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1 th trials combined (P < 0.01 with or without adjustment for age).
2 manifest Parkinson's disease, p=0.01), after adjustment for age.
3 risk of CNV and macular atrophy, even after adjustment for age.
4 t this association was not significant after adjustment for age.
5 gnificant association with incident HF after adjustment for age.
6 erences in mortality became attenuated after adjustment for age.
7 1,000 person-years), which disappeared after adjustment for age.
8 n group; 6) minimum 1-year follow-up; and 7) adjustment for age.
11 ays (-5.3 days; 95% CI, -7.7 to -3.0), after adjustment for age, academic degree, specialty, and numb
14 utcome in this context, still observed after adjustment for age and after censoring patients who rece
17 n females compared with males and, following adjustment for age and body size, these differences beca
33 dverse prognostic factor after multivariable adjustment for age and hypertension (HR = 5.95; 95% CI,
34 h increased odds of severe D(3) SA-AKI after adjustment for age and illness severity (odds ratio, 1.4
35 0 to 1995 to 5.9% in 2009 to 2010, but after adjustment for age and indication, a modest decrease was
37 significant during the EDIC follow-up after adjustment for age and mean HbA(1c) (HR = 1.20; P = 0.00
38 rates of SICH and poor 3-month outcome after adjustment for age and National Institutes of Health Str
39 This association remained significant after adjustment for age and other factors associated with mal
40 1.28-1.58) compared with never smokers after adjustment for age and other potential risk factors.
43 re compared between strains and seasons with adjustment for age and prior LAIV (n = 436), inactivated
44 , 0.94-1.26), which remained unchanged after adjustment for age and race (hazard ratio, 1.00; 95% con
47 R], 3.36; 95% CI, 1.07-10.59; P = .04) after adjustment for age and sex and a 14-fold increase in odd
48 were at an increased risk of mortality after adjustment for age and sex compared with stage I patient
55 d higher long-term all-cause mortality after adjustment for age and sex, driven by early and noncardi
70 sociated with relative telomere length after adjustment for age and the length of follow-up (for each
75 of mortality and cardiovascular events after adjustments for age and sex; cholesterol, systolic BP, a
77 eriod from 3 to 16 days after diagnosis with adjustments for age and the baseline cycle-threshold val
78 essed risk factors for hospitalization after adjustment for age- and sex-specific prevalence of risk
81 atio (HR) was 2.66 (95% CI, 1.30-5.43) after adjustment for age at diagnosis, sex, and medical center
82 in intake with BMI, weight, and height, with adjustment for age at diet diary, sex, total energy inta
85 ards regression under an additive model with adjustment for age at onset, sex, and the first 4 princi
86 active TB disease remained significant after adjustment for age, biomass fuel (BMF) use, and presence
87 sm according to MMR vaccination status, with adjustment for age, birth year, sex, other childhood vac
90 mL compared with 74.4 pg/mL, P = 0.01) after adjustment for age, BMI, race, dietary factors, and phys
93 espectively (P-linear trend < 0.0001), after adjustment for age, body mass index, alcohol use, smokin
97 associated with lower recurrence risk after adjustment for age, body mass index, number of AF episod
98 -.82) than PLWH with other haplogroups after adjustments for age, body mass index, combination antire
99 ment for covariates had little effect except adjustment for age category (fully adjusted model HR, 1.
103 th versus without a pregnancy history, after adjustment for age, CHD severity, comorbidities, and adm
105 p Interference Test -2.6, -7.4 to 2.3) after adjustment for age, education, and baseline cognitive fu
107 tly associated with better PCS and MCS after adjustment for age, education, marital status, number of
108 hese estimates were similar after additional adjustment for age, education, smoking, use of alcohol,
115 e interval, 0.32-0.87; P = 0.01 period after adjustment for age, from the first 5-year interval betwe
116 n meta-analysis (P-value=3.28 x 10(-9) after adjustment for age, gender and education) in an intron o
119 ith overall retransplant-free survival after adjustment for age, gender, and transplant indication.
