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1 o, 1.48; 95% confidence interval, 1.03-2.13; adjusted analysis).
2 that retained full coverage (P<0.001 for the adjusted analysis).
3 sits variation) of the untreated fellow eye (adjusted analysis).
4 [CI], 5.2-40.8;P= .0109, using a propensity-adjusted analysis).
5 Antibody responses were compared in adjusted analysis.
6 re TBI (HR 5 11.4, 95% CI 5 7.4-17.5) in age-adjusted analysis.
7 were tested using multivariate time-to-event adjusted analysis.
8 CI, 1.03-1.74; P = 0.02) in propensity score-adjusted analysis.
9 ly associated with cancer mortality in fully adjusted analysis.
10 ) in East Europe compared to South Europe in adjusted analysis.
11 between time to intubation and mortality in adjusted analysis.
12 lihood of hyperphosphatemia in multivariable-adjusted analysis.
13 pared with higher levels of each variable in adjusted analysis.
14 sians or Hispanics compared with NHWs in the adjusted analysis.
15 9, 95% CI 1.50-6.37, overall P=0.007) in the adjusted analysis.
16 and race-adjusted analysis to 0.89 in fully adjusted analysis.
17 found to be associated with infection in the adjusted analysis.
18 (34.4%; P=.04) by the prespecified covariate-adjusted analysis.
19 ]: 0.97 [0.71-1.31]) in the propensity score-adjusted analysis.
20 the odds of a complication (P < 0.001) in an adjusted analysis.
21 I, 1.59 to 3.71) compared with usual care in adjusted analysis.
22 interval [CI], 1.04-3.89) in a multivariable-adjusted analysis.
23 remained independent determinants of LVFP in adjusted analysis.
24 ciated with greater ICU-onset BSI risk after adjusted analysis.
25 (RR, .20; 95% CI, .09-.44; P < .0001) in the adjusted analysis.
26 DGF was similar on adjusted analysis.
27 irst 18 months of life in a propensity score-adjusted analysis.
28 in the superficial than deep cortex, even in adjusted analysis.
29 dropout and overall survival through a risk-adjusted analysis.
30 20-13.33]) were associated with blindness in adjusted analysis.
31 with all cognitive measures in multivariable-adjusted analysis.
32 ificantly lower in the intervention group in adjusted analysis.
33 d odds of nvAMD in unadjusted and confounder-adjusted analysis.
34 ations between GB and CPQ11-14 scores in the adjusted analysis.
35 nd 0.74 (0.46-1.20) for the propensity score-adjusted analysis.
36 ted only with higher risk of ESRD in a fully adjusted analysis.
37 , but the association was not significant in adjusted analysis.
38 cant comorbid conditions through matched and adjusted analysis.
39 and WRF on outcome was not significant in an adjusted analysis.
40 postoperative pressure ulcer development on adjusted analysis.
43 ervention and control periods, respectively (adjusted analysis: 26.7/1,000 versus 26.2/1,000, adjuste
52 stimate was attenuated and nonsignificant in adjusted analysis (adjusted hazard ratio = 0.77, 95% con
53 ype seemed to be associated with worse OS on adjusted analysis (adjusted hazard ratio = 1.57; 95% CI,
54 gh this was not statistically significant in adjusted analysis (adjusted HR, 0.36; 95% CI, 0.05 to 2.
55 p = 0.02), and this difference persisted in adjusted analysis (adjusted odds ratio [AOR] = 0.51, 95%
56 talizations without cannabis use disorder in adjusted analysis (adjusted odds ratio, 1.19; 95% CI, 1.
