ライフサイエンスコーパス: adenosine triphosphatase

1 anslocation proposed for other AAA+ ATPases (adenosine triphosphatases).

2 t the KIF5-SNPH coupling inhibited the motor adenosine triphosphatase.

3 ccurs on the level of the vacuolar-type H(+)-adenosine triphosphatase.

4 the proton pumping enzyme vacuolar-type H(+)-adenosine triphosphatase.

5 n ATP7B, which encodes a copper-transporting adenosine triphosphatase.

6 requires Vps4, a member of the AAA family of adenosine triphosphatases.

7 pression of cell surface-associated vacuolar adenosine triphosphatases.

8 uses phosphorylation of plasma membrane H(+)-adenosine triphosphatase 2 at Ser(899), mediating the in

9 ession of a critical proteasome subunit, non-adenosine triphosphatase 4 (PSMD4), was reduced in old m

10 ntermediates (AIs) that contain the cellular adenosine triphosphatase ABCE1 (ATP-binding cassette pro

11 ATP-binding cassette (ABC) adenosine triphosphatases actively transport a wide vari

12 Observations on the adenosine triphosphatase activities and protease suscept

13 Motor adenosine triphosphatase activities and their associatio

14 that reduces contractility by decreasing the adenosine triphosphatase activity of the cardiac myosin

15 y interfered with the microtubule-stimulated adenosine triphosphatase activity of the kinesin motor,

16 A lack of VPS4 adenosine triphosphatase activity reduced budding by up

17 nucleosome binding, nucleosome sliding, and adenosine triphosphatase activity, but ARID1A is require

18 dehydrogenase, inhibition of membrane Na /K adenosine triphosphatase activity, inactivation of membr

19 ) generated by microtubule-activated kinesin adenosine triphosphatase activity.

20 ssed for Na/K/2Cl cotransporter and Na+,K(+)-adenosine triphosphatase activity.

21 stimulated its double-stranded DNA-dependent adenosine triphosphatase activity.

22 variants in ATP1A3-encoded sodium-potassium adenosine triphosphatase alpha 3 (ATP1A3).

23 lation of the Na(+)/K(+) exchanger ATPalpha (adenosine triphosphatase alpha) in glia may be modulated

24 ori represses expression of the gastric H, K-adenosine triphosphatase alpha-subunit (HKalpha), which

25 we showed that astrocytic alpha2-Na(+)/K(+) adenosine triphosphatase (alpha2-NKA) is elevated in pos

26 on markers including GATA4, GATA6, and H+/K+-adenosine triphosphatase and abnormal expression of memb

27 ssociate with wild-type CLPB and inhibit its adenosine triphosphatase and disaggregase activity in a

28 fect the distribution and abundance of the P-adenosine triphosphatase and Pinformed 1 auxin efflux fa

29 l domain and forms a hexameric core of fused adenosine triphosphatase and protease domains.

30 ns in Ser-23, Lys-148, and Arg-321 uncoupled adenosine triphosphatase and Rca activity, implicating t

31 taining of the cells both with anti-H(+)K(+)-adenosine triphosphatase antibodies and with Texas Red-l

32 olated flagella when exogenous ubiquitin and adenosine triphosphatase are added, suggesting that the

33 Here, using helicase and adenosine triphosphatase assays we show that a complex c

34 airpin RNA (shRNA) screen, we identified the adenosine triphosphatase associated with diverse cellula

35 domain through canonical sites at the AAA+ (adenosine triphosphatases associated with diverse cellul

36 es are conserved members of the AAA+ family (adenosine triphosphatases associated with diverse cellul

37 uride's cyclohexylurea group, which binds to adenosine triphosphatase (ATP)-sensitive K(+) (K(ATP)) c

38 d H(+) secretion by the nongastric H(+)/K(+) adenosine triphosphatase (ATP12A) acidified airway surfa

39 We found that the vacuolar H(+)-adenosine triphosphatase ATPase (v-ATPase) is necessary

40 and dissociation between the electron donor adenosine triphosphatase (ATPase) (Fe-protein) and its t

41 The Cdc48 adenosine triphosphatase (ATPase) (p97 or valosin-contai

42 The vesicular adenosine triphosphatase (ATPase) acidifies intracellula

43 /X comprised of the Mre11 nuclease and Rad50 adenosine triphosphatase (ATPase) active sites dimerizes

44 The kinase, phosphatase, and adenosine triphosphatase (ATPase) activities of anKaiC w

45 n acridine orange fluorescence and H(+),K(+)-adenosine triphosphatase (ATPase) activity in isolated t