120 [odds ratio, 2.16 (95% CI 1.10-4.26)], after adjustment for age, gender, body mass index, diabetes du
121 ssociation between gender and survival after adjustment for age, gender, cardiac arrest rhythm, witne
122 VF test (P = 0.18) or the MD (P = 0.7) after adjustment for age, gender, CCT and history of glaucoma
128 were used to estimate odds ratios (ORs) with adjustment for age, gender, smoking, education, setting,
129 s independently associated with asthma after adjustment for age, gender, socio-economic stratum, city
130 ptic ulcer (HR 2.24; CI 95% 1.16:4.35) after adjustment for age, gender, socioeconomic status, non-st
131 ated and accident-related index injury after adjustment for age group, socioeconomic status, and chro
132 used to compare the CRC incidence rates with adjustment for age, history of lower gastrointestinal en
138 f combined death and rehospitalization after adjustment for age, left ventricular ejection fraction,
139 d using conditional logistic regression with adjustment for age, marital status, prescription drug mo
141 ith mortality and remained independent after adjustment for age, N-terminal pro-B-type natriuretic pe
142 , 3.1; 95% CI, 2.1-4.4; P < .001) even after adjustment for age, National Institute of Health Stroke
143 ith change in ED:EI (r=0.650, p=0.006) after adjustment for age (odds ratio 1.12, 95% CI 1.02-1.24; p
144 er, this association was not sustained after adjustment for age or additional adjustment for cardiova
146 iver fibrosis lack serial fibrosis measures, adjustment for age, or longitudinal observations in coin
147 ependently predicted 30-day mortality (after adjustment for age, PSI score, and preexisting comorbid
148 sk factors and SSB intake were examined with adjustment for age, pubertal stage, physical fitness, so
150 ciation between AMH and CRP without and with adjustment for age, race, body mass index (BMI), smoking
153 CI: 0.179, 0.194 ng/dL), respectively] after adjustment for age, race, percentage of body fat, daily
154 (95% CI: 2.02%, 10.52%), respectively, after adjustment for age, race, percentage of body fat, percei
155 se in risk factor area under the curve after adjustment for age, race, sex, and education (P<0.05 for
156 ociated with better cognitive function after adjustment for age, race, sex, and total calorie intake
157 decisional conflict and distress, even after adjustment for age, race, sex, education, employment, an
161 atio, 2.5; 95% CI, 1.5-4.2; p = 0.001) after adjustment for age, region, baseline disability, maligna
165 d -0.56 (-0.96, -0.17), respectively), after adjustments for age, service specialty, waist circumfere
166 marked reduction in hospital mortality after adjustment for age, severity of illness, and comorbiditi
167 (OS) using a log-additive genetic model with adjustment for age, sex, and age-adjusted International
169 tically significant even after multivariable adjustment for age, sex, and BMI (adjusted OR, 3.36; 95%
178 among post-operative conduction groups after adjustment for age, sex, and concomitant procedures (p =
180 erse relationship remained significant after adjustment for age, sex, and conventional childhood risk
182 viduals versus nondiabetic individuals after adjustment for age, sex, and education and after additio
183 ed with an increased risk for dementia after adjustment for age, sex, and educational level (hazard r
189 measured by using FreeSurfer software, with adjustment for age, sex, and intracranial volume, and su
190 a higher risk of in-hospital mortality after adjustment for age, sex, and measured comorbidities.
191 t this association was not significant after adjustment for age, sex, and medical history (adjusted h
193 ith future death/myocardial infarction after adjustment for age, sex, and race (odds ratio, 2.05; 95%
194 01-2.50; P=0.04) in unadjusted analyses, but adjustment for age, sex, and race attenuated association
199 have a history of one or more tattoos after adjustment for age, sex, and race/ethnicity (OR, 5.17; 9
204 Findings were irrespective of multivariable adjustment for age, sex, and surgical/transcatheter aort
206 tion (shift analysis) and in subgroups after adjustment for age, sex, baseline stroke severity (Natio
207 ing repeated-measures linear regression with adjustment for age, sex, birth weight, maternal educatio
208 nfidence interval, 1.05-1.21; P<0.001) after adjustment for age, sex, body mass index, and ASA-perfor
211 ibrosis (OR, 2.08; 95% CI: 1.20-3.55), after adjustment for age, sex, body mass index, fasting hyperg
212 ) and remained a significant predictor after adjustment for age, sex, body mass index, N-terminal pro
213 efore (54.7% vs. 44.7%; p < 0.001) and after adjustment for age, sex, body mass index, pre-existing m
214 ression under an additive genetic model with adjustment for age, sex, body mass index, smoking status
215 r trunk muscle endurance in models including adjustment for age, sex, body mass index, socioeconomic
