ライフサイエンスコーパス: adolescent-onset

1 ychiatric disorders with a high incidence of adolescent onset.

2 The disorder has an early childhood or adolescent onset.

3 eletal muscle degeneration with childhood to adolescent onset.

4 fied: (1) stable low, (2) teacher-identified adolescent onset, (3) moderate, and (4) stable high.

5 at-onset groups: childhood onset (ages <12), adolescent onset (ages 12-17), early adult onset (ages 1

6 volumes of adolescents and young adults with adolescent-onset alcohol use disorders to those of healt

7 h poor disease control, psychosocial issues, adolescent-onset allergic disease and female sex; (b) Ps

8 isms underlying schizophrenia's typical post-adolescent onset also remain elusive.

9 highest detection rate was in those with an adolescent onset and a family history (75%).

10 150-1151del) was associated with juvenile or adolescent onset and mental retardation, whereas we show

11 with bipolar disorder, especially those with adolescent onset and the above-noted factors, appear to

12 Ketamine users were categorized as adolescent-onset and adult-onset based on when they were

13 Adolescent (35-day-old at start; adolescent-onset) and adult (86-day-old at start) male r

14 ding to age at onset (childhood-onset versus adolescent-onset) and the presence or absence of callous

15 rmalities may contribute to the emergence of adolescent-onset as well as early-onset conduct disorder

16 95% CI, 0.213-4.404) and teacher-identified adolescent-onset (B, 2.114; 95% CI, 0.725-3.503) groups

17 ional anisotropy (FA), have been reported in adolescent-onset bipolar disorder and in youth at famili

18 Although adolescent-onset bipolar disorder is associated with sig

19 oes not support a causal association linking adolescent-onset cannabis problems with young adult depr

20 sitive association between, on the one hand, adolescent-onset cannabis use and dependence and, on the

21 Impairment was concentrated among adolescent-onset cannabis users, with more persistent us

22 ermine whether decline is concentrated among adolescent-onset cannabis users.

23 restore neuropsychological functioning among adolescent-onset cannabis users.

24 ogy of early-onset conduct disorder, whereas adolescent-onset conduct disorder arises as a result of

25 ress; thus, arg(-/-) mice present a model of adolescent-onset dendritic simplification.

26 mpared the social functioning of adults with adolescent-onset depression (ages 10-20) to those with a

27 ts, little is known about the association of adolescent-onset depression and development of T2DM in y

28 blunted neural response to rewards precedes adolescent-onset depression and symptom emergence.

29 ophysiology of depression is associated with adolescent-onset depression.

30 city, including myelination, are affected by adolescent-onset depression.

31 Rates of adolescent onset disorder dropped sharply by the late 20

32 more to recurrence than chronicity of child-adolescent onset disorders.

33 omplications and relapses that are common in adolescent-onset drug addiction.

34 Diagnostic stability was high for child- and adolescent-onset DSM-III-R schizophrenia (positive predi

35 Although most patients report adolescent onset, existing HS diagnostic criteria may no

36 rating that adolescents with both early- and adolescent-onset forms of conduct disorder show impaired

37 Adolescent-onset gambling is associated with more severe

38 Similarly, the adolescent-onset group failed to show significant neural

39 etamine users, with a larger decrease in the adolescent-onset group.

40 The stable-high and teacher-identified adolescent-onset groups had elevated risk for all-cause

41 ralimbic cortex was greater in rats from the adolescent-onset groups than adults.

42 bid delinquency were identified: stable low, adolescent-onset high to moderate, stable moderate, and

43 We investigated families with adolescent-onset idiopathic generalized epilepsy (IGE),

44 ound evidence for linkage to chromosome 8 in adolescent-onset IGE families in which JME was not prese

45 families ascertained through a proband with adolescent-onset IGE.

46 ssible candidate for these specific forms of adolescent-onset IGE.

47 omosome 18 locus predisposing to a number of adolescent-onset IGEs.

