which represses mTOR signaling by activating adenosine monophosphate-activated protein kinase, has be
68 response, leading to decreased activation of adenosine monophosphate-activated protein kinase, total
69 ng from the master energy-regulating kinase, adenosine monophosphate-activated protein kinase, while
71 lmodulin-dependent kinase kinase-beta and 5' adenosine monophosphate-activated protein kinase-depende
74 onse is cyclic guanosine monophosphate (GMP)-adenosine monophosphate (AMP) (cGAMP) synthase (cGAS), w
75 adenosine triphosphate (ATP) hydrolysis into adenosine monophosphate (AMP) and 2) AMP into adenosine
78 w that while Gh can form hydrogen bonds with adenosine monophosphate (AMP) during incorporation, this
79 ediates AMPylation, a covalent attachment of adenosine monophosphate (AMP) from ATP to hydroxyl side
81 transcription factor 2 (ATF2) to the cyclic adenosine monophosphate (AMP) response element (CRE) in
83 lasmic sensor cyclic guanosine monophosphate-adenosine monophosphate (AMP) synthase (cGAS) drives IRF
87 ein, HYPE/FicD, catalyzes the addition of an adenosine monophosphate (AMP) to the ER chaperone, BiP,
89 idly hydrolyzed by the ecto-ATPase CD39 into adenosine monophosphate (AMP), and it is AMP that regula
90 ve catabolites: adenosine diphosphate (ADP), adenosine monophosphate (AMP), inosine monophosphate (IM
91 precursors, adenosine diphosphate (ADP) and adenosine monophosphate (AMP), using mouse heart, kidney
92 ow expression of SIRT1 and PGC1alpha and low adenosine monophosphate (AMP)-activated kinase (AMPK) ac
95 longed metformin treatment on phosphorylated adenosine monophosphate (AMP)-activated protein kinase (
101 hate (ATP), adenosine diphosphate (ADP), and adenosine monophosphate (AMP); and antioxidants, the sum
102 reduction in plasma adenosine (P = 0.03) and adenosine monophosphate (AMP; P < 0.0001) concentrations
103 iphosphate [ATP]/adenosine diphosphate [ADP]/adenosine monophosphate [AMP], nicotinamide adenine dinu
106 nopus oocytes in the presence of both cyclic adenosine monophosphate and Ca(2+) results in Ca(2+) inf
107 enzyme involved in the regulation of cyclic adenosine monophosphate and cyclic guanosine monophospha
108 culture and decidualized with 8-bromo-cyclic adenosine monophosphate and medroxyprogesterone acetate.
109 Nucleotide (ATP, adenosine diphosphate, adenosine monophosphate) and nucleoside (adenosine and i
112 ly, we demonstrate that Akt up-regulates the adenosine monophosphate-associated kinase (AMPK)-related
113 Strikingly, the measured on-rate for cyclic adenosine monophosphate binding is two orders of magnitu
114 educed renal ATP, adenosine diphosphate, and adenosine monophosphate, but not adenosine levels, durin
122 n ADCY5 was studied by measurement of cyclic adenosine monophosphate (cAMP) accumulation under stimul
123 l had an impaired capacity to degrade cyclic adenosine monophosphate (cAMP) and a blunted pharmacolog
124 mediated initially by an increase in cyclic adenosine monophosphate (cAMP) and a subsequent inactiva
128 lation of the intracellular levels of cyclic adenosine monophosphate (cAMP) and cyclic guanosine mono
130 The spatiotemporal regulation of cyclic adenosine monophosphate (cAMP) and its dynamic interacti