216 ntal disorders and medical conditions, after adjustment for age, sex, calendar time, and previous men
220 al and facility-based care, persisting after adjustment for age, sex, comorbidities, and insurance ty
222 l, 1.3-1.8]; P < 0.0001, respectively) after adjustment for age, sex, comorbidities, diagnosis, creat
225 neralised linear modelling, with and without adjustment for age, sex, diabetes diagnosis, systolic bl
226 ratio 1.58 [1.10-2.31], P=0.014) even after adjustment for age, sex, diabetes mellitus, and ischemic
227 al length of stay (p < 0.001) remained after adjustment for age, sex, diagnoses, sedation, and ventil
230 isolation was 1.73 (95% CI 1.65-1.82) after adjustment for age, sex, ethnic origin, and chronic dise
231 nalyzed using Cox proportional hazards, with adjustment for age, sex, ethnicity, alcohol use, CD4(+)
232 d cravings and appetite scores at 6 mo after adjustment for age, sex, ethnicity, baseline body mass i
233 link function with robust error variance and adjustment for age, sex, health care use because of AR,
235 sociation that persisted after multivariable adjustment for age, sex, heart rate, hypertension, systo
236 ndently related to FeNO (all P < 0.05) after adjustment for age, sex, height, smoking history and med
237 ssociated with LV mass-to-volume ratio after adjustment for age, sex, hypertension, race, and dyslipi
238 idence intervals [1.29-6.75]; P=0.011) after adjustment for age, sex, hypertension, smoking, sodium l
239 hese associations remained significant after adjustment for age, sex, inflammatory markers, and cardi
240 1.71) had an increased risk of death despite adjustment for age, sex, insurance status, etiology of c
242 ; 95% confidence interval: 1.10-1.87), after adjustment for age, sex, living alone, education, lifest
243 od (beta=0.16; P<0.001) that persisted after adjustment for age, sex, medication use, and cardiovascu
245 portional hazards regression analysis (after adjustment for age, sex, numbers of annual medical visit
246 low socioeconomic status group was robust to adjustment for age, sex, obesity, and physical activity,
247 These differences remained significant after adjustment for age, sex, parental history of myopia, and
249 dence interval, 1.32 to 2.41; P<0.001) after adjustment for age, sex, presence of diabetes mellitus,
253 erence, 18 m [95% CI, 6-30]; P = .30), after adjustment for age, sex, race, body mass index, forced e
255 ith adiposity phenotypes were examined after adjustment for age, sex, race, comorbidities, and body m
256 V structure and function were examined after adjustment for age, sex, race, comorbidities, and lean m
258 ting equation Poisson models were used, with adjustment for age, sex, race, educational level, income
261 interval duration (QTcorr) was determined by adjustment for age, sex, race/ethnicity, and RR interval
262 s or differentially methylated probes, after adjustment for age, sex, race/ethnicity, batch effects,
264 using Cox proportional hazards models, with adjustment for age, sex, race/ethnicity, body mass index
266 using Cox proportional hazards models, with adjustment for age, sex, race/ethnicity, smoking, diagno
267 using Cox proportional hazards models, with adjustment for age, sex, race/ethnicity, smoking, diagno
268 le with and without asthma, before and after adjustment for age, sex, social deprivation and smoking
269 alls) (HR = 0.79, 95% CI: 0.44, 1.42), after adjustment for age, sex, socioeconomic position, alcohol
272 egression for each health-care system, after adjustment for age, sex, year, and Charlson comorbidity
273 type and baseline cognitive performance with adjustment for age, sex, years of education, disease dur
276 15 years) prior to dementia diagnosis, with adjustments for age, sex, education, apolipoprotein E (A
278 s than periodontally healthy controls, after adjustments for age, sex, physical activity, systolic bl
281 ified analysis revealed that the study type, adjustment for age/sex, treatment duration, cumulative d
284 ting for potential confounding factors.After adjustment for age, smoking, and other factors, women wi
286 CVD was quantified with joint modeling, with adjustment for age, smoking, oral contraceptive use, bod
287 fidence intervals (CI) were calculated after adjustment for age, smoking, physical activity, socioeco
290 k, and Asian women than in white women after adjustment for age, socioeconomic status (income and edu
291 sion analyses before and after multivariable adjustment for age, socioeconomic status, depressive sym
292 Cancer type, data source, reporting quality, adjustment for age, stage, or comorbidities, use of prop
297 ared with women in the lowest quintile after adjustment for age, total energy, race, income, smoking,
299 h year as independent variables (i.e., after adjustment for age, we were able to analyze how LTL corr
300 3.1; 95% confidence interval, 1.2-8.2) after adjustment for age, year of diagnosis, septic complicati