61 dence interval], 8.7 [2.0-37.3]; P=0.004 for adjusted analysis and 3.4 [0.8-13.8]; P=0.07 for the una
62 as associated with increased mortality in an adjusted analysis and contained a distinct repertoire of
63 duce the likelihood of a worse outcome in an adjusted analysis and was associated with postoperative
72 requency repeated measures did not differ in adjusted analysis between groups post baseline (mean dif
82 I, 1.14-1.28) than were white patients in an adjusted analysis, but there were no significant racial
83 to the current practice of prospective risk-adjusted analysis by a National Surgical Quality Improve
94 d 14.1% (95% CI, 10.8%-18.0%) of inpatients; adjusted analysis demonstrated a similar difference in i
97 >/=15 mm in thickness and in those with LGE; adjusted analysis demonstrated that segmental presence o
109 ted in 29 (13%) patients; immortal time bias adjusted analysis found no significant difference betwee
110 ociated with higher in-hospital mortality in adjusted analysis (GFR, 60-89; odds ratio [OR], 1.5; 95%
113 tality was observed for the entire cohort on adjusted analysis (hazard ratio, 0.99; 95% CI, 0.94-1.04
114 associated with cardiovascular events in an adjusted analysis (hazard ratio, 1.08; 95% confidence in
115 ot associated with risk for heart failure in adjusted analysis (hazard ratios, 1.0 [reference], 0.77
124 nterval [CI] 0.62 to 0.87; p < 0.001) and in adjusted analysis (HR 0.80, 95% CI 0.66 to 0.97; p = 0.0
128 1 to 0.95; p < 0.001) and multivariable risk-adjusted analysis (HR per unit change for mortality risk
130 or trend < 0.01) but not in the multivariate-adjusted analysis (HR: 1.09; 95% CI: 0.98, 1.21; P for t
131 y associated with type 2 diabetes in the age-adjusted analysis (HR: 1.91; 95% CI: 1.72, 2.11; P for t
133 rmore, the pattern of variation changed: the adjusted analysis identified three new outliers, and two
134 or of outcome for the composite end point in adjusted analysis III (hazard ratio=0.808; 95% CI, 0.689
136 her systolic blood pressure was confirmed in adjusted analysis in the Chicago Genetics of Hypertensio
137 ve implantation devices were observed at the adjusted analysis in Valve Academic Research Consortium
146 ent predictors of the composite end point in adjusted analysis (LVH hazard ratio [HR], 3.0; 95% confi
162 sociated with 30-day readmission; in a fully adjusted analysis, none of the 9 SDOH were individually
163 ifferentially abundant in aoCRC vs. eoCRC in adjusted analysis (Odds Ratio = 21.8, FDR P = 0.04).
164 ociated with decreased odds of death in site-adjusted analysis (odds ratio [OR] 0.82 [95% CI 0.71-0.9
165 6%]; P = 0.06) and increased mortality in an adjusted analysis (odds ratio of 5.85; 95% confidence in
166 vorable neurocognitive outcome in propensity-adjusted analysis (odds ratio, 1.61; 95% confidence inte
170 , we propose a method for age-sex population-adjusted analysis of disease severity in epidemics that
173 (95% CI, 0.26-0.84; P = .01) for a covariate-adjusted analysis of propensity-matched data to 0.61 (95
183 gnificantly associated with ASD in partially adjusted analysis (OR, 1.20; 95% CI, 1.06-1.36), but thi
188 rtality based failure to rescue in the fully adjusted analysis (P<0.05); however, the extended stay b
202 nteraction.Measurements and Main Results: In adjusted analysis, PM(2.5) (CMAQ) (odds ratio [OR], 1.12
207 d properly, then the resulting ascertainment-adjusted analysis produces parameter estimates that gene
213 this difference remained significant in the adjusted analysis (relapse rate ratio 1.27, P=0.0305).
215 and 1.06 (95% CI, 0.93 to 1.22) in the fully adjusted analysis restricted to women with depression.
216 be correctly modeled, then an ascertainment-adjusted analysis returns population-based parameter est
223 and nonischemic cardiomyopathy groups after adjusted analysis (RR 0.99, 95% CI 0.86 to 1.15; p = 0.9
224 not present in those taking pravastatin (age-adjusted analysis: RR, 0.98; 95% CI, 0.47-2.04; P =.046
227 uary and May 2021 in Maryland, in regression-adjusted analysis, SARS-CoV-2 viruses carrying the spike
235 ort with ENE-negative disease, multivariable adjusted analysis showed nonsignificant improvements wit
247 2.29), and in the maximally (multivariate-) adjusted analysis the relative risk was 1.59 (95% CI: 1.
263 s (95% PI, 6.0-9.3 percentage points) in the adjusted analysis; the excess death rate among Republica
272 Cox-regression analysis demonstrated that in adjusted analysis those in the N-SLK group were signific
276 education changed from 0.79 in age- and race-adjusted analysis to 0.89 in fully adjusted analysis.
280 associated with incident T2D in multivariate-adjusted analysis until body mass index was adjusted: od
290 associated with multiple acts of violence in adjusted analysis were male gender (OR, 3.37; 95% CI, 1.
292 5-0.82, P < 0.001) had lesser Star scores on adjusted analysis, whereas patients with a cancer diagno
294 ormed within the overall cohort and using an adjusted analysis with 1:1 propensity score matching for
295 HR], 1.60; 95% CI, 1.04-2.47; p = 0.033) and adjusted analysis with overlap weighting (adjusted HR, 1
296 2-way analysis of variance mixed-effects sex-adjusted analysis with post hoc Sidak correlation for mu
300 r incidence of HF, and this held true in the adjusted analysis yielding a hazard ratio of 1.54 (95% c