132 s contributed to an increase in basal cyclic adenosine monophosphate (cAMP) and vasodilator-stimulate
135 prostaglandin E1-induced increase in cyclic adenosine monophosphate (cAMP) by ADP was impaired, wher
136 udies exploring the importance of the cyclic adenosine monophosphate (cAMP) cascade in major depressi
137 bunit through formation of a PDE-PKAR-cyclic adenosine monophosphate (cAMP) complex (the termination
138 cally, coronin 1-deficiency increased cyclic adenosine monophosphate (cAMP) concentrations to suppres
141 mooth muscle cells (hCASMCs) to 3',5'-cyclic adenosine monophosphate (cAMP) generation and phosphoryl
142 phodiesterase (PDE4) and elevation of cyclic adenosine monophosphate (cAMP) has emerged as a promisin
143 s associated with increased levels of cyclic adenosine monophosphate (cAMP) in cholangiocytes lining
144 hnology have revealed oscillations of cyclic adenosine monophosphate (cAMP) in insulin-secreting cell
145 centration of the secondary messenger cyclic adenosine monophosphate (cAMP) in MLT cells, in response
146 rom the canalicular membrane, whereas cyclic adenosine monophosphate (cAMP) increases plasma membrane
153 a PDE that regulates cardiac myocyte cyclic adenosine monophosphate (cAMP) levels and myocardial con
154 hibitory protein Galpha(o1) to reduce cyclic adenosine monophosphate (cAMP) levels in mice and in GPR
155 and kidney (PKD) diseases, increased cyclic adenosine monophosphate (cAMP) levels trigger hepatorena
157 ds such as elevation of intracellular cyclic adenosine monophosphate (cAMP) levels, and depends on up
161 he GNAS gene, which encodes the 3',5'-cyclic adenosine monophosphate (cAMP) pathway-associated G-prot
164 o have taken effect through increased cyclic adenosine monophosphate (cAMP) response element binding
166 ond, we found that phosphorylation of cyclic adenosine monophosphate (cAMP) responsive-element-bindin
167 A (PKA) is the major receptor for the cyclic adenosine monophosphate (cAMP) secondary messenger in eu
168 FTO augmented second messenger 3', 5'-cyclic adenosine monophosphate (cAMP) signaling and suppressed
169 demonstrate a differential effect of cyclic adenosine monophosphate (cAMP) signaling between normal
176 ent increased intracellular levels of cyclic adenosine monophosphate (cAMP) that turned on protein ki
178 or D1 (DRD1) via the second messenger cyclic adenosine monophosphate (cAMP) to synthetic promoters co
179 were treated with Angiopoietin 1 and cyclic adenosine monophosphate (cAMP) to vary the Pd of the HUV
182 f phenylalanine, acetylhistidine, and cyclic adenosine monophosphate (cAMP) were found in urine sampl
184 Here we examine whether increases in cyclic adenosine monophosphate (cAMP), an intracellular signali
185 th a small molecule second messenger, cyclic adenosine monophosphate (cAMP), and a downstream cell-se
186 y modulated by a HCN channel blocker, cyclic adenosine monophosphate (cAMP), and neuromodulators.
187 rchestrated by waves of extracellular cyclic adenosine monophosphate (cAMP), and previous theory sugg
189 s have implicated defective dopamine, cyclic adenosine monophosphate (cAMP), and Ras homeostasis.