46 se IV substrate inactivated bile canalicular adenosine triphosphatase (ATPase) activity in normal but

47 Na,K-adenosine triphosphatase (ATPase) activity is elevated i

48 gold electron microscopy and kinetics of the adenosine triphosphatase (ATPase) activity of purified R

49 -expressing cells, stimulates Pgp-associated adenosine triphosphatase (ATPase) activity, and causes c

50 of IFN-gamma on T84 barrier function, Na+,K+-adenosine triphosphatase (ATPase) activity, and certain

51 depending on its dinucleotide loading state, adenosine triphosphatase (ATPase) activity, interactions

52 ion) and displays low microtubule-stimulated adenosine triphosphatase (ATPase) activity.

53 l-permeable, specific inhibitor of myosin II adenosine triphosphatase (ATPase) activity.

54 nique relative orientation of the N-terminal adenosine triphosphatase (ATPase) and C-terminal nucleas

55 es of 60 and 130 nM in microtubule-dependent adenosine triphosphatase (ATPase) and cell-based cytotox

56 sin heavy chain, sarcoplasmic reticulum Ca2+-adenosine triphosphatase (ATPase) and Na+-Ca2+ exchanger

57 ant baculovirus, possesses RNA/DNA helicase, adenosine triphosphatase (ATPase) and single-stranded (s

58 pic properties related to inhibition of Na/K adenosine triphosphatase (ATPase) and stimulation of sar

59 t of SpoIIIE was shown to be a DNA-dependent adenosine triphosphatase (ATPase) capable of tracking al

60 Here we present evidence that the adenosine triphosphatase (ATPase) cycle of the SF1 helic

61 c environment in the catalytic space of gp17-adenosine triphosphatase (ATPase) determines the rate at

62 we functionally characterized the DExD/H box adenosine triphosphatase (ATPase) Dhh1, a critical regul

63 MTBD) is separated from its ring-shaped AAA+ adenosine triphosphatase (ATPase) domain by a 15-nanomet

64 ide exchange factor GrpE in complex with the adenosine triphosphatase (ATPase) domain of Escherichia

65 hia coli chromosomal replication, has in its adenosine triphosphatase (ATPase) domain residues requir

66 BP72, an editing auxiliary factor of the ABC adenosine triphosphatase (ATPase) family that exhibits R

67 members of the conserved Microrchidia (MORC) adenosine triphosphatase (ATPase) family, which are pred

68 are covalent inhibitors of the gastric H+,K+-adenosine triphosphatase (ATPase) forming disulfide bond

69 We examined whether ITP can be used by adenosine triphosphatase (ATPase) in human erythrocytes

70 The enzyme F1-adenosine triphosphatase (ATPase) is a molecular motor t

71 ss animal species and cell types, Na(+),K(+)-adenosine triphosphatase (ATPase) is arguably the most p

72 Na,K-adenosine triphosphatase (ATPase) is essential for the r

73 Because Na,K-adenosine triphosphatase (ATPase) is involved in aqueous

74 ntified the chromatin-remodeling nucleosomal adenosine triphosphatase (ATPase) ISWI as a key molecule

75 As a ring-shaped adenosine triphosphatase (ATPase) machine, cohesin organ

76 The SecA adenosine triphosphatase (ATPase) mediates extrusion of

77 hitecture of MetNI reveals two copies of the adenosine triphosphatase (ATPase) MetN in complex with t

78 rough attached ubiquitin chains and uses its adenosine triphosphatase (ATPase) motor for mechanical u

79 y component of the system is the proteasomal adenosine triphosphatase (ATPase) Mpa, which captures, u

80 The H,K-adenosine triphosphatase (ATPase) of gastric parietal ce

81 croscopy structures portraying the hexameric adenosine triphosphatase (ATPase) p on a pathway to term

82 i release (corresponding to the steady-state adenosine triphosphatase (ATPase) rate of actomyosin (AM

83 with acto-myosin function through the myosin adenosine triphosphatase (ATPase) reaction; 1-(5-isoquin

84 Prp5 protein (Prp5p) is an RNA-stimulated adenosine triphosphatase (ATPase) required for presplice

85 croscopy structures portraying the hexameric adenosine triphosphatase (ATPase) rho on a pathway to te