48 Both the early-onset high-persistent and adolescent-onset increasing trajectory classes were asso

49 The adolescent-onset increasing trajectory was associated wi

50 al encephalopathy to milder manifestation of adolescent-onset, isolated hereditary spastic paraplegia

51 Six patients presented as an adult or adolescent-onset limb-girdle muscular dystrophy, one pre

52 A total of 274 participants with adolescent-onset major depressive disorder were assessed

53 opmental pathways that lead to first-episode adolescent-onset MDD (incident cases) in those at high f

54 us testing of different pathways to incident adolescent-onset MDD composed of contributions from fami

55 Clinical outcomes of adolescent-onset MDD into adulthood compared with contro

56 ial mortality from suicide into adulthood in adolescent-onset MDD patients.

57 cant independent clinical antecedents of new adolescent-onset MDD, but disruptive behavior (beta = -0

58 noid system as a notable research target for adolescent-onset mental health disorders.

59 nt research target for the neuropathology of adolescent-onset mental health disorders.

60 Individuals were excluded for adolescent-onset MOGAD or short disease duration.

61 iological research progress in childhood-and adolescent-onset multiple sclerosis have been informed b

62 male adolescents with early-onset (N=36) or adolescent-onset (N=27) conduct disorder and in healthy

63 Dopamine system dysfunction is implicated in adolescent-onset neuropsychiatric disorders.

64 incubation of nicotine craving occurs after adolescent-onset nicotine self-administration and that n

65 n of nicotine craving is also observed after adolescent-onset nicotine self-administration and that n

66 ng whether this incubation is observed after adolescent-onset nicotine self-administration.

67 ther incubation of craving also occurs after adolescent-onset nicotine self-administration.

68 er 4 (19.4%) female-undiagnosed-wheezers had adolescent-onset non-atopic wheeze, low BDR and BHR, imp

69 s sharing autism spectrum disorder, NDD, and adolescent-onset obesity.

70 d with developmental tooth abnormalities and adolescent onset of a broad range of ectodermal defects.

71 We next asked whether the late-adolescent onset of a7nAChR modulation of hippocampal in

72 ory circuits is thought to contribute to the adolescent onset of cognitive deficits observed in schiz

73 FSIN development may contribute to the post-adolescent onset of schizophrenia endophenotypes.

74 The disease is characterized by adolescent onset of weakness, and atrophy of thenar and

75 rodevelopmental theory to address causes for adolescent onsets of clinical depressive disorders.

76 rative manifestations in our cohort included adolescent-onset parkinsonism and dystonia with cognitiv

77 ealthy subjects with a longitudinal study of adolescent-onset patients (n = 12, representative subset

78 cross-sectional study of brain structure for adolescent-onset patients (n = 25) and adult-onset patie

79 Upon combined genetic loss of Urp1 and Urp2, adolescent-onset planar curves manifested in the caudal

80 disorder to 40% in those with a childhood or adolescent onset progressive disorder.

81 The phenotype is characterized by adolescent-onset progressive spastic ataxia with frequen

82 ain function, a trajectory likely underlying adolescent-onset psychiatric conditions such as schizoph

83 ciencies in this circuit are associated with adolescent-onset psychiatric disorders in humans.

84 of frontostriatal networks may contribute to adolescent-onset psychopathology.

85 cutive patients with first-episode child- or adolescent-onset psychosis (mean age at onset=14.2 years

86 utation at the endogenous locus, there is an adolescent-onset reduction in dendritic length and compl

87 Structural abnormalities specific to adolescent-onset schizophrenia in the sensori-motor cort

88 ervations suggest that the neuropathology of adolescent-onset schizophrenia involves larger and wides

89 Adolescent-onset schizophrenia provides an exceptional o

90 Compared with other psychoses, child- or adolescent-onset schizophrenia was associated with signi

91 ocial and symptomatic outcomes of child- and adolescent-onset schizophrenia with those of nonschizoph

92 altered white matter maturation specific to adolescent-onset schizophrenia.

93 tural grey and white matter abnormalities in adolescent-onset schizophrenia.

94 ractional Anisotropy) were carried out on 25 adolescent-onset schizophrenic patients and 25 healthy a

95 roups and reduced right insula volume in the adolescent-onset subgroup.

96 variants in USH2A causing progressive early adolescent onset visual and hearing impairment consisten

97 ation associated with hyperphagic obesity of adolescent onset with variable NDD.