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1 s the GCN5 acetyltransferase during a normal adenovirus infection.
2 were unchanged after tyrphostin treatment or adenovirus infection.
3 e in calpain protein was observed after LacZ adenovirus infection.
4 artments with no effect on the efficiency of adenovirus infection.
5 ive treatment, if possible, in patients with adenovirus infection.
6 n on cell surfaces is required for efficient adenovirus infection.
7  be important in cutaneous wound healing and adenovirus infection.
8 in three of four woodchucks at 2 weeks after adenovirus infection.
9 er to cells that are relatively resistant to adenovirus infection.
10 involved in the induction of JNK activity by adenovirus infection.
11 ne expression in cells that are resistant to adenovirus infection.
12 etically normal cells outside the context of adenovirus infection.
13 ned the activation of this pathway following adenovirus infection.
14 hich were null for both alleles of p53, upon adenovirus infection.
15 s, but not IgG antibody molecules, inhibited adenovirus infection.
16  normally inhibits DNA replication following Adenovirus infection.
17 HeLa cells, which are commonly used to study adenovirus infection.
18 e system can instruct the innate response to adenovirus infection.
19  in the host neutralizing immune response to adenovirus infection.
20 e interact specifically during the course of adenovirus infection.
21  their impact on protein VII function during adenovirus infection.
22  for production of infectious progeny during adenovirus infection.
23 cines against coronavirus disease 2019 or by adenovirus infection.
24 c cells proceed with a lytic or a persistent adenovirus infection.
25 roducing cells in mouse liver within 24 h of adenovirus infection.
26  of fusing with autophagosomes which enhance adenovirus infection.
27 nity in humanized mice during a hepatotropic adenovirus infection.
28 yncytial virus infection and higher rates of adenovirus infection.
29 h clinical data on treatment and outcomes of adenovirus infection.
30  reduces cell surface CAR(Ex8) abundance and adenovirus infection.
31 hology associated with replication-defective adenovirus infection.
32 nt of rejection did not increase the risk of adenovirus infection.
33 bial peptides are potent inhibitors of human adenovirus infection.
34 show that this prevents ATR signaling during adenovirus infection.
35 23 patients with KD and 7 of 8 patients with adenovirus infection.
36 n nonpermissive 293 cells can be overcome by adenovirus infection.
37 apical surface and was highly susceptible to adenovirus infection.
38 f neonates undergoing examinations developed adenovirus infection.
39 tep of NHEJ, is degraded as a consequence of adenovirus infection.
40 l cellular function necessary for productive adenovirus infection.
41 on hinders RIDalpha activity during an acute adenovirus infection.
42 1 repair complex (MRN) to promote productive adenovirus infection.
43  and induction of the DNA damage response by adenovirus infection.
44 kDa proteins interact with each other during adenovirus infection.
45 piratory syncytial virus, parainfluenza, and adenovirus infection.
46  is accompanied by reduced susceptibility to adenovirus infection.
47 ellular repair complex is inactivated during adenovirus infection.
48  thus enhancing the cell's susceptibility to adenovirus infection.
49 njugation, which were not found with control adenovirus infections.
50 as antiviral agents for treatment of serious adenovirus infections.
51 are believed to prolong acute and persistent adenovirus infections.
52 onarily conserved target of E4-ORF3 in human adenovirus infections.
53 little is known about the immune response to adenovirus infections.
54 hile they are potent, they also risk causing adenovirus infections.
55 e as a vector for the transmission of ocular adenovirus infections.
56 an patients can develop severe, often lethal adenovirus infections.
57  prevalence behind RSV infection (15.7%) and adenovirus infection (10.3%).