190 MRE-269 increased intracellular 3',5'-cyclic adenosine monophosphate (cAMP), augmented glucose-stimul
193 R reduced ability of SCT to stimulate cyclic adenosine monophosphate (cAMP), with signaling augmented
194 conductance regulator (CFTR) gene, a cyclic Adenosine MonoPhosphate (cAMP)-dependent chloride channe
195 helial barrier can be up-regulated by cyclic adenosine monophosphate (cAMP)-dependent mechanisms thro
196 e regulatory subunit (PRKAR1A) of the cyclic adenosine monophosphate (cAMP)-dependent protein kinase
197 rmore, we demonstrated that Tregs use cyclic adenosine monophosphate (cAMP)-dependent protein kinase
198 spatiotemporal signaling control, the cyclic adenosine monophosphate (cAMP)-dependent protein kinase
199 roach was applied to the prototypical cyclic adenosine monophosphate (cAMP)-dependent protein kinase
200 coding the gamma-catalytic subunit of cyclic adenosine monophosphate (cAMP)-dependent protein kinase
203 motes beta cell Tcf7 expression via a cyclic adenosine monophosphate (cAMP)-independent and extracell
204 (CT)-induced diarrhea is mediated by cyclic adenosine monophosphate (cAMP)-mediated active Cl- secre
205 odendritic domain, depends on ongoing cyclic adenosine monophosphate (cAMP)-protein kinase A (PKA) le
206 lation of the prostaglandin E2 (PGE2)-cyclic adenosine monophosphate (cAMP)-protein kinase A (PKA) si
214 P-ribose) polymerase 1 (PARP1) by the cyclic adenosine monophosphate (cAMP)/protein kinase A (PKA) sy
215 ntified as full antagonist ligands on cyclic adenosine monophosphate (cAMP, KB = 4.9 and 5.9 nM, resp
216 aling pathway function (Ras activity, cyclic adenosine monophosphate [cAMP], and dopamine levels).
217 lutamatergic, monoaminergic, calcium, cyclic adenosine monophosphate [cAMP], dopamine- and cAMP-regul
218 horylation of DARPP-32 (dopamine- and cyclic adenosine monophosphate [cAMP]-regulated phospho-protein
220 s as a direct cyclic guanosine monophosphate-adenosine monophosphate (cGAMP) mimetic that induces the
221 ne stimulates cyclic guanosine monophosphate-adenosine monophosphate (cGAMP) production in vitro more
222 tification of cyclic guanosine monophosphate-adenosine monophosphate (cGAMP) synthase (cGAS) as a cyt
223 he DNA sensor cyclic guanosine monophosphate-adenosine monophosphate (cGAMP) synthase (cGAS) binds to
224 sis activated cyclic guanosine monophosphate-adenosine monophosphate (cGAMP) synthase (cGAS) in macro
225 ion activates cyclic guanosine monophosphate-adenosine monophosphate (cGAMP) synthase (cGAS) to produ
226 ulating 2',3'-cyclic guanosine monophosphate-adenosine monophosphate (cGAMP), an agonist of the inter
227 STING), 2'3'- cyclic guanosine monophosphate-adenosine monophosphate (cGAMP), robustly augmented and
228 STING ligand cyclic guanosine monophosphate-adenosine monophosphate (cGAMP), we stimulated periphera
230 production of cyclic guanosine monophosphate-adenosine monophosphate (cyclic GMP-AMP, or cGAMP), whic
231 rase 10A (PDE10A), a dual-specificity cyclic adenosine monophosphate/cyclic guanosine monophosphate-i
233 c adenosine monophosphate levels; and cyclic adenosine monophosphate-dependent protein kinase A-media
234 ng the gamma-catalytic subunit of the cyclic adenosine monophosphate-dependent protein kinase, the mu
241 e protein directly activated by cyclic-3',5'-adenosine monophosphate (Epac) on Nav1.4 currents from i
244 easing the pathway flux, and influencing the adenosine monophosphate/guanosine monophosphate ratio.