86 Mutations in the AAA adenosine triphosphatase (ATPase) Spastin (SPG4) cause a

87 Katanin, a member of the AAA adenosine triphosphatase (ATPase) superfamily, uses nucl

88 SF (N-ethylmaleimide-sensitive factor) is an adenosine triphosphatase (ATPase) that contributes to a

89 p97 is a hexameric AAA+ adenosine triphosphatase (ATPase) that is an attractive

90 necessary for packaging in such viruses: the adenosine triphosphatase (ATPase) that powers DNA transl

91 Here, we found that the orphan P5A-adenosine triphosphatase (ATPase) transporter ATP13A1 (S

92 ctivity of the hepatocyte basolateral Na+,K+-adenosine triphosphatase (ATPase) was also decreased by

93 1) Hsp90 facilitates both recruitment of the adenosine triphosphatase (ATPase)-activating cochaperone

94 onsequences of SFX binding, we engineered an adenosine triphosphatase (ATPase)-competent, hexameric 2

95 ombin significantly reduces the rate of Na,K-adenosine triphosphatase (ATPase)-mediated ion transport

96 ereas Kar2p reciprocally activated the Lhs1p adenosine triphosphatase (ATPase).

97 rboxy terminal two thirds is comprised of an adenosine triphosphatase (ATPase)/helicase domain.

98 ium iodide symporter, and hydrogen potassium adenosine triphosphatase [ATPase]) showed reduced expres

99 As related P-type adenosine triphosphatases (ATPases) are not known to oli

100 lectron microscopy structure reveals how the adenosine triphosphatases (ATPases) form a closed spiral

101 In eukaryotes, P-type adenosine triphosphatases (ATPases) generate the plasma

102 DNA-dependent adenosine triphosphatases (ATPases) participate in a bro

103 previously unrecognized subfamily of P-type adenosine triphosphatases (ATPases) that may have diverg

104 volved from ring-shaped hexameric AAA-family adenosine triphosphatases (ATPases), dynein's large size

105 member of the microrchidia (MORC) family of adenosine triphosphatases (ATPases), has been shown to b

106 , an inhibitor of endoplasmic reticulum Ca2+-adenosine triphosphatases (ATPases).

107 er complexes belonging to the AAA+ family of adenosine triphosphatases (ATPases).

108 Here we show that rotary adenosine triphosphatases (ATPases)/synthases from Therm

109 d a peptide cleaved from a2 isoform vacuolar adenosine triphosphatase called a2NTD.

110 report that in living cells the cytoplasmic adenosine triphosphatase called ClpV specifically recogn

111 ffinity catalytic site 1 of chloroplast F(1) adenosine triphosphatase (CF(1) ATPase) were characteriz

112 al outer membrane by the p97/Cdc48-Ufd1-Npl4 adenosine triphosphatase complex is essential for mitoch

113 Cohesin is a chromosome-bound, multisubunit adenosine triphosphatase complex.

114 s and the neuron-specific proton pump and V0 adenosine triphosphatase component V100.

115 nteraction of BRG1-also known as SMARCA4, an adenosine triphosphatase-containing chromatin remodeler-

116 It is caused by mutations in the adenosine triphosphatase copper transporting beta gene (

117 RCA (sarcoplasmic-endoplasmic reticulum Ca2+ adenosine triphosphatase) corrected [Ca2+]er and mitocho

118 ticles and how guanosine triphosphatases and adenosine triphosphatase couple irreversible nucleotide

119 ports of membrane-bound, copper-transporting adenosine triphosphatases (Cu-ATPases) selective for cop

120 e catalysis on membranes and suppress futile adenosine triphosphatase cycles.

121 an inhibitor of the vacuolar proton-pumping adenosine triphosphatase, cytosolic calcein fluorescence

122 on supported Trio-dependent Rac1 activation, adenosine triphosphatase-deficient Hsc70 (D10N) abrogate

123 imulate, in a Rag-, Ragulator-, and vacuolar adenosine triphosphatase-dependent fashion, the transloc

124 tide that is essential for stimulating HSP70 adenosine triphosphatase diverges in candidate orthologu

125 that the conserved Swc2/YL1 subunit and the adenosine triphosphatase domain of Swr1 are mainly respo

126 ured regions within Hebo: a TUDOR domain, an adenosine triphosphatase domain, and a new domain, HEBO,

127 complex with a nucleosome and show that the adenosine triphosphatase domains engage their substrate

128 tone acetyltransferase dTip60 as well as the adenosine triphosphatase Domino/p400 catalyze the exchan

129 examined the expression of canalicular ecto-adenosine triphosphatase (ecto-ATPase) and mdr P-glycopr

130 d cells have implicated the canalicular ecto-adenosine triphosphatase (ecto-ATPase) in adenosine trip

131 x, which appears to load/unload RuvBL AAA(+) adenosine triphosphatase from pre-snoRNPs; and (d) a pot

132 riggers translocation of the proton pump, HK-adenosine triphosphatase, from cytoplasmic tubulovesicle

133 of fusiform cells, and it inhibited H(+)K(+)-adenosine triphosphatase gene expression.

134 al transformation and inhibition of H + K + -adenosine triphosphatase gene expression.

135 th factor for 7-16 hours stimulates H(+)K(+)-adenosine triphosphatase gene expression.

136 caused by mutations in a copper-transporting adenosine triphosphatase gene, ATP7A.

137 le with the coiled coil emerging from Smc3's adenosine triphosphatase head.

138 eport that the Caenorhabditis elegans P-type adenosine triphosphatase homolog, TAT-1, is critical for

139 Studies examining the development of H, K-adenosine triphosphatase in infants and the role of ente

140 riphosphate through the action of Na(+)/K(+) adenosine triphosphatases in an integrated in vitro lipi

141 s in autophagy, we silenced VCP or expressed adenosine triphosphatase-inactive VCP.

142 hibition was combined with the vacuolar H(+)-adenosine triphosphatase inhibitor bafilomycin A1 or wit

143 adenosine 5'-triphosphate and ARL-67156, an adenosine triphosphatase inhibitor, and were attenuated

144 The use of MLC kinase (MLCK) and myosin adenosine triphosphatase inhibitors led us to propose a

145 Proton pump (H(+)/K(+)-adenosine triphosphatase) inhibitors (PPIs) are widely u

146 The a2 isoform vacuolar adenosine triphosphatase is found on the surface on many

147 conclude that the a3-subunit of the vacuolar adenosine triphosphatase is not only responsible for the

148 t the a3-subunit of the vacuolar-type (H(+))-adenosine triphosphatase is required for establishing a

149 We have found that Swr1, a Swi2/Snf2-related adenosine triphosphatase, is the catalytic core of a mul

150 RSC is delineated into the adenosine triphosphatase motor, the actin-related protei

151 and RMES1B] lack signaling domains and have adenosine triphosphatase mutations expected to abrogate

152 ions and that expression of sodium potassium adenosine triphosphatase (Na(+)-K(+)-ATPase) correlates

153 Genetic differences in sodium-potassium adenosine triphosphatase (Na(+)-K(+)ATPase) could explai

154 ly believed that sodium, potassium-activated adenosine triphosphatase (Na+, K+-ATPase) is localized o

155 he vacuolar-type (V-type) sodium ion-pumping adenosine triphosphatase (Na+-ATPase) from Enterococcus

156 hyperplasia with increased sodium-potassium-adenosine triphosphatase (Na,K-ATPase) activity, attenua

157 ty of lens epithelium to synthesize new Na,K-adenosine triphosphatase (Na,K-ATPase) catalytic subunit

158 The rate of Na,K-adenosine triphosphatase (Na,K-ATPase) dependent potassi

159 Sodium and potassium-exchanging adenosine triphosphatase (Na,K-ATPase) in the kidney is

160 + channels, the basolateral sodium potassium-adenosine triphosphatase (Na,K-ATPase), and possibly chl

161 Inhibitors of sodium-potassium-activated adenosine triphosphatase (Na-K-ATPase) have been implica

162 reviously reported that the sodium potassium adenosine triphosphatase (Na/K-ATPase) can effect the am

163 that the alpha1 subunit of sodium potassium adenosine triphosphatase (Na/K-ATPase), acts as a recept

164 ular bile acid transporter Ca2+, Mg(2+)-ecto-adenosine triphosphatase, now facilitates such studies.

165 mmune systems, Hachiman and AVAST (antiviral adenosine triphosphatase/nucleoside triphosphatase of th

166 cytochrome c oxidase, inhibition of F(1)F(0) adenosine triphosphatase, or replacement of all mtDNA-en

167 ligase Hrd1, its partner Sel1, the cytosolic adenosine triphosphatase p97, and degradation by the pro

168 Many members of the SWI2/SNF2 family of adenosine triphosphatases participate in the assembly/di

169 eased expression of the plasma membrane Ca2+-adenosine triphosphatase (PMCA) efflux pump.