58 of late adenovirus mRNA in the late phase of adenovirus infection; (4) repression of host mRNA and tr
59 tuzumab in vivo were at the greatest risk of adenovirus infection (45% probability) regardless of don
60                                              Adenovirus infection activates cellular DNA damage respo
61                                        Human adenovirus infection activates intracellular signaling,
62                           Here, we show that adenovirus infection activates the c-Jun N-terminal kina
63 ice (transthyretin HNF-3beta) or recombinant adenovirus infection (AdHNF3beta), and observed diminish
64                                              Adenovirus infections affected 24% of recipients and dev
65        We describe the first case of a fatal adenovirus infection after several years of immunosuppre
66 ctively studied the incidence and outcome of adenovirus infections after SCT using preemptive screeni
67 y(A) sites is found during the late stage of adenovirus infection, after viral DNA replication has be
68                                  Respiratory adenovirus infection among military recruits is a seriou
69  in the initiation of inflammation following adenovirus infection and adenovirus-mediated gene transf
70                                              Adenovirus infection and E1A expression sensitize cells
71                                              Adenovirus infection and expression of E1A induces both
72 he intracellular response that are unique to adenovirus infection and how adenoviral proteins produce
73 inant poly(A) site during the early stage of adenovirus infection and in plasmid transfections when m
74  production of chemokines by monocytes after adenovirus infection and increases monocyte migration.
75 K protein) is synthesized abundantly in late adenovirus infection and is required for efficient lysis
76 ed to viral replication centers early during adenovirus infection and is then targeted in an E1B-55K/
77 ding definition of how p53 is inactivated in adenovirus infection and provides key insights that coul
78 negative cells) increased its sensitivity to adenovirus infection and significantly inhibited its in
79 d to inhibit apoptosis induced by E1A during adenovirus infection and transformation.
80  overlap between the metabolic phenotypes of adenovirus infection and transformed tumor cells may pro
81 nsitive to cell stresses, namely heat shock, adenovirus infection and treatment with cycloheximide, w
82       Hence, a possible relationship between adenovirus infection and tyrosine phosphorylation of FAK
83 bronchoalveolar lavage fluid after pulmonary adenovirus infection, and all were significantly elevate
84 I3K) and Akt and their downstream targets in adenovirus infection, and here we report the novel findi
85                 We have also discovered that adenovirus infections are tightly regulated by endosome
86  be employed to develop novel drugs to treat adenovirus infection as well as be used as tools for gen
87 ctive therapeutic strategies to treat severe adenovirus infections as well as improved adenovirus vec
88         These lymphocytes are susceptible to adenovirus infection, as demonstrated by reporter green
89              We detected unsuspected mucosal adenovirus infection associated with enteritis as well a
90                     These results reveal how adenovirus infection attenuates LMP2 expression, thereby
91 es, including lymphocytes, are refractory to adenovirus infection because they lack the Coxsackie/ade
92  grossly rearranged and stabilized following adenovirus infection, but paclitaxel does not increase t
93  antihexon monoclonal antibody 9C12 inhibits adenovirus infection by blocking microtubule-dependent t
94                            C4 inhibits human adenovirus infection by directly inactivating the virus
95 ct evidence that human alpha-defensins block adenovirus infection by preventing uncoating during cell
96                           These data suggest adenovirus infection can activate beta-cell survival and
97              Together, these data argue that adenovirus infection can result in inhibition of RNAi an
98 ranslation of mRNAs during the late phase of adenovirus infection, can also modulate mRNA export from
99                                              Adenovirus infections cause significant morbidity and mo
100 s the first report describing an outbreak of adenovirus infection causing diarrhea among adult hemato
101 eline indicated that the parainfluenza 3 and adenovirus infections could have been linked to some of
102 rexpression of CREM-17X in intact islets via adenovirus infection decreased islet insulin mRNA levels
103 ent with previous studies demonstrating that adenovirus infection depends on attachment of a viral fi
104  characteristics of 5 children admitted with adenovirus infection during 2018-2019 who developed HLH
105 pithelial cells obtained from null mice show adenovirus infection efficiency equal to that from wild-
106 alysis from a prospective study of high-risk adenovirus infections following hematopoietic progenitor
107 pulations required stimulation subsequent to adenovirus infection for reporter expression.
108                        Numerous outbreaks of adenovirus infection from different types of health care
109 e PDZ1 domain is able to rescue CAR(Ex8) and adenovirus infection from MAGI-1-mediated suppression.
110  findings explain the relative resistance to adenovirus infection from the apical surface.
111                               In addition to adenovirus infection, group 3 ILCs were also found in mo
112 tein that is produced at high levels in late adenovirus infection (>24 h postinfection).