248 beta) is highly associated with cAMP (cyclic adenosine monophosphate)-independent dopamine D(2) recep
250 )-TOC demonstrated higher potency for cyclic adenosine monophosphate inhibition (half maximal effecti
252 is a key molecule, since via degradation of adenosine monophosphate into adenosine, endorses the gen
253 (GP)IIb/IIIa activation and decreased cyclic adenosine monophosphate levels (n = 6, P < .01) in plate
254 n of the cardiac stress marker NR4A1; cyclic adenosine monophosphate levels; and cyclic adenosine mon
255 STING agonist cyclic guanosine monophosphate-adenosine monophosphate (NP-cGAMP) in mouse models of lu
256 that regulates the phosphodiesterase/cyclic adenosine monophosphate pathway and modulates T cell res
257 ability of RvD5n-3 DPA to upregulate cyclic adenosine monophosphate, phagocytosis of bacteria, and e
258 n-activated protein kinase) and cAMP (cyclic adenosine monophosphate)-PKA (protein kinase A) cascades
259 (UPR), intracellular ion homeostasis, cyclic adenosine monophosphate production and regulation of ins
260 nosine uptake by red blood cells, and cyclic adenosine monophosphate production by cells overexpressi
261 teoclast differentiation by enhancing cyclic adenosine monophosphate production through an unidentifi
262 her than canonical Galpha(s)-mediated cyclic adenosine-monophosphate production, explained 88% of the
263 giocytes show increased production of cyclic adenosine monophosphate, protein kinase A-dependent acti
264 m of ICAM-4 activation occurs via the cyclic adenosine monophosphate-protein kinase A (cAMP-PKA)-depe
265 therapy, which elevates intracellular cyclic adenosine monophosphate/protein kinase A (cAMP-PKA) sign
266 acyclin which stimulates the platelet cyclic adenosine monophosphate/protein kinase A (cAMP/PKA)-sign
267 Previous studies have implicated the cyclic adenosine monophosphate/protein kinase A pathway as well
268 nitines, including inosine monophosphate and adenosine monophosphate (purine metabolism), malic acid
269 ed the role of DARPP-32 (dopamine and cyclic adenosine monophosphate-regulated phosphoprotein, Mr 320
270 trophic factor and phosphorylation of cyclic adenosine monophosphate response element binding and neu
271 expression of the binding protein of cyclic adenosine monophosphate response element binding protein
272 own that nuclear transcription factor cyclic adenosine monophosphate response element binding protein
273 ated the functional regulation of the cyclic adenosine monophosphate response element binding protein
274 ional activity and phosphorylation of cyclic adenosine monophosphate response element binding protein
275 tion and is required for signaling to cyclic adenosine monophosphate response element-binding protein
276 r regions of the transcription factor cyclic adenosine monophosphate response element-binding protein
277 logous protein, and activation of the cyclic adenosine monophosphate response element-binding protein
278 ISC1 caused a significant increase of cyclic adenosine monophosphate response element-binding protein
279 o evaluation of H(3)R agonism using a cyclic adenosine monophosphate response element-luciferase repo
280 promoter region as well as increased cyclic adenosine monophosphate response element-mediated transc
282 ranscription coupling caused by CREB (cyclic adenosine monophosphate-responsive element-binding prote
283 phosphate, deoxyadenosine monophosphate, and adenosine monophosphate), results in ring opening to lin
284 he current study examined whether D1R-cyclic adenosine monophosphate signaling reduces neuronal firin
285 amine D1 receptor (D1R) activation of cyclic adenosine monophosphate signaling, which reduces PFC neu
286 cts of dopamine receptor D2 (DRD2) on cyclic adenosine monophosphate signaling; PDAC tissues had a sl
287 to ion channel Kir7.1, while lacking cyclic adenosine monophosphate stimulation, highlights a hetero
288 ed generation of the second messenger cyclic adenosine monophosphate, suggesting that alterations in
289 e (3polyP), adenosine triphosphate (ATP) and adenosine monophosphate supported equivalent growth rate
290 on of nuclear cyclic guanosine monophosphate-adenosine monophosphate synthase (cGAS) and enhanced int
293 he DNA sensor cyclic guanosine monophosphate-adenosine monophosphate synthase and the downstream adap
294 king STING or cyclic guanosine monophosphate-adenosine monophosphate synthase exhibit unaltered abili
295 he DNA sensor cyclic guanosine monophosphate-adenosine monophosphate synthase in micronuclei, leading
297 well-studied system is the binding of cyclic adenosine monophosphate to the cyclic nucleotide binding
298 exchange factor directly activated by cyclic adenosine monophosphate, which maintains vascular integr
300 osine 3',5'-cyclic monophosphate, and cyclic adenosine monophosphate with reduced spreading on collag