170 isk-like structures through a unidirectional adenosine triphosphatase polymerization, primed with a s

171 inding portion binds the polypeptide, and an adenosine triphosphatase portion facilitates substrate e

172 H(+)K(+)-adenosine triphosphatase protein and gene expression wer

173 portional disassembly of both ezrin and Na/K adenosine triphosphatase proteins from their cytoskeleta

174 nts are irreversible inhibitors of the H+/K+ adenosine triphosphatase proton pump.

175 ion at the final common pathway of the H+/K+ adenosine triphosphatase proton pump.

176 ulation of the sarcoplasmic reticulum Ca(2+)-adenosine triphosphatase pump and by augmented levels of

177 Curcumin is a nontoxic Ca-adenosine triphosphatase pump inhibitor that can be admi

178 omerization preceding it may be limiting the adenosine triphosphatase rate.

179 o be significantly regulated by the enzymes' adenosine triphosphatase regions.

180 ion and canalicular localization of the ecto-adenosine triphosphatase remained unchanged.

181 mic reticulum (SR) and reuptake by the Ca(2+)adenosine triphosphatase SERCA.

182 Abundance of sarcoplasmic reticulum Ca(2+) adenosine triphosphatase (SERCA), Na(+)/Ca(2+) exchanger

183 Sarcoplasmic/endoplasmic reticulum Ca(2+) adenosine triphosphatase (SERCA)2a, a critical regulator

184 bits the cardiac sarcoplasmic reticular Ca2+-adenosine triphosphatase (SERCA2a) pump.

185 sion of sarcoplasmic reticulum Ca(2+) uptake adenosine triphosphatase (SERCA2a), phospholamban (PLB),

186 to be catalyzed by katanin, a heterodimeric adenosine triphosphatase that can remove tubulin subunit

187 CLPB encodes an adenosine triphosphatase that is implicated in protein f

188 Mtr4 is an essential RNA-dependent adenosine triphosphatase that is required for all of the

189 om pH 7 to 3, and the gene encoding a P-type adenosine triphosphatase that may catalyze NH4+/H+ excha

190 d in WD, encodes a multitransmembrane domain adenosine triphosphatase that traffics from the trans-Go

191 nded DNA coil, akin to the way AAA+ ATPases (adenosine triphosphatases) unfold peptides.

192 ted the effect of directly inhibiting myosin adenosine triphosphatase using 2,3-butanedione monoxime

193 protons, together with reduced vacuolar H(+)-adenosine triphosphatase (V-ATPase) activity, accounts f

194 etaCTF) to disrupt lysosomal vacuolar (H(+))-adenosine triphosphatase (v-ATPase) and acidification.

195 ysosomes, leading to the removal of vacuolar adenosine triphosphatase (V-ATPase) and the neutralizati

196 et was enriched for subunits of the vacuolar adenosine triphosphatase (V-ATPase) complex, a proton pu

197 multiple subunits of the ubiquitous vacuolar adenosine triphosphatase (V-ATPase) complex, which is es

198 tion through inhibition of the vacuolar H(+)-adenosine triphosphatase (V-ATPase) increased the lumina

199 The vesicular adenosine triphosphatase (v-ATPase) is a proton pump tha

200 that the V0 domain of the vacuolar-type H(+)-adenosine triphosphatase (V-ATPase) is directly implicat

201 , lysosomes never recycle vacuolar-type H(+)-adenosine triphosphatase (V-ATPase) or neutralize to for

202 g sites of the vacuolar proton-translocating adenosine triphosphatase (V-ATPase), cysteine scanning m

203 the Ragulator complex, and the vacuolar H(+)-adenosine triphosphatase (v-ATPase).

204 Vesicular- or vacuolar-type adenosine triphosphatases (V-ATPases) are ATP-driven pro

205 Vacuolar-type adenosine triphosphatases (V-ATPases) are rotary proton

206 Vacuolar-type adenosine triphosphatases (V-ATPases)(1-3) are electroge

207 m proton-pumping vesicular- or vacuolar-type adenosine triphosphatases (V-ATPases).

208 We show that the neuronal vacuolar-type H(+)-adenosine triphosphatase V0 subunit a1 (V100) can regula

209 Binding to the AAA adenosine triphosphatase valosin-containing protein (VCP

210 The AAA-adenosine triphosphatase Vps4 disassembles and recycles

211 ion of ESCRT-III polymers induced by the AAA-adenosine triphosphatase Vps4 have been shown to remodel

212 utant deficient in a presumptive proteasomal adenosine triphosphatase was attenuated in mice, and exp

213 ndent functions of human copper-transporting adenosine triphosphatases (Wilson's and Menkes disease p