113 his study was aimed at investigating whether adenovirus infection has any impact on the potential of
114 m and the metabolic fate of glutamine during adenovirus infection have remained elusive.
115 neumoniae, Mycoplasma pneumoniae, and latent adenovirus infections have been correlated with asthma c
116                                 Unlike human adenovirus infections, however, mouse adenovirus type 1
117              We demonstrate here that during adenovirus infection, immediate early viral proteins fro
118 rotected mice from a subsequent OVA-encoding adenovirus infection in a CD8(+) cell-dependent manner a
119 of pneumonia developed during an outbreak of adenovirus infection in a chronic psychiatric care facil
120 o treat maternally-transmitted, disseminated adenovirus infection in a premature infant.
121                              IgA neutralizes adenovirus infection in a TRIM21- and proteasome-depende
122  GFAT1 and GFAT1Alt expressed by recombinant adenovirus infection in COS-7 cells displayed robust enz
123 cious in minimizing the clinical symptoms of adenovirus infection in rabbit eyes.
124  study, we recommend active surveillance for adenovirus infection in T-cell-depleted SCT and withdraw
125 e the incidence and clinical significance of adenovirus infection in this population.
126 l localization, it affects both adhesion and adenovirus infection in unique ways.
127  fatal disseminated disease resembling human adenovirus infections in immunocompromised patients.
128 lovirus (CMV), Epstein-Barr virus (EBV), and adenovirus infections in immunosuppressed patients.
129 lymphocytes are not generally susceptible to adenovirus infection, in part because of the absence of
130                                       SERCA2 adenovirus infection increased SERCA2 mRNA expression to
131                                              Adenovirus infection induced cytotoxic T lymphocytes (CT
132                                              Adenovirus infection induced the rapid activation of Raf
133                                              Adenovirus infection induces a cellular DNA damage respo
134               These results demonstrate that adenovirus infection induces a significant stabilizing e
135                        E1A expression during adenovirus infection induces apoptosis.
136 sion of the E4-6/7 protein in the context of adenovirus infection induces E2F-1 protein accumulation.
137                                 We show that adenovirus infection induces global changes in SUMO loca
138 ajor part of the mechanism by which systemic adenovirus infection induces pathology, as opposed to th
139                                              Adenovirus infection inhibits synthesis and processing o
140                                              Adenovirus infection is a potentially serious complicati
141                                              Adenovirus infection is an important cause of morbidity
142                                              Adenovirus infection is becoming increasingly recognized
143 ha)14iNKT cells during replication-defective adenovirus infection is not known and is the main focus
144   In the nontransplant pediatric population, adenovirus infection is the most common cause.
145 es, which are almost completely resistant to adenovirus infection, is sufficient to facilitate the ef
146                                              Adenovirus infection leads to increased apoptosis by the
147 n a murine model of intravenous hepatotropic adenovirus infection, liver-primed antiviral CD8(+) T ce
148 enovirus-induced myocarditis, and persistent adenovirus infection may contribute to ongoing cardiac d
149 nscriptional activation in the late phase of adenovirus infection, newly synthesized viral early E1A
150 the AAV2 capsid gene promoter (P40), neither adenovirus infection nor the large Rep protein was requi
151                      At the late stage after adenovirus infection, numerous autophagic vacuoles accom
152                                              Adenovirus infections occur in 5% to 21% of patients fol
153                                              Adenovirus infection occurs in 10% of pediatric orthotop
154 age at transplantation was a risk factor for adenovirus infection (odds ratio=0.81, 95% confidence in
155                                              Adenovirus infection of hematopoietic cells frequently r
156 rly dynamics in an experimental system using adenovirus infection of human embryonic kidney (293) cel
157                                    Efficient adenovirus infection of target cells depends upon the pr
158 One patient had primary graft failure due to adenovirus infection of the donor lungs, and required pr
159 y contribute to the inflammatory response to adenovirus infection of the human cornea.
160                          We report a case of adenovirus infection of the renal allograft in a combine
161 bstantial morbidity and mortality from human adenovirus infections, often in the setting of reactivat
162 bstantial morbidity and mortality from human adenovirus infections, often in the setting of reactivat
163 effect on transfection by either recombinant adenovirus infection or electroporation.
164 ses a ribosome jumping mechanism during late adenovirus infection or heat shock (stress) of mammalian
165  overexpression of the active form of Akt by adenovirus infection or inhibition of the Akt downstream
166 f the mutant p53(175H) allele by recombinant adenovirus infection or stable transfection also stabili
167 when cells are sensitized to TRAIL either by adenovirus infection or treatment with cycloheximide.
168 patients with KD than in patients with acute adenovirus infections or systemic adverse drug reactions
169 orylation of the ssD-BP also correlates with adenovirus infection, or expression of the adenovirus E4
170 00k protein displaces Mnk1 from eIF4G during adenovirus infection, or in transfected cells.
171 nd ARF levels by DNA damage, recombinant ARF adenovirus infection, or inducible MDM2 expression leads
172 ential for normal development, reproduction, adenovirus infection, or the healing of cutaneous wounds
173                       Here, we show that the adenovirus infection process has a significant impact on
174 has been analyzed in detail, the dynamics of adenovirus infection remain largely unknown due to techn
175 s dying a nonapoptotic cell death induced by adenovirus infection repressed macrophage proinflammator
176                                              Adenovirus infection resulted in a transient enhancement
177 s in the presence of doxycycline followed by adenovirus infection resulted in helper and vector gene
178 ession, DNA damage down-regulates MCL-1, and adenovirus infection resulted in the accumulation of pho
179 , Cryptosporidium, rotavirus, norovirus, and adenovirus infections resulting from indirect wastewater
180 evented activation of p40 transcription with adenovirus infection, resulting in a reduced level of ca
181                                              Adenovirus infection should be considered when evaluatin
182 om this study, the primary control of falcon adenovirus infections should be based on segregation of
183                 These data shed new light on adenovirus infection strategies and provide insights for
184 d more than cccDNA and mRNA levels following adenovirus infection suggests that the former decline ei
185                    During the late stages of adenovirus infection, the 100K protein (100K) inhibits t
186                                       During adenovirus infection, the emphasis of gene expression sw
187 e estrogen exposure, and through recombinant adenovirus infection, the introduction and stable expres
188                    During the early phase of adenovirus infection, the promoter-proximal L1 poly(A) s
189                                       During adenovirus infection, the viral protein E4orf3 associate
190                                   Late in an adenovirus infection, the viral proteinase (AVP) becomes
191 ndings validate a novel approach to treating adenovirus infections through the modulation of host cel
192 tion by cycloheximide and at a late stage of adenovirus infection, thus accounting for the loss of RN
193 mplished either through cell treatment or by adenovirus infection to express dominant-negative AMPK,
194 udies we used transgenic mice or recombinant adenovirus infection to increase hepatic expression of f
195 a)14iNKT cells were activated in response to adenovirus infection to induce significant levels of hep
196                            We also show that adenovirus infection triggers a response mediated by the
197             We explored the possibility that adenovirus infection triggers signal transduction pathwa
198                                       During adenovirus infection, viral proteins are expressed that
199                             The incidence of adenovirus infection was 19.7% (15 of 76), and the virus
200                    FR901228 treatment before adenovirus infection was associated with at least a 10-f
201                                              Adenovirus infection was classified as either disease or
202                                              Adenovirus infection was diagnosed in 17 (94%) persons w
203                                Recovery from adenovirus infection was marked by elevation of sorbitol
204                                              Adenovirus infection was very efficient, even at very lo
205 ytoplasm during the late phase of subgroup C adenovirus infection, we have examined the metabolism of
206                Two hallmarks of a successful adenovirus infection were abolished by the NO donors: th
207  age at transplantation is a risk factor for adenovirus infection; whereas cytomegalovirus D+/R- sero
208                                              Adenovirus infection, which is a waterborne viral diseas
209 lu RNAs and that these RNPs accumulate after adenovirus infection, while levels of SRP9, SRP14, SRP54
210 onors were beta-galactosidase-positive after adenovirus infection with a multiplicity of infection of
211  be phosphorylated by ATR during a wild-type adenovirus infection, with some contribution from ATM an